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1 c-ischaemic neuronal injury, consistent with acute infarction.
2 he extent of microvascular obstruction after acute infarction.
3 and enhancer RNAs in a large animal model of acute infarction.
4 time to be limited to hypoxic regions during acute infarction.
5 y flow as well as five key parameters during acute infarction.
6 h bypass surgery performed in the setting of acute infarction.
7 ction against intraventricular thrombi after acute infarction.
8 ce (3 days after occlusion for assessment of acute infarction; a mean of 50 days after occlusion +/-
10 roups according to the clinical diagnosis of acute infarction (Al) (n = 20), stable angina (SA) (n =
12 r compensatory responses of the rat brain to acute infarction and to the development and expression o
13 s reduce myocyte apoptosis in the setting of acute infarction, and this effect can be detected by in
14 r agent produced differential enhancement of acute infarctions at 3 days (SI ratio 5.8 +/- 1.3) but n
16 cable findings in all categories, except for acute infarction, compared with the other two ICUs (p <
17 nrolled in the SHOCK Trial Registry of CS in acute infarction, comparing 55 patients whose shock was
19 ocardiographic criteria for the diagnosis of acute infarction in the presence of left bundle-branch b
20 a with independent value in the diagnosis of acute infarction in these patients were an ST-segment el
21 effect of bFGF on infarct size in a model of acute infarction in which coronary occlusion was followe
22 and bypass surgery in select patients after acute infarction is associated with low repeat procedure
29 hypothesis that restraining expansion of an acute infarction preserves LV geometry and resting funct
31 be safely operated on early in the course of acute infarction, risk factors for hospital mortality ar
32 ted animals with chronic, but not those with acute, infarction showed the following differences compa
33 ients admitted with a tentative diagnosis of acute infarction, the initial impression was confirmed.
35 When evaluating a patient who has survived acute infarction treated with thrombolysis, clinicians c
41 ts who underwent thrombolysis within 12 h of acute infarction with confirmed cardiogenic shock, 27 un
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