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1 c tumors, controlled the expression of ATF2 (activating transcription factor 2).
2 sponse element-binding protein 2 (CREB2) and activating transcription factor 2.
3 PK, c-Jun NH2-terminal kinase, p38 MAPK, and activating transcription factor 2.
4 he gamma1 promoter binds and is activated by activating transcription factor 2.
5 content of c-Jun but not that of c-Fos or of activating transcription factor 2.
6 ns and in its interaction with the mammalian activating transcription factor 2.
7 maintenance of chromatin protein 1, but not activating transcription factor 2.
8 eam of the JNK and p38 pathways as c-Jun and activating transcription factor-2.
9 tors NF-kappaB, IFN regulatory factor-1, and activating transcription factor-2.
10 ases c-Jun N-terminal kinase and p38, and of activating transcription factor-2.
11 or, blocked p38-dependent phosphorylation of activating transcription factor-2.
12 ate with increased serine phosphorylation of activating transcription factor-2.
13 ing nuclear proteins of the AP-1 complex and activating transcription factor-2.
14 /CRE bound to nuclear proteins that included activating transcription factor-2.
17 ed ASK1-mediated c-Jun N-terminal kinase and activating transcription factor 2 activities in intact c
18 IFN regulatory factor-1 and upregulation of activating transcription factor-2 and c-Jun, transcripti
19 d activation of transcription factors c-Jun, activating transcription factor-2 and, in addition, NF k
21 nstream effectors: phosphorylation of c-Jun, activating transcription factor 2, and E twenty-six-like
22 lastine-induced phosphorylation of c-Jun and activating transcription factor-2, and AP-1 activation,
24 y mouse aortic endothelial cells identifying activating transcription factor 2 as a new target for PK
27 rect regulation of transcription mediated by activating transcription factor 2 (ATF-2) and (ii) H(2)O
29 cAMP response element-binding protein (CREB)/activating transcription factor 2 (ATF-2) binding site.
30 sphorylation of JNK, PKCdelta, p38 MAPK, and activating transcription factor 2 (ATF-2) in RA ST fibro
31 t the attachment of CREB binding protein and activating transcription factor 2 (ATF-2) to the cAMP-re
32 gen-activated protein kinase (p38 MAPK), and activating transcription factor 2 (ATF-2) were detected
33 by activated transcription factors c-Jun and activating transcription factor 2 (ATF-2), which bound t
36 at down-stream effectors of p38 MAPK, namely activating transcription factor-2 (ATF-2) and cyclic AMP
37 hibits the phosphorylation of EGF-stimulated activating transcription factor-2 (ATF-2) and signal tra
39 found that p38 target transcriptional factor activating transcription factor-2 (ATF-2) bound to ATF s
42 n transcription factors, including c-Jun and activating transcription factor-2 (ATF-2) upon activatio
47 r protein 1 (AP-1) binding proteins, namely, activating transcription factor 2 (ATF2) and c-Jun, bind
49 er showed an increase of Thr(P)-71-Thr(P)-69-activating transcription factor 2 (ATF2) binding in resp
56 y suppressing expression via binding through activating transcription factor 2 (ATF2) to the cyclic a
57 in transcription factors including c-jun and activating transcription factor 2 (ATF2) upon activation
58 uitination of its other substrates (Elk1 and activating transcription factor 2 (ATF2)) and associated
59 -response element binding protein (CREB) and activating transcription factor 2 (ATF2), function as a
60 tion of cell growth and apoptosis, including activating transcription factor 2 (ATF2), Max, cAMP resp
61 we identify four transcription factors, JUN, activating transcription factor 2 (ATF2), myocyte-specif
66 we show that under non-stressed conditions, activating transcription factor-2 (ATF2) in cooperation
69 E(2) or Bt(2)cAMP + PDA stimulated c-Jun and activating transcription factor-2 (ATF2) phosphorylation
71 d activities of transcription factors ELK-1, activating transcription factor-2, c-Fos, c-Jun, activat
72 CpG DNA also induces the phosphorylation of activating transcription factor-2, c-Jun, and mitogen-ac
73 cAMP response element modulator (CREB/CREM), activating transcription factor-2/c-Jun, and Jun-Jun/Oct
74 er, both phosphorylated and unphosphorylated activating transcription factor 2 DNA-binding activities
75 , and its subline SW1-P2, which expresses an activating transcription factor 2-driven peptide, were t
76 se 3/6, and downstream transcription factors activating transcription factor 2, ETS domain transcript
77 reduced, there was no reduction in pulmonary activating transcription factor-2 expression, arguing ag
78 protein substrate, glutathione S-transferase-activating transcription factor 2 (GST-ATF2), binding be
79 on protein of the trans-activation domain of activating transcription factor-2 (GST-ATF2-(1-115)) by
81 timulated FLS and the complex phosphorylated activating transcription factor-2 in an in vitro kinase
82 lation of the transcription factors, Jun and activating transcription factor 2, in addition to activa
83 Investigation of JNK targets revealed that activating transcription factor-2 is phosphorylated unde
84 vation specifically activated c-Jun, but not activating transcription factor-2 or JunD, and increased
85 1) the trans-phosphorylation of recombinant activating transcription factor-2 substrate by immunopre
89 tors activator protein 1, ELK-1, p90RSK, and activating transcription factor 2 was also observed in c
90 ation of the transcription factors c-Jun and activating transcription factor 2, which in turn stimula
91 ylation of cAMP response-element binding and activating transcription factor 2, which leads to transa
92 of p38 mitogen-activated protein kinase and activating transcription factor-2, which were blocked by
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