ライフサイエンスコーパス: acquired immune response

1 or to the onset of IFN counteraction and the acquired immune response.

2 o define the magnitude and the nature of the acquired immune response.

3 t specific components of the host innate and acquired immune response.

4 ys a crucial role in both the innate and the acquired immune response.

5 nnate immunity that profoundly influences an acquired immune response.

6 viable bacteria together with an attenuated acquired immune response.

7 ), mediate innate responses, and initiate an acquired immune response.

8 e miRNAs in the regulation of the innate and acquired immune response.

9 eficient bone formation and activation of an acquired immune response.

10 e an antigen-specific T helper type 1 (Th1)--acquired immune response.

11 on TCR stimulation and thus may modulate the acquired immune response.

12 ing immunoglobulin gene diversity during the acquired immune response.

13 t TNF- alpha is a necessary component of the acquired immune response.

14 Less is known regarding the role of the acquired immune response.

15 he innate immune response in addition to the acquired immune response.

16 the innate as well as the early phase of the acquired immune response.

17 most certainly preceded the emergence of the acquired immune response.

18 e required for the induction of a Th1 T-cell acquired immune response.

19 ism by which motile bacteria can initiate an acquired immune response.

20 endritic cell (DC) is a critical step of the acquired immune response.

21 levels in tissues is not due to an impaired acquired immune response.

22 re may contribute to the polarization of the acquired immune response.

23 igration, a key process governing innate and acquired immune responses.

24 id metabolism and trafficking and influences acquired immune responses.

25 nate cytokine production, leading to altered acquired immune responses.

26 rpreting activation states of the innate and acquired immune responses.

27 omodulatory cytokine that affects innate and acquired immune responses.

28 lopment of colitis by activating deleterious acquired immune responses.

29 roteins during the transition from innate to acquired immune responses.

30 n of the airways and induction of innate and acquired immune responses.

31 e of PPARgamma in regulating both native and acquired immune responses.

32 r, insulin sensitivity, bone metabolism, and acquired immune responses.

33 strongest balancing selection from naturally acquired immune responses.

34 ost defense mechanisms leading to innate and acquired immune responses.

35 cytokines and to exert helper functions for acquired immune responses.

36 etory pathway is crucial for both innate and acquired immune responses.

37 Host innate and acquired immune responses activated following RSV infect

38 nflammation and the modulation of innate and acquired immune responses after binding to their G prote

39 ncode molecules that lie at the heart of the acquired immune response against infectious diseases.

40 to the central role of DCs in initiating the acquired immune response against M. tuberculosis infecti

41 bscess formation, yet in humans, a naturally acquired immune response against the CRD did not persist

42 Naturally acquired immune responses against human cancers often in

43 nd play an important role in both innate and acquired immune responses against infectious diseases an

44 ion may enhance Mphi-mediated innate and Th1-acquired immune responses against intracellular infectio

45 jor role in the induction of both innate and acquired immune responses against pathogenic invaders.

46 Absence of a properly functioning acquired immune response allows MAC persistence within m

47 infection is not necessarily dependent on an acquired immune response and can occur despite the prese

48 nesis is dependent upon inflammation, a Th-1 acquired immune response and hormonal changes including

49 ive vaccine that improves upon the naturally acquired immune response and induces rapid and long-last

50 gnificant impact on our understanding of the acquired immune response and may provide insight concern

51 by ANCA may contribute to modulation of the acquired immune response and to granuloma formation.

52 -18) is an important regulator of innate and acquired immune responses and plays an important role in

53 , it represents a potent target for the host acquired immune response, and constitutive expression of

54 he host innate immune response, modulate the acquired immune response, and evade intracellular destru

55 uces both a strong inflammatory and a strong acquired immune response, and Mtb then actively regulate

56 nce the sensitization phase of at least some acquired immune responses, and can contribute to the pat

57 t Candida infection involves both innate and acquired immune responses, and cytokines produced by mon

58 Thus, the acquired immune response appeared to be functional in My

59 nate/proinflammatory and some aspects of the acquired immune response are up-regulated to maintain a

60 The acquired immune responses are crucial to the survival of

61 Our data indicated that vigorous innate and acquired immune responses are elicited, activated, and r

62 ammatory pathways, and the host's innate and acquired immune responses are leading to identification

63 of macrophages as participants in innate and acquired immune responses are regulated by the specific

64 CNS inflammation associated with adaptive or acquired immune responses are uncertain.

65 In summary, CCR4 modulates both innate and acquired immune responses associated with chronic fungal

66 , development, expression, and regulation of acquired immune responses, both those associated with Ig

67 HLA class I molecules are essential to the acquired immune response, but they are also important in

68 drivers of tissue remodeling in established acquired immune responses, but also may contribute to th

69 ue environment essential for generating this acquired immune response by reversing the LN defect in l

70 es during the innate response may modify the acquired immune response by T cells.

71 type-I IFNs can influence the generation of acquired immune responses by modifying T-helper cell dif

72 In addition to the conventional innate and acquired immune responses, complex organisms have evolve

73 To investigate how the acquired immune response could contribute to osteolytic

74 st defense until the slower, more definitive acquired immune response develops.

75 , we investigated the role of the innate and acquired immune responses elicited by Chlamydophila pneu

76 , including mechanisms related to innate and acquired immune responses following gamma radiation.

77 CpG ODN also enhance the development of acquired immune responses for prophylactic and therapeut

78 Evasion of the acquired immune response in African trypanosomes is prin

79 Evidence for a CD8 antigen-driven acquired immune response in psoriatic synovium and blood

80 esent study addresses the causal role of the acquired immune response in the selection of TprK varian

81 rtance of cross-talk between host innate and acquired immune responses in host mucosal defense.

82 of malarial Ags that are capable of inducing acquired immune responses in the fetus.

83 ctivation of key genes involved in adaptive (acquired) immune responses, including major histocompati

84 Activation of the acquired immune response increased osteoclastogenesis an

85 The role of PKCtheta for the acquired-immune responses involved in the development of

86 Development of the acquired immune response is dependent on the signaling o

87 components provides a mechanism by which the acquired immune response may be tailored to specific pat

88 Understanding the basis of these acquired immune responses may be critical in developing

89 Activation of mammalian innate and acquired immune responses must be tightly regulated by e

90 ve antigens are specifically targeted by the acquired immune response of the host and are able to ind

91 b) is able to manipulate both the innate and acquired immune response of the host.

92 ear to be required for the development of an acquired immune response, presumably by functioning in a

93 involved in controlling/resolving innate or acquired immune responses so as to provide a framework f

94 orders and other immunoglobulin E-associated acquired immune responses that it can be difficult to th

95 We propose that, in addition to acquired immune responses, the innate immune system and

96 m, has been implicated in the development of acquired immune responses, though its roles are largely

97 w that IFN-beta influences the generation of acquired immune responses through its regulation of CD40

98 al chitin, such as involvement in innate and acquired immune responses, tissue remodeling, fibrosis,

99 ciated with DHF than primary infections, the acquired immune response to dengue, both B cells and T c

100 e of CD4(+) helper T cells in modulating the acquired immune response to herpes simplex virus type 1

101 s of this study to dissect the nature of the acquired immune response to infection with Listeria mono

102 r gammadelta receptor-bearing T cells in the acquired immune response to infection with Mycobacterium

103 An effective acquired immune response to infectious agents mediated b

104 crucial role of the spleen in the innate and acquired immune response to malaria, there is little inf

105 paB (NF-kappaB) is central to the innate and acquired immune response to microbial pathogens, coordin

106 To investigate mechanisms by which the acquired immune response to Porphyromonas gingivalis cou

107 dicated that deficiency of DbpA/B allows the acquired immune response to restrict early dissemination

108 im of this study was to determine whether an acquired immune response to toxin A, during an episode o

109 ion that play an important role in governing acquired immune responses to a variety of foreign antige

110 IL-10 could have an effect on innate and acquired immune responses to B. burgdorferi and influenc

111 nnate immune responses, but they also affect acquired immune responses to infection.

112 of PA-X in counteracting the host innate and acquired immune responses to influenza virus, an importa

113 pact on viral growth and the host innate and acquired immune responses to influenza virus.

114 Acquired immune responses to malaria have widely been pe

115 In acquired immune responses to parasites or allergens, the

116 ion is only transient due to host innate and acquired immune responses to the virus.

117 cause NS5 may interfere with both innate and acquired immune responses to virus infection, this prote

118 Additionally, the strength of vertebrate acquired immune responses varies dramatically depending

119 Once an acquired immune response was initiated, the P. carinii f

120 Their presence seems to reflect an acquired immune response, which is not translated into a

121 smodium infections trigger strong innate and acquired immune responses, which can lead to severe comp

122 An acquired immune response--with the participation of CD4+