ライフサイエンスコーパス: activating transcription factor 3

1 ugh interaction with transcription repressor activating transcription factor 3.

2 e induction of another transcription factor, activating transcription factor 3.

3 anti-inflammatory drug-activated gene-1 and activating transcription factor-3.

4 amma, insulin-like growth factor I receptor, activating transcription factor 3, aldehyde dehydrogenas

5 ssociated with nerve injury and cell stress, activating transcription factor 3 and growth-associated

6 gly, four other genes, cylindromatosis, CD9, activating transcription factor 3 and oxytocin receptor,

7 d to activation of the transcription factors activating transcription factor 3 and protooncogene c-fo

8 totic responses, including induction of p27, activating transcription factor 3, and nonsteroidal anti

9 er transcripts, including neuropeptide Y and activating transcription factor-3, are upregulated norma

10 ressor of cytokine signaling 3 (SOCS-3), and activating transcription factor 3 (ATF-3), which termina

11 te the induction of the stress/injury marker activating transcription factor 3 (ATF-3).

12 From the candidates selected, activating transcription factor-3 (ATF-3), caveolin-1, d

13 ic stress-induced UPR and cell death through activating transcription factor 3 (ATF3) and C/EBP homol

14 ound that microRNA-494 binds to the 3'UTR of activating transcription factor 3 (ATF3) and decreases i

15 C irradiation, immediate early genes such as activating transcription factor 3 (ATF3) are overexpress

16 Here we report that activating transcription factor 3 (ATF3) bound common mu

17 Activating transcription factor 3 (ATF3) can repress Nrf

18 8 mitogen activated protein kinase (p38MAPK)-activating transcription factor 3 (ATF3) dependent pathw

19 1) phosphorylation and subsequently enhances activating transcription factor 3 (ATF3) expression inde

20 Expression of the activating transcription factor 3 (ATF3) gene is induced

21 Activating transcription factor 3 (ATF3) has been propos

22 Demyelination induced activating transcription factor 3 (ATF3) in DRG neurons.

23 A recent study showed the role of activating transcription factor 3 (ATF3) in SCC developm

24 Activating transcription factor 3 (ATF3) is a basic leuc

25 Activating transcription factor 3 (ATF3) is a common med

26 Activating transcription factor 3 (ATF3) is a common str

27 Activating transcription factor 3 (ATF3) is a key mediat

28 Activating transcription factor 3 (ATF3) is a member of

29 Activating transcription factor 3 (ATF3) is a member of

30 Activating transcription factor 3 (ATF3) is a member of

31 Activating transcription factor 3 (ATF3) is a member of

32 Activating transcription factor 3 (ATF3) is a negative r

33 Activating transcription factor 3 (ATF3) is a prime cand

34 Activating transcription factor 3 (ATF3) is a stress-ind

35 Activating transcription factor 3 (ATF3) is an important

36 Activating transcription factor 3 (ATF3) is another tran

37 Activating transcription factor 3 (Atf3) is rapidly and

38 Activating transcription factor 3 (ATF3) is rapidly indu

39 Activating transcription factor 3 (ATF3) promotes neuron

40 Activating transcription factor 3 (ATF3) responds to div

41 Here we report that activating transcription factor 3 (ATF3), a broad DNA da

42 Here we report that activating transcription factor 3 (ATF3), a common stres

43 Previous studies demonstrate that activating transcription factor 3 (ATF3), a common stres

44 In this report, we demonstrate that activating transcription factor 3 (ATF3), a hub of the c

45 issue of Blood, Boespflug et al report that activating transcription factor 3 (ATF3), a member of th

46 roup of genes that appear to be regulated by activating transcription factor 3 (ATF3), a member of th

47 We find that expression of activating transcription factor 3 (ATF3), a member of th

48 Activating transcription factor 3 (ATF3), a member of th

49 Expression of activating transcription factor 3 (ATF3), a nuclear calc

50 Here we report the discovery that activating transcription factor 3 (ATF3), a stress respo

51 We tested the hypothesis that activating transcription factor 3 (ATF3), a stress-induc

52 Activating transcription factor 3 (ATF3), an NMD target

53 with rapid induction of heat shock proteins, activating transcription factor 3 (ATF3), and CHOP.

54 or p75NTR, choline acetyltransferase (ChAT), activating transcription factor 3 (ATF3), and cleaved ca

55 itor of differentiation/DNA binding 2 (Id2), activating transcription factor 3 (Atf3), and the phosph

56 72 h after surgery, there is an increase in activating transcription factor 3 (ATF3), the neuropepti

57 expression of the transcriptional regulator activating transcription factor 3 (ATF3), which we show

58 Here, we report that activating transcription factor 3 (ATF3)-a broad stress

59 or-activated receptor-gamma (PPARgamma), and activating transcription factor 3 (ATF3).

60 abeling for the stress and injury-associated activating transcription factor 3 (ATF3).

61 and increased the mRNA and protein levels of activating transcription factor 3 (ATF3).

62 One novel GH-induced gene encodes activating transcription factor 3 (ATF3).

63 e (jun B), jun D proto-oncogene (jun D), and activating transcription factor 3 (ATF3).

64 Importantly, transcripts for the activating transcription factor-3 (Atf3) and mitochondri

65 Activating transcription factor-3 (ATF3) is rapidly indu

66 amined a marker of cell injury/regeneration (activating transcription factor 3; ATF3), macrophage hyp

67 Activating transcription factor 3 attenuates chemokine a

68 xpression, and nerve damage was evaluated by activating transcription factor 3 expression.

69 apoptosis in prostate cancer cells by ATF3 (activating transcription factor 3) expression.

70 ansection models, the upregulation of c-Jun, Activating transcription factor 3, Heat shock protein 27

71 Previously, we demonstrated that ATF3 (activating transcription factor-3) is a stress-inducible

72 f the transcription factors c-Jun and ATF-3 (activating transcription factor 3), known regulators of

73 dent, whereas induction of cystathionase and activating transcription factor 3 was Nur77 independent.