ライフサイエンスコーパス: activity control

1 ting the MID1-PP2A protein complex with GLI3 activity control.

2 brings essential elements to understand its activity control.

3 expected spatial separation of stability and activity control.

4 -treated mice without wheel access served as activity controls.

5 advanced inflammatory activity and 220 mild activity controls.

6 y decreased baseline renal sympathetic nerve activity (control, 68.5+/-7.1% of Max; 10(10) particles

7 altered, our results suggest that E protein activity controls a novel checkpoint that regulates the

8 We propose that PLK1 activity controls a polarity checkpoint and compensates

9 mitter release, indicating that postsynaptic activity controls a retrograde signal that regulates pre

10 coding level or sparseness of these neurons' activity controls a trade-off between generalization and

11 oreactors for two-phase reactions with water activity control; and environmental bioreactors.

12 re, we report how Src family tyrosine kinase activity controls apCAM-mediated growth cone steering by

13 tightly regulated molecules known: neuronal activity controls Arc mRNA induction, trafficking and ac

14 n (1:1:1) to receive routine polio programme activities (control, arm A), additional interventions wi

15 he opposite phenotype, indicating that cup-5 activity controls aspects of endocytosis.

16 n three transgenic mouse lines, two with Cre activity controlled at the transcriptional level (Ahcre,

17 results suggest a model whereby Mek1 kinase activity controls axial element assembly by regulating t

18 Understanding how dynamic changes in brain activity control behavior is a major challenge of cognit

19 upling (NVC) is the process whereby neuronal activity controls blood vessel diameter.

20 We show balanced Rac1 activity controls both leading edge protrusion and point

21 and Ste2p required the vacuolar proteolytic activities controlled by the PEP4 gene.

22 action prevents the regulation of BK channel activity controlled by changes in actin cytoskeletal dyn

23 el enhancer has been characterized, with its activity controlled by Dorsocross and Tinman transcripti

24 ecular switches, with target protein-binding activity controlled by prior binding to specific input s

25 nregulation reflects a decrease in endocytic activity controlled by Rho family GTPases, especially Cd

26 shock might be regulated by changes in PP2A activity controlled by the SET protein.

27 - L-type-Ca(2+) channels and D2-autoreceptor activity, controlled by NCS-1, and indicate that this ad

28 ivo cellular experiments indicate that TIM-1 activity controls CD4(+) T cell activity.

29 s provide a mechanism by which PP2A and DAPK activities control cell adhesion and anoikis.

30 These results show that CDK5 activity controls cell motility and metastatic potential

31 cterization of this mutant showed that ClpXP activity controls cell size and is required for growth a

32 est that the balance between Akt and caspase activity controls cell survival.

33 embrane helices, is subject to a complicated activity-control circuit involving two other proteins wi

34 t to cell culture systems, cortical synaptic activity controls CRE-mediated gene expression without a

35 ck, and from BCI/BMI studies in which neural activity controls cursors or peripheral devices.

36 ur results illustrate how a gradient of MAPK activity controls differential gene expression and, thus

37 gradient of cell movement, with WNT and FGF activities controlling direction and velocity, respectiv

38 The ATPase activity controls dissociation of an MVH complex contain

39 chanism underlying precise MCAK depolymerase activity control during mitosis remains elusive.

40 tor (TCR)-dependent regulatory T cell (Treg) activity controls effector T cell (Teff) function and is

41 its Ras guanosine triphosphatase -activating activity, controls ERK1/2-dependent fibroblast growth fa

42 , by pathways that stimulate phospholipase C activity, controls excitability throughout the nervous s

43 ta-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis prom

44 nism by which cell spreading and RhoA GTPase activity control FA formation through YAP to stabilize t

45 The results suggest that sympathetic activity controls fat-induced satiety by enabling the co

46 Because plasmin activity controls fibrinolysis in a variety of pathologi

47 We propose that neuronal activity controls filopodial motility in a developmental

48 esults suggest the importance of prestimulus activity control for improving sensory coding within the

49 tic evidence that PTEN's protein phosphatase activity controls FYN kinase function in glioma cells an

50 tructs, we found that Akt (protein kinase B) activity controlled gap junction stability and was neces

51 Neuronal NMDA receptor activity controls glial motility through intercellular a

52 However, the mechanism by which neuronal activity controls glucose influx via GLUT3 is unknown.

53 lease, but the mechanism by which electrical activity controls GnRH secretion is not well characteriz

54 An OT group, a social activity control group, and a nontreatment control group

55 No mutant alleles were found in the high activity control groups.

56 emory formation to decision making and motor activity control--have inspired their re-creation in a w

57 in suggests that it may be involved in motor activity control in basal ganglia as well as higher cent

58 We show that AKAP79/150-anchored PKA activity controls Kv4.2 surface expression in heterologo

59 n the chick limb bud, the zone of polarizing activity controls limb patterning along the anteroposter

60 se exciting results and discuss how synaptic activity controls local translation, the proteins that a

61 LRRK2 Roc-COR tandem domain exhibits GTPase activity controlling LRRK2 kinase activity via an intram

62 We propose that LKB1 kinase activity controls membrane dynamics through FAK since lo

63 regulator of midzone plus-end dynamics whose activity controls midzone length but not stability.

64 promoting factor (MPF), the major enzymatic activity controlling mitotic cycles in frog eggs, early

65 As a motor activity control [motor control (MC)], lesion and sham a

66 s" in the hippocampus of rodents to cortical activity controlling movement, temporal sequence generat

67 e randomized 296 patients to normal physical activity (control; n=145) or walking exercise (n=151); 2

68 Here we demonstrate that activity controls NMDAR synaptic accumulation by regulat

69 ipheral, segmental, and supraspinal neuronal activities control nociceptive processing at all levels

70 center and program characteristics, clinical activities, control of clinical activities, and needs an

71 een reported to be involved in recombination activity, control of gene expression of nearby gene(s) (

72 reprogramming of an organizing center whose activity controls outgrowth and patterning of the mid an

73 pylase Dop, affording a rapid and reversible activity control over Mpa function.

74 taxin (ATX), through its lysophospholipase D activity controls physiological levels of lysophosphatid

75 anti-silencer (pp52 anti-NRE) with opposing activities controlling pp52 gene expression.

76 ial activation and destruction of CDK-cyclin activities controls progression through the cell cycle.

77 Wnt16, which is also downstream of muscle activity, controls proliferation and migration, but play

78 s, and increase or decrease of KCNQ5 channel activity controlled release probability through alterati

79 ion-specific, axon repulsive and stimulatory activities control retinal axon patterning in the embryo

80 reatly improve our understanding of how PARG activity controls reversible protein poly(ADP-ribosyl)at

81 in the cNTS during dehydration, sympathetic activity control reverts back to forebrain regions.

82 tivity of Runx3 expression, and its level of activity, control sensory afferent targeting in the deve

83 Thus, Clr4 activity controls siRNA amplification from the different

84 f spatial and temporal control of the kinase activity controlling spatial patterning during multicell

85 Furthermore, neuronal activity controls synaptic AMPA receptor trafficking, an

86 We found that sensory-independent electrical activity controls synaptic maturation in IHCs.

87 Whereas previous work has focused on activities controlling TGF-beta signaling, more recent s

88 ous studies have investigated the regulatory activities controlling TGF-beta signaling, there is rela

89 tion and subsequent reactivation of cellular activities controlling the cycling of Golgi components i

90 ntial phases of Hox-c protein expression and activity control the columnar differentiation of spinal

91 tica to the ocean, and fluctuations in their activity control the mass balance of the ice sheet.

92 cetylcholine-dependent spontaneous bursts of activity control the outgrowth of receptive-field areas

93 gulated by LPC, rather than direct agonistic activity control the signaling responses of murine G2A t

94 lobal levels of adhesion strength and myosin activity control the stability of the stationary state:

95 preferentially the conditioned neurons whose activity controlled the BMI actuator during training.

96 this is achieved in part by postmeiotic gene activity controlling the development of the haploid fema

97 nhibitor of IRAK4, we show that IRAK4 kinase activity controls the activation of interferon regulator

98 We conclude that although Nkx2-3 activity controls the addressin balance of HEVs in GALT,

99 aded by the proteasome, indicating that Rbd2 activity controls the balance between SREBP activation a

100 at after postnatal day 3, glutamate-mediated activity controls the development of their axons and den

101 show that spatial regulation of ADF/cofilin activity controls the directional responses of the growt

102 ng cultured neurons have suggested that Cdk5 activity controls the efficiency of neurite extension [3

103 Collectively, these data showed that PAP activity controls the expression of PSS for membrane pho

104 Thus, telomerase activity controls the glycolytic pathway, potentially al

105 Thus, spatiotemporal ULP-4 activity controls the HMGS-1 sumoylation state in a mech

106 r olive is thought to act as a teacher whose activity controls the induction of motor learning.

107 ntegration of Bmp4 signaling and Gata factor activity controls the progression of hematopoiesis, as e

108 In this process, Lkb1 activity controls the progression of mitotic chondrocyte

109 Thus, high-frequency neuronal activity controls the ratio of extracellular proBDNF/mBD

110 During postnatal development, neuronal activity controls the remodeling of initially imprecise

111 We propose that a graded distribution of BMP activity controls the specification of several cell type

112 In addition, CK2 activity controls the subcellular organization of indivi

113 er understand how this intrinsic oscillatory activity controls the susceptibility of the brain to sti

114 We propose Cdk1 activity controls the timing of telomere elongation by r

115 We show that PP1beta, by regulating myosin activity, controls the generation of the polarizing sign

116 fluxes integrate mitochondria into cellular activities, controlling their volume homeostasis and str

117 ich to understand how the dynamics of neural activity control this search behaviour.

118 reveals how spatiotemporal patterns of gene activity control tissue shape by introducing several typ

119 genome is recombinogenic, with DNA exchange activity controlled to a large extent by nuclear gene pr

120 complex animal behaviors depends on precise activity control tools, as well as compatible readout me

121 , our results suggest that WAT hematopoietic activity controls WAT inflammatory processes and also su