ライフサイエンスコーパス: acute myocarditis

1 se Criteria is used to make the diagnosis of acute myocarditis.

2 rence standard was the clinical evidence for acute myocarditis.

3 R approaches in patients suspected of having acute myocarditis.

4 and global myocardium edema in patients with acute myocarditis.

5 s T. cruzi control and protection from fatal acute myocarditis.

6 acute from chronic MI and identification of acute myocarditis.

7 L-1beta, and IL-6 levels in the heart during acute myocarditis.

8 lar magnetic resonance to detect and monitor acute myocarditis.

9 ting that IL-17A plays a minimal role during acute myocarditis.

10 min in cardiac myocytes, results in a lethal acute myocarditis.

11 rdiovirulent phenotype in a murine model for acute myocarditis.

12 nt myocarditis, and 132 met the criteria for acute myocarditis.

13 lminant myocarditis compared with those with acute myocarditis.

14 ction at six months compared with those with acute myocarditis.

15 f interferon, may determine reovirus-induced acute myocarditis.

16 minant (fractional shortening 19 +/- 4%) and acute myocarditis (17 +/- 7%) had LV systolic dysfunctio

17 ompared with no improvement in patients with acute myocarditis (19 +/- 7%, p < 0.01 for interaction b

18 Eighteen patients with clinical diagnosis of acute myocarditis (25 years [23-38 years]; 78% males) we

19 compared with only 45 percent of those with acute myocarditis (95 percent confidence interval, 30 to

20 ction is one of the most prevalent causes of acute myocarditis, a disease that frequently is identifi

21 nfection is one of the most common causes of acute myocarditis, a serious and sometimes fatal disease

22 Coxsackievirus B3 (CVB3) is a major cause of acute myocarditis, a serious condition that is refractor

23 enhancement (LGE) has not been clarified in acute myocarditis (AM) with preserved left ventricular (

24 ome sequencing of 42 unrelated children with acute myocarditis (AM), some with proven viral causes.

25 Thirteen patients with acute myocarditis and a control group of seven healthy a

26 ations for the pathogenesis and treatment of acute myocarditis and allograft rejection.

27 Chagas disease, characterized by acute myocarditis and chronic cardiomyopathy, is caused

28 curacy to discriminate between patients with acute myocarditis and healthy controls was 86% for T2>52

29 t as BTR should be considered in the care of acute myocarditis and PPCM.

30 ata from ITAMY (ITalian multicenter study on Acute MYocarditis) and evaluated CMR results from 386 pa

31 Diagnosis, prognosis, and management of acute myocarditis are also discussed.

32 cted with Brazil strain of T cruzi developed acute myocarditis by day 21 after infection, consisting

33 ulminant myocarditis is distinguishable from acute myocarditis by echocardiography.

34 role of viral RNA synthesis in induction of acute myocarditis by infecting primary cultures of cardi

35 Th2 responses reduce acute myocarditis by inhibiting Th1 responses via regula

36 cate that Th1-type immunity protects against acute myocarditis by reducing viral replication and prev

37 mouse models with opposite susceptibility to acute myocarditis caused by the myotropic Colombiana str

38 lammatory microRNA-155 is upregulated during acute myocarditis, contributes to the adverse inflammato

39 Patients with acute myocarditis did not have these features.

40 se Abs may contribute to the pathogenesis of acute myocarditis found in patients with KS.

41 /- 0.2 cm) at presentation, while those with acute myocarditis had increased diastolic dimensions (6.

42 s and severe hemodynamic compromise, whereas acute myocarditis has an indistinct presentation, less s

43 t in the human heart long after the signs of acute myocarditis have abated are still not completely u

44 tions regarding athletic participation after acute myocarditis have heightened the importance of earl

45 consistently and strongly upregulated during acute myocarditis in both humans and susceptible mice.

46 among the most commonly identified causes of acute myocarditis in children and adults and have been i

47 irus B3 (CVB3) is a principal viral cause of acute myocarditis in humans and has been implicated in t

48 erent reovirus reassortant viruses to induce acute myocarditis in mice correlates with cytopathogenic

49 Reovirus-induced acute myocarditis in mice serves as a model to investiga

50 vation, and reduced myocardial damage during acute myocarditis in mice.

51 a provide two insights into reovirus-induced acute myocarditis in mice.

52 enation for 147 patients with a diagnosis of acute myocarditis in the Extracorporeal Life Support Org

53 ear diagnostic and management guidelines for acute myocarditis in the pediatric population.

54 Acute myocarditis is characterized by the rapid developm

55 Although acute myocarditis is observed in most patients with KS,

56 The short-term prognosis of acute myocarditis is usually good, but varies widely by

57 Twenty four patients with acute myocarditis (mean age +/- standard deviation, 34.7

58 localized granzyme B activity in hearts with acute myocarditis monitored by fluorescent molecular tom

59 Acute myocarditis must be considered in patients who pre

60 Patients with acute myocarditis (n=514) were identified from April 200

61 ugh both T2 and T1 mapping reliably detected acute myocarditis, only T2 mapping discriminated between

62 y 2001 and November 2016 with a diagnosis of acute myocarditis (onset of symptoms <1 month) of whom 5

63 identify myocardial injury in patients with acute myocarditis or TTCM.

64 s were significantly longer in patients with acute myocarditis than in control subjects (1185.3 msec

65 rotozoan parasite Trypanosoma cruzi leads to acute myocarditis that is accompanied by autoimmunity to

66 hile viral RNA synthesis is a determinant of acute myocarditis, this is not due to generation of infe

67 The severity of acute myocarditis uniquely correlated with the proportio

68 hophysiological mechanisms, and treatment of acute myocarditis were gained during the last years, no

69 Intense immunostaining was observed in acute myocarditis, whereas VP1 was detected in scattered