ライフサイエンスコーパス: acute HIV infection

1 D14, D-dimer, and HA levels were elevated in acute HIV infection.

2 mune pressure in a cohort of 17 persons with acute HIV infection.

3 ing has not routinely included screening for acute HIV infection.

4 ing of populations with a high prevalence of acute HIV infection.

5 s at mucosal sites are essential targets for acute HIV infection.

6 dies, p24 antigen and RNA typically indicate acute HIV infection.

7 163 had established HIV infection, and 8 had acute HIV infection.

8 analysis and MRS in some individuals during acute HIV infection.

9 infection and poor performance for detecting acute HIV infection.

10 making clinical decisions for patients with acute HIV infection.

11 HIV testing: rapid testing and detection of acute HIV infection.

12 entations from the meeting on the Biology of Acute HIV Infection.

13 macytoid DC levels decline very early during acute HIV infection.

14 t disease, tumor immunotherapy regimens, and acute HIV infection.

15 nterventions that modify the consequences of acute HIV infection.

16 role, we examined plasma from patients with acute HIV infection.

17 mian immunodeficiency virus macaque model of acute HIV infection.

18 assays as primary tools for the diagnosis of acute HIV infection.

19 modest effects on overall cell death during acute HIV infection.

20 emergence of drug resistance and to diagnose acute HIV infections.

21 ilable therapeutic options; 9) treatment for acute HIV infection; 10) considerations for antiretrovir

22 Of the 23 acute HIV infections, 16 were detected at sexually trans

23 nce estimates were based on (1) detection of acute HIV infection, (2) documentation of HIV seroconver

24 ial increase in plasma virus associated with acute HIV infection (3, 7, 20, 35, 48).

25 A 128-member specimen pooling scheme for acute HIV infection (AHI) detection was evaluated using

26 prior to initiating therapy in patients with acute HIV infection, although therapy should not be dela

27 icians should be alert to the possibility of acute HIV infection and promptly pursue diagnostic testi

28 t cytolytic CD4+ T cells emerge early during acute HIV infection and tightly follow acute viral load

29 hould be preferred to avoid missing cases of acute HIV infection and to decrease the related risks of

30 subjects generated these responses early in acute HIV infection and were able to control HIV replica

31 iral RNA technologies and initiating ART for acute HIV infection appears cost effective.

32 s, these factors indicate that events during acute HIV infection are likely to include distortions in

33 Two women had acute HIV infection at enrollment, 4 seroconverted, and

34 he TDF-FTC group who had had an unrecognized acute HIV infection at enrollment.

35 Ten patients were treated during acute HIV infection before complete Western blot (WB) se

36 racterize the early plasma viral dynamics in acute HIV infection better than it has been possible thu

37 42 women were diagnosed with acute HIV infection between Dec 1, 2012, and June 30, 20

38 s may modulate the innate immune response to acute HIV infection by reducing viral replication.

39 e HIV diagnostic yield (both established and acute HIV infections) by 10.4% (95% CI, 8.8%-12.2%) and

40 Difficulties inherent in studying acute HIV infection can be overcome by modeling virus-ho

41 In seronegative acute HIV infection, CD8(+) T cell counts increased in t

42 After acute HIV infection, CD8(+) T cells are able to control

43 context of light chain use in subjects with acute HIV infection, chronic HIV infection, and among HI

44 Although acute HIV infection contributes disproportionately to on

45 In acute HIV infection, CSF NFL levels are inversely associ

46 IV Ag/Ab combination testing detected 82% of acute HIV infections detectable by pooled HIV RNA testin

47 lowing a negative rapid test detected 82% of acute HIV infections detectable by pooled HIV RNA testin

48 Number and proportion with acute HIV infections detected.

49 Twenty individuals who had acute HIV infection (Fiebig stages I-IV), with average 1

50 subjects studied included two subjects with acute HIV infection, five subjects with chronic HIV infe

51 the presentations on clinical management of acute HIV infection from the 2009 Acute HIV Infection Me

52 People with acute HIV infection have demonstrated enhanced transmiss

53 Fourteen people with acute HIV infection (HIV antibody negative/NAT positive)

54 f young women detected during Fiebig stage I acute HIV infection in whom treatment was initiated imme

55 findings suggest the widespread diagnosis of acute HIV infections in a routine testing population is

56 Acute HIV infection induced a rapid depletion of M. tube

57 of HIV-specific CD4 T cell responses during acute HIV infection influences disease progression and w

58 Acute HIV infection is associated with a vigorous immune

59 of HIV-specific CD4 T cell responses during acute HIV infection is beneficial overall and does not f

60 Acute HIV infection is characterized by massive loss of

61 time pooled RNA testing for the detection of acute HIV infection is feasible in resource-limited sett

62 The literature on acute HIV infection is predominantly small nonrandomized

63 Acute HIV infection lasts approximately 3 weeks and earl

64 eeks and early HIV infection, which includes acute HIV infection, lasts approximately 7 weeks.

65 We conclude that early treatment of acute HIV infection leads to a more narrowly directed CT

66 The detection of acute HIV infections may need to rely on other testing s

67 size and the relationships to viral load in acute HIV infection, measurements of the latent reservoi

68 agement of acute HIV infection from the 2009 Acute HIV Infection Meeting in Boston, Massachusetts.

69 In an in vitro acute HIV infection model, MTB increased HIV replication

70 e cohorts in KwaZulu-Natal were assessed for acute HIV infection monthly (n = 245) or every 3 months

71 Few instances of typhoid fever (n = 8), acute HIV infection (n = 5), and dengue (n = 2) were enc

72 Here, we demonstrate that while acute HIV infection of human microglia/macrophages resul

73 Here virologic and immunologic aspects of acute HIV infection of humans and SIV infection of Asian

74 ions, we have used an in vitro cell model of acute HIV infection of quiescent, primary CD4 lymphocyte

75 In a longitudinal study, the effect of acute HIV infection on M. tuberculosis-specific Th1 cell

76 CSF NFL levels are not elevated in untreated acute HIV infection or after 6 months of immediately ini

77 ogeneity in transmission, for example due to acute HIV infection or the presence of 'core groups' wit

78 ial advantages and disadvantages of treating acute HIV infection outlined in treatment guidelines, an

79 tion of ART at one of the earliest stages of acute HIV infection possible.

80 sponses dominantly targeting Gag over Env in acute HIV infection remained off antiretroviral therapy

81 on of the vivo inhibitory effect of IL-10 on acute HIV infection suggests that further studies may be

82 For detecting acute HIV infection, the antigen portion had a sensitivi

83 barrier already in the seronegative phase of acute HIV infection, thereby inducing microbial transloc

84 is an important IRF3 regulator that supports acute HIV infection through innate immune suppression.

85 ensively investigated in eight patients with acute HIV infection to define the earliest rates of chan

86 ART initiated in early acute HIV infection was associated with normalization of

87 Acute HIV infection was detected in 134 participants wit

88 s diagnosed in 1158 participants (1.33%) and acute HIV infection was diagnosed in 168 participants (0

89 tion of Gag-specific CD4 T cell responses in acute HIV infection was significantly inversely correlat

90 tiating potent antiretroviral therapy during acute HIV infection was similar to estimates from treate

91 The antigen portion of the Combo RT (for acute HIV infection) was compared with a Roche Monitor H

92 tudying individuals in the first few days of acute HIV infection, we detected the emergence of a uniq

93 Patients with possible acute HIV infection were followed to confirm seroconvers

94 the epitopes targeted at a high frequency in acute HIV infection were recognized at the same frequenc

95 No acute HIV infections were identified.

96 ent a highly reactive cell population during acute HIV infection, which responds independently from t

97 rd HIV antibody tests to detect persons with acute HIV infection who are viremic but antibody-negativ

98 nt is the identification of individuals with acute HIV infection whose contribution to HIV transmissi

99 tive rapid HIV test result were screened for acute HIV infection with an HIV Ag/Ab combination assay

100 on of activated CD8(+)T cells appears during acute HIV infection with diminished capacity to inhibit

101 d to determine whether ART initiation during acute HIV infection would attenuate changes in these bio