1  IL-10 and attenuated renal tissue damage in acute glomerulonephritis.

2 strains that cause impetigo, pharyngitis and acute glomerulonephritis.

3 equelae, including acute rheumatic fever and acute glomerulonephritis.

4 teristics and involved in rheumatic fever or acute glomerulonephritis.

5 ularly when associated with pre-eclampsia or acute glomerulonephritis.

6 enal disease (2 with lupus nephritis, 1 with acute glomerulonephritis, and 1 with acetaminophen-induc

7 om important differential diagnoses, such as acute glomerulonephritis (GN).

8  glomerulonephritis, the most common form of acute glomerulonephritis in children, continues to be a

9 administration of IL-10 has shown benefit in acute glomerulonephritis, no studies have addressed the

10 tacrolimus toxicity], nontubular injury (eg, acute glomerulonephritis), or physiological stress (eg,

11 f a large 1998 outbreak of poststreptococcal acute glomerulonephritis (PSGN) in Nova Serrana, Brazil,

12 C repeat region peptides of M protein, while acute glomerulonephritis sera were not as reactive.

13 break the stability of the Th17 phenotype in acute glomerulonephritis, we subjected nephritic mice to