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1 ypermutation and class switch recombination (activation-induced cytidine deaminase).
2 ccur in genomic DNA similar to the action of activation-induced cytidine deaminase.
3 factors, Pax5, IRF4, and Blimp-1, and of the activation-induced cytidine deaminase.
4 when their antigen receptors are damaged by activation-induced cytidine deaminase.
5 during the repair of DNA breaks initiated by activation-induced cytidine deaminase.
6 suggesting that p53 inhibits the activity of activation-induced cytidine deaminase.
7 o impaired upregulation of the gene encoding activation-induced cytidine deaminase.
8 l activity, or off-targeting activity of the activation-induced cytidine deaminase.
9 CSR, at least in part via expression of the activation-induced cytidine deaminase.
10 eptin and Pontin were shown to interact with activation-induced cytidine deaminase.
11 nsducer and activator of transcription 3 and activation-induced cytidine deaminase.
12 switch through regulating the expression of activation-induced cytidine deaminase.
13 after the generation of dG:dU mismatches by activation-induced cytidine deaminase.
14 onally activating the B cell-specific factor activation-induced cytidine deaminase.
15 ength and repeat density that are targets of activation-induced cytidine deaminase.
16 tream CH genes as well as expression of AID (activation-induced cytidine deaminase), a DNA-editing en
17 cription is associated with up-regulation of activation-induced cytidine deaminase, a key element of
19 intron, perhaps a self-regulating aspect of activation-induced cytidine deaminase action that is con
21 at specific genes involved in SHM, including activation-induced cytidine deaminase (AICDA) and uracil
22 genes, both X-linked; and mutations of CD40, activation-induced cytidine deaminase (AICDA), and uraci
25 s in IL6 transgenic mice that are mutant for activation induced cytidine deaminase (AID), the enzyme
28 L5, shows a mutational pattern suggestive of activation-induced cytidine deaminase (AID) activity.
30 ds to rapid induction of the genomic mutator activation-induced cytidine deaminase (AID) and AID-depe
31 B cells triggered the enhanced expression of activation-induced cytidine deaminase (AID) and also sti
33 c acid-dependent cytidine deaminases include activation-induced cytidine deaminase (AID) and Apobec3G
35 at BRIT1 is recruited to the Igh locus in an activation-induced cytidine deaminase (AID) and H2AX-dep
36 ombination (CSR) despite normal induction of activation-induced cytidine deaminase (AID) and Iepsilon
38 genesis, we used MEFs ectopically expressing activation-induced cytidine deaminase (AID) and observed
39 F directly controlled the expression of both activation-induced cytidine deaminase (AID) and of germl
41 The introduction of DSBs is initiated by activation-induced cytidine deaminase (AID) and requires
44 ermutation in the germinal center (GC), with activation-induced cytidine deaminase (AID) as a prerequ
45 occurs as a consequence of the expression of activation-induced cytidine deaminase (AID) by Ag-activa
50 by processing G.U mismatches generated when activation-induced cytidine deaminase (AID) deaminates C
52 g to c-myc/IgH translocations are created by activation-induced cytidine deaminase (AID) during antib
53 imulation of B cell proliferation, repressed activation-induced cytidine deaminase (AID) expression,
54 demethylation and genome rearrangements via activation-induced cytidine deaminase (AID) followed by
58 f autoimmune disease, elevated expression of activation-induced cytidine deaminase (AID) in recircula
78 ns convert single-strand nicks instigated by activation-induced cytidine deaminase (AID) into the dou
114 d presenting as GC B cells with constitutive activation-induced cytidine deaminase (AID) mutator acti
120 s switch recombination (CSR) is initiated by activation-induced cytidine deaminase (AID) that catalyz
121 measured translocations in mice deficient in activation-induced cytidine deaminase (AID) that lack cl
122 Secondary Ig gene diversification relies on activation-induced cytidine deaminase (AID) to create U:
124 cells prevent harmful R loops, we used human activation-induced cytidine deaminase (AID) to identify
125 by the transcription-coupled recruitment of activation-induced cytidine deaminase (AID) to Ig switch
126 nthesis of high affinity antibodies requires activation-induced cytidine deaminase (AID) to initiate
128 by the transcription-coupled recruitment of activation-induced cytidine deaminase (AID) to switch re
130 This was associated with less targeting of activation-induced cytidine deaminase (AID) to the Igh l
131 somatic hypermutation (SHM) is initiated by activation-induced cytidine deaminase (AID) upon deamina
132 Ig variable regions requires the activity of activation-induced cytidine deaminase (AID) which has pr
135 cting directly with the C-terminal region of activation-induced cytidine deaminase (AID), 14-3-3gamma
136 s undergo rapid clonal expansion and express activation-induced cytidine deaminase (AID), a DNA mutat
137 (MCL), we analyzed the expression levels of activation-induced cytidine deaminase (AID), a key playe
138 inciple experiments, we apply this screen to activation-induced cytidine deaminase (AID), a poorly so
139 c diversification processes are catalyzed by activation-induced cytidine deaminase (AID), a potent DN
141 rmed pockets populated by B cells expressing activation-induced cytidine deaminase (AID), an enzyme a
145 o-step process: (i) DNA lesions initiated by activation-induced cytidine deaminase (AID), and (ii) le
146 on the action of the B cell specific enzyme, activation-induced cytidine deaminase (AID), and can be
147 ases in Ig class switch recombination (CSR), activation-induced cytidine deaminase (AID), and E47 tra
148 re transcription and the trans-acting factor activation-induced cytidine deaminase (AID), and must be
150 d somatic hypermutation (SHM), which require activation-induced cytidine deaminase (AID), and plasma
151 tation (SHM) of Ig genes is initiated by the activation-induced cytidine deaminase (AID), and require
152 creased in class switch recombination (CSR), activation-induced cytidine deaminase (AID), and stabili
153 critical elements of that machinery, such as activation-induced cytidine deaminase (AID), as well as
154 inding site in the 3'-untranslated region of activation-induced cytidine deaminase (AID), designated
155 ients with mutations in AICDA, which encodes activation-induced cytidine deaminase (AID), display an
156 cell cycle, proliferation and expression of activation-induced cytidine deaminase (AID), DNA repair
157 r joining of distal DNA lesions initiated by activation-induced cytidine deaminase (AID), in the abse
160 s switch recombination (CSR) is initiated by activation-induced cytidine deaminase (AID), the activit
165 s switch recombination (CSR) is initiated by activation-induced cytidine deaminase (AID), which conve
166 unoglobulin switch (S) regions, and requires activation-induced cytidine deaminase (AID), which conve
167 s requires transcription and is triggered by activation-induced cytidine deaminase (AID), which conve
168 bination is initiated by the B cell-specific activation-induced cytidine deaminase (AID), which deami
171 anonical NF-kappaB pathway, thereby inducing activation-induced cytidine deaminase (AID), which is cr
172 l center B cells including the expression of activation-induced cytidine deaminase (AID), which is es
174 ith hyper-IgM syndromes who are deficient in activation-induced cytidine deaminase (AID), which is re
175 ion transcription is insufficient to attract activation-induced cytidine deaminase (AID), which is re
177 o generate genomic instability in B cells as activation-induced cytidine deaminase (AID), which media
178 n and somatic hypermutation are initiated by activation-induced cytidine deaminase (AID), which prefe
179 switch recombination (CSR) are initiated by activation-induced cytidine deaminase (AID), which prefe
182 lass switch recombination (CSR), which joins activation-induced cytidine deaminase (AID)-dependent do
183 ne or IgH locus of B lymphocytes induced for activation-induced cytidine deaminase (AID)-dependent Ig
184 SBs in splenic IgM(+) B cells stimulated for activation-induced cytidine deaminase (AID)-dependent Ig
185 nse, B cells undergo rapid proliferation and activation-induced cytidine deaminase (AID)-dependent re
187 ination (CSR) of Ig genes are dependent upon activation-induced cytidine deaminase (AID)-induced muta
189 ccurs through the deliberate introduction of activation-induced cytidine deaminase (AID)-instigated D
208 g RNA (siRNA)-mediated knockdown showed that activation-induced cytidine deaminase (AID, also known a
209 ng gene segment (V(D)J) recombination, or by activation-induced cytidine deaminase (AID, also known a
214 distinct but homologous cytidine deaminases-activation-induced cytidine deaminase and apolipoprotein
215 shall review the functions and regulation of activation-induced cytidine deaminase and apolipoprotein
216 (immature/T1) B cells constitutively express activation-induced cytidine deaminase and B lymphocyte-i
217 These requisite switching factors include activation-induced cytidine deaminase and components of
219 n-associated DNA-modifying events, involving activation-induced cytidine deaminase and DNA polymerase
220 lucidated, involving cytosine deamination by activation-induced cytidine deaminase and generation of
221 ner dependent on CD40L and ICOS and inducing activation-induced cytidine deaminase and Ig class switc
222 nses and are orchestrated by the activity of activation-induced cytidine deaminase and many proteins
224 n the Ig loci are responsible for recruiting activation-induced cytidine deaminase and promoting its
225 tors IRF4 and Blimp-1, and altered levels of activation-induced cytidine deaminase and sphingosine-1-
226 ermutation (SHM), deamination of cytidine by activation-induced cytidine deaminase and subsequent DNA
227 cally enriched for autoreactivity, expresses activation-induced cytidine deaminase and T-bet, and exh
228 genotoxic stress and liganded AR, including activation-induced cytidine deaminase and the LINE-1 rep
229 3K signaling enhanced the expression of AID (activation-induced cytidine deaminase) and accelerated C
230 ith germinal center formation, expression of activation-induced cytidine deaminase, and affinity matu
231 te defects in Ig class switch recombination, activation-induced cytidine deaminase, and E47 transcrip
232 er centroblasts, follicular dendritic cells, activation-induced cytidine deaminase, and IL-21(+)PD1(+
233 urn, this led to proliferation, induction of activation-induced cytidine deaminase, and the productio
234 B cells up-regulate the CSR-inducing enzyme, activation-induced cytidine deaminase, and undergo CSR f
235 TET oxidases and, more controversially, the activation-induced cytidine deaminase/APOBEC deaminases
236 both cDNA sequence and protein expression of activation-induced cytidine deaminase appear normal, the
237 immunoglobulin genes, uracils introduced by activation-induced cytidine deaminase are processed by u
238 y conservation in jawed vertebrates, we used activation-induced cytidine deaminase as a marker to mon
239 e-induced maturation protein-1 (BLIMP-1) and activation-induced cytidine deaminase as well as the pro
240 ctional B cell activation with expression of activation-induced cytidine deaminase, as well as local
241 lls with the generation of U:G mismatches by activation-induced cytidine deaminase but differ in thei
242 nal evolution of Ig V regions, expression of activation-induced cytidine deaminase, clonal H chain sw
243 impaired induction of E47, and subsequently activation-induced cytidine deaminase, contribute to poo
244 as well as transgenic mice (quasimonoclonal, activation-induced [cytidine] deaminase-Cre-tamoxifen-de
246 yelinating disease, germline IgM produced in activation-induced cytidine deaminase-deficient mice (ai
247 plasmacytomas from uracil N-glycosylase and activation-induced cytidine deaminase-deficient mice.
248 ient), in switched Igs and hypermutated IgM (activation-induced cytidine deaminase-deficient), or ful
249 FtL-specific B-1a to mount dominant IgM and activation-induced cytidine deaminase-dependent IgG anti
250 consistently accumulated high frequencies of activation-induced cytidine deaminase-dependent IgH locu
251 were accompanied by ongoing duplications and activation-induced cytidine deaminase-dependent somatic
253 fficult to uncouple because a single enzyme, activation-induced cytidine deaminase (encoded by Aicda)
254 ass switch recombination are mediated by the activation-induced cytidine deaminase enzyme and under A
255 ind to murine B lymphocytes thereby inducing activation-induced cytidine deaminase expression and Ig
256 e that oncogenic viruses can directly induce activation-induced cytidine deaminase expression and onc
257 cl-2 function, are associated with increased activation-induced cytidine deaminase expression, and co
260 o steps: the generation of uracils in DNA by activation-induced cytidine deaminase, followed by their
261 nd that CRISPR/Cas9-mediated ablation of the activation-induced cytidine deaminase gene required for
262 and circle transcripts and to upregulate the activation-induced cytidine deaminase gene through in vi
263 e ELS features and support functional GL7(+)/activation-induced cytidine deaminase(+) germinal center
264 hough the enzyme critical to both processes, activation-induced cytidine deaminase, has been identifi
266 , Teng et al. and Dorsett et al. report that activation-induced cytidine deaminase in B cells is repr
267 firmed the role of the miR-155 target Aicda (activation-induced cytidine deaminase) in this process a
268 hese germinal center-like tumors arose by an activation-induced cytidine deaminase-independent pathwa
270 ally delete Dicer in activated B cells where activation-induced cytidine deaminase is highly expresse
271 analyzed these data and found evidence that activation-induced cytidine deaminase is the major sourc
273 are recruited to dU:dG mispairs generated by activation-induced cytidine deaminase-mediated deaminati
274 -acting DNA elements essential for targeting activation-induced cytidine deaminase-mediated sequence
275 e primarily synonymous, and likely caused by activation-induced cytidine deaminase-mediated somatic h
276 an agonistic anti-TACI antibody could induce activation-induced cytidine deaminase mRNA in those with
277 TACI by an agonistic antibody showed loss of activation-induced cytidine deaminase mRNA induction in
278 Furthermore, pax-5 mRNA was decreased and activation-induced cytidine deaminase mRNA was increased
280 A and C-to-T substitutions that parallel the activation-induced cytidine deaminase nucleotide exchang
282 rentiation), and a 2- to 6-fold increase for activation-induced cytidine deaminase, PAX5, and the non
284 ion and CSR both require the B-cell-specific activation-induced cytidine deaminase protein (AID), whi
286 mutations, expressed germ-line Cgamma1- and activation-induced cytidine deaminase-specific transcrip
287 cells from an immunized individual to study activation-induced cytidine deaminase targeting and foun
288 s S site location because sequences with few activation-induced cytidine deaminase targets generate m
291 rprisingly, however, the cell line expresses activation-induced cytidine deaminase, the enzyme that m
292 eficiency is combined with overexpression of activation-induced cytidine deaminase, the hotspot lengt
293 SR and, thus, is not essential for targeting activation-induced cytidine deaminase to S regions, as w
295 ough p110delta can regulate transcription of activation-induced cytidine deaminase via Akt, repressio
296 s were found within ectopic lymphoid tissue, activation-induced cytidine deaminase was expressed, and
297 switch recombination (CSR) is instigated by activation-induced cytidine deaminase, which converts cy
298 ass switch recombination (CSR) is induced by activation-induced cytidine deaminase, which initiates a
300 ne DNA polymerase zeta, polymerase iota, and activation-induced cytidine deaminase, which together, c
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