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1 n into toxic products that ultimately causes acute renal failure.
2 ic or end-stage renal disease, and seven had acute renal failure.
3 li infection is a leading cause of pediatric acute renal failure.
4 (IRI), which is the major cause of intrinsic acute renal failure.
5 nfarction, diabetes, sepsis, and hepatic and acute renal failure.
6 n of xenon before renal ischemia can prevent acute renal failure.
7 iitis, resulting in pulmonary hemorrhage and acute renal failure.
8 pathogen that causes hemorrhagic colitis and acute renal failure.
9 US is similar to TTP, but is associated with acute renal failure.
10 thic hemolytic anemia, thrombocytopenia, and acute renal failure.
11 mber of new therapies are in development for acute renal failure.
12 y provide a breakthrough in the treatment of acute renal failure.
13 eins, cyclophilin, attenuated sepsis-induced acute renal failure.
14 y and apoptotic insult during sepsis-induced acute renal failure.
15 athophysiology, prevention, and treatment of acute renal failure.
16 Attempts have been made to clearly define acute renal failure.
17 erular cells and is the most common cause of acute renal failure.
18 ons, are associated with a high incidence of acute renal failure.
19 urrent infection, cardiovascular events, and acute renal failure.
20 eday offer the best option for recovery from acute renal failure.
21 olestatic liver diseases and the associating acute renal failure.
22 l animal models during or after the onset of acute renal failure.
23 of stem cell research might be used to treat acute renal failure.
24 promise for the proactive treatment of human acute renal failure.
25 a on current and promising new therapies for acute renal failure.
26 of IL-6 was investigated in murine ischemic acute renal failure.
27 thy, hemolytic anemia, thrombocytopenia, and acute renal failure.
28 6, and this IL-6 exacerbates ischemic murine acute renal failure.
29 ded clues towards accelerating recovery from acute renal failure.
30 ators of tissue damage in ischemic and toxic acute renal failure.
31 ay be useful for cell replacement therapy of acute renal failure.
32 es renal I/R injury and severity of ischemic acute renal failure.
33 ion injury (I/R injury) is a common cause of acute renal failure.
34 ated with several renal syndromes, including acute renal failure.
35 is common, and in some cases, can present as acute renal failure.
36 herapy; the most common of these effects was acute renal failure.
37 nfection, bleeding or transfusion event, and acute renal failure.
38 d a 2.5- to 4-fold higher risk of developing acute renal failure.
39 tor antagonist was associated with decreased acute renal failure.
40 d by hemolytic anemia, thrombocytopenia, and acute renal failure.
41 enia, microangiopathic hemolytic anemia, and acute renal failure.
42 ) infections are leading causes of pediatric acute renal failure.
43 y diarrhea with the potential to progress to acute renal failure.
44 alkalinization, and monitoring for signs of acute renal failure.
45 l hemorrhage (1.6% versus 0.2%; P=0.03), and acute renal failure (13.9% versus 9.4%; P=0.02) were sig
46 7%), paraplegia (8.5%), bowel ischemia (7%), acute renal failure (21%), dialysis requirement (13%), a
47 related to treatment), pneumonia (27 [11%]), acute renal failure (25 [10%]; five related to treatment
51 rculatory support-related complications were acute renal failure 41%, bleeding 25%, neurologic damage
52 The most common serious adverse events were acute renal failure (41 [3.2%] vs 33 [2.5%]) and suprave
53 troke (14 [2.0%] and 15 [2.2%], P=0.79), and acute renal failure (42 [6.1%] and 35 [5.1%], P=0.45).
54 of death (2.3%, 0.8%, and 0.6%; P = .02) and acute renal failure (6.2%, 7.6%, and 2.4%; P < .001) aft
55 N1 influenza (8%), respiratory failure (8%), acute renal failure (7%), and acute respiratory distress
58 r than ischemic or multifactorial) origin of acute renal failure, acute respiratory failure, and lowe
60 living donor kidney recipients who developed acute renal failure after exposure to sirolimus-tacrolim
62 Y720 reduced IRI and prevented unrecoverable acute renal failure after significant ischemic injury.
67 ns (eg, myocardial infarction, pneumonia, or acute renal failure and a length of stay >75th percentil
68 ns (eg, pneumonia, myocardial infarction, or acute renal failure and a length of stay >75th percentil
69 y diarrhea with the potential to progress to acute renal failure and central nervous system complicat
70 part D; two adverse events leading to death (acute renal failure and death, cause unknown) were possi
72 isk of postcolonoscopy complications such as acute renal failure and GI bleeding, especially in patie
74 iations were strongest between ED visits for acute renal failure and heat waves defined by maximum ap
75 fusion injury (IRI) is a feature of ischemic acute renal failure and it impacts both short- and long-
78 for these patients to prevent or ameliorate acute renal failure and reduce the need for RRT postoper
79 rhoea and urinary sepsis in one patient, and acute renal failure and respiratory failure in one patie
80 y well tolerated, with similar occurrence of acute renal failure and treatment-emergent serious adver
82 ts (pneumonia, myelodysplastic syndrome, and acute renal failure) and two in the treatment of physici
83 at care for a larger number of patients with acute renal failure, and black patients had lower in-hos
84 smoking, steroid, congestive heart failure, acute renal failure, and dyspnea) were analyzed in terms
85 acheitis, encephalopathy, bacteremia/sepsis, acute renal failure, and myocarditis were rare (each </=
86 in-hospital events (death, stroke, bleeding, acute renal failure, and need for permanent pacemaker) w
90 Renal ischemia-reperfusion injury causes acute renal failure, and the hallmarks of renal ischemia
91 imately 50%), ileus ( approximately 2 days), acute renal failure ( approximately 30%), and blood loss
93 ims databases may be useful for the study of acute renal failure (ARF) and ARF that requires dialysis
96 R) injury of the kidney is a common cause of acute renal failure (ARF) and is associated with high mo
100 nown that, among human patients with sepsis, acute renal failure (ARF) dramatically increases mortali
104 e prediction of adverse clinical outcomes in acute renal failure (ARF) has not been well described.
105 have concluded that outcomes associated with acute renal failure (ARF) have not improved significantl
106 to determine the incidence and mortality of acute renal failure (ARF) in Medicare beneficiaries.
108 nd to bovine serum albumin ameliorate murine acute renal failure (ARF) induced by temporary occlusion
113 kidneys recovered from deceased donors with acute renal failure (ARF) is higher compared with those
116 We therefore hypothesized that LPS-induced acute renal failure (ARF) requires systemic TNF release
123 ction of postoperative 180-day mortality and acute renal failure (ARF), improving upon predictions th
126 Among the 31 patients, 21 presented with acute renal failure (ARF), were normocalcemic, and had a
131 can cause bloody diarrhea and, occasionally, acute renal failure as a consequence of Shiga toxin (Stx
132 tested the hypothesis that the diagnosis of acute renal failure associated with coronary artery bypa
133 We conclude that the nationwide trend of acute renal failure associated with coronary artery bypa
134 EPO can ameliorate ischaemic and nephrotoxic acute renal failure, Bahlmann's work is the first eviden
135 a novel therapeutic intervention in ischemic acute renal failure, based at least in part on its abili
136 In stratified analyses of patients with acute renal failure, black patients had significantly lo
137 rest, coma >24 hours, myocardial infarction, acute renal failure, bleeding requiring >4 units of red
139 udy, the role of exogenous HGF in preventing acute renal failure by systemic administration of naked
141 % (95% confidence interval [CI], 67%-91%) of acute renal failure cases compared with 38% (95% CI, 25%
144 isease, the incidence of secondary causes of acute renal failure continue to grow, especially in pati
146 s grafted into WT recipients (n=7) developed acute renal failure (control group), WT grafts transplan
147 mposite of mortality, myocardial infarction, acute renal failure, coronary revascularization, or stro
149 30 mL/min who received a high dose of GBCA, acute renal failure, delayed hemodialysis after contrast
152 ndings suggest that the observed increase in acute renal failure diagnosis rates may be partly attrib
153 ttributable to less restrictive criteria for acute renal failure diagnosis, consistent with acute ren
154 further assess the role of BAK in sepsis in acute renal failure, dogs were nephrectomized and 48 h l
155 5) on deviant days for heat-related illness, acute renal failure, electrolyte imbalance, and nephriti
156 tions, particularly in splenectomized cases, acute renal failure, Evans syndrome, and multitreatment
157 reased susceptibility to hospitalization for acute renal failure for blacks, Hispanics, people aged 2
160 eceived N-acetylcysteine had an incidence of acute renal failure (>or=0.5 mg/dL increase in creatinin
161 diffuse neurologic deficit, amaurosis fugax, acute renal failure, gut ischemia, livedo reticularis an
166 ology and insight into mechanisms leading to acute renal failure have triggered investigators to eval
167 luded congenital heart disease, age >/=1 yr, acute renal failure, hepatic insufficiency, and sepsis.
172 yndrome, which is the most frequent cause of acute renal failure in children in the Americas and Euro
173 ome (HUS), which is the most common cause of acute renal failure in children in the United States.
178 IAH/ACS is an important possible cause of acute renal failure in critically ill patients and scree
180 c syndrome (HUS) is the most common cause of acute renal failure in infants and young children, and i
182 here were trends toward reduced incidence of acute renal failure in patients with baseline Sequential
186 our understanding of the pathophysiology of acute renal failure in pregnancy where plasma androgen l
188 replacement therapy over the past few years, acute renal failure in the intensive care unit remains a
189 s, the most common condition associated with acute renal failure in the intensive care unit, may alte
190 udies have suggested a variable incidence of acute renal failure in this population, with an associat
198 nces in understanding the pathophysiology of acute renal failure, little progress has been made in it
200 pment of sensitive, predictive biomarkers of acute renal failure may help to diagnose the syndrome ea
201 Secondary outcomes included encephalopathy, acute renal failure, mechanical ventilation, and dischar
202 (n = 2), gastric outlet obstruction (n = 1), acute renal failure (n = 2), pneumonia (n = 2), respirat
203 possible exception of radio-contrast-induced acute renal failure, no drugs are capable of preventing
206 scitation on multivariable analysis included acute renal failure (odds ratio 1.5, 95% confidence inte
207 io 36.6, 95% confidence interval 21.9-61.0), acute renal failure (odds ratio 21.6, 95% confidence int
208 ly associated with clinical failure included acute renal failure (odds ratio [OR], 3.91 [95% confiden
212 rate (eGFR) lower than 30 mL/min, and 11 had acute renal failure or acute deterioration of chronic re
213 inflammatory state, more specific effects of acute renal failure or chronic kidney disease, and effec
215 en expended to develop techniques to prevent acute renal failure or to facilitate its resolution.
216 th (OR 7.8, 95% CI 4.2 to 14.7; p < 0.0001), acute renal failure (OR 2.8, 95% CI 1.4 to 5.7; p = 0.00
217 y (OR, 8.6; 95% CI, 3.9-18.8; P < .001), and acute renal failure (OR, 10.5; 95% CI, 3.8-29.3; P < .00
218 mia (OR, 9.03; 95% CI, 3.49-23.38; P<0.001), acute renal failure (OR, 3.61; 95% CI, 1.68-7.75; P=0.00
219 hromboembolism (OR:2.11; 95% CI: 1.70-2.61), acute renal failure (OR: 1.34; 95% CI; 1.22-1.47), and s
220 peracillin" and "tazobactam"] and ["AKI" or "acute renal failure" or "nephrotoxicity"] and registered
221 peracillin" and "tazobactam"] and ["AKI" or "acute renal failure" or "nephrotoxicity"] and registered
222 ents with a subarachnoid hemorrhage, trauma, acute renal failure, or severe community-acquired pneumo
224 e (P<0.0001), need for laparotomy (P<0.008), acute renal failure (P<0.0001), need for dialysis (P<0.0
225 ute renal failure diagnosis, consistent with acute renal failure patterns observed in other clinical
226 l 9% increase in odds of hospitalization for acute renal failure per 5 degrees F (2.78 degrees C) was
227 nts that were deemed unrelated to alectinib: acute renal failure; pleural effusion and pericardial ef
228 k for postoperative complications, including acute renal failure, pneumonia, bleeding, septicemia, st
230 tive risk, 1.42; P<0.001), and postoperative acute renal failure (relative risk, 2.13; P<0.001).
233 Acute tubular necrosis secondary to ischemic acute renal failure remains a common clinical problem wi
238 ents for each sample was 2% (39 of 1924) for acute renal failure requiring dialysis, 0.7% (18 of 2327
245 ute respiratory distress syndrome (ARDS) and acute renal failure, requiring mechanical ventilation, v
247 enal solid organ transplants are at risk for acute renal failure resulting from cardiac or hepatic fa
250 (RR=12.17 [95% CI 1.3-117.2], P=0.007), and acute renal failure (RR=11.8 [95% CI 2.9-48.8], P<0.001)
252 se, presenting with profound hypotension and acute renal failure, secondary to hypovolemic shock.
253 , including myocardial infarction, diabetes, acute renal failure, sepsis, and acute lung injury.
254 s associated with failure to rescue included acute renal failure, septic shock, and postoperative pul
255 ncluded bacteremia, pulmonary complications, acute renal failure, shock, intensive care unit admissio
258 placement therapy) to $876,539 (data from an acute renal failure study in which continuous renal repl
259 in comorbid disease burden, mortality in the acute renal failure subgroup declined from 39.5% to 17.9
261 one (0.7%) group were less likely to develop acute renal failure than those randomized to placebo (5.
264 We found no increase in the incidence of acute renal failure, the frequency of intensive care uni
265 herapies for the treatment and prevention of acute renal failure, there are reasons to be optimistic.
266 microvasculature by platelet-fibrin thrombi, acute renal failure, thrombocytopenia, microvascular hem
269 ow- and high-intensity treatment arms of the Acute Renal Failure Trial Network and RENAL Replacement
270 Demographic data and effluent rates from the Acute Renal Failure Trial Network and RENAL Replacement
273 of death, myocardial infarction, stroke, or acute renal failure up to the time of hospital discharge
274 a relatively clear benchmark for diagnosing acute renal failure, use of alternate criteria to define
275 ife-threatening disease often accompanied by acute renal failure, usually occurs after gastrointestin
276 lung transplant recipients, the incidence of acute renal failure was 25%, with 8% of patients requiri
281 =7), where the protection from postoperative acute renal failure was no greater than in mice with MAS
283 ients who did not require renal replacement, acute renal failure was strongly associated with increas
285 eptor activity is required for recovery from acute renal failure, we examined the role of the EGF rec
286 esult of diabetic nephropathy presented with acute renal failure, weakness, myalgia, and pigmented ur
288 ry failure, major cardiac complications, and acute renal failure were associated with an increased mo
289 tus, congestive heart failure, or chronic or acute renal failure were identified as high-risk patient
290 dothelial injury, glomerular thrombosis, and acute renal failure were markedly attenuated despite the
291 , septicemia, acute respiratory failure, and acute renal failure were the most common causes for read
293 h no-balanced fluids: in-hospital mortality, acute renal failure with and without dialysis, and hospi
294 pio) developed thrombocytopenia, anemia, and acute renal failure with loss of glomerular function, in
295 ed by hemolytic anemia, thrombocytopenia and acute renal failure with multiple organ involvement.
296 its contribution to acute kidney injury and acute renal failure with regard to intra-abdominal press
297 significant differences in the prevalence of acute renal failure (with and without dialysis) or in-ho
298 nia, dehydration, heat stroke, diabetes, and acute renal failure, with a 10 degrees F increase in sam
299 of a pathogenic role for B cells in ischemic acute renal failure, with a serum factor as a potential
300 a, approximately 10% have dialysis-dependent acute renal failure, with cast nephropathy, caused by mo