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2 hat, during activation by wild-type CRP, the activatory and inhibitory determinants of AR3 balance ea
5 hat Dok-3/Grb2 modulates the balance between activatory and inhibitory Lyn functions with the aim to
6 lobulin-like receptors encode MHC-I-specific activatory and inhibitory receptors, implying that MHC-I
7 age ligands, facilitating the integration of activatory and inhibitory signals from multiple receptor
8 ms, including NtrC (where phosphorylation is activatory), and indicate that recognition properties, w
9 ependent promoters by making three different activatory contacts with different surfaces of holo RNA
11 nitiation assays were used to show that this activatory determinant accelerates the rate of isomerisa
12 de that these residues in AR3 of CRP form an activatory determinant for Class II transcription activa
13 lts provide a structural explanation for the activatory effect of this mutation and provide a greater
14 ngage with CD14(+) monocytes in a reciprocal activatory fashion, thereby amplifying the inflammatory
16 bitory FcgammaRIIB in 3/23 activity, whereas activatory FcgammaR (FcgammaRI, -III, and -IV) was dispe
18 cytidylic acid or were given subcutaneously, activatory FcgammaR could also provide cross-linking.
23 ants like STINGa, which can improve FcgammaR activatory:inhibitory (A:I) ratios on TAM, are appealing
26 NKp30 that upon interaction with its cognate activatory ligand B7-H6 induces rapid production of type
28 nition of tumor cells expressing the NK cell-activatory ligands H60 or CD137L by mouse natural killer
32 ity of MT1-MMP from its enzymatic toward its activatory mode, suggesting a tight mechanosensitive reg
34 s that contain immunoreceptor tyrosine-based activatory motifs to mediate NK direct lysis of tumor ce
35 ne the structural changes that arise from an activatory mutation (S205L) located in the cytoplasmic d
40 ectively, our findings identify a novel auto-activatory pathway that is essential for optimal NK cell
41 dSTRIPAK depletion leads to increased Hpo activatory phosphorylation and repression of Yki target
44 ations (inhibitory monoubiquitination versus activatory phosphorylation), and kinetic/allosteric prop
45 This astrocyte-specific regulation involves activatory protein-1 (AP-1) because overexpression of do
46 Fc receptor (FcR) gamma-chain complex, a key activatory receptor for collagen on platelet surface mem
48 n epithelia, and is the major ligand for the activatory receptor, NKG2D, both attributes relevant to
55 of cellular proteins including c-Src at the activatory site, and this effect is blocked when the 5'-
57 ell priming if provided appropriately phased activatory stimuli, an observation that could be factore
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