ライフサイエンスコーパス: acrodermatitis

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1 ons; in the case of one patient two separate acrodermatitis chronica atrophicans lesions were culture

2 re isolated in culture from skin biopsies of acrodermatitis chronica atrophicans lesions; in the case

3 cells and macrophages in the skin lesions of acrodermatitis chronica atrophicans patients had very li

4 s with erythema migrans and 27 patients with acrodermatitis chronica atrophicans were analyzed for mR

5 onic skin manifestation of Lyme borreliosis, acrodermatitis chronica atrophicans, and infection by B.

6 restricted pattern of cytokine expression in acrodermatitis chronica atrophicans, including the lack

7 In contrast, acrodermatitis chronica atrophicans, the typical cutaneo

8 es collected from patients with well-defined acrodermatitis chronica atrophicans.

9 t, but not the exclusive, etiologic agent of acrodermatitis chronica atrophicans.

10 localized (palmoplantar pustular psoriasis, acrodermatitis continua of Hallopeau) and generalized va

11 psoriasis, palmoplantar pustular psoriasis, acrodermatitis continua of Hallopeau, and acute generali

12 h a clinical presentation similar to that of acrodermatitis enteropathica (AE) in humans.

13 Acrodermatitis enteropathica (AE) is a rare autosomal re

14 The rare inherited condition acrodermatitis enteropathica (AE) results from a defect

15 s identified because of its association with acrodermatitis enteropathica (AE), a genetic disorder of

16 strated dramatically by the genetic disorder acrodermatitis enteropathica (AE).

17 homeostasis, a process that is defective in acrodermatitis enteropathica in humans.

18 Ectodomain cleavage is inhibited by acrodermatitis enteropathica mutations near a predicted

19 ecessive genetic disorder of zinc metabolism acrodermatitis enteropathica, but the physiological func

20 porter mutated in the human genetic disorder acrodermatitis enteropathica.

21 for the genetic disorder of zinc metabolism acrodermatitis enteropathica.

22 pellagra, necrolytic migratory erythema, and acrodermatitis enteropathica.

23 f inherited nutritional deficiencies such as acrodermatitis enteropathica.

24 he rare, autosomal recessive genetic disease acrodermatitis enteropathica.

25 nt in humans is the zinc deficiency disease, acrodermatitis enteropathica.

26 s suggest that heterozygous mutations in the acrodermatitis gene Zip4 may be associated with a wider

27 deficiencies causing a periorificial and/or acrodermatitis: zinc deficiency, biotin deficiency, kwas

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