ライフサイエンスコーパス: actinic

1 Actinic damage and squamous atypia were found in grafts

2 posure instrument and histologic evidence of actinic damage in a population-based study of keratinocy

3 radiation and therefore develop accelerated actinic damage, including cancer, on sun-exposed tissue.

4 e exposure related to histologic evidence of actinic damage.

5 Chronic actinic dermatitis (CAD) is classically described in old

6 Chronic actinic dermatitis presents with an earlier age at onset

7 The primary donor recovery kinetics after an actinic flash revealed a pronounced dependence on the ti

8 hange in absorbance at 478 nm after a single actinic flash was delivered to the sample.

9 lengthy preillumination of a sample and the actinic flash.

10 These states were detected both by short actinic flashes and by the switching of the actinic illu

11 n cool Earth's surface, they also change the actinic flux of ultraviolet (UV) radiation.

12 B <-- X spectrum is combined with the solar actinic flux to estimate an atmospheric lifetime for CH(

13 Conjunctival actinic granuloma is an underrecognized entity.

14 Diagnosis of actinic granuloma of the conjunctiva was confirmed.

15 This led to the final diagnoses of actinic granuloma of the conjunctiva.

16 ), but were diagnosed histopathologically as actinic granuloma.

17 -type bacteriorhodopsin photocycle formed by actinic illumination at 230 K has been determined by x-r

18 vailability of inorganic carbon, and various actinic illumination conditions.

19 actinic flashes and by the switching of the actinic illumination level between different stationary

20 thylakoid PMF is actually present long after actinic illumination of the thylakoids ceases and that t

21 PMF decays rapidly after the termination of actinic illumination, and it has been well established v

22 conditions and in air (21 percent O2) at an actinic intensity of 200 microeinsteins per square meter

23 This was achieved by applying actinic irradiance perpendicular to one side of thallus

24 nd/or overestimations, depending on incident actinic irradiance.

25 Precancerous actinic keratoses (AKs) (17) and SCCs (27) expressed two

26 Actinic keratoses (AKs) are small scaly red areas of ski

27 ed to be effective in reducing the number of actinic keratoses (AKs) for up to 6 months, but no rando

28 pical methyl 5-aminolevulinic acid (MAL) for actinic keratoses (AKs) is as effective as conventional

29 osmetically favorable treatment modality for actinic keratoses (AKs).

30 SCCs (n = 11) than in cSCC in situ (n = 69), actinic keratoses (n = 63), and normal skin (n = 5).

31 The percentage of reduction in the number of actinic keratoses (primary outcome), local skin reaction

32 At 3-monthly visits, new actinic keratoses and basal-cell carcinomas were identif

33 ilar mutations are seen in later precancers (actinic keratoses and Bowen's disease).

34 veness of promoting sunscreen use to prevent actinic keratoses and nonmelanoma skin cancer.

35 rom noncancerous skin abnormalities, such as actinic keratoses and seborrheic keratoses, achieving 92

36 s of the mutant p53 clusters associated with actinic keratoses and squamous cell carcinomas.

37 Treating actinic keratoses and the surrounding skin area (i.e., f

38 ting a CD4+ T cell-mediated immunity against actinic keratoses and, potentially, cancers of the skin

39 Actinic keratoses arose in 9% of grafts treated with ult

40 itu SCCs in the 5 years prior to enrollment, actinic keratoses count at enrollment, a history of ever

41 ia verruciformis keratosis and in some cases actinic keratoses demonstrated similar histology associa

42 and basal-cell carcinomas and the number of actinic keratoses during the 12-month intervention perio

43 prevalence of untreated skin malignancy and actinic keratoses in high-risk kidney and liver transpla

44 he rates of new nonmelanoma skin cancers and actinic keratoses in high-risk patients.

45 versus 26.3% mean reduction in the number of actinic keratoses in participants (P < 0.0001).

46 Actinic keratoses on the face and ears were counted by s

47 studies, we randomly assigned patients with actinic keratoses on the face or scalp or on the trunk o

48 i67 positive cells, unlike the situation for actinic keratoses or basal cell carcinomas.

49 therapy for 2 or 3 consecutive days to treat actinic keratoses produced clinically relevant sustained

50 a promising option to evaluate emerging new actinic keratoses therapies.

51 Dermatologists treat actinic keratoses to prevent non-melanoma skin cancer.

52 The number of actinic keratoses was 11% lower in the nicotinamide grou

53 The annualised rate of new actinic keratoses was 8.2 among the patients assigned T4

54 protective measures, other skin cancers, and actinic keratoses were not.

55 c therapy (PDT) is used extensively to treat actinic keratoses(AKs).

56 and 5 of 11 patients [45.5%] diagnosed with actinic keratoses).

57 Actinic keratoses, a precancerous benign skin growth and

58 cinomas, five squamous cell carcinomas, five actinic keratoses, and one case of Bowen's disease) and

59 l cell carcinomas, squamous cell carcinomas, actinic keratoses, atypical nevi, melanocytic nevi, blue

60 hat both methods are viable ways to evaluate actinic keratoses, even when the investigators differ at

61 , scaly plaques, and large tumors resembling actinic keratoses, SCC in situ, and SCCs, respectively.

62 all and those for presumptive skin cancer or actinic keratoses, skin biopsies, or PCP diagnostic accu

63 gonist approved for the topical treatment of actinic keratoses, superficial basal cell carcinoma, and

64 nic NRF2 target genes were activated in some actinic keratoses, the major precancerous lesion in huma

65 gnificantly younger, had more nevi but fewer actinic keratoses, were more likely to report a family h

66 ociated with premalignant lesions resembling actinic keratoses, whereas those in the heterozygous and

67 CC) as well as dysplastic precursor lesions, actinic keratoses.

68 ture abnormalities, pigment dyschromias, and actinic keratoses.

69 ent, with approximately 30% having 5 or more actinic keratoses.

70 ar lentigines or seborrheic keratoses, and 3 actinic keratoses.

71 n and to reduce the rate of new premalignant actinic keratoses.

72 proteins in human skin SCC and precancerous actinic keratoses.

73 Cs and their premalignant precursor lesions, actinic keratoses.

74 o 3 days is effective for field treatment of actinic keratoses.

75 rapy) can eradicate clinical and subclinical actinic keratoses.

76 l diagnosis, followed by pterygium (36%) and actinic keratosis (19%).

77 Actinic keratosis (AK) is a skin growth induced by UV li

78 (mean time, 6 months [range, 0-18 months]), actinic keratosis (AK) of the same sun-exposed skin area

79 anced malignancies, ranging from a precursor actinic keratosis (AK) to squamous cell carcinoma (SCC)

80 Benefit and harm associated with treating actinic keratosis (AK) with the immune response modifier

81 Actinic keratosis (AK), a skin growth induced by ultravi

82 ion with a distinct precancerous lesion, the actinic keratosis (AK).

83 , squamous cell carcinoma (SCC; n = 28), and actinic keratosis [AK (a precursor to SCC); n = 38].

84 Lastly, we evaluated the Raman signal in actinic keratosis and basal cell carcinoma lesions and p

85 te' stages of epidermal carcinogenesis; late actinic keratosis and cSCC.

86 duction in PTEN levels in human premalignant actinic keratosis and malignant SCCs, supporting a key r

87 riasis and show promise for the treatment of actinic keratosis and squamous cell carcinoma.

88 at TRAF1 expression is up-regulated in human actinic keratosis and squamous cell carcinoma.

89 erved in any cancer-related end points or in actinic keratosis counts.

90 aseline plus 5-FU for the field treatment of actinic keratosis in a randomized, double-blind clinical

91 Actinic keratosis is a common precursor to sun-related s

92 Actinic keratosis is a precursor to cutaneous squamous c

93 rds cSCC: Normal epidermis, solar elastosis, actinic keratosis KIN1-2, advanced actinic keratosis KIN

94 lastosis, actinic keratosis KIN1-2, advanced actinic keratosis KIN3 and well-differentiated cSCC.

95 lated only in invasive SCC nests, but not in actinic keratosis or in situ SCC, compared with normal e

96 Over 80% had actinic keratosis present, with approximately 30% having

97 Evaluation of actinic keratosis therapy depends on reliable measures o

98 normal epidermis, solar elastosis and early actinic keratosis to the 'late' stages of epidermal carc

99 effects have limited the efficacy of current actinic keratosis treatments.

100 ential expression of CD200 in normal skin vs actinic keratosis vs SCC in situ vs invasive SCC.

101 Actinic keratosis was the most common diagnosis associat

102 nd skin lesions resulting from sun exposure (actinic keratosis), and individual Neandertal alleles we

103 treatment of a precancerous skin condition (actinic keratosis), and phorbol derivatives such as resi

104 ogenesis--changes that resemble hyperplastic actinic keratosis, a commonly observed human precancerou

105 tate, which is approved for the treatment of actinic keratosis, a precancerous skin condition.

106 ormal skin, PTEN levels are reduced in human actinic keratosis, a precancerous skin lesion caused by

107 in skin biopsies from 40 healthy donors, 13 actinic keratosis, and 74 cSCC patients.

108 perproliferative diseases such as psoriasis, actinic keratosis, and certain neoplasias.

109 5% hyaluronan inhibits basal cell carcinoma, actinic keratosis, and murine colon-26 growth in vivo.

110 trials for eradicating basal cell carcinoma, actinic keratosis, and squamous cell carcinoma (SCC) in

111 approved for the treatment of genital warts, actinic keratosis, and superficial basal cell carcinoma.

112 oid concentrations in basal cell carcinomas, actinic keratosis, and their perilesional skin demonstra

113 Sun-induced skin lesions, in particular actinic keratosis, are generally considered as premalign

114 nd safety of a new topical field therapy for actinic keratosis, ingenol mebutate gel (0.015% for face

115 were thought clinically to be pingueculitis, actinic keratosis, or ocular surface squamous neoplasia

116 d included the search terms solar keratosis, actinic keratosis, photodynamic therapy, and photochemot

117 n hyperproliferative skin diseases including actinic keratosis, squamous and basal cell carcinoma as

118 -fluorouracil (5-FU) as an immunotherapy for actinic keratosis.

119 treatment of the precancerous skin condition actinic keratosis.

120 *-) radicals ( approximately 5 mus after the actinic laser flash) the transient absorbance of neutral

121 With increasing actinic laser strength, the M-fast cycle at first rises

122 hose with a self-reported history of treated actinic lesions (group 2), and those without either (gro

123 melanoma (group 1; n = 1433) and/or treated actinic lesions (group 2; n = 24,006) were more likely t

124 lly controlled, spatially separated beams of actinic light and a fluorescence imaging system.

125 sses that occur upon rapid shuttering of the actinic light are analyzed.

126 l crystals were trapped by illuminating with actinic light at 230 K, followed by quenching in liquid

127 e (PM) to dilute Triton X-100 eliminates the actinic light effect on the relative amounts of fast M (

128 PAM fluorescence parameters under a range of actinic light intensities (steady-state fluorescence yie

129 ng the same sample to a sequence of distinct actinic light intensities.

130 s fixed) with induction/relaxation kinetics (actinic light intensity is fixed and the response is fol

131 sample by combining the measurement of LCs (actinic light intensity is varied while measuring time i

132 rties of the photocycle caused by changes in actinic light intensity.

133 ifteen-day-old cultures were exposed to blue actinic light or to white fluorescent light for 24 hours

134 n chlorophyll fluorescence after turning off actinic light reflects nonphotochemical reduction of the

135 n of a whole LC, including a large number of actinic light steps and adequate replication, within the

136 as possible explanations for the ability of actinic light to modify the kinetics and pathways of the

137 suggest that this perceptive site gives the actinic light which has traveled downward through the le

138 ht response curves measured under controlled actinic light with the same leaves.

139 ld, (2) the mole fraction of M-fast (at high actinic light) decreased from 50 to 20%, and (3) the tim

140 , biosensor film thickness, intensity of the actinic light, illumination cycle, and temperature on th

141 derived from cells differentially exposed to actinic light, where cells located deeper inside tissues

142 of the BR photocycle to different levels of actinic light.

143 hyll fluorescence increase after turning off actinic light.

144 following excitation with a pulse of 477-nm actinic light.

145 easured quantum yields with even exposure to actinic light.

146 vement in multiple intact plants under white actinic light.

147 These tumours appeared to arise earlier in actinic progression and grew more rapidly than tumours o

148 Actinic prurigo and polymorphic light eruption are two o

149 genetic background, supporting the view that actinic prurigo may represent a human leukocyte antigen-

150 ained through polymorphic light eruption and actinic prurigo probands using segregation analysis.

151 polymorphic light eruption) in relatives of actinic prurigo probands was 23.7%, with a relative risk

152 ght eruption in families ascertained through actinic prurigo probands, as evidence of a shared pathog

153 suggests that polymorphic light eruption and actinic prurigo share a common genetic background, suppo

154 No appreciable absorption in the solar actinic region (lambda > 290 nm) was observed, leaving t

155 protect the retina from damage by absorbing actinic short-wave light (analogous to internal sunglass

156 (represented by prior sunburns and signs of actinic skin damage identified by dermatologists), age,

157 ranging from <.001 to .04 when adjusted for actinic skin damage on the hands).

158 sex, age, sunburns in the past, and signs of actinic skin damage, the associations remained significa

159 g from <.001 to .01 when adjusted for dorsal actinic skin damage; and OR, 2.54 [95% CI, 1.76-3.67] fo

160 g from <.001 to .04 when adjusted for facial actinic skin damage; OR, 2.36 [95% CI, 1.62-3.43] for R/

161 and no more than five destructively treated actinic skin lesions.

162 ect seen particularly in those with previous actinic skin tumors.

163 Here we characterize the actinic step of DASA-photoswitching and its key intermed

164 unds that readily absorb in the tropospheric actinic window (ionic organic complexes, PAHs, aromatic