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1 osis of carditis as a major manifestation of acute rheumatic fever.
2 abscesses and 2 (0.28%) were diagnosed with acute rheumatic fever.
3 mporally with a decrease in the incidence of acute rheumatic fever.
4 i.e., skin infection and pharyngitis-induced acute rheumatic fever.
5 atment in timely fashion in order to prevent acute rheumatic fever.
6 n that at which group A streptococcus causes acute rheumatic fever.
7 quelae of group A streptococcal infection in acute rheumatic fever.
8 fulfill the Jones Criteria for diagnosis of acute rheumatic fever.
9 They had had a total of 20 attacks of acute rheumatic fever.
10 and absence of other major manifestations of acute rheumatic fever.
11 eptococcal throat culture who are at risk of acute rheumatic fever.
12 es: uncomplicated pharyngitis, impetigo, and acute rheumatic fever.
13 1000 patient-years), 19 (0.6%) had recurrent acute rheumatic fever (3.49/1000 patient-years), and 20
14 The pathogenic mechanisms for developing acute rheumatic fever after group A streptococcal pharyn
16 The intervals between the last attack of acute rheumatic fever and operation ranged from 2 to 8 y
17 f 7 strategies for the primary prevention of acute rheumatic fever and rheumatic heart disease in chi
21 with documented GAS infections (pharyngitis, acute rheumatic fever, and severe invasive disease) also
24 nvestigated adverse outcomes for people with acute rheumatic fever (ARF) and rheumatic heart disease
30 l M protein is an epidemiological marker for acute rheumatic fever (ARF)-associated serotypes of grou
31 d States, Kawasaki disease has now surpassed acute rheumatic fever as the leading cause of acquired h
34 nternational guidelines for the diagnosis of acute rheumatic fever by defining high-risk populations,
35 drome was considered part of the spectrum of acute rheumatic fever by some, whereas others stressed t
36 only have the diagnostic Jones criteria for acute rheumatic fever changed, but substantial advances
39 s a means to diagnose cardiac involvement in acute rheumatic fever, even when overt clinical findings
40 icated that serum samples from patients with acute rheumatic fever have higher levels of antibodies t
41 espite a recent increase in the incidence of acute rheumatic fever in North America, rheumatic heart
42 the historic Jones criteria used to diagnose acute rheumatic fever in the context of the current epid
43 th post-streptococcal glomerulonephritis and acute rheumatic fever indicated that FBP54 is expressed
44 impact of population-specific differences in acute rheumatic fever presentation and changes in presen
49 of 5 of 13 KS sera, as compared with 5 of 8 acute rheumatic fever sera, contained Ab titers to human
50 d had a different pattern of reactivity than acute rheumatic fever sera, further supporting the assoc
51 coccal homologue in certain diseases such as acute rheumatic fever, suggest that SOK plays an importa
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