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1 EV) infection typically causes self-limiting acute viral hepatitis.
2 ne components of the liver parenchyma during acute viral hepatitis.
3 timulator and a hepatoprotective cytokine in acute viral hepatitis.
4 rly if clinical features are compatible with acute viral hepatitis.
5 s (CTLs) and organ damage in mouse models of acute viral hepatitis.
6 e patients in the Sentinel Counties Study of Acute Viral Hepatitis.
7 rum aminotransferases, clinically resembling acute viral hepatitis.
8 ecific cytotoxic T lymphocytes (CTLs) during acute viral hepatitis.
9 er how it contributes to the pathogenesis of acute viral hepatitis.
10 virus (HCV), this has not been evaluated in acute viral hepatitis.
11 se RNA virus, generally causes self-limiting acute viral hepatitis, although chronic HEV infection ha
12 tudy to estimate morbidity and mortality for acute viral hepatitis, and for cirrhosis and liver cance
14 patologic evaluation revealed no evidence of acute viral hepatitis, autoimmune, metabolic or alcohol-
15 hanisms contribute to viral clearance during acute viral hepatitis by purging HBV replicative interme
21 lected patients from a surveillance study of acute viral hepatitis in four U.S. counties who had acut
22 robably contribute to viral clearance during acute viral hepatitis in humans, and they imply that ind
23 Hepatitis E virus (HEV) is a major cause of acute viral hepatitis in people in many developing count
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