ライフサイエンスコーパス: actin microfilament

1 everse transcription was dependent on intact actin microfilaments.

2 leocapsids are specifically localized on the actin microfilaments.

3 mmunolocalization of the junction-associated actin microfilaments.

4 a occludens 1 and of the junction-associated actin microfilaments.

5 like bodies (I-LBs) move in association with actin microfilaments.

6 and (5) an association of the receptor with actin microfilaments.

7 which contained fine fibers the diameter of actin microfilaments.

8 PCs) involve the assembly and disassembly of actin microfilaments.

9 Dys-ABD alone associated with actin microfilaments.

10 omplement factor C3 and that uptake requires actin microfilaments.

11 otubules was independent of the integrity of actin microfilaments.

12 uently associated with microtubules and with actin microfilaments.

13 on of spyA in HeLa cells resulted in loss of actin microfilaments.

14 uding the CH domain interacted directly with actin microfilaments.

15 ulating filaments had the same dimensions as actin microfilaments.

16 eletal proteins is implicated in stabilizing actin microfilaments.

17 ut not alphavbeta3 particle binding required actin microfilaments.

18 a colocalization of the BCCV N protein with actin microfilaments.

19 s are dependent upon cytoplasmic networks of actin microfilaments (6 nm), intermediate filaments (10

20 that axial strains caused by the sliding of actin microfilaments about the fixed integrin attachment

21 ntral surface and exhibits a localization of actin microfilaments along the free edges of the cells,

22 While disruption of the cellular actin microfilament and microtubule by cytochalasin D an

23 Coordinated actin microfilament and microtubule dynamics is required

24 is originated mostly from the remodeling of actin microfilaments and adhesion complexes, to less ext

25 Junction-associated actin microfilaments and cell density were unchanged.

26 the peripheral cytoskeleton, disassembly of actin microfilaments and disaggregation of microtubules

27 stent and involved extensive organization of actin microfilaments and focal adhesions.

28 Depolymerization of the actin microfilaments and inhibition of the Arp2/3 comple

29 li cell injury through disruptive effects on actin microfilaments and microtubule (MT) organization a

30 plants may employ unique KCHs to coordinate actin microfilaments and microtubules during cell growth

31 erted its regulatory effect by disorganizing actin microfilaments and microtubules in Sertoli cells s

32 res composed of an interconnected network of actin microfilaments and microtubules when mechanical st

33 t trajectories, and is dependent upon intact actin microfilaments and myosin motors, since treatment

34 nism that was dependent on polymerization of actin microfilaments and on a functional cytoskeleton, a

35 is effect does not require interactions with actin microfilaments, and it is possible that other acti

36 e, chromatin condensation, reorganization of actin microfilament architecture, and extensive detachme

37 When LIMKs are inhibited, actin microfilaments are disorganized and microtubules a

38 ominant, but both intermediate filaments and actin microfilaments are involved in dynamic cross-linki

39 centriolar material (PCM) fails to assemble, actin microfilaments are not organized into furrows at t

40 major protein of muscle thin filaments, and actin microfilaments are the main component of the eukar

41 Disruption of the actin microfilament array by cytochalasin D treatment of

42 me and nuclear envelope motility depended on actin microfilaments as well as tubulin.

43 and Arg colocalize with each other and with actin microfilaments at the apical surface of the develo

44 In summary, plastin 3 is a regulator of actin microfilament bundles at the ES in which it dictat

45 These actin microfilament bundles require rapid debundling to

46 increased interprocess spacing and haphazard actin microfilament bundles.

47 ndicating a requirement for rearrangement of actin microfilaments but less dependence on tyrosine kin

48 nd to be associated with transverse-cortical actin microfilaments, but never with axial actin cables

49 rms motile granules that are associated with actin microfilaments, but not with microtubules.

50 nges were the result of an alteration of the actin microfilaments, converting from their bundled to b

51 red in the absence of intact microtubule and actin microfilament cytoskeletal elements.

52 nd their structure is regulated primarily by actin microfilaments, cytoskeletal proteins present in h

53 reading is dependent on the integrity of the actin microfilament cytoskeleton, we sought to determine

54 The small G-protein Rho regulates the actin microfilament-dependent cytoskeleton.

55 ultinucleate cells, likely by inhibiting the actin microfilament-dependent step of cytokinesis.

56 Finally, the effect of actin microfilament depolymerization on total release is

57 nsitive to treatment with cytochalasin D, an actin microfilament-depolymerizing drug.

58 In contrast, the depolymerization of actin microfilaments did not have any effect on virus bi

59 -dependent process, since treatment with the actin microfilament disrupter cytochalasin D prevented i

60 rane properties with cholesterol removal and actin microfilament disruption.

61 reversibly stabilized microtubules, blocked actin microfilament dynamics, inhibited cell motility in

62 t study was to evaluate the effect of BFT on actin microfilaments (F-actin) and cell volume.

63 ons requires the formation of filopodia from actin microfilaments (F-actin) and their engorgement wit

64 their ECM, the attached ECs rearrange their actin microfilaments first into peripheral stress fibers

65 Actin microfilaments form a three-dimensional cytoskelet

66 Inhibition of actin microfilaments had the greatest effect on bulk com

67 Our results support the notion that actin microfilaments impose a barrier for mobilization o

68 -permeability barrier, causing disruption of actin microfilaments in cell cytosol, perturbing the loc

69 dynamic interaction between microtubules and actin microfilaments in cotton fibers.

70 al tight junctional ring and thickening of F-actin microfilaments in focal contacts at the basolatera

71 d cell death, highlighting the importance of actin microfilaments in rituximab/milatuzumab-mediated c

72 in roles in other cell types, is to assemble actin microfilaments in support of photoreceptor disk mo

73 The role of actin microfilaments in synaptic transmission was tested

74 th F-actin in vivo and that can cross-link F-actin microfilaments in vitro.

75 result of CRB3 KD-induced re-organization of actin microfilaments, in which actin microfilaments were

76 endocytosis via the accumulation of cortical actin microfilaments induced by the ROP2 effector protei

77 Cytochalasin D, an agent which disrupts actin microfilaments, inhibited BN- and TPA-induced tyro

78 crylamide, or colchicine was used to disrupt actin microfilaments, intermediate filaments, or microtu

79 an actin-dependent manner and to cross-link actin microfilaments into higher-order structures has be

80 ccur either through directed transport along actin microfilaments into one daughter cell or through c

81 These results suggest that actin microfilaments may interact, either directly or in

82 Actin microfilaments mediated force transfer to the nucl

83 levation, apical recruitment of p150(Glued), actin microfilament meshwork organization, and ultrastru

84 ors embedded in membrane microdomains induce actin-microfilament meshwork formation, anchoring microt

85 Actin microfilament (MF) organization and remodelling is

86 To determine whether actin microfilament (MF) organization is correlated with

87 l changes due to structural modifications in actin microfilaments (MFs) and microtubules (MTs).

88 s is structured by a scaffolding composed of actin microfilaments, microtubules, and intermediate fil

89 ed by the presence of an array of bundles of actin microfilaments near the Sertoli cell plasma membra

90 related to its known ability to disrupt the actin microfilament network and consequently to affect c

91 ized cell phenotype and the integrity of the actin microfilament network are important cellular deter

92 With a close examination of the F-actin microfilament network, these findings show that Pa

93 Unstretched HTM cells displayed a diffuse F-actin microfilament network, whereas stretched cells exh

94 mechanism that utilizes the microtubule and actin microfilament network.

95 le of the polymeric form of actin, i.e., the actin microfilaments of the cytoskeletal framework, in t

96 M to the nucleus, endoplasmic reticulum, and actin microfilaments of the cytoskeleton in response to

97 domain of vitronectin resulted in changes in actin microfilament organization and the subcellular dis

98 o act by depolymerizing actin and disrupting actin microfilament organization.

99 eleton, having a role in adhesion-plaque and actin-microfilament organization.

100 These results strongly suggest that actin microfilaments play an important role in the repli

101 plants with cytochalasin E, an inhibitor of actin microfilament polymerisation.

102 reatment with cytochalasin D, which disrupts actin microfilaments, prevented the calcitonin-induced H

103 In summary, CRB3 is an actin microfilament regulator, playing a pivotal role in

104 sassemble actin microfilaments, we show that actin microfilament remodeling is part of fenestra bioge

105 milarly, wortmannin inhibited hep I-mediated actin microfilament reorganization and the hep I-induced

106 tes RIC4 to promote the assembly of cortical actin microfilaments required for localized outgrowth.

107 s concomitant to increased polymerization of actin microfilaments resulting in decreased G- to F-acti

108 ves the elongated oocyte microvilli, rich in actin microfilaments, since it can be blocked by the mic

109 titatively controlled conditions, to perturb actin microfilament structure and assembly in an attempt

110 of these channels requires interactions with actin microfilaments subjacent to the plasma membrane.

111 ngle-domain proteins which directly link the actin microfilament system to a variety of signalling pa

112 ulated by both phospholipase D (PLD) and the actin microfilament system.

113 n yeast, plant and animal cells that confers actin microfilaments their bundled configuration.

114 Sertoli cell cytosol, causing truncation of actin microfilament, thereby failing to support the Sert

115 the cytoskeletal protein vinculin, connects actin microfilaments to the intercalated disk and membra

116 , are large cytoskeletal proteins which link actin microfilaments to the plasma membrane.

117 (1) cell phenotype using antibodies to alpha-actin (microfilaments), vimentin and desmin (intermediat

118 eover, the association of the N protein with actin microfilaments was confirmed by coimmunoprecipitat

119 The localization of reverse transcription to actin microfilaments was mediated by the interaction of

120 use of agents that stabilize or disassemble actin microfilaments, we show that actin microfilament r

121 ganization of actin microfilaments, in which actin microfilaments were truncated, and extensively bra

122 termined by centrifugal cosedimentation with actin microfilaments, where bound protein is separated f

123 Actin microfilaments, which are prominent in pollen tube

124 Disruption of actin microfilaments, which causes delocalization of Bif

125 ication, resulting in the re-organization of actin microfilaments, which rendered them similar to tho

126 n of microtubules with colchicine (Colch) or actin microfilaments with cytochalasin D (CD) dramatical

127 cone actin cytoskeleton, because disrupting actin microfilaments with cytochalasin D or stabilizing