ライフサイエンスコーパス: activator protein-1

1 ctivator, but not activator Rap1p (repressor-activator protein 1).

2 CCL2-regulating transcription factor, AP-1 (activator protein-1).

3 eta V1, likely via nuclear factor kappaB and activator protein 1.

4 ays, and this induction of activity requires activator protein 1.

5 transcription factors such as NF-kappaB and activator protein 1.

6 hat COX-2 gene transcription is regulated by activator protein-1.

7 -responsive transcription factors, including activator protein-1.

8 of the transcription factors NF- kappa B and activator protein-1.

9 ppaB (NF-kappaB) and c-Jun N-terminal kinase/activator protein-1.

10 ed to involve transcription factors, Sp1 and activator protein-1.

11 ytokine secretion, nuclear factor kappaB and activator protein 1 activation, mitogen-activated protei

12 ediated mitogen-activated protein kinase and activator protein-1 activation, and epithelial to mesenc

13 L2 and c-fos promoters, as well as increased activator protein 1 activity, causing normalization of I

14 ady-state level of c-Jun, thereby inhibiting activator protein-1 activity and cell transformation cau

15 In addition, activator protein-1 activity in H3 WT-overexpressing cel

16 phosphorylation is critical for controlling activator protein-1 activity, which is a major driver in

17 ed for nuclear factor kappaB (NF-kappaB) and activator protein-1 activity.

18 AP-1 (activator protein 1) activity is strongly induced in res

19 V6 expression is through EGR1-mediated AP-1 (activator protein-1) activity and that the EGR1- and AP-

20 on levels of IL-6 by reducing the ability of activator protein-1, an important mediator of IL-6 expre

21 by multiple transcription factors, including activator protein 1 and cAMP response element-binding pr

22 es activities of other DNA-bound regulators, activator protein 1 and nuclear factor kappaB, utilizing

23 and miR-466l regulated transcription factors activator protein 1 and nuclear factor kappaB1 in miRNA

24 rosis factor alpha expression, and increased activator protein 1 and nuclear factor-kappaB transcript

25 s: extracellular signal-regulated kinase 1/2-activator protein 1 and signal transducer and activator

26 d a trend for increased NF-AT, but decreased activator protein 1 and similar NF-kappaB, activity in C

27 ndently attenuated TPA-induced activation of activator protein-1 and c-fos, whereas daidzein did not

28 dose, many of which are under the control of activator protein-1 and ERK signaling.

29 he +1123/+1134 region harbored non-consensus activator protein-1 and Ets1 binding sites bound with c-

30 Furthermore, the activation of activator protein-1 and interleukin-2 induced by CD3 was

31 otein-1 (GRIP1), GR tethers to the DNA-bound activator protein-1 and NF-kappaB and represses transcri

32 The activation of activator protein-1 and nuclear factor-kappaB induced by

33 The activation of activator protein-1 and nuclear factor-kappaB induced by

34 9 in vitro, which required activation of the activator protein-1 and nuclear factor-kappaB signaling

35 ivation was specific, inasmuch as binding of activator protein-1 and octameric transcription factor 1

36 tant synapses, c-Jun N-terminal kinase (JNK)/activator protein-1 and TGF-beta signaling were overacti

37 rotein carbonyls and reduced the activity of activator protein-1 and the level of proliferating cellu

38 ogic events through the transcription factor activator protein-1 and transcription-independent contro

39 ly conserved JNK/AP-1 (Jun N-terminal kinase/activator protein 1) and BMP (Bone Morphogenetic Protein

40 g CD4(+) T cell activation: NF-kappaB, AP-1 (activator protein 1), and NFAT (nuclear factor of activa

41 h the JNK pathway, reduced the activation of activator protein 1, and decreased the expression of MMP

42 g of transcription factors nuclear factor-Y, activator protein 1, and specificity protein 1, respecti

43 s binding to transcription factors NFkappaB, Activator Protein-1, and CCAAT/enhancer-binding protein

44 55,212-2 acts via PPARalpha to activate JNK, activator protein-1, and positive regulatory domain IV t

45 presence of putative nuclear factor kappaB, activator protein-1, and STAT-1 response elements.

46 r; competition with the transcription factor activator protein 1; and reduced expression of histone d

47 The signal-dependent transcription factor activator protein 1 (AP-1) activates Notch4 transcriptio

48 ted T-cells (NFAT) c1, whereas NF-kappaB and activator protein 1 (AP-1) activation were not adversely

49 e effect of Tcl1 expression on NF-kappaB and activator protein 1 (AP-1) activity.

50 nase, and phospho-cJun, as well as decreased activator protein 1 (AP-1) activity.

51 cription factors nuclear factor (NF)-kappaB, activator protein 1 (AP-1) and CCAAT/enhancer binding pr

52 Activation of activator protein 1 (AP-1) and nuclear factor kappaB (NF

53 marked increase in UV-induced activation of activator protein 1 (AP-1) and nuclear factor kappaB (NF

54 ed HSPC specification by activating a unique activator protein 1 (AP-1) and runx1 transcription progr

55 nhancer of activated B cells (NF-kappaB) and activator protein 1 (AP-1) are transcription factors act

56 Activator protein 1 (AP-1) binding and transcriptional a

57 Radiation-induced AC gene transactivation by activator protein 1 (AP-1) binding on the proximal promo

58 In contrast, methylation of CpGs in the activator protein 1 (AP-1) binding sites was low but was

59 c-Jun transcription factors, members of the activator protein 1 (AP-1) complex, form heterodimers an

60 Cell-type-specific activator protein 1 (AP-1) complexes occupied NOTCH4 chr

61 2-related factor 2 (Nrf2) and members of the activator protein 1 (AP-1) family of proteins.

62 f interferon regulatory factor 4 (IRF4) with activator protein 1 (AP-1) family proteins and STAT3 in

63 ctor 7) are highly homologous members of the activator protein 1 (AP-1) family.

64 Activator protein 1 (AP-1) is a pivotal transcription fa

65 T: Activator protein 1 (AP-1) is a transcription factor tha

66 The transcription factor activator protein 1 (AP-1) is formed through the dimeriz

67 The transcription factor activator protein 1 (AP-1) is involved in cellular proli

68 d to measure the effect of Smad3, MAPKs, and activator protein 1 (AP-1) on TGFbeta-mediated CCN3 prom

69 measure the effects of Smads 2, 3, and 7 and activator protein 1 (AP-1) on TGFbeta-mediated CTGF prom

70 factors nuclear factor kappaB (NFkappaB) and activator protein 1 (AP-1) regulate key proinflammatory

71 a cyclic AMP response element (CRE) and two activator protein 1 (AP-1) response elements in the muri

72 oter by H. pylori involved occupation of the activator protein 1 (AP-1) sites at -72 and -181 and, su

73 FAT), nuclear factor kappaB (NF-kappaB), and activator protein 1 (AP-1) to induce type 2 cytokines.

74 ctivation of specific signaling proteins and activator protein 1 (AP-1) to regulate cell cycle regres

75 Hypoxia-inducible factor 1 (HIF-1) and activator protein 1 (AP-1) transcription complexes were

76 show that in Drosophila, quenching of basal activator protein 1 (AP-1) transcription factor activity

77 The actin-binding protein Ezrin and the activator protein 1 (AP-1) transcription factor are impl

78 Activation of the activator protein 1 (AP-1) transcription factor as well

79 ding enhancer element that contains multiple activator protein 1 (AP-1) transcription factor binding

80 d, and normal cellular FOS forms part of the activator protein 1 (AP-1) transcription factor complex,

81 JUN proto-oncogene, which forms part of the activator protein 1 (AP-1) transcription factor complex.

82 The Activator Protein 1 (AP-1) transcription factor subunit

83 K activation was required for the binding of activator protein 1 (AP-1) transcription factor to the M

84 Activator protein 1 (AP-1) transcription factors become

85 y, COP1 was also shown to be able to inhibit activator protein 1 (AP-1) transcription.

86 Following shear-flow exposure, six activator protein 1 (AP-1) transcripts (ATF4,JUNB,JUN,FO

87 Activation of NF-kappaB and activator protein 1 (AP-1) was evaluated by electrophore

88 The expression of the activator protein 1 (AP-1) was increased in lung tissues

89 Additionally, activator protein 1 (AP-1) was shown to be essential in

90 he nuclear factor kappaB (NF-kappaB) and the activator protein 1 (AP-1) with a corresponding depletio

91 ription factors, nuclear factor (NF)-kappaB, activator protein 1 (AP-1), and interferon regulatory fa

92 mponent of the transcription factor complex, activator protein 1 (AP-1), and promoted SIRT1 associati

93 -activated protein (MAP) kinase signaling to activator protein 1 (AP-1), and thus contribute to resis

94 We found that activator protein 1 (AP-1), Ets related gene (Erg) and G

95 ellular signal-regulated kinase 1/2 (Erk1/2)-activator protein 1 (AP-1), known collectively as the Ra

96 downstream target gene c-fos, a component of activator protein 1 (AP-1), that directly regulates epit

97 inal kinase (JNK), results in stimulation of activator protein 1 (AP-1), which promotes gene transcri

98 promoter and exhibit a strong enrichment for activator protein 1 (AP-1)-binding events, suggesting th

99 gene regulation by the transcription factor activator protein 1 (AP-1).

100 Activator protein 1 (AP-1, also known as JUN) transcript

101 bital-responsive enhancer module (PREM), and activator protein 1 (AP-1-) -driven pathways.

102 proximal cAMP-response element (-45/-38) or activator protein-1 (AP-1) (-207/-201) sites in the 9-kb

103 ear factor of activated T cells (NFATs), and activator protein-1 (AP-1) activation and preceded by a

104 ies Sulfiredoxin (Srx) as a unique target of activator protein-1 (AP-1) activation and TAM67 inhibiti

105 uggest that p38 is necessary for Nef-induced activator protein-1 (AP-1) activation, as inhibition lea

106 inhibition was associated with induction of activator protein-1 (AP-1) activity and stimulation of a

107 , SAG inhibited TPA-induced c-Jun levels and activator protein-1 (AP-1) activity in both in vitro pri

108 ffect in inducing ERK1/2 phosphorylation and activator protein-1 (AP-1) activity in both primary Syri

109 enzo(a)pyrene diol-epoxide [B(a)PDE]-induced activator protein-1 (AP-1) activity in mouse epidermal C

110 ated with an increase in VEGF transcription, activator protein-1 (AP-1) activity, and JunB accumulati

111 GSK3beta activation significantly stimulated activator protein-1 (AP-1) activity.

112 Six of these factors, including activator protein-1 (AP-1) and cAMP responsive element b

113 rapy-induced HB-EGF was largely dependent on activator protein-1 (AP-1) and NF-kappaB activation.

114 factor of activated T-cells (NFAT), but not activator protein-1 (AP-1) and NFkappaB, suggesting that

115 Fos and Jun are components of activator protein-1 (AP-1) and play crucial roles in the

116 Previously, we implicated Akt and activator protein-1 (AP-1) as mediators of growth and ge

117 ent on nuclear factor-kappaB (NF-kappaB) and activator protein-1 (AP-1) binding and sensitive to phar

118 region of CXCL16 and identified a functional activator protein-1 (AP-1) binding motif.

119 wo DHSs at -13 kb and -11.5 kb which contain activator protein-1 (AP-1) binding sites, both of which

120 n addition, we found that MTA1/polymerase II/activator protein-1 (AP-1) co-activator complex interact

121 hown that COBRA1 can negatively regulate the activator protein-1 (AP-1) complex at the TFF1 promoter

122 c-Jun is a component of the activator protein-1 (AP-1) complex, which plays a crucia

123 and site-directed mutagenesis identified an activator protein-1 (AP-1) consensus site at -76 relativ

124 vels of nuclear factor kappaB (NFkappaB) and activator protein-1 (AP-1) DNA binding were increased by

125 -/-)/Apc(Min/+) showed dramatic increases in activator protein-1 (AP-1) DNA binding, and SOCS2 overex

126 Here we show the activator protein-1 (AP-1) factor JunB is an essential r

127 LIP(L) is transcriptionally regulated by the activator protein-1 (AP-1) family member protein c-Fos.

128 The activator protein-1 (AP-1) family transcription factor,

129 The transcription factor activator protein-1 (AP-1) has been implicated as a driv

130 n about the role of the transcription factor activator protein-1 (AP-1) in ABC DLBCL.

131 ssociated with activation of NF- kappa B and activator protein-1 (AP-1) in HEK293A cells but only wit

132 sensus binding site for transcription factor activator protein-1 (AP-1) is required for promoter acti

133 Our studies continue with the Jun/Fos activator protein-1 (AP-1) leucine zipper complex, as it

134 that inhibition of the transcription factor activator protein-1 (AP-1) may contribute to the chemopr

135 in a dose-dependent manner by activating the activator protein-1 (AP-1) member proteins c-FOS, JunD,

136 The levels of activator protein-1 (AP-1) mRNA and protein, as well as

137 Additionally, JunD but not JunB formed an activator protein-1 (AP-1) oligomeric complex to augment

138 not binding of the corresponding proteins to activator protein-1 (AP-1) or nuclear factor-kappaB (NF-

139 Activator protein-1 (AP-1) regulates a wide range of cel

140 Activator protein-1 (AP-1) regulates diverse gene respon

141 0 and IL-12Rbeta2 via inhibition of the MAPK-activator protein-1 (AP-1) signaling pathway.

142 Furthermore, deletion studies revealed an activator protein-1 (AP-1) site in the ceruloplasmin pro

143 f transcription factors to the NF-kappaB and activator protein-1 (AP-1) sites on the IL-6 promoter.

144 ven genes by attenuating the availability of activator protein-1 (AP-1) sites to Jun family signal-de

145 omoting redistribution of TAp73 from TP53 to activator protein-1 (AP-1) sites.

146 to the pro-inflammatory transcription factor activator protein-1 (AP-1) through protein-protein inter

147 proteins NFAT1-4 and NF-kappaB complex with activator protein-1 (AP-1) to transactivate target genes

148 to the c-fos promoter resulting in increased activator protein-1 (AP-1) transactivation.

149 gh EGF receptor and Akt results in increased activator protein-1 (AP-1) transcription factor activity

150 Although induction of activator protein-1 (AP-1) transcription factor activity

151 The proto-oncogene c-Jun is a component of activator protein-1 (AP-1) transcription factor complexe

152 Because of the important role of the activator protein-1 (AP-1) transcription factor in cance

153 nic, linkage and microarray studies that the activator protein-1 (AP-1) transcription factor JunD is

154 is an IDP that acts as a potentiator of the Activator Protein-1 (AP-1) transcription factor.

155 ty, which was associated with recruitment of activator protein-1 (AP-1) transcription factors and was

156 To examine the consequences of inhibiting activator protein-1 (AP-1) transcription factors in skin

157 o the nuclear factor kappa B (NF-kappaB) and activator protein-1 (AP-1) transcriptional activators.

158 ated TF expression depended most strongly on activator protein-1 (AP-1) transcriptional activity and

159 sphorylation and a corresponding increase in activator protein-1 (AP-1) transcriptional activity.

160 ranscriptional activation of c-Fos-dependent activator protein-1 (AP-1) via serum response factor (SR

161 e sulfate activates the transcription factor activator protein-1 (AP-1) via stimulation of transient

162 haracteristically contained motifs that bind activator protein-1 (AP-1), a pivotal regulator of infla

163 TNF is a potent inducer of activator protein-1 (AP-1), and flavopiridol abrogated t

164 -1), Sp1, nuclear factor-kappaB (NF-kappaB), activator protein-1 (AP-1), and hypoxia-inducible factor

165 , such as nuclear factor kappaB (NF-kappaB), activator protein-1 (AP-1), and signal transduction and

166 The transcription factor, activator protein-1 (AP-1), binds to cognate DNA under r

167 cer of activated B cells (NF-kappaB), STAT3, activator protein-1 (AP-1), hypoxia-inducible factor-1 (

168 e-induced promoter activity of NF-kappaB and activator protein-1 (AP-1), indicating that NF-kappaB an

169 which is a component of transcription factor activator protein-1 (AP-1), to the promoter region of mi

170 elevated CYR61 induced transcription factor activator protein-1 (AP-1), which functions to stimulate

171 Pdcd4 inhibited by 46% TPA-induced activator protein-1 (AP-1)-dependent transcription, an e

172 ranscription factor-2 or JunD, and increased activator protein-1 (AP-1)-mediated endogenous transcrip

173 Pdcd4 also suppresses the transactivation of activator protein-1 (AP-1)-responsive promoters by c-Jun

174 e transcription factors NF-kappaB itself and activator protein-1 (AP-1).

175 ctors nuclear factor-kappa B (NF-kappaB) and activator protein-1 (AP-1).

176 C2/Rbx2 is a novel transcriptional target of activator protein-1 (AP-1).

177 S-2 and IGF-I, requires transcription factor activator protein-1 (AP-1).

178 oblast differentiation via the activation of activator protein-1 (AP-1).

179 receptor 1) was identified as a novel c-Fos/activator protein-1(AP-1)-regulated gene.

180 es which also contain many binding sites for activator protein 1 (AP1) and cyclic AMP response elemen

181 ensus DNA sequence for transcription factors activator protein 1 (AP1) and specificity protein 1 (SP1

182 tion factor (GABP-alpha) to their respective activator protein 1 (AP1) and v-ets erythroblastosis vir

183 high-throughput sequencing, we identify the activator protein 1 (AP1) as a major partner for product

184 increased in SUM44/LCCTam, and inhibition of activator protein 1 (AP1) can restore or enhance TAM sen

185 ase kinase 7 (MKK7)/c-Jun-NH(2)-kinase (JNK)/activator protein 1 (AP1) cascade.

186 Deregulation of the activator protein 1 (AP1) family gene regulators has bee

187 The activator protein 1 (AP1) family transcription factor BA

188 its relationship to the transcription factor activator protein 1 (AP1) in CHF rabbits and in the CATH

189 extracellular signal-regulated kinase (Erk)-activator protein 1 (Ap1) pathway seems to mediate the e

190 obility supershift assay using an involucrin activator protein 1 (AP1) response element sequence reve

191 including nuclear factor-kappaB (NF-kappaB), activator protein 1 (AP1), signal transducer and activat

192 ein (CHOP), X2-Box-binding protein 1 (XBP1), activator protein 1 (AP1), SMAD, CCAAT/enhancer-binding

193 we identified several functional NF-kappaB, activator protein 1 (AP1), STAT, and Smad DBS in the TSL

194 on in nonsteroidogenic cells, likely through activator protein 1 (AP1).

195 affect the ubiquitin-editing enzyme A20 and activator protein 1 (AP1).

196 sion that is mediated by DNA-bound NF-kappaB/activator protein 1 (AP1)/STAT3 activators and instrumen

197 tion factors such as nuclear factor B (NFB), activator protein-1 (AP1) and heat shock factor 1 (HSF1)

198 ressed, IRF4 unexpectedly can cooperate with activator protein-1 (AP1) complexes to bind to AP1-IRF4

199 ERK activity promoted the expression of the activator protein-1 (AP1) components Fra-1 and c-Jun, bo

200 such as nuclear factor-kappaB (NFkappaB) and activator protein-1 (AP1) for 24 hours after separation

201 The activator protein-1 (AP1) transcription complex remains

202 ear factor of activated T cells c1 (NFATc1), activator protein-1 (AP1), and NF-kappaB.

203 nscription factors nuclear factor-kappaB and activator protein-1 are activated in acne lesions with c

204 3beta (hepatocyte nuclear factor 3) and AP-1(activator protein 1) as proteins likely to be involved i

205 late Mmp13 expression via the P2rx7/MAPK/ERK/activator protein 1 axis.

206 atin immunoprecipitation (ChIP), we detected activator protein-1 binding within an evolutionarily con

207 by COOH-truncated HBx was abolished when the activator protein 1-binding sites on the MMP10 promoter

208 on of the inflammatory transcription factor, activator protein 1, but not NF-kappaB was observed.

209 n oxidant-mediated phenomenon acting through activator protein-1, but not nuclear factor kappaB, path

210 otein-1 reporter activity, but activation of activator protein-1 by the three SLK mutants was ineffec

211 HGF treatment caused activation of the activator protein-1, CCAAT/enhancer-binding protein, and

212 possibly because of decreased activation of activator protein-1, compared with control cells overexp

213 ing of a phosphorylated c-Jun containing the activator protein 1 complex to the PUMA promoter was ide

214 FRA-1 forms activator protein-1 complexes in association with member

215 X1, PREP1 induces the expression of multiple activator protein 1 components including the proinvasive

216 th the inflammatory cytokines and/or various activator protein-1 constructs.

217 ions involving several signaling modulators, activator protein-1-dependent gene expression remains hi

218 d, in turn, to reduced expression of several activator protein-1-dependent genes (COX-2, cyclin D1, c

219 was not due to an increase in NF-kappaB- or activator protein-1-dependent IL-8 promoter transcriptio

220 the nuclear factor of activated T cell- and activator protein-1-dependent signaling pathways, which

221 y phosphorylation of c-Jun and activation of activator protein-1-dependent transcription.

222 RK2), in turn leading to inhibition of c-Jun/activator protein-1-dependent transcriptional activity.

223 oblast mmp9 and mmp13 promoters, established activator protein 1 effectors.

224 kappaB) and epidermal growth factor receptor-activator protein-1 (EGFR-AP1) pathways are often co-act

225 transcription through the recruitment of an activator protein-1/estrogen receptor-alpha (ER alpha) c

226 ivated involucrin promoter activity, nuclear activator protein-1 factor accumulation and binding to D

227 ene c-jun encodes the founding member of the activator protein-1 family and is required for cellular

228 ization of their regulators (Rap1 [repressor activator protein 1], Fhl1, Ifh1, Sfp1, and Hmo1), the t

229 We verified activation of CREB and activator protein 1 in polarized cells by gel shift assa

230 ncreases the expression of ET(A) R, OBRb and activator protein-1 in HSCs.

231 ion of mitogen-activated protein kinases and activator protein-1 in myofibers of mdx mice.

232 anscriptional activity of both NF-kappaB and activator protein 1, in part by means of recruitment of

233 AR-small interfering RNA or treated with the activator protein-1 inhibitor SR-11302 [3-methyl-7-(4-me

234 Activator protein 1 is known as a pivotal regulator of m

235 Activator protein-1 is a key participant in Smad-depende

236 Rap1 (repressor activator protein 1) is a conserved multifunctional prot

237 Rap1 (repressor-activator protein 1) is a multifunctional protein that c

238 d by the nuclear factor of activated T cells.activator protein 1 (NF-AT.AP-1) complex.

239 ing from GPCRs and RhoA to the regulation of activator protein-1, NFkappaB (nuclear factor kappa-ligh

240 xtracellular signal-regulated kinase 1/2 and activator protein-1 nuclear factors in IL-13Ralpha2-posi

241 NA-binding activity of transcription factors activator protein-1, nuclear factor-kappaB, and Stat3; a

242 ygenase-2 expression, the transactivation of activator protein-1 or nuclear factor-kappaB, or MEK.

243 No significant alterations were observed for activator protein 1, p53 or Akt activity.

244 These results demonstrate that the c-Jun/activator protein 1 pathway is critical for maintaining

245 tivated the mitogen-activated protein kinase/activator protein 1 pathway, together with the inflammas

246 its target gene programmed cell death 4 and activator protein 1 pathway.

247 llular injury response through PDCD4 and the activator protein 1 pathway.

248 kinase 1/2 mitogen-activated protein kinase/activator protein-1 pathway activation.

249 s Hoxc6 with oncogenic signaling through the activator protein-1 pathway in carcinoid tumorigenesis.

250 ceptor gamma agonist rosiglitazone decreased activator protein 1 promoter activity.

251 Repressor activator protein 1 (RAP1) and telomeric repeat-binding

252 Repressor/activator protein 1 (RAP1) is a highly conserved telomer

253 Repressor activator protein 1 (Rap1) performs multiple vital cellu

254 We show that repressor-activator protein 1 (Rap1), a master regulator of yeast

255 ide direct evidence that the yeast repressor/activator protein 1 (Rap1), tightly bound to its consens

256 ct contact with the transactivator repressor activator protein 1 (Rap1).

257 rolled by the transcription factor repressor activator protein 1 (Rap1p) in a TFIID-dependent fashion

258 ssive activation of the transcription factor activator protein 1, reduced histone deacetylase-2 (HDAC

259 the telomere-binding protein Rap1 (repressor activator protein 1) relocalizes to the upstream promote

260 Similarly, SLK WT stimulated activator protein-1 reporter activity, but activation of

261 ERbeta-dependent retardation of migration of activator protein-1 response elements in EMSA.

262 Our findings demonstrate that the JNK/activator protein 1 signaling pathway underlies the mela

263 ated by cyclooxygenase/nuclear factor-kappaB/activator protein 1 signaling pathways.

264 xpression is up-regulated by ROS through the activator protein-1 signaling pathway and promotes cell

265 Hoxc6 activated the oncogenic activator protein-1 signaling pathway through a physical

266 dermal cells by targeting the MEK/ERK/p90RSK/activator protein-1 signaling pathway.

267 l-regulated kinase 1/2 pathway, involving an activator protein 1 site in MMP10 gene promoter.

268 the region of the PR gene containing the +90 activator protein 1 site, but not with the ERE-containin

269 et psoriasis and are close to the functional activator protein-1 site (+419) through which retinoids,

270 d transcriptional activation of the relevant activator protein-1 site in the human TGFbeta1 promoter.

271 enhanced c-Fos/c-Jun binding to the proximal activator protein-1 site of the StAR promoter in HPAECs,

272 binding of c-Fos and c-Jun to Mmp-9 promoter activator protein 1 sites.

273 rotein partner of DeltaFosB binding to AP-1 (activator protein-1) sites of genes, remained unchanged

274 Batf belongs to the activator protein 1 superfamily of basic leucine zipper

275 TbPIP5Pase associates with repressor/activator protein 1 (TbRAP1), and their telomeric silenc

276 Tax is shown to activate activator protein-1 through the phosphatidylinositol 3-k

277 absence of NF-kappaB signaling, can activate activator protein-1 to promote cellular proliferation an

278 and E7 were mediated by enhanced binding of activator protein-1 to the cyclic AMP (cAMP)-responsive

279 Ser(63) and Ser(73), resulting in increased activator protein-1 transactivation.

280 a highly conserved member of the multimeric activator protein 1 transcription factor complex and pla

281 suprabasal epidermis-specific inhibition of activator protein 1 transcription factor signaling.

282 kappaB p65, and the nuclear translocation of activator protein 1 transcription factor.

283 -13 promoters that contain binding sites for activator protein 1 transcription factors.

284 ough serine 84 phosphorylation and promoting activator protein 1 transcription.

285 on, emphasizing compositional differences in activator protein-1 transcription factor activity and ex

286 The oncoprotein c-Jun is a component of the activator protein-1 transcription factor complex, which

287 V-1 and HTLV-2 but instead contains a unique activator protein-1 transcription factor upstream of the

288 y our study indicated that MAPK pathways and activator protein-1 transcription factor were involved i

289 xpression had no effect on activation of the activator protein-1 transcription factor.

290 st, MG132 and LC potentiated the activity of activator protein-1 transcription factor.

291 ell as their major downstream effectors--the activator protein-1 transcription factors c-Fos and c-Ju

292 with increased expression of Fra-1 and JunD, activator protein-1 transcription factors known to be re

293 IL-1beta signaling converges upon the activator protein-1 transcription factors, which have be

294 ng protein kinase Calpha and is dependent on activator protein-1 transcription factors.

295 tion and nuclear factor of activated T cells/activator protein 1 transcriptional activity.

296 Although the c-Jun/activator protein 1 transcriptional factor regulates cel

297 by Cav1 knockdown to increased expression of activator protein-1 transcriptional targets, including c

298 tor, endothelin-1 type A receptor (ET(A) R), activator protein-1, transforming growth factor beta (TG

299 Moreover, activator protein 1 was a downstream signaling molecule

300 Soluble LT also led to the activation of activator protein 1, whereas LT(+) vesicle IL-6 response