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1 s with different mobilities occur during the acute stage.
2 roup of 160 stroke patients scanned at a sub-acute stage.
3 th the 562-bp promoter in neurons during the acute stage.
4 the severity of metabolic derangement in the acute stage.
5 f a test to diagnose this disease during the acute stages.
6 reated routinely with simvastatin during the acute stages.
7 associated acute pancreatitis survived their acute stage; 151 had peripancreatic fluid collections.
8 t of blood throughout gray matter; (2) a sub-acute stage, 4-5 days after the onset of clinical signs,
11 ozing, erythematous, pruritic lesions in the acute stage and xerotic, lichenified plaques in the chro
12 tosis were similar in both models during the acute stage, and the number of apoptotic nuclei increase
13 associated with neglect symptoms at the sub-acute stage but only some of these are critical for pred
14 have biphasic properties-deleterious in the acute stage but potentially beneficial in the delayed st
15 ation and demyelination was noted during the acute stage, but only marginal effects on these disease
17 heterosexual transmission that precede this acute stage could facilitate the development of effectiv
20 the primary cause of muscle paralysis in the acute stage, during which impaired muscle membrane excit
21 in various lymphoid compartments during the acute stage (first 4 months) of feline immunodeficiency
22 lasma and cell-associated viremia during the acute-stage FIV infection appears to precede the appeara
24 ite strikingly, the expression of nearly all acute stage genes return to baseline levels in the asymp
29 nd nifurtimox are more effective against the acute stage in both clinical and experimental settings.
31 red on admission (baseline) and at 48 hours (acute stage) in 126 patients with acute coronary syndrom
32 mox as curative treatments, particularly for acute stage infections, most likely as a result of the h
35 rate that high transmission rates during the acute stage is not the main cause of excess clustering o
37 own extensive astrogliosis and cell death at acute stages (<7 days) but the temporal response at a ch
40 he efficacy of high-dose antioxidants in the acute stage of central serous chorioretinopathy (CSC).
41 ction of Ito in chagasic myocytes during the acute stage of Chagas' disease may reflect the lack of t
44 t IL-2 mRNA is markedly increased during the acute stage of collagen-induced arthritis (CIA), an anim
45 usceptible (SJL/J and B10.S) mice during the acute stage of DA and DAL*-1 infection was characterized
50 gan and site of viral replication during the acute stage of feline immunodeficiency virus (FIV) infec
54 tic gene expression changes during the early-acute stage of infection (8 weeks), suggesting it is a s
55 totoxic effect on cells collected during the acute stage of infection and an inhibition of proliferat
56 of strong Th2 responses that peak during the acute stage of infection and then decline despite ongoin
57 erferon by splenocytes challenged during the acute stage of infection but with a diminished recall re
58 es in gene expression were determined as the acute stage of infection developed into the chronic stag
61 ver, the presence of escape mutations in the acute stage of infection has raised a concern that accel
62 of viral antigen to white matter during the acute stage of infection is essential for the induction
63 ocytes obtained from C57BL/6 mice during the acute stage of infection were able to mediate both patho
65 ced in canine epicardial myocytes during the acute stage of infection with Trypanosoma cruzi (Chagas'
67 or 14 weeks of age (i.e. , the early- or mid-acute stage of infection), whole blood and large surgica
68 difficile bacteria and, in comparison to the acute stage of infection, have persistent, albeit amelio
69 anulomatous inflammation was enhanced at the acute stage of infection, the livers of IL-10T mice disp
70 nt increase in hepatic granuloma size at the acute stage of infection, which was associated with incr
74 mplaints using resting state fMRI in the sub-acute stage of injury as well as a group of control part
75 g complex plays a protective role during the acute stage of intestinal inflammation and contributes t
78 nervous system (ENS) of macaques during the acute stage of simian immunodeficiency virus (SIV) infec
81 recovery and that inhibition of VEGF at the acute stage of stroke may reduce the BBB permeability an
82 or hearts transplanted into mice during the acute stage of T. cruzi infection become parasitized and
83 f the immune system is known to occur in the acute stage of the disease and plays an important role i
84 mice, an altered tropism was seen during the acute stage of the disease and the chronic demyelinating
85 ence of possible pain experienced during the acute stage of the disease but not at the chronic stage
86 cardial coronary artery abnormalities at the acute stage of the disease or during follow-up, as asses
87 inst the P35 antigen are produced during the acute stage of the infection but are uncommon in the lat
88 the ME49 strain, all muMT mice survived the acute stage of the infection but died between 3 and 4 wk
89 y the P35-IgM-ELISA is more specific for the acute stage of the infection than demonstration of IgM a
91 the action of BK and KD, participates in the acute stage of the inflammatory and pain response, where
95 ed chronic cystitis, arguing that during the acute stages of infection, a higher number of IBCs prece
99 re one of many antigens that are secreted in acute stages of inflammation after left ventricle assist
100 eeded in order to optimize inhibition during acute stages of injury without interfering with benefici
101 that Fn14 plays a protective role during the acute stages of intestinal inflammation, and its absence
102 chondrial impairment and liver damage in the acute stages of liver copper accumulation compared with
104 sclerosis, yet potential swelling during the acute stages of optic neuritis may confound baseline mea
108 hepatitis B virus (HBV) dynamics during the acute stages of the infection and analyze the immune mec
110 ion and play a deterministic role during the acute stages on the rate of infection and disease progre
111 munoreactivity in convalescent compared with acute-stage or healthy control samples (34 in plasma, 39
112 egeneration was more limited overall than at acute stages, our combinatorial strategy in the chronica
114 d and unprotected animals are present during acute stages postchallenge, before different antigenic l
117 nalysis of these biopsy specimens during the acute stage revealed a massive infiltration of T cells,
118 ctious agent whose diagnosis at least in the acute stage, susceptibility testing, and management are
119 sibility of intervening in the acute and sub-acute stages to protect specific brain systems, such as
120 cutaneous anthrax and the first to associate acute stage toxemia with subsequent antitoxin antibody r
121 response to Francisella infection during the acute stage, transcriptomic analysis was performed on lu
123 ering statistics to be used for inference of acute stage transmission rates and found that no single
125 in lung B and T cell populations seen in the acute stage were also significantly reduced in the chron
126 c and egocentric neglect symptoms at the sub-acute stage were associated with lesions to dissociated
128 sis, the patients had a lower slope 5 in the acute stage, which then gradually increased during the f
129 ed with behavioural performance (i) at a sub-acute stage (within 3 months of the stroke) and (ii) at
130 nts had significantly lower DFAalpha2 in the acute stage (within 72 hours) and lower DFAalpha1 at 3 m
131 l necrosis consisted of three stages: (1) an acute stage, within 24 h of motor regression, characteri
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