ライフサイエンスコーパス: acute-phase reactant

1 r of the pentraxin family of proteins and an acute phase reactant.

2 s a unique serum pentraxin and the prototype acute phase reactant.

3 nate immune recognition molecule and classic acute-phase reactant.

4 monstrating that human hepcidin is a type II acute-phase reactant.

5 pain, HAQ, patient's global assessment, and acute-phase reactant.

6 ssment Questionnaire (HAQ), and levels of an acute-phase reactant.

7 erum levels of proinflammatory cytokines and acute phase reactants.

8 uring each period and measured for levels of acute-phase reactants.

9 signs and symptoms of RA and normalized the acute-phase reactants.

10 erum levels of proinflammatory cytokines and acute-phase reactants.

11 ng cell and by producing cytokines and other acute-phase reactants.

12 r factor (NF)-kappaB or transcription of the acute phase reactant alpha1-acid glycoprotein (AGP), wou

13 Chronic elevation of systemic levels of acute phase reactants and inflammatory cytokines found i

14 r (PPAR) signalling and lipid metabolism, of acute phase reactants and of two adipocytokines, leptin

15 ean concentrations of C-reactive protein, an acute phase reactant, and tissue plasminogen activator (

16 g syndrome (electrolytes and phosphate), and acute phase reactants, and recorded the nutritional ther

17 ssion of GST isozymes, positive and negative acute-phase reactants, and antioxidant enzymes were dete

18 d a much greater lymphocytosis, elevation in acute-phase reactants, and fall in serum iron and albumi

19 eft hindpaw volumes, circulating leukocytes, acute-phase reactants, and urinary collagen crosslinks t

20 lipopolysaccharide binding protein (LBP), an acute phase reactant; and plasma levels of bactericidal-

21 ; persistence for mean of 2 months; elevated acute phase reactants; and laboratory (usually serologic

22 of inflammation and vascular insufficiency, acute-phase reactants (APRs), and imaging including conv

23 Increases in acute phase reactants are typical of polymyalgia rheumat

24 Acute-phase reactants are, by far, the strongest determi

25 adipose tissue to the systemic elevation of acute phase reactants associated with chronic hyperglyce

26 te that adipose tissue expresses a number of acute phase reactants at high levels, including serum am

27 an indicator of functional ID that is not an acute-phase reactant, but challenges in its interpretati

28 of human neutrophils, it was found that the acute phase reactant C-reactive protein (CRP) activates

29 The human acute-phase reactant C-reactive protein (CRP) also prote

30 The acute-phase reactant C-reactive protein (CRP) is an impo

31 d high-density lipoprotein), serum levels of acute-phase reactants (C-reactive protein), erythrocyte

32 Serum levels of the acute-phase reactant, C-reactive protein (CRP), increase

33 Serum concentrations of the classic acute-phase reactants, C-reactive protein and serum amyl

34 est that LBP levels, which vary as LBP is an acute phase reactant, could be relevant to initiating in

35 Furthermore, levels of the acute-phase reactants ESR, hsCRP, and SAA were lower at

36 ammatory cytokine TNF-alpha and the positive acute phase reactant fibrinogen are also elevated in the

37 A small cluster included acute-phase reactant genes (SAA1, SAA2, and SAA2-SAA4).

38 serum protein that is >90% homologous to the acute-phase reactant haptoglobin (Hp).

39 Ferritin, an acute-phase reactant, has been found to be elevated in m

40 Because IL-22 induces the expression of acute phase reactants, IL-22RA2 may play an important ro

41 C-reactive protein (CRP) is the classic acute phase reactant in humans, with serum levels elevat

42 ChoP is the target of an acute phase reactant in serum, C-reactive protein (CRP),

43 C-reactive protein (CRP), the prototypic acute-phase reactant in humans, is synthesized in liver

44 raxin, C-reactive protein (CRP), is a strong acute-phase reactant in man and an opsonin.

45 physiological importance of the prototypical acute-phase reactant in the etiology of the disease is u

46 ced by T cells and induces the production of acute-phase reactants in vitro and in vivo, suggesting i

47 nd diminished insulin sensitivity (increased acute phase reactants) in patients with histologically p

48 The function of this acute-phase reactant includes activation of complement a

49 ence that ceruloplasmin, a copper-containing acute phase reactant, is secreted by zymosan-activated U

50 Serum amyloid A protein (SAA) is an acute-phase reactant, known to mediate pro-inflammatory

51 rrelation overall between EMP counts and the acute-phase reactant levels in the febrile disease contr

52 Birmingham Vasculitis Activity Score and the acute-phase reactant levels in the patients with systemi

53 nt's and physician's global assessments, and acute-phase reactant levels).

54 All individual ASAS components, acute-phase reactant levels, and spinal mobility measure

55 ut does not completely normalize symptoms or acute-phase reactant levels.

56 esis in HD patients) and plasma levels of an acute phase reactant, lipopolysaccharide binding protein

57 C-reactive protein (CRP), an acute-phase reactant long considered merely an innocent

58 dult liver specific gene, and its role as an acute phase reactant may well be more diverse than previ

59 mor necrosis factor-alpha (TNFalpha) and the acute phase reactant orosomucoid-1 (ORM1).

60 proinflammatory syndrome with alterations in acute-phase reactants, particularly at doses >50 mg/ m2/

61 Etanercept also reduced levels of acute-phase reactants, particularly during asymptomatic

62 evels of C-reactive protein (CRP), a hepatic acute phase reactant, predict risk for coronary heart di

63 onse, C-reactive protein being the classical acute-phase reactant produced in response to tissue dama

64 C-reactive protein (CRP) is an acute phase reactant protein produced primarily by the l

65 Serum amyloid A (SAA) is a highly conserved acute phase reactant protein, and its concentration in s

66 Determination of types I and II acute phase reactant proteins, constitutive serum protei

67 2, 5, and 7 and serum glucose, electrolytes, acute phase reactant proteins, tumor necrosis factor alp

68 differential nasal airway response including acute-phase reactants proteins (fibrinogen, haptoglobin

69 serum ferritin is a secreted protein and an acute phase reactant regulated by inflammatory hormones

70 ator may be limited by the lack of available acute-phase reactant results at the time of the clinical

71 ntrations of their most sensitive endogenous acute phase reactants, SAA and serum amyloid P component

72 racterized by the systemic deposition of the acute-phase reactant serum amyloid A (SAA).

73 oid fibrils are derived from the circulating acute-phase reactant serum amyloid A protein (SAA), but

74 of extracellular aggregates derived from the acute-phase reactant serum amyloid A protein.

75 Hepatic induction of the acute-phase reactant serum amyloid A1 resulted in its in

76 The circulating cytokine milieu (IL-6) and acute phase reactants such as SAA may reflect alteration

77 ocument the marked elevations of nonspecific acute-phase reactants, such as C-reactive protein and er

78 saccharide (LPS)-binding protein (LBP) is an acute phase reactant that may play a dual role in vivo,

79 C-reactive protein (CRP) is an acute-phase reactant that is positively associated with

80 C-reactive protein (CRP) is an acute-phase reactant that is positively correlated with

81 C-reactive protein (CRP) is an acute-phase reactant that is positively correlated with

82 A-SAAs are major acute-phase reactants, the in vivo concentrations of whi

83 Since D-dimer and fibrinogen are acute phase reactants, these data support the hypothesis

84 as a minimum, joint counts and levels of an acute-phase reactant to define remission.

85 Acute-phase reactants were elevated with worsening of sy

86 hepatic gene expression, but in one class of acute-phase reactants, which includes fibrinogen, IL-1be

87 We here show that ceruloplasmin (Cp), an acute phase reactant with important functions in iron ho

88 C-reactive protein (CRP) is an acute phase reactant with roles in innate host defense,

89 orrelation of symptoms, signs, and levels of acute-phase reactants with disease activity in approxima