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1 corticosteroids reduce pain in patients with acute gout.
2  analgesic effectiveness among patients with acute gout.
3  corticosteroids relieve pain in adults with acute gout.
4 corticosteroids reduce pain in patients with acute gout.
5 effective first-line option for treatment of acute gout.
6  inhibitors and glucocorticoids for treating acute gout.
7 well as traditional NSAIDs, are effective in acute gout.
8 s confirmed to be an effective treatment for acute gout.
9 ptomatic hyperuricemia and the resolution of acute gout.
10 tained regarding the inflammatory process in acute gout.
11 SAIDs), or colchicine to treat patients with acute gout.
12 ess the role of CXCR-2 ligands in a model of acute gout.
13 trophil-dependent inflammation is central to acute gout.
14  corticosteroids relieve pain in adults with acute gout.
15 se colchicine when using colchicine to treat acute gout.
16 sting is necessary in patients with possible acute gout.
17 -1 inhibitors or placebo in the treatment of acute gout?
18 se reductions of 33-66% for the treatment of acute gout and 50-75% for prophylaxis were calculated fo
19                                      Because acute gout and pseudogout appear to be dependent on inte
20 yrophosphate dihydrate (CPPD) crystals cause acute gout and pseudogout, respectively.
21 eroids are increasingly accepted in treating acute gout, and the role of colchicine in acute and inte
22 ol or febuxostat) reduces long-term risk for acute gout attacks after 1 year or more.
23 ly colchicine or NSAIDs reduces the risk for acute gout attacks by at least half in patients starting
24                                     Although acute gout attacks can be treated, eliminating gout requ
25        To review evidence about treatment of acute gout attacks, management of hyperuricemia to preve
26 rpose: To review evidence about treatment of acute gout attacks, management of hyperuricemia to preve
27 ases serum urate levels and reduces risk for acute gout attacks.
28 nted on appropriate dosing of colchicine for acute gout flares and potential toxicities of combining
29 o play a key role in the resolution phase of acute gout has also been demonstrated.
30  important in controlling the development of acute gout in patients with hyperuricemia.
31                    Unlike IL-8, the roles in acute gout of individual complement-derived peptides and
32 is for oral colchicine therapy and dosing in acute gout remains limited.
33  investigated both for the management of the acute gout symptoms, targeting interleukin-1beta, as wel
34                   Limitation: Few studies of acute gout treatments, no placebo-controlled trials of m

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