ライフサイエンスコーパス: active oxygen

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1 iOO(-)), which can be described as adsorbed "active oxygen".

2 sites relative to Co(3+) and by the shift of active oxygen 2p states.

3 nd productive orientation between heme-bound active oxygen and acceptor carbon bond(s).

4 ressing VU-3 calmodulin exhibited a stronger active oxygen burst that occurred more rapidly than in n

5 verall, our study highlights a novel, highly active oxygen evolution catalyst; moreover, it provides

6 A highly active oxygen-evolving photosystem II (PSII) complex was

7 moval of this extension is necessary to form active oxygen-evolving photosystem II centers.

8 trocatalyst for those reactions that involve active oxygen intermediates, such as reduction of oxygen

9 late the properties of both the monoxygenase active-oxygen intermediates and the proton-delivery netw

10 ectroscopy has been used to confirm that the active oxygen of HSO(5-) is nonexchanging (t(1/2) >> 1 h

11 d responses including programmed cell death, active oxygen production and transcription of the pathog

12 reasing the surface density of catalytically active oxygen radical sites on a MoVTeNb oxide (M1 phase

13 a previously unknown role for pyridoxine in active oxygen resistance.

14 O(3)(001) involving surface peroxides as the active oxygen source is suggested.

15 timing and magnitude with development of the active oxygen species (AOS) burst.

16 Active oxygen species (AOS) generated in response to sti

17 The use of relatively low amounts of active oxygen species (such as hypochlorite), followed b

18 eviously reported that hydrogen peroxide, an active oxygen species and a cellular oxidant, induces c-

19 recently reported that hydrogen peroxide, an active oxygen species and a cellular oxidant, stimulates

20 stant attack from spontaneous hydrolysis and active oxygen species and from other intracellular metab

21 r9, respectively, triggered the synthesis of active oxygen species and MAP kinase activation.

22 and by ozone treatment is the production of active oxygen species and the accumulation of hydrogen p

23 ctors in addition to ceruloplasmin, possibly active oxygen species and/or lipoxygenases, are essentia

24 The cumulative results suggest that active oxygen species are generated near cell walls of v

25 ls and yeast DNA repair mutants sensitive to active oxygen species are not sensitive to this agent, t

26 , indicating that the peroxide is likely the active oxygen species attacking the aromatic substrates.

27 synthesis and phosphorylation in response to active oxygen species could play a role in anti-oxidativ

28 in is likely to have a role in metabolism of active oxygen species derived from internal body metabol

29 yrin radical cation intermediate that is the active oxygen species in these hydroxylation reactions.

30 his paper two hypotheses are tested: (i) the active oxygen species is similar in energetics for all c

31 ch once released can be readily converted to active oxygen species or to dissolved dioxygen.

32 in vitro or in cultured cells also generates active oxygen species such as superoxide, which can indi

33 ed NrdI, responsible for the formation of an active oxygen species suggested to be either a superoxid

34 rate from heat shock protein induction, ABA, active oxygen species, and salicylic acid pathways are i

35 ed changes in SA levels, the accumulation of active oxygen species, defense gene expression, and the

36 of plant cell death and the induction of an active oxygen species-responsive promoter (AoPR1-GUS) we

37 erate atomic oxygen [O(3P)] or an equivalent active oxygen species.

38 athway that is required for the Avr9-induced active oxygen species.

39 lular toxic species in the medium, including active oxygen species.

40 se observations show that more electrophilic active-oxygen species (i.e., lower-energy LMCT) are more

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