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1 athway that is required for the Avr9-induced active oxygen species.
2 lular toxic species in the medium, including active oxygen species.
3 erate atomic oxygen [O(3P)] or an equivalent active oxygen species.
4 eviously reported that hydrogen peroxide, an active oxygen species and a cellular oxidant, induces c-
5 recently reported that hydrogen peroxide, an active oxygen species and a cellular oxidant, stimulates
6 stant attack from spontaneous hydrolysis and active oxygen species and from other intracellular metab
8 and by ozone treatment is the production of active oxygen species and the accumulation of hydrogen p
9 ctors in addition to ceruloplasmin, possibly active oxygen species and/or lipoxygenases, are essentia
10 rate from heat shock protein induction, ABA, active oxygen species, and salicylic acid pathways are i
14 ls and yeast DNA repair mutants sensitive to active oxygen species are not sensitive to this agent, t
15 , indicating that the peroxide is likely the active oxygen species attacking the aromatic substrates.
16 synthesis and phosphorylation in response to active oxygen species could play a role in anti-oxidativ
17 ed changes in SA levels, the accumulation of active oxygen species, defense gene expression, and the
18 in is likely to have a role in metabolism of active oxygen species derived from internal body metabol
19 se observations show that more electrophilic active-oxygen species (i.e., lower-energy LMCT) are more
20 yrin radical cation intermediate that is the active oxygen species in these hydroxylation reactions.
21 his paper two hypotheses are tested: (i) the active oxygen species is similar in energetics for all c
23 of plant cell death and the induction of an active oxygen species-responsive promoter (AoPR1-GUS) we
24 in vitro or in cultured cells also generates active oxygen species such as superoxide, which can indi
26 ed NrdI, responsible for the formation of an active oxygen species suggested to be either a superoxid
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