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1 ts targeted at the underlying disease (e.g., acute coronary occlusion).
2 al with STEMI and mapped the location of the acute coronary occlusion.
3 te the restoration of cardiac function after acute coronary occlusion.
4 nt detection of the onset of ischemia during acute coronary occlusion.
5 articipate in the arrhythmogenic response to acute coronary occlusion.
6 otherwise healthy men even in the absence of acute coronary occlusion.
7 tomography/magnetic resonance imaging after acute coronary occlusion and interventional reperfusion.
8 ular infarct size was assessed at 24 h after acute coronary occlusion by triphenyltetrazolium chlorid
16 e extent of microvascular obstruction during acute coronary occlusion may determine the eventual magn
18 before vs. 0.93 +/- 0.41 mm Hg.ml(-1) during acute coronary occlusion [p < 0.05] and 7.9 +/- 3.1 m.s(
19 tested in an ambulatory porcine model, with acute coronary occlusion precipitated by stent thrombosi
20 pen-chest dogs were subjected to a 10-minute acute coronary occlusion (proximal left anterior descend
21 arrhythmias similar to those observed during acute coronary occlusion/reperfusion in intact hearts.
24 veloped a spatial map of the distribution of acute coronary occlusions to test our hypothesis that pl
26 in distance from the ostium, the risk of an acute coronary occlusion was significantly decreased by
27 Identification of these high-risk zones for acute coronary occlusions will lead to future advances i
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