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1 osphate response element binding protein and activating transcription factor.
2 me stimulates monocytes through induction of activating transcription factor 1 (ATF-1).
3 sponse element binding protein 1 (CREB1) and activating transcription factor 1 (ATF1) are closely rel
4 rthermore, we found that cdk3 phosphorylates activating transcription factor 1 (ATF1) at serine 63 an
5 sponsive element binding protein (CREB), and activating transcription factor 1 (ATF1) pathway is invo
6 esponsive element binding protein (CREB) and activating transcription factor 1 (ATF1), transcription
7                     Here we found that ATF1 (activating transcription factor 1) is a transcriptional
8 ic protein EWS-ATF1 (Ewing's sarcoma protein-activating transcription factor 1), which contains the D
9                    The 5' element binds CREB/activating transcription factor 1, and the 3' element is
10 ent of CREB transcription factors (CREB1 and activating transcription factor-1) in this functional re
11 nse element-binding protein (CREB) and ATF1 (activating transcription factor-1).
12 3) cAMP-responsive binding protein (P-CREB), activating transcription factor-1, and CREB-induced CRE
13  chemokine promoter association of CREB1 and activating transcription factor-1.
14 rect regulation of transcription mediated by activating transcription factor 2 (ATF-2) and (ii) H(2)O
15 sphorylation of JNK, PKCdelta, p38 MAPK, and activating transcription factor 2 (ATF-2) in RA ST fibro
16 t the attachment of CREB binding protein and activating transcription factor 2 (ATF-2) to the cAMP-re
17                                              Activating transcription factor 2 (ATF2) belongs to the
18 er showed an increase of Thr(P)-71-Thr(P)-69-activating transcription factor 2 (ATF2) binding in resp
19                                              Activating transcription factor 2 (ATF2) is regulated by
20                                              Activating transcription factor 2 (ATF2) regulates trans
21 y suppressing expression via binding through activating transcription factor 2 (ATF2) to the cyclic a
22 in transcription factors including c-jun and activating transcription factor 2 (ATF2) upon activation
23 -response element binding protein (CREB) and activating transcription factor 2 (ATF2), function as a
24 we identify four transcription factors, JUN, activating transcription factor 2 (ATF2), myocyte-specif
25 am transcription factor substrates c-Jun and activating transcription factor 2 (ATF2).
26 phages, only BPPcysMPEG enhanced p38MAPK and activating transcription factor 2 activation.
27 of interferon regulatory factor 3 (IRF3) and activating transcription factor 2 in DCs.
28                Instead, low-dose LPS induces activating transcription factor 2 through Toll-interacti
29                  Transcription factors ATF2 (activating transcription factor 2) and ATF7 (activating
30 c tumors, controlled the expression of ATF2 (activating transcription factor 2).
31 nstream effectors: phosphorylation of c-Jun, activating transcription factor 2, and E twenty-six-like
32 lation of the transcription factors, Jun and activating transcription factor 2, in addition to activa
33 ation of the transcription factors c-Jun and activating transcription factor 2, which in turn stimula
34 ylation of cAMP response-element binding and activating transcription factor 2, which leads to transa
35  maintenance of chromatin protein 1, but not activating transcription factor 2.
36 at down-stream effectors of p38 MAPK, namely activating transcription factor-2 (ATF-2) and cyclic AMP
37 found that p38 target transcriptional factor activating transcription factor-2 (ATF-2) bound to ATF s
38                                          The activating transcription factor-2 (ATF-2) mRNA encodes a
39 n transcription factors, including c-Jun and activating transcription factor-2 (ATF-2) upon activatio
40 ow that this is dependent on both ERK1/2 and activating transcription factor-2 (ATF-2).
41                                     However, activating transcription factor-2 (ATF2) expression was
42  we show that under non-stressed conditions, activating transcription factor-2 (ATF2) in cooperation
43                     The crucial functions of activating transcription factor-2 (ATF2) in the developm
44 E(2) or Bt(2)cAMP + PDA stimulated c-Jun and activating transcription factor-2 (ATF2) phosphorylation
45  IFN regulatory factor-1 and upregulation of activating transcription factor-2 and c-Jun, transcripti
46 d activation of transcription factors c-Jun, activating transcription factor-2 and, in addition, NF k
47 o kinase assay with [gamma-(32)P]ATP and GST-activating transcription factor-2 as substrate.
48 reduced, there was no reduction in pulmonary activating transcription factor-2 expression, arguing ag
49   Investigation of JNK targets revealed that activating transcription factor-2 is phosphorylated unde
50 vation specifically activated c-Jun, but not activating transcription factor-2 or JunD, and increased
51                          IFN-gamma recruited activating transcription factor-2 via p38 to the TNF-alp
52                              Although dermal activating transcription factor-2, a downstream p38 MAPK
53  of p38 mitogen-activated protein kinase and activating transcription factor-2, which were blocked by
54 eam of the JNK and p38 pathways as c-Jun and activating transcription factor-2.
55 ressor of cytokine signaling 3 (SOCS-3), and activating transcription factor 3 (ATF-3), which termina
56 te the induction of the stress/injury marker activating transcription factor 3 (ATF-3).
57 ic stress-induced UPR and cell death through activating transcription factor 3 (ATF3) and C/EBP homol
58 ound that microRNA-494 binds to the 3'UTR of activating transcription factor 3 (ATF3) and decreases i
59 C irradiation, immediate early genes such as activating transcription factor 3 (ATF3) are overexpress
60                          Here we report that activating transcription factor 3 (ATF3) bound common mu
61                                              Activating transcription factor 3 (ATF3) can repress Nrf
62 8 mitogen activated protein kinase (p38MAPK)-activating transcription factor 3 (ATF3) dependent pathw
63 1) phosphorylation and subsequently enhances activating transcription factor 3 (ATF3) expression inde
64                            Expression of the activating transcription factor 3 (ATF3) gene is induced
65                                              Activating transcription factor 3 (ATF3) has been propos
66                        Demyelination induced activating transcription factor 3 (ATF3) in DRG neurons.
67            A recent study showed the role of activating transcription factor 3 (ATF3) in SCC developm
68                                              Activating transcription factor 3 (ATF3) is a basic leuc
69                                              Activating transcription factor 3 (ATF3) is a common med
70                                              Activating transcription factor 3 (ATF3) is a common str
71                                              Activating transcription factor 3 (ATF3) is a key mediat
72                                              Activating transcription factor 3 (ATF3) is a member of
73                                              Activating transcription factor 3 (ATF3) is a member of
74                                              Activating transcription factor 3 (ATF3) is a negative r
75                                              Activating transcription factor 3 (ATF3) is a prime cand
76                                              Activating transcription factor 3 (ATF3) is an important
77                                              Activating transcription factor 3 (ATF3) is another tran
78                                              Activating transcription factor 3 (Atf3) is rapidly and
79                                              Activating transcription factor 3 (ATF3) promotes neuron
80                                              Activating transcription factor 3 (ATF3) responds to div
81                          Here we report that activating transcription factor 3 (ATF3), a broad DNA da
82            Previous studies demonstrate that activating transcription factor 3 (ATF3), a common stres
83                          Here we report that activating transcription factor 3 (ATF3), a common stres
84          In this report, we demonstrate that activating transcription factor 3 (ATF3), a hub of the c
85 roup of genes that appear to be regulated by activating transcription factor 3 (ATF3), a member of th
86  issue of Blood, Boespflug et al report that activating transcription factor 3 (ATF3), a member of th
87                                Expression of activating transcription factor 3 (ATF3), a nuclear calc
88            Here we report the discovery that activating transcription factor 3 (ATF3), a stress respo
89                We tested the hypothesis that activating transcription factor 3 (ATF3), a stress-induc
90                                              Activating transcription factor 3 (ATF3), an NMD target
91 with rapid induction of heat shock proteins, activating transcription factor 3 (ATF3), and CHOP.
92 or p75NTR, choline acetyltransferase (ChAT), activating transcription factor 3 (ATF3), and cleaved ca
93 itor of differentiation/DNA binding 2 (Id2), activating transcription factor 3 (Atf3), and the phosph
94  72 h after surgery, there is an increase in activating transcription factor 3 (ATF3), the neuropepti
95  expression of the transcriptional regulator activating transcription factor 3 (ATF3), which we show
96                         Here, we report that activating transcription factor 3 (ATF3)-a broad stress
97 or-activated receptor-gamma (PPARgamma), and activating transcription factor 3 (ATF3).
98 abeling for the stress and injury-associated activating transcription factor 3 (ATF3).
99 and increased the mRNA and protein levels of activating transcription factor 3 (ATF3).
100 ssociated with nerve injury and cell stress, activating transcription factor 3 and growth-associated
101 gly, four other genes, cylindromatosis, CD9, activating transcription factor 3 and oxytocin receptor,
102 d to activation of the transcription factors activating transcription factor 3 and protooncogene c-fo
103                                              Activating transcription factor 3 attenuates chemokine a
104 xpression, and nerve damage was evaluated by activating transcription factor 3 expression.
105 dent, whereas induction of cystathionase and activating transcription factor 3 was Nur77 independent.
106  apoptosis in prostate cancer cells by ATF3 (activating transcription factor 3) expression.
107 f the transcription factors c-Jun and ATF-3 (activating transcription factor 3), known regulators of
108 totic responses, including induction of p27, activating transcription factor 3, and nonsteroidal anti
109 ansection models, the upregulation of c-Jun, Activating transcription factor 3, Heat shock protein 27
110 ugh interaction with transcription repressor activating transcription factor 3.
111 e induction of another transcription factor, activating transcription factor 3.
112 amined a marker of cell injury/regeneration (activating transcription factor 3; ATF3), macrophage hyp
113                From the candidates selected, activating transcription factor-3 (ATF-3), caveolin-1, d
114             Importantly, transcripts for the activating transcription factor-3 (Atf3) and mitochondri
115                                              Activating transcription factor-3 (ATF3) is rapidly indu
116 er transcripts, including neuropeptide Y and activating transcription factor-3, are upregulated norma
117  anti-inflammatory drug-activated gene-1 and activating transcription factor-3.
118  Several ER stress/UPR genes, including BiP, activating transcription factor 4 (ATF-4), ATF-6, and a
119  mammalian stress response pathway regulator activating transcription factor 4 (ATF-4).
120 trends toward increasing downstream signals, activating transcription factor 4 (ATF4) and ATF6 indica
121 h preferential translation of mRNAs, such as activating transcription factor 4 (ATF4) and C/EBP-homol
122 UPR was activated, as the expression of both activating transcription factor 4 (ATF4) and CHOP (DDIT3
123 rotein synthesis but enhances translation of activating transcription factor 4 (ATF4) and is a crucia
124                In silico analysis identified activating transcription factor 4 (ATF4) as a potential
125 ypes of mitochondrial stressors, we identify activating transcription factor 4 (ATF4) as the main reg
126  of FGF21 transcription was conferred by two activating transcription factor 4 (ATF4) binding sites i
127 sphorylation of eIF2alpha, which upregulates activating transcription factor 4 (ATF4) expression.
128 culum stress pathway, linked functionally to activating transcription factor 4 (ATF4) following treat
129 ed protein response (UPR) and an increase in activating transcription factor 4 (ATF4) has been previo
130 ve was to investigate the protective role of activating transcription factor 4 (ATF4) in controlling
131              We evaluate a potential role of activating transcription factor 4 (Atf4) in invertebrate
132 d the signaling pathway of ER stress-induced activating transcription factor 4 (ATF4) in the regulati
133                                              Activating transcription factor 4 (ATF4) is a critical t
134                                              Activating transcription factor 4 (Atf4) is a leucine-zi
135                                              Activating transcription factor 4 (ATF4) is a mediator o
136                                              Activating transcription factor 4 (ATF4) is a transcript
137                                              Activating transcription factor 4 (ATF4) is a transcript
138                                              Activating transcription factor 4 (ATF4) is an osteoblas
139 ently that HRI also activates the eIF2alphaP-activating transcription factor 4 (ATF4) ISR in primary
140                                              Activating transcription factor 4 (ATF4) mRNA and protei
141  and selectively enhances the translation of activating transcription factor 4 (ATF4) mRNA to induce
142      In contrast, obese mice lacking hepatic activating transcription factor 4 (Atf4) showed an exagg
143  a heme-regulated inhibitor kinase/eIF2alpha/activating transcription factor 4 (ATF4) signaling modul
144  protein kinase RNA-like ER kinase (PERK) or activating transcription factor 4 (ATF4) significantly r
145 istration of AP20187 significantly increased activating transcription factor 4 (ATF4) translation and
146 zed enzymes are transcriptionally induced by activating transcription factor 4 (Atf4) via C/ebp-Atf-R
147  expression of the bZip transcription factor activating transcription factor 4 (ATF4) was induced by
148            eIF2alpha phosphorylation induces activating transcription factor 4 (ATF4), a basic leucin
149                     Here, we have shown that activating transcription factor 4 (ATF4), a master trans
150 ancer-derived cells stimulated expression of activating transcription factor 4 (ATF4), a master trans
151 regulation of triglyceride metabolism by the activating transcription factor 4 (ATF4), a member of th
152          An important effector of the ISR is activating transcription factor 4 (ATF4), a transcriptio
153 P response element binding protein 2 (CREB2)/activating transcription factor 4 (ATF4), a transcriptio
154 ulated by arsenite, in a manner dependent on activating transcription factor 4 (ATF4), an important m
155  X-box-binding protein-1 mRNA, expression of activating transcription factor 4 (ATF4), and cleavage o
156  master regulator of amino acid homeostasis, activating transcription factor 4 (ATF4), is dysfunction
157                      Increased expression of activating transcription factor 4 (Atf4), which indicate
158 ISC1 interacts with the transcription factor Activating Transcription Factor 4 (ATF4), which is invol
159 sion and purine synthesis were stimulated by activating transcription factor 4 (ATF4), which was acti
160 nts that the histone demethylase JMJD3 is an activating transcription factor 4 (ATF4)-dependent targe
161 ion initiation factor 2alpha (eIF2alpha) and activating transcription factor 4 (ATF4).
162 ins that impact neuronal function, including activating transcription factor 4 (ATF4).
163 nt-binding protein 1 (CREB) and another with activating transcription factor 4 (ATF4).
164 anslation of selected mRNAs such as that for activating transcription factor 4 (ATF4).
165  PERK- and ISR-dependent upregulation of the Activating Transcription Factor 4 (ATF4).
166 bility, in part through direct regulation of activating transcription factor 4 (Atf4).
167 e in aged muscle are positively regulated by activating transcription factor 4 (ATF4).
168 selected stress-related transcripts, such as activating transcription factor 4 (ATF4).
169 34 expression by other stresses that involve activating transcription factor 4 (ATF4).
170 on general control nonderepressible 2 kinase-activating transcription factor 4 (GCN2-ATF4) pathway ac
171 d early activation of the PKR-like ER kinase/activating transcription factor 4 (PERK/ATF4) ER stress
172 ed to persistent eif2-alpha phosphorylation (activating transcription factor 4 [ATF-4], C/EBP homolog
173 down of inositol-requiring enzyme 1alpha and activating transcription factor 4 abrogates autophagosom
174 initiation factor 2alpha phosphorylation and activating transcription factor 4 and C/EBP homologous p
175  factor 2alpha (eiF2alpha) and expression of activating transcription factor 4 and tribbles 3 were el
176 eens and by implication of the ISR-inducible activating transcription factor 4 in the process.
177 ugh arsenite treatment and is independent of activating transcription factor 4 signaling and protein
178                                  Since ATF4 (activating transcription factor 4) and CCAAT/enhancer-bi
179 ulator homolog 1) was regulated by the ATF4 (activating transcription factor 4) arm of the unfolded p
180 f genes enriched for components of the ATF4 (activating transcription factor 4) arm of the unfolded p
181                                        ATF4 (activating transcription factor 4) is an osteoblast-enri
182               The transcription factor ATF4 (activating transcription factor 4) is induced by multipl
183 response genes (C/EBP homologous protein and activating transcription factor 4), accumulation of cera
184 on by amino acid deprivation did not require activating transcription factor 4, a critical component
185 alpha, inositol requiring kinase 1alpha, and activating transcription factor 4, all of which are feat
186 such as the pseudokinase tribbles homolog 3, activating transcription factor 4, and transcription fac
187 er binding of the transcriptional activators activating transcription factor 4, C/EBPalpha, Runx1, an
188 luding glucose-regulated protein-78 (GRP78), activating transcription factor 4, growth arrest and DNA
189 rylated eukaryotic initiation factor-2alpha, activating transcription factor 4, inositol requiring ki
190 iation factor 2 (eIF2alpha) phosphorylation, activating transcription factor-4 (ATF4) induction, and
191 e proteasome inhibitor bortezomib, levels of activating transcription factor-4 increase dramatically
192 UPR induction, particularly up-regulation of activating transcription factor-4, CHOP (C/EBP homologou
193 en and nitrogen species and regulated by the activating-transcription factor-4.
194             Nucleophosmin (NPM1/B23) and the activating transcription factor 5 (ATF5) are both known
195                            The expression of activating transcription factor 5 (ATF5) correlates nega
196 th the heat shock protein 70 (HSP70) and the activating transcription factor 5 (ATF5) have been shown
197                                              Activating transcription factor 5 (ATF5) is a member of
198                                              Activating transcription factor 5 (ATF5) is a stress-res
199 h mouse and human BCR-ABL-transformed cells, activating transcription factor 5 (ATF5), a prosurvival
200     As a result, seven transcription factors-activating transcription factor 5 (ATF5), early growth r
201 r eif2alpha causing selective translation of activating transcription factor 5 (ATF5).
202 glia, require the transcription factor ATF5 (activating transcription factor 5) for survival.
203                                              Activating transcription factor-5 (ATF5) is highly expre
204                                              Activating transcription factor 6 (ATF6) and protein kin
205 nositol-requiring protein-1 (IRE1) alpha and activating transcription factor 6 (ATF6) arms of the UPR
206                       We recently identified activating transcription factor 6 (ATF6) as a genetic ca
207                            Activation of the activating transcription factor 6 (ATF6) branch of the U
208                                              Activating transcription factor 6 (ATF6) is important fo
209                                              Activating transcription factor 6 (ATF6) is located with
210 ections were stained with antibodies against activating transcription factor 6 (ATF6) or activated ca
211 th ER resident kinase (PERK) activation, and activating transcription factor 6 (ATF6) splicing.
212 tases (OAS) genes, and selectively activated activating transcription factor 6 (ATF6), an unfolded pr
213 ol-requiring 1A/X box binding protein 1, and activating transcription factor 6 (ATF6), and each of th
214 binding protein 1 (XBP1) mRNA, activation of activating transcription factor 6 (ATF6), and protein ki
215 ions of three proximal sensors of ER stress: activating transcription factor 6 (ATF6), inositol requi
216 ates that DKK3 added as a cytokine activates activating transcription factor 6 (ATF6), leading to the
217 d accompanied by a reduction in the level of activating transcription factor 6 (ATF6), one of the tra
218 a cells induced the nuclear translocation of activating transcription factor 6 (ATF6), which is part
219 K), inositol requiring kinase-1 (IRE-1), and activating transcription factor 6 (ATF6).
220 d the unfolded protein response (UPR) sensor activating transcription factor 6 (ATF6).
221 in 78 (GRP 78), and nuclear translocation of activating transcription factor 6 (ATF6).
222 sion of several SKI-1 target genes including activating transcription factor 6 (ATF6).
223 ress indexes in vivo and in vitro, decreased activating transcription factor 6 activation (cleavage,
224 nd X-box-binding protein 1 (XBP1) but not by activating transcription factor 6 alpha (ATF6alpha).
225 ty control genes through an association with activating transcription factor 6 alpha (ATF6alpha, also
226 , activating the transcription factor, ATF6 (activating transcription factor 6 alpha), which induces
227 ess proteins inositol-requiring enzyme 1 and activating transcription factor 6 and greater increases
228 ), which involves activation of the Ire1 and activating transcription factor 6 branches, but not the
229 tions of spliced X-box binding protein 1 and activating transcription factor 6 levels in affected liv
230 ide, a serine protease inhibitor that blocks activating transcription factor 6 processing.
231 ion primary response gene 88), but not ATF6 (activating transcription factor 6) or XBP1 (X-box-bindin
232 lucose-regulated protein was down-regulated, activating transcription factor 6, and eIF2alpha were ac
233              Direct miR-199a-5p targeting of activating transcription factor 6, p50, and p65 by miR-1
234                                       GRP78, activating transcription factor 6, p50, and p65 were inc
235 e of dominant-negative versions of XBP-1 and activating transcription factor 6, the kinetics of class
236 onary circulation involves the activation of activating transcription factor 6, which, via induction
237 found to play a critical role in driving the activating transcription factor 6-mediated arm of this r
238 ER signal kinase 1alpha but did not activate activating transcription factor 6.
239 or of caspases, HtrA serine peptidase 2, and activating transcription factor 6.
240 BP1) is selectively impaired in DN, inducing activating transcription factor-6 (ATF6) and C/EBP homol
241  augmented tunicamycin-induced activation of activating transcription factor-6 (ATF6) and induction o
242 ER kinase (PERK; official name EIF2AK3), and activating transcription factor-6, control the UPR.
243 nase (PERK), inositol-requiring enzyme-1 and activating transcription factor-6.
244 ozygous mutations in the ATF6 gene (encoding activating transcription factor 6A), a key regulator of
245           Thbs bind the ER lumenal domain of activating transcription factor 6alpha (Atf6alpha) to pr
246 tion factor involved in ER stress signaling, activating transcription factor 6alpha (ATF6alpha), thro
247 lasmic reticulum (ER) stress pathway through activating transcription factor 6alpha (Atf6alpha).
248 tol-requiring enzyme-1alpha (IRE1alpha), and activating transcription factor-6alpha (ATF6alpha)-were
249 tol-requiring protein-1alpha (IRE1alpha) and activating transcription factor-6alphaalpha (ATF6alpha).
250 activating transcription factor 2) and ATF7 (activating transcription factor 7) are highly homologous
251  miRNA activity, possibly via the downstream Activating Transcription Factor-7 (ATF-7).
252  the downstream 36-bp region containing CREB/activating transcription factor and kappaB6 sites.
253  pathways and the transcription factors ATF (activating transcription factor) and CREB (cyclic AMP re
254  PYD, pyrin N-terminal homology domain; ATF, activating transcription factor; and PTEN, phosphatase a
255 luding the bZip transcription factor atfs-1 (activating transcription factor associated with stress).
256 , we examined the mechanism by which ATFS-1 (activating transcription factor associated with stress-1
257 nse element (CRE)-binding protein (CREB) and activating transcription factor (ATF) 1 contributes to m
258 eport that anisomycin, a potent activator of activating transcription factor (ATF) 2, and c-Jun-NH(2)
259 meric complexes with the AP-1 family members activating transcription factor (ATF) 2, ATF3, and ATF7.
260 -1 significantly inhibited the expression of activating transcription factor (ATF) 3, a member of the
261 tiation factor 2, and increased synthesis of activating transcription factor (ATF) 4 by a translation
262 identified CHOP as an interacting partner of activating transcription factor (ATF) 4 in a yeast two-h
263 anced levels of phosphorylated eIF2alpha and activating transcription factor (ATF) 4, which is essent
264 ern and the role of different members of the activating transcription factor (ATF) family in survival
265 tion factors are dimers of JUN, FOS, MAF and activating transcription factor (ATF) family proteins ch
266 CREB3L1), a transcription factor of the CREB/activating transcription factor (ATF) family, increases
267 -AMP response element binding protein (CREB)/activating transcription factor (ATF) site overlapping a
268 -related protein (SMAD)-3 (nt -584 to -581), activating transcription factor (ATF)-2 (nt -571 to -568
269        The p38 target transcriptional factor activating transcription factor (ATF)-2 bound to the PTE
270  site 2 bound CRE-binding protein (CREB) and activating transcription factor (ATF)-2 in vitro and was
271 tion and activation of p38 kinase substrate, activating transcription factor (ATF)-2.
272 equires binding sites for sequence-dependent activating transcription factor (ATF)-adenosine 3',5'-mo
273 s belonging to three families: jun, fos, and activating transcription factor (ATF).
274                                          Two activating transcription factor (ATF)/CREB family member
275 developing heart and somites via one or more activating transcription factor (ATF)/cyclic AMP respons
276  kinase and downstream components, including activating transcription factor (ATF)/cyclic AMP-respons
277                                              Activating transcription factor (ATF)3 regulates the exp
278 f type I IFN gene expression, interacts with activating transcription factor (ATF)4, a key component
279 RAD is increased by the ER stress modulator, activating transcription factor (ATF)6, which can induce
280  expression of the transcriptional repressor activating transcription factor (ATF-3) in a STAT1-depen
281 kinase and PKR-like ER kinase (PERK)-induced activating transcription factor (ATF3) binding to its pr
282  transcription by a mechanism dependent upon activating transcription factor ATF4.
283 alpha and upregulation of gene expression of activating transcription factors ATF4 and ATF6.
284 c (Tg) mice constitutively expressing B cell-activating transcription factor (BATF), a basic leucine
285  homology to the cyclic AMP response element/activating transcription factor binding sites.
286                                  It binds an activating transcription factor complex in erythroid cel
287 cts of cAMP response element binding protein/activating transcription factor (CREB/ATF) and AP-1 tran
288               WT.BRCA1 selectively binds OPN-activating transcription factors estrogen receptor alpha
289  (cAMP response element modulator), and atf (activating transcription factor) genes.
290 1) family transcription factor BATF (B cell, activating transcription factor-like) was among the gene
291 ression mediated, in part, by members of the activating transcription factor protein (AP1) group.
292 h defects in IFN regulatory factor 4, B cell-activating transcription factor, retinoic acid-related o
293                Subsequent recruitment of the activating transcription factor Sp1 to the remodeled pro
294  Hep3B cells, IL-6 induces CRP expression by activating transcription factors STAT3 and C/EBPbeta.
295 nhibition can increase cAMP and cGMP levels, activating transcription factors such as the cAMP respon
296 ndelian disease, we show that DNA binding of activating transcription factor (TF) determines histone
297 reasing histone deacetylase activity, or (e) activating transcription factors that antagonize chronic
298 drial ROS augmented the expression of B cell-activating transcription factor, which may contribute to
299 ified a cellular repressor of transcription, activating transcription factor x (ATFx), as a novel Tax
300                Ectopic expression of the EMT-activating transcription factor ZEB1 stimulated Golgi co

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