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1 e myelitis (TM); 15% simultaneous TM&ON; 10% Acute disseminated encephalomyelitis.
2 classify subtypes of transverse myelitis and acute disseminated encephalomyelitis.
3 elate of depressed level of consciousness in acute disseminated encephalomyelitis.
4 clinical syndromes were encephalitis (52%), acute disseminated encephalomyelitis (12%), transverse m
5 rlying diagnosis (39% multiple sclerosis, 7% acute disseminated encephalomyelitis, 7% neuromyelitis o
8 ) disorders like multiple sclerosis (MS) and acute disseminated encephalomyelitis (ADEM) have been di
10 %) for children older than 11 years with non-acute disseminated encephalomyelitis (ADEM) presentation
11 g syndromes and with encephalitis other than acute disseminated encephalomyelitis (ADEM) recruited fr
13 ients with presentation data [43.3%]), while acute disseminated encephalomyelitis (ADEM), brain, or b
14 ines may trigger transverse myelitis (TM) or acute disseminated encephalomyelitis (ADEM), but the evi
15 n MOG, but had different diseases, including acute disseminated encephalomyelitis (ADEM), one episode
16 sseminated demyelination of the CNS, 28 with acute disseminated encephalomyelitis (ADEM), seven with
17 ein (MOG)-Ab was detected in seven; two with acute disseminated encephalomyelitis (ADEM), two with ON
20 16/25 [64%] vs 129/391 [33%]; p = 0.002), or acute disseminated encephalomyelitis (ADEM; 8/25 [32%] v
21 (perivenous demyelination), but misdiagnosed acute disseminated encephalomyelitis among 9% of patient
22 is than encephalopathy, which over-diagnosed acute disseminated encephalomyelitis among multiple scle
23 clude encephalitis, transverse myelitis, and acute disseminated encephalomyelitis among others; histo
25 ividuals suggests pathogenic overlap between acute disseminated encephalomyelitis and multiple sclero
26 , neuromyelitis optica spectrum disorder and acute disseminated encephalomyelitis) and from non-demye
27 %-30%) had immune-mediated encephalitis, 18% acute disseminated encephalomyelitis, and 6% anti-N-meth
28 y disorders, including neuromyelitis optica, acute disseminated encephalomyelitis, and Balo's concent
29 itis optica spectrum disorders and relapsing acute disseminated encephalomyelitis, and characterizing
30 serious; six (fever, gastroenteritis (n=3), acute disseminated encephalomyelitis, and encephalitis)
31 nt for these donors included stroke, anoxia, acute disseminated encephalomyelitis, and meningoencepha
32 wn, but possible links with Devic's disease, acute disseminated encephalomyelitis, and neurotoxicity
33 rotein antibody disease, multiple sclerosis, acute disseminated encephalomyelitis, and postinfectious
34 matter injury, including multiple sclerosis, acute disseminated encephalomyelitis, and spinal cord in
35 an typical patients with transverse myelitis/acute disseminated encephalomyelitis, and these observat
38 ng on their presenting phenotype: those with acute disseminated encephalomyelitis demonstrated abnorm
39 versus confluent demyelination distinguishes acute disseminated encephalomyelitis from multiple scler
40 ar coagulation, encephalomyelitis (including acute disseminated encephalomyelitis), Guillain-Barre sy
41 essary on data from related diseases such as acute disseminated encephalomyelitis, Guillain-Barre syn
43 age 56, range 18-84 years; 22 samples), six acute disseminated encephalomyelitis (mean age 25, range
44 isease include encephalopathy, encephalitis, acute disseminated encephalomyelitis, meningitis, ischem
46 ephalitis (n = 2, para- or post-infectious), acute disseminated encephalomyelitis (n = 9), with haemo
47 and understanding the disease mechanisms of acute disseminated encephalomyelitis, neuromyelitis opti
48 criteria, MOGAD is typically associated with acute disseminated encephalomyelitis, optic neuritis, or
50 and 91% specific for pathologically defined acute disseminated encephalomyelitis (perivenous demyeli
51 atric participants, as did patients with non-acute disseminated encephalomyelitis presentations assoc
53 n (40.8%) and adults (55.9%, p = 0.013), and acute disseminated encephalomyelitis syndrome was more f
54 nical criterion for pathologically confirmed acute disseminated encephalomyelitis than encephalopathy
55 superior to clinical criteria for diagnosing acute disseminated encephalomyelitis, the co-occurrence
56 itical neuroinflammatory diagnoses including acute disseminated encephalomyelitis, transverse myeliti
57 lude neuromyelitis optica spectrum disorder, acute disseminated encephalomyelitis, tumefactive demyel
59 Sclerosis Study Group clinical criteria for acute disseminated encephalomyelitis were assessed in bo
60 emyelination is the pathological hallmark of acute disseminated encephalomyelitis, whereas confluent