ライフサイエンスコーパス: acquired tolerance

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1 ly defined mechanisms of alloengraftment and acquired tolerance.

2 f both neonatal tolerance and other forms of acquired tolerance.

3 plays an important role in the induction of acquired tolerance.

4 of allopeptides in the adult animal induces acquired tolerance.

5 tory T cells in the development of naturally acquired tolerance.

6 We set out to characterize the retention of acquired tolerance and in the process uncovered two dist

7 echanisms are necessary for the induction of acquired tolerance and prevention of CAV in porcine hear

8 e mechanisms of alloengraftment and variable acquired tolerance can be facilitated by minimum posttra

9 into the mechanisms of organ engraftment and acquired tolerance has made it possible to facilitate th

10 er-regulation (controlling inflammation) and acquired tolerance in T cells.

11 e signaling may be required for induction of acquired tolerance in vivo.

12 o investigate the role of B cells as APCs in acquired tolerance induced by low dose soluble protein A

13 One hallmark of acquired tolerance is bystander suppression, a process w

14 ion of the mechanisms of alloengraftment and acquired tolerance, it was realized that such heavy prop

15 for relapse after bone marrow transplant is acquired tolerance of allogeneic minor histocompatibilit

16 rance) that linked organ engraftment and the acquired tolerance of bone marrow transplantation and ev

17 e same organ donor, and (3) the induction of acquired tolerance prevents the development of CAV.

18 This systemic acquired tolerance (SAT) consists of reduced tissue dama

19 G-derived MHC class I peptide induces active acquired tolerance similar to results obtained with the

20 allopeptide-primed host T cells might induce acquired tolerance through their interaction with thymic

21 o identification of transplant patients with acquired tolerance to an allograft that can be studied.

22 Thus, acquired tolerance to arsenic is associated with increas

23 We found that de novo and acquired tolerance to BETi is driven by the robustness o

24 rafted without causing GVHD, suggesting that acquired tolerance to disparate unrelated HLA antigens w

25 pathogenesis of CAV and to determine whether acquired tolerance to donor antigen can prevent the deve

26 fetus and confirms the potential of actively acquired tolerance to facilitate prenatal therapeutic ap

27 As a consequence, Nf1-/- cells acquired tolerance to proteotoxic stress.

28 On the other hand, acquired tolerance to severe heat shock was associated w

29 beta1 (TGFbeta-1), indicating that they have acquired tolerance to this normally growth inhibitory cy

30 As in conventional mice, acquired tolerance was dominant, such that naive monospe

31 Armed with the knowledge of actively acquired tolerance, we attempted to prenatally abolish o

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