ライフサイエンスコーパス: acute

1 ionally, fish were subjected to one of three acute (24 h) temperature treatments: cold stress (18 deg

2 acutely activated by caloric need, and this acute activation promotes increased food intake and decr

3 Also, acute administration of psychostimulants, at levels repo

4 dings suggest that the beneficial effects of acute aerobic exercise on inhibitory control are sustain

5 ne whether recAP prevents the progression of acute and acute-on-chronic liver failure (ACLF).

6 an unanticipated role of HIF2alpha, and link acute and chronic adaptive responses to hypoxia.

7 We used state-of-the-art models for both acute and chronic HBV infection to study the pathogen-cr

8 , we analyzed multiple brain regions through acute and chronic infection (90 days postinfection [dpi]

9 Rat miRNA profiles identified TBI across all acute and chronic intervals.

10 ve capacity, and increased susceptibility to acute and chronic lung diseases.

11 e are clinically relevant sex differences in acute and chronic pain mechanisms, but we are only begin

12 Activity-escalated acute and chronic toxicity studies of (225)Ac-L1 reveale

13 ause these compounds have been associated to acute and chronic toxicity, mainly in the liver.

14 is and persistence as well as arthralgias in acute and convalescent filovirus disease.

15 s up to day 84 from 10 outbreaks, as well as acute and convalescent sera were collected.

16 aced with a plethora of challenges including acute and critical care management, long-term care and r

17 functional ganglionic CD8(+) T cells during acute and latent HSV-1 infection.

18 t or treatment regimens that may affect both acute and long-term effects of manipulating the activity

19 d clinical characteristics and self-reported acute and persistent symptoms were assessed using a stan

20 This decrease is due to acute and reversible action of TNF-alpha and is not asso

21 stent with this, ex vivo exposure of LSVs to acute arterial WSS promoted monocyte interactions with t

22 Sixteen adults presenting to the ED with acute asthma exacerbations were recruited after giving i

23 atory cells invade only the stroma during an acute attack.

24 ged mice, the latter group developed greater acute axonal and myelin loss attributed to elevated oxid

25 ia (PNA), urinary tract infection (UTI), and acute bacterial skin and skin structure infection (ABSSS

26 ong-range traffic of DAT in intact brain and acute brain slices from the knock-in mouse expressing ep

27 an FEV(1) >100% of predicted, but during an acute bronchospastic episode can experience a life-threa

28 Acute cardiorespiratory responses to O(2) deficiency are

29 into the inpatient management of adults with acute cardiovascular events.

30 worldwide, yet most attention has focused on acute care and the impact on long-term health is poorly

31 d patient-level characteristics and transfer acute care hospitals (ACHs) as risk factors for coloniza

32 identified; cases were attributed mostly to acute care hospitals (ACHs; 141, 50%) and skilled nursin

33 113, 40%), and less frequently to long-term acute care hospitals (LTACHs; 29, 10%).

34 domised, placebo-controlled study done in 48 acute care hospitals in eight countries, we enrolled pat

35 sentative database of all discharges from US acute care hospitals.

36 sions, inpatient professional fees, and post-acute care utilization.

37 ation of the medial meniscus (DMM) and after acute cartilage injury and repair.

38 gulated on renal tubular epithelial cells by acute cell-mediated rejection (ACR.

39 The possible increased risk of acute cerebrovascular disease in patients with dual infe

40 We used rat hippocampal cultures and their acute cholesterol depletion by methyl-beta-cyclodextrin

41 Acute choroidal ischemia, defined as any new choroidal i

42 terruptions in healthcare will contribute to acute chronic disease complications.

43 ce maintenance is disrupted by an episode of acute CMV infection.

44 Delirium is an acute confusional state that is common and costly and is

45 Patients with AA were older, presented with acute congestive heart failure or non-ST-segment-elevati

46 F-COAST, a prospective multicenter cohort of acute coronary syndrome (ACS), in relation to BB use: pr

47 cific thresholds, in patients with suspected acute coronary syndrome admitted to Swedish coronary car

48 n A-I were associated with increased odds of acute coronary syndrome and its manifestations in indivi

49 es in patients with non-ST-segment elevation acute coronary syndrome undergoing invasive treatment.

50 scular death, myocardial infarction or other acute coronary syndrome, stroke, or coronary revasculari

51 onary artery disease and 18 046 (56.1%) with acute coronary syndrome.

52 P2Y(12) Receptor Blockers In Non-ST Elevated Acute Coronary Syndromes With Initial Invasive Indicatio

53 ing Computerized Tomography in Patients With Acute Coronary Syndromes) trial (NCT02061891) evaluated

54 n beds were most needed for the treatment of acute COVID-19 cases, private providers suffered a liqui

55 Neutrophils from patients with acute Crohn's disease, rheumatoid arthritis, or sepsis w

56 sociopolitical events, perceived stress, and acute CVD events.

57 patocytes in zone 3 of the Rappaport acinus (acute damage), supporting the hypothesis of ACLF.

58 Acute decompensated heart failure (ADHF) is a highly mor

59 937 weighted hospitalizations) classified as acute decompensated HF had available ejection fraction d

60 cells expressing IL-2 receptor beta cause an acute decrease in cellularity of Peyer's Patches while c

61 line or localized conduit necrosis (9%), and acute delirium (5%).

62 Acute desensitization of the potassium conductance induc

63 ished during development, or if they reflect acute differences in activity-dependent regulation of cr

64 d by transient dFB activation, confirming an acute disconnect from the external environment.

65 nalogues, can be used to monitor episodes of acute disease activity.

66 extent globally (1,310 km(2) ) following an acute disturbance.

67 Extrapolation to all 12 acute/early participants estimated a much smaller reduct

68 No acute effect on passive muscle stiffness was observed.

69 Acute epiploic appendagitis (EA) is a relatively rare, b

70 of functional antibodies follows survival of acute EVD.

71 nosinusitis (CRS) is complicated by frequent acute exacerbations leading to significant health care b

72 ge sample of community-dwelling individuals, acute exercise elicits widespread changes in the circula

73 versible tissue damage under dysregulated or acute exposure conditions.

74 (-/-) and TLR4(-/-) RAGE(-/-) mice following acute exposure to cigarette smoke (CS).

75 In terms of cardiac physiology, acute exposure to e-cigarette aerosols in human subjects

76 of symptomatic dengue patients experience an acute febrile illness, 5-20% progress to severe infectio

77 e extent of resistance of E. dermatitidis to acute gamma-radiation exposure and the major mechanisms

78 Noroviruses are a leading cause of acute gastroenteritis.

79 Acute genetic perturbations that eliminated caveolae in

80 disease (OR = 2.67; 95% CI = 1.12-6.32), and acute graft rejection (OR = 3.01; 95% CI = 1.78-5.09).

81 Steroid-resistant or steroid-refractory acute graft-versus-host disease (SR-aGVHD) poses one of

82 e serum (L1S1) antibody completely prevented acute GVHD.

83 nts were enrolled during hospitalisation for acute heart failure from 358 centres in 44 countries on

84 nts have higher rates of hospitalization for acute heart failure than other race/ethnic groups.

85 Notably, short-term acute heat stress assays resolved per-colony (genotype)

86 scopy in primary cortical cultures, and from acute hippocampal and cortical slices from male wild-typ

87 Electrophysiological recordings on acute hippocampal slices showed that exogenous Amh prote

88 ibing older patients' experiences of care in acute hospital settings.

89 ckade of the IL-1 receptor is therapeutic in acute hyperinflammatory respiratory failure in COVID-19

90 During acute illness, antibodies predominate to VP40 and glycop

91 osed by 2018 but gender disparities remained acute in parts of sub-Saharan Africa, and North Africa a

92 ted to CRP and IL (interleukin)-1 changes in acute infection patients.

93 l numbers in the spleen were observed during acute infection with myeloid-restricted ZIKV that preclu

94 starting antiretroviral therapy (ART) during acute infection, 2% of proviruses that persist on ART ar

95 (NAATs) are the primary means of identifying acute infections caused by severe acute respiratory synd

96 hypoxic stress manifest within monocytes in acute inflammatory lesions.

97 it of cardiac cell therapy is thus due to an acute inflammatory-based wound-healing response that rej

98 Constitutive loss or acute inhibition of the Arp2/3 regulator, N-WASP, which

99 Following acute injection, MCAM and naltrexone decreased fentanyl

100 onation patterns under homeostasis and after acute injury.

101 This compensatory mechanism is lost with acute irradiation injury, resulting in a paucity of tuft

102 n eight countries, we enrolled patients with acute ischaemic stroke due to large vessel occlusion wit

103 ter symptom onset (58.9%), including 17 with acute ischemic infarct (23.3%), one with a deep venous t

104 with faster thrombolytic treatment times for acute ischemic stroke and modestly lower 1-year all-caus

105 issue-type plasminogen activator; IV tPA) in acute ischemic stroke patients with prior ischemic strok

106 In acute ischemic stroke with unknown time of onset, magnet

107 Complications during admission included: acute kidney injury (63%), transaminitis (31%), shock (3

108 ociated with mortality in patients with both acute kidney injury (adjusted relative risk, 2.38; 95% C

109 Malnutrition is common in patients with acute kidney injury (AKI) and the risk of mortality is h

110 cutive patients with ACLF diagnosed with HRS acute kidney injury (AKI) were randomized to albumin wit

111 repair process can result in recovery after acute kidney injury (AKI) with adaptive proliferation of

112 vels were associated with the development of acute kidney injury and decreased survival.

113 sion correlates with increased mortality and acute kidney injury early after transcatheter aortic val

114 red in 48 of 116 patients (41.4%) and severe acute kidney injury in 32 of 116 (27.6%) patients, which

115 uPAR) as a therapeutic strategy to attenuate acute kidney injury in transgenic mice receiving contras

116 Any-stage acute kidney injury occurred in 48 of 116 patients (41.4

117 There was a trend toward higher risk of acute kidney injury requiring renal replacement therapy

118 The risk of acute kidney injury was lower between those who either u

119 Autophagy has been implicated in acute kidney injury, which can arise in response to neph

120 ey disease, and ischemia/reperfusion-induced acute kidney injury.

121 ular proliferation, lobular cholestasis, and acute liver cell necrosis, together with central vein th

122 e injected with azoxymethane (AOM) to induce acute liver failure and HE.

123 1) has been shown to contribute to HE during acute liver failure; however, TGFbeta1 must be activated

124 disease and COVID-19 (n = 375), incidence of acute liver injury was lower in LT recipients (47.5% vs.

125 1s are critical for tissue protection during acute liver injury.

126 ry syncytial virus (RSV) is a major cause of acute lower respiratory infection (ALRI) in young childr

127 ped in the Rapid Diagnostics in Categorizing Acute Lung Infections (RADICAL) study.

128 ve the potential to prevent and to treat the acute lung injury after SARS-CoV-2 infection, especially

129 In both zebrafish tail injury and murine acute lung injury models of neutrophilic inflammation, o

130 After acute lung injury, they are preferentially localized in

131 e (female BALB/c strain) with an LPS-induced acute lung injury.

132 of inappropriate NOTCH1 signalling in T-cell acute lymphoblastic leukaemia (T-ALL), and the involveme

133 Patients undergoing treatment of acute lymphoblastic leukemia (ALL) are at risk for throm

134 Relapsed acute lymphoblastic leukemia (ALL) has remained challeng

135 plantation (HSCT) in pediatric patients with acute lymphoblastic leukemia (ALL) is efficacious, but l

136 expression are commonly altered in pediatric acute lymphoblastic leukemia (PALL).

137 lts with Philadelphia chromosome-like B cell acute lymphoblastic leukemia (Ph-like B-ALL) experience

138 omponent of therapy for patients with T cell acute lymphoblastic leukemia (T-ALL), and although resis

139 Analysis of 13 T-lineage acute lymphoblastic leukemias identified a recurrent int

140 patients) and PDGFRB fusion-positive B-cell acute lymphocytic leukaemia (43 [88%] of 49 patients).

141 Children with ABL-class fusion B-cell acute lymphocytic leukaemia have poor outcomes when trea

142 ses chronic myeloid leukemia and a subset of acute lymphoid leukemias.

143 Severe acute malnutrition (SAM) is the most serious form of und

144 were all higher in the setting of prolonged acute mechanical ventilation than short-term mechanical

145 rovide novel insight regarding (2R,6R)-HNK's acute mechanism of action, and may inform novel antidepr

146 f most other genes was largely unaffected by acute Mediator ablation.

147 stress response, and higher activation with acute mental stress may indicate a more severe stress re

148 ith stable coronary artery disease underwent acute mental stress testing using a series of standardiz

149 n effective non-sanguineous, LVR solution in acute models of HS through mechanisms targeting cell swe

150 rature-matched passive heat stress ABSTRACT: Acute moderate-intensity exercise increases core tempera

151 DF cell lines, and on neoplastic cell lines: acute monocytic leukemia THP-1 and lung adenocarcinoma A

152 pliant patients, to compare the incidence of acute mucositis (Radiation Therapy Oncology Group and WH

153 Anthracyclines used in the treatment of acute myelogenous leukemia (AML) inhibit the activity of

154 been observed in about 35% of patients with acute myeloid leukaemia (AML).

155 se-risk karyotype, the presence of secondary acute myeloid leukaemia arising from previous myelodyspl

156 tracted data on myelodysplastic syndrome and acute myeloid leukaemia cases from ClinicalTrials.gov.

157 mes for children with relapsed or refractory acute myeloid leukaemia remain poor.

158 Eligible patients had previously untreated acute myeloid leukaemia, an Eastern Cooperative Oncology

159 ve neoplasm, the presence of therapy-related acute myeloid leukaemia, or being 65 years or older.

160 We tested MASQ in a pilot study in acute myeloid leukemia (AML) patients who entered comple

161 BC transfusion-independence in patients with acute myeloid leukemia (AML) treated with the isocitrate

162 Acute myeloid leukemia (AML) with mixed lineage leukemia

163 te alteration during leukemogenesis of human acute myeloid leukemia (AML), and ALKBH5 is required for

164 rement for Runx1 in a mouse model of inv(16) acute myeloid leukemia (AML).

165 pproved for treatment of IDH1-mutant (mIDH1) acute myeloid leukemia (AML).

166 the most frequent chromosome aberrations in acute myeloid leukemia (AML).

167 Several acute myeloid leukemia genetic sub-types converge on hig

168 In a xenograft model of acute myeloid leukemia, a single injection of 10 million

169 Secondary acute myeloid leukemias (AMLs) evolving from an antecede

170 Hospitalizations after acute myocardial infarction are commonly underreported i

171 hy, and by primary CR-qualifying event type (acute myocardial infarction hospitalization; coronary ar

172 r heart failure, 1.76 (95% CI 1.51-2.05) for acute myocardial infarction, 1.78 (95% CI 1.53-2.07) for

173 for acute nonischemic myocardial injury and acute myocardial infarction, particularly type 2 myocard

174 e as potential immunomodulatory treatment in acute myocardial infarction.

175 irect cardiovascular complications including acute myocardial injury, myocarditis, arrhythmias, and v

176 Background Hyperemia is a key component of acute myocarditis (AM).

177 An understanding of alcohol's acute neural effects could augment our knowledge of mech

178 rocnemius model, we evaluated the effects of acute nitrite infusion on muscle force and skeletal musc

179 (SARS-CoV-2) infection increase the risk for acute nonischemic myocardial injury and acute myocardial

180 gs provide a mechanistic explanation for the acute O(2) regulation of breathing, reveal an unanticipa

181 tcomes questions) on the management acute or acute on chronic liver failure in the ICU, related to fi

182 ity of these factors to predict survival and acute-on-chronic liver failure (ACLF) in patients awaiti

183 recAP prevents the progression of acute and acute-on-chronic liver failure (ACLF).

184 Either genetically directed deletion or acute optogenetic silencing of DR(Sert) neurons dramatic

185 n, and outcomes questions) on the management acute or acute on chronic liver failure in the ICU, rela

186 , a small molecule activator of PKM2, during acute outer retinal stress.

187 In this cohort study, prediction models of acute ovarian failure risk were developed using eligible

188 her TRV130 maintenance would protect against acute oxycodone-induced decreases in nucleus accumbens o

189 We induced severe acute pancreatitis by partial duct ligation with caerule

190 cally occurs 3 to 5 weeks after the onset of acute pancreatitis.

191 r several pathological conditions, including acute pancreatitis.

192 adult myocardium, and compares the effect of acute (perfusion only) versus prolonged (2 weeks pre-tre

193 cantly higher in the olanzapine group in the acute period (74% v 52%; P < .001), delayed period (74%

194 CD14 + + classical monocytes isolated in an acute phase from DENV-infected pediatric patients correl

195 drome encompassing events before, during the acute phase, and subsequently in the chronic phase is pr

196 CIV control was more likely in patients with acute-phase complete CIV control (RR, 1.19; 95% CI, 1.06

197 Viral load was quantified in acute-phase serum by real-time reverse transcription pol

198 d in patients with longer ICU stay and lower Acute Physiology and Chronic Health Evaluation II scores

199 lyses were conducted using an ICU cohort and Acute Physiology and Chronic Health Evaluation III score

200 ity paradox was not observed after including Acute Physiology and Chronic Health Evaluation III score

201 n +/- SD age: 59 +/- 16; and mean Simplified Acute Physiology Score 2: 54 +/- 20), 53 (54%) experienc

202 9-2.29) and, after adjustment for Simplified Acute Physiology Score II, with mortality (odds ratio, 2

203 his l-fucose dose, complement activation and acute post-ischemic kidney injury are prevented, with ad

204 plement-mediated glomerulopathy that, unlike acute postinfectious GN, has a poor prognosis.

205 This remains a particularly acute problem for triploids, which produce desirable see

206 These patterns likely distinguish acute (protective) and chronic (damaging) associated cha

207 eviates LPS-/IgG immune complexes-stimulated acute pulmonary damage through reducing vascular permeab

208 e patients are hemodynamically unstable with acute pulmonary embolism, and a minority of them have pr

209 el of uropathogenic Escherichia coli-induced acute pyelonephritis to determine the contribution of ne

210 s that are psychiatric (e.g. schizophrenia), acute rather than chronic, or benefitting little from se

211 s 83%, respectively) and consisted mainly of acute reactions (defined as symptoms within 3 days of ad

212 68 [1.08-2.62]; P = 0.022) and biopsy-proven acute rejection (aHR, 1.71 [1.13-2.60]; P = 0.012).

213 V-infected (HIV+) persons are excellent, yet acute rejection (AR) is common and optimal immunosuppres

214 The primary endpoint was biopsy-confirmed acute rejection (BCAR) within 60 weeks after transplanta

215 Biopsy-proven acute rejection (BPAR) rates and types were compared bet

216 ients who received ATG were at lower risk of acute rejection compared to those who received IL2RA (1-

217 as compared to IL2RA, may lower the risk of acute rejection without increasing hepatic complications

218 points were treatment failure (biopsy-proven acute rejection, graft loss, or death), delayed graft fu

219 5 to an increased incidence of biopsy-proven acute rejection.

220 hospitalization (OR, 2.9; 95% CI, 1.3-6.7), acute renal injury (OR, 2.7; 95% CI, 1.3-5.6), and CRP o

221 ury (63%), transaminitis (31%), shock (31%), acute respiratory distress syndrome (25%), neurological

222 respiratory symptoms, which can progress to acute respiratory distress syndrome (ARDS) in the most s

223 motif) receptor 4 (CXCR4) agonists in a rat acute respiratory distress syndrome (ARDS) model utilizi

224 d during autopsy from patients who died from acute respiratory distress syndrome (ARDS) secondary to

225 MNs), which serves as the first event in the acute respiratory distress syndrome (ARDS).

226 mptoms or progress toward a life-threatening acute respiratory distress syndrome (ARDS).

227 del demonstrated good discrimination between acute respiratory distress syndrome and nonacute respira

228 A Streptococcus coinfection, progressing to acute respiratory distress syndrome and shock.

229 e corona virus 2 pneumonia is linked to both acute respiratory distress syndrome and systemic hyperco

230 The 2015 definition for pediatric acute respiratory distress syndrome did not require the

231 Clinician recognition of acute respiratory distress syndrome is associated with b

232 Survival from acute respiratory distress syndrome is improving, and ou

233 However, acute respiratory distress syndrome often goes unrecogni

234 ssed by a CT scan in mechanically ventilated acute respiratory distress syndrome patients.

235 .6%) related to sepsis, respiratory failure, acute respiratory distress syndrome, or multiple organ d

236 SARS-CoV-2 predicted the odds of developing acute respiratory distress syndrome, which increased by

237 anical ventilation via endotracheal tube for acute respiratory failure.

238 for signs and symptoms of diarrheal disease, acute respiratory illness, and anemia.

239 older adults hospitalized with influenza and acute respiratory illness.

240 ibe the epidemiology of hospitalizations for acute respiratory infection or febrile illness (ARFI) an

241 fficult to distinguish clinically from other acute respiratory infections.

242 Dysregulated immunothrombosis in severe acute respiratory syndrome corona virus 2 pneumonia is l

243 tion-based serosurveys measuring anti-severe acute respiratory syndrome coronavirus 2 (anti-SARS-CoV-

244 body-based drugs and vaccines against severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) ar

245 Tests for severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) ba

246 rus disease 2019 (COVID-19) caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) em

247 Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) em

248 The rapid spread of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) ha

249 source and number of introductions of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) in

250 sease 2019 (COVID-19) or detection of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) in

251 A safe and effective vaccine for severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) ma

252 The severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) pa

253 ern for healthcare workers during the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) pa

254 Preliminary data from severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) pn

255 y tumour subtype, age, and sex and on severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) pr

256 id amplification for the detection of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) RN

257 alizing monoclonal antibodies against severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) sp

258 where, and how the earliest sustained severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) tr

259 This pathogen, named severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), c

260 The causative agent, severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), i

261 Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), t

262 tion of individuals who may carry the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), w

263 responded following the emergence of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2).

264 dentifying acute infections caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2).

265 e risk for exposure to infection with severe acute respiratory syndrome coronavirus 2 during the coro

266 dysarthria with positive findings for severe acute respiratory syndrome coronavirus 2 from nasopharyn

267 conditions have been associated with severe acute respiratory syndrome coronavirus 2 illness, but it

268 coronavirus disease 2019 out of 1,788 severe acute respiratory syndrome coronavirus 2 positive cases,

269 lism events in patients infected with severe acute respiratory syndrome coronavirus 2 requiring venov

270 subsequent family members exposure to severe acute respiratory syndrome coronavirus 2.

271 and procoagulant responses following severe acute respiratory syndrome-coronavirus-2 (SARS-CoV-2) in

272 The inflammatory response to severe acute respiratory syndrome-related coronavirus 2 infecti

273 the Study to Understand the Genetics of the Acute Response to Metformin and Glipizide in Humans (SUG

274 Acute retinal vascular occlusions are common causes of v

275 history presented to the Emergency Room with acute right flank pain.

276 Nonetheless, it is noteworthy that an acute septic episode may harbor antitumoral properties u

277 Myocardial damage due to acute ST-segment elevation myocardial infarction (STEMI)

278 CD4(+) T cells are critical for controlling acute-stage poliomyelitis (gray matter inflammation), ch

279 sought to examine how trait impulsivity and acute stress exposure affect participants' choice behavi

280 voidance of stress-related stimuli following acute stress exposure.SIGNIFICANCE STATEMENT This study

281 pathways responsible for protection against acute stressors.

282 esults The COMPASS study (Comprehensive Post-Acute Stroke Services) was a pragmatic cluster-randomize

283 figurations of motor network connectivity in acute stroke.

284 hdrawal trial of rilonacept in patients with acute symptoms of recurrent pericarditis (as assessed on

285 biophysical feature for engendering inherent acute synaptotoxicity, with preformed oligomers found in

286 d CD11c-expressing IMs in the development of acute T(H)2-cell-dependent allergic airway inflammation.

287 rdinating physiology and behavior both on an acute timescale to adapt to rapid fluctuations in calori

288 ation of oxidants by immune cells results in acute tissue damage.

289 cals to extrapolate chronic information from acute toxicity data.

290 Nevertheless, acute toxicity experiments with (212)Pb-anti-mCD38 estab

291 ntiinflammatory capabilities, increased with acute treatment and was 35% higher upon completion of th

292 hibitor, ABD-110207, which is active in vivo Acute treatment of WT mice with ABD-110207 resulted in e

293 The acute treatment period was 2 weeks.

294 predominant finding correlating with AKI was acute tubular injury.

295 ble rapid diagnostic tests (RDTs) for common acute undifferentiated febrile illnesses are often used

296 had the lowest complication, ED visit, post-acute utilization, and readmission rates (P < 0.001).

297 ulation most prominent in the early phase of acute viral and bacterial infections and a molecularly d

298 microbiome differences after chronic versus acute viral infections and identify CD8 T cell responses

299 conazole in subjects with moderate-to-severe acute VVC.

300 s revealed that short Abeta oligomers induce acute wakefulness through Adrenergic receptor b2 (Adrb2)