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1 cteristic that is usually not seen during an acute exacerbation.
2 tinuation due to clinical deterioration, and acute exacerbation.
3  risk for death after hospitalization for an acute exacerbation.
4 ignificant increase in SNOT-22 scores during acute exacerbation.
5 ical changes locally and systemically during acute exacerbation.
6 and the most common (40%) cause of death was acute exacerbation.
7     Surgical intervention prevents recurrent acute exacerbations.
8  whose disease is characterized by recurrent acute exacerbations.
9  about the details of managing patients with acute exacerbations.
10 cially studies focusing on the management of acute exacerbations.
11 or disease symptoms, including treatment for acute exacerbations.
12 onic course of COPD is worsened by recurrent acute exacerbations.
13 r interleukin-8 concentrations, than were no acute exacerbations.
14 f smoking cessation will be needed to reduce acute exacerbations.
15 9% vs. 2.5%, P=0.30 by the log-rank test) or acute exacerbation (2.3% in each group, P>0.99).
16          Withdrawal of phenytoin resulted in acute exacerbation, accompanied by a significantly incre
17                                              Acute exacerbations adversely affect patients with chron
18 ronic obstructive asthma (COA) and during an acute exacerbation (AE) in patients without airflow obst
19               The mean number of episodes of acute exacerbation after surgery was 1.6 +/- 2.3 events
20 ited 18 asthmatics admitted to hospital with acute exacerbation and 18 healthy nonsmoking controls ma
21  pro-MMP-9 were also significantly higher in acute exacerbation and decreased in remission but remain
22 stane and CRP were significantly elevated in acute exacerbation and decreased in remission but remain
23 and tissue damage in patients with asthma in acute exacerbation and remission.
24 ondary end points were the time to the first acute exacerbation and the change from baseline in the t
25 ower respiratory tract colonization and with acute exacerbations and disease progression in chronic o
26 l pathogens from sputum were the same during acute exacerbations and during stable disease.
27            Research into methods to decrease acute exacerbations and improve emergency and in-hospita
28 n of lung function, reduction in episodes of acute exacerbation, and enhanced longevity.
29 diagnosis, asthma phenotypes, severe asthma, acute exacerbations, and clinical management of disease
30                     INTERPRETATION: Although acute exacerbations are common, the exacerbation status
31                 While the factors leading to acute exacerbations are poorly understood, antibiotic tr
32                                              Acute exacerbations are the major cause of asthma morbid
33 re likely to have respiratory infections and acute exacerbations at baseline or to develop them subse
34 AE management involves not only treatment of acute exacerbations but also individualized patient pref
35 o control daily symptoms and prevent serious acute exacerbations, but chronic SCS use is associated w
36 tive measures, but admission to hospital for acute exacerbations can be expected to remain common.
37 of COPD at week 56, defined as the number of acute exacerbations divided by total duration of person-
38                                              Acute exacerbation during pregnancy is the most importan
39  538 (49%) of 1105 patients had at least one acute exacerbation during the 3 years of follow-up, wher
40    82 (7%) of 1105 patients had at least one acute exacerbation each year, whereas only 23 (2%) had t
41 py significantly decreased the occurrence of acute exacerbation events, which is consistent with esta
42 C therapy had no effect on the occurrence of acute exacerbation events.
43 one (group 1), severe pain with intermittent acute exacerbations (group 2), and intermittent acute ex
44           In logistic regression, consistent acute exacerbations (>/=1 event per year for 3 years) we
45 ve pulmonary disease admitted to the ICU for acute exacerbations had abnormal breathing-swallowing in
46  and placebo groups in the time to the first acute exacerbation (hazard ratio with nintedanib, 1.15;
47 e 2 inflammation during a rhinovirus-induced acute exacerbation; however, only anti-IL-33 boosted ant
48 atic subjects of similar ages can experience acute exacerbation in an environmental chamber that rese
49 atinine; range, 70 to 110) (p < 0.001) as an acute exacerbation in their clinical condition resolved.
50 learance and pulmonary function, and reduces acute exacerbations in CF patients.
51  more than 1000 genes was upregulated during acute exacerbations in comparison with 7 to 14 days late
52 h year, whereas only 23 (2%) had two or more acute exacerbations in each year.
53 ommonly causing otitis media in children and acute exacerbations in patients suffering from chronic o
54 g cause of lung infections and contribute to acute exacerbations in patients with chronic respiratory
55 wice daily reduced lung-function decline and acute exacerbations in patients with idiopathic pulmonar
56 ability (outpatients) and 13 patients during acute exacerbation (inpatients).
57 , the use of macrolides in chronic asthma or acute exacerbations is not justified.
58 iversity of airway microbiota, whose role in acute exacerbations is unclear.
59 -free survival was defined as time to death, acute exacerbation, lung transplant, or decrease in forc
60                                     Although acute exacerbations, mostly triggered by viruses, accoun
61        An estimated 7,511,267 admissions for acute exacerbations occurred from 1998 to 2008.
62                            After surgery, no acute exacerbations occurred in 42 of 64 (66%) group 2 p
63 producing: [1] hyperacusis, together with an acute exacerbation of [2] chronic aberrant Type-I neural
64  characterized by bacterial colonization and acute exacerbation of airway infections, we assessed whe
65 te overlapping risk factors and symptoms, an acute exacerbation of asthma or an episode of acute ches
66 sthma were enrolled within 24 hours after an acute exacerbation of asthma requiring short-term medica
67 e in-hospital management of patients with an acute exacerbation of chronic heart failure.
68 , and in group 3 (n = 10) were patients with acute exacerbation of chronic liver disease.
69 re beneficial for patients hospitalized with acute exacerbation of chronic obstructive pulmonary dise
70 n, ischemic stroke, urinary tract infection, acute exacerbation of chronic obstructive pulmonary dise
71 s with cardiogenic pulmonary edema (n = 97), acute exacerbation of chronic obstructive pulmonary dise
72 ciated with community acquired pneumonia and acute exacerbation of chronic obstructive pulmonary dise
73                                              Acute exacerbation of chronic obstructive pulmonary dise
74 re often used in the outpatient treatment of acute exacerbation of chronic obstructive pulmonary dise
75 ents with acute HF, but not in patients with acute exacerbation of chronic obstructive pulmonary dise
76  We studied 1,298 patients hospitalized with acute exacerbation of congestive heart failure who were
77 ing cardiologist care among patients with an acute exacerbation of congestive heart failure.
78 -intensity adverse event in both studies was acute exacerbation of COPD (1-4 [<1-2%] patients across
79 in both systemic and airway inflammation and acute exacerbation of COPD (AECOPD) has been reported by
80 S hospitals involving patients admitted with acute exacerbation of COPD in 2006 and 2007 to a non-int
81              Among patients hospitalized for acute exacerbation of COPD low-dose steroids administere
82 (P=0.01), and the hazard ratio for having an acute exacerbation of COPD per patient-year in the azith
83 nts presenting with signs and symptoms of an acute exacerbation of COPD were prospectively randomized
84 nts with persistent hypercapnia following an acute exacerbation of COPD, adding home noninvasive vent
85 , with moderate-to-severe COPD, at least one acute exacerbation of COPD, and a sputum eosinophil coun
86  acceptable, and transportable definition of acute exacerbation of COPD, as well as improved methods
87  admitted to hospital for more than 24 h for acute exacerbation of COPD.
88 result from either acute limb ischemia or an acute exacerbation of critical limb ischemia.
89                 Immunological changes during acute exacerbation of CRS may provide valuable clues to
90 motherapy and can effectively prevent severe acute exacerbation of HBV infection in hospitals among H
91 ng PMX-DHP and SHEDD-fA may be used to treat acute exacerbation of idiopathic interstitial pneumonias
92  outcomes of patients with steroid-resistant acute exacerbation of idiopathic interstitial pneumonias
93                                              Acute exacerbation of IIPs is now well defined.
94 failure in clinical AA amyloidosis following acute exacerbation of inflammation.
95 at the beginning and end of treatment for an acute exacerbation of lung infection and again 3 wk late
96 elapse or insufficient clinical response and acute exacerbation of OCD symptoms.
97  due to relapse or insufficient response, or acute exacerbation of OCD symptoms.
98 stress occurs in the sickle kidney, and that acute exacerbation of oxidative stress in the sickle mou
99 tus and antipsychotic treatment following an acute exacerbation of psychosis.
100  status and antipsychotic treatment after an acute exacerbation of psychosis.
101 mitted inpatients with schizophrenia with an acute exacerbation of psychotic symptoms, were randomly
102 olerability of aripiprazole in patients with acute exacerbation of schizophrenia or schizoaffective d
103 he outcomes of patients hospitalized with an acute exacerbation of severe chronic obstructive pulmona
104 l response (9% versus 24%, respectively) and acute exacerbation of symptoms (12% versus 35%).
105 ypothesis) that ACLF is the expression of an acute exacerbation of the SI already present in decompen
106 in the United States each year experience an acute exacerbation of their asthma, a quarter of which r
107  up-regulated by rhinovirus infection during acute exacerbations of asthma and chronic obstructive pu
108                 Air pollution contributes to acute exacerbations of asthma and the development of ast
109  identify pregnancy-related risk factors for acute exacerbations of asthma during pregnancy.
110 n is now recognized as an important cause of acute exacerbations of asthma in school-aged children.
111  for chronic treatment and intravenously for acute exacerbations of asthma).
112 al infections contribute to the causation of acute exacerbations of asthma, but that additional cofac
113       Viruses are frequently associated with acute exacerbations of asthma, but the extent to which t
114 rove the pulmonary function of patients with acute exacerbations of asthma, but their effect on hospi
115 e efficacy of telithromycin in patients with acute exacerbations of asthma.
116 he benefit of telithromycin in patients with acute exacerbations of asthma; the mechanisms of benefit
117 etiologies of diseases such as pneumonia and acute exacerbations of chronic bronchitis.
118 infections are associated with morbidity and acute exacerbations of chronic lung diseases, such as cy
119 ion (NIPPV) use in patients hospitalized for acute exacerbations of chronic obstructive pulmonary dis
120  Guidelines recommend antibiotic therapy for acute exacerbations of chronic obstructive pulmonary dis
121                  It has been associated with acute exacerbations of chronic obstructive pulmonary dis
122 oids are routinely used for the treatment of acute exacerbations of chronic obstructive pulmonary dis
123           The role of bacterial pathogens in acute exacerbations of chronic obstructive pulmonary dis
124 g a new test lung model designed to simulate acute exacerbations of chronic obstructive pulmonary dis
125                                   RATIONALE: Acute exacerbations of chronic obstructive pulmonary dis
126                 Daily azithromycin decreases acute exacerbations of chronic obstructive pulmonary dis
127  an important cause of acute lung injury and acute exacerbations of chronic obstructive pulmonary dis
128             From 1998 to 2010, patients with acute exacerbations of chronic obstructive pulmonary dis
129 course of systemic glucocorticoid therapy in acute exacerbations of chronic obstructive pulmonary dis
130 een 1998 and 2010, severity and mortality of acute exacerbations of chronic obstructive pulmonary dis
131         Fourteen thousand four hundred forty acute exacerbations of chronic obstructive pulmonary dis
132 , long appreciated as one of the triggers of acute exacerbations of chronic pulmonary diseases, has r
133 iolitis in infancy, childhood pneumonia, and acute exacerbations of chronic respiratory diseases such
134 61%), and had lower rates of readmission for acute exacerbations of COPD (7.91%; 95% CI, 7.89%-7.94%
135 ould be associated with an increased risk of acute exacerbations of COPD (AECOPD).
136         To determine the association between acute exacerbations of COPD (and acute respiratory event
137 uary 1, 2006, through December 31, 2007, for acute exacerbations of COPD at 413 acute care facilities
138  primary endpoint was the annualised rate of acute exacerbations of COPD at week 56, defined as the n
139 izumab did not reduce the annualised rate of acute exacerbations of COPD compared with placebo in the
140  compare factors associated with one or more acute exacerbations of COPD every year for 3 years versu
141 ss the pattern and outcomes of NIPPV use for acute exacerbations of COPD from 1998 to 2008.
142 ed to establish whether benralizumab reduces acute exacerbations of COPD in patients with eosinophili
143 uality of care for patients hospitalized for acute exacerbations of COPD may be improved by increasin
144 ved outcomes among patients hospitalized for acute exacerbations of COPD regardless of the risk of tr
145 r the hypercarbia developed by patients with acute exacerbations of COPD when treated with supplement
146  presenting to the emergency department with acute exacerbations of COPD, 5-day treatment with system
147 ical, albeit non-significant, improvement in acute exacerbations of COPD, SGRQ-C, CRQ-SAS, and FEV1 w
148 ly over time among patients hospitalized for acute exacerbations of COPD, whereas the need for intuba
149 intubation and the mortality associated with acute exacerbations of COPD.
150 ntion due to oxygen therapy in patients with acute exacerbations of COPD.
151 ebo, benralizumab did not reduce the rate of acute exacerbations of COPD.
152 e use of a 5-day glucocorticoid treatment in acute exacerbations of COPD.
153 ro studies, fibrosis reduces the severity of acute exacerbations of CP by reducing lipolytic flux bet
154 le to respiratory infections contributing to acute exacerbations of disease.
155                              Inpatients with acute exacerbations of DSM-III schizophrenia (N = 66) we
156 B coinfection were more likely to experience acute exacerbations of hepatitis, HBeAg seroconversion,
157  of 434 children (2 to 18 years old) who had acute exacerbations of moderate or severe asthma treated
158 dinal studies of antipsychotic treatment for acute exacerbations of psychosis (p < .01 for each).
159 arameters (CD4/CD8) may be state markers for acute exacerbations of psychosis, others (CD56) may be t
160 d plasma nitrite) might be state markers for acute exacerbations of psychosis, others (RBC superoxide
161 reased following antipsychotic treatment for acute exacerbations of psychosis.
162                                          For acute exacerbations of schizoaffective disorder or of sc
163 l placebo-controlled trials in patients with acute exacerbations of schizophrenia, and they investiga
164 odel and supports its potential use to treat acute exacerbations of the disease.
165                                              Acute exacerbations of underlying COPD are a common caus
166 subset of patients with CP have intermittent acute exacerbations, often with increasing frequency and
167 te exacerbations (group 2), and intermittent acute exacerbations only (group 3).
168 ession of such diseases, often punctuated by acute exacerbations or secondary illnesses, can lead to
169                   Among patients who had any acute exacerbation over 3 years, very few repeatedly had
170  who died (P = 0.008), and 70% of those with acute exacerbations (P = 0.0005).
171  pulmonary artery hypertension (P = 0.01) or acute exacerbations (P = 0.002).
172                                       During acute exacerbation, peripheral blood SAA levels increase
173           No differences in the frequency of acute exacerbations, pneumonia, or hospitalization were
174                 Additionally, the severe HBV acute exacerbation rate in the therapeutic control stage
175  rate, HBV prophylactic rate, and severe HBV acute exacerbation rate were compared between stages wit
176 nol abuse, pain, narcotic use, and recurrent acute exacerbations requiring hospital admission before
177  safe and well tolerated in CF patients with acute exacerbations requiring hospitalization, but the s
178 s and controls, which rose sharply during an acute exacerbation suggesting that galectin-3 may be a m
179 atment for patients with severe COPD with an acute exacerbation that includes increased sputum purule
180 t complication of advanced COPD and predicts acute exacerbations, though pulmonary vascular abnormali
181  in 54 patients with COPD examined during an acute exacerbation (V1) and 2 months afterward (V2) and
182 hat in men, the incidence of death caused by acute exacerbation was higher and that caused by cardiov
183 d participants to determine if treatment for acute exacerbation was required.
184                             The mean rate of acute exacerbations was 6.3 +/- 2.1 events per year befo
185 om asthmatic patients with stable disease or acute exacerbations was further studied to determine the
186 sistent pattern (both years with and without acute exacerbations) was common (456 [41%] of the group)
187 obiome before, at the onset of, and after an acute exacerbation were examined in 60 sputum samples co
188  Patients with schizophrenia experiencing an acute exacerbation were randomly assigned to daily brexp
189                                              Acute exacerbations were primarily respiratory (five pat
190 ional morbidity and mortality are related to acute exacerbations, which are associated with further m

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