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1 ave a direct role in adipocyte metabolism or adaptive thermogenesis.
2 by basal metabolism, physical activity, and adaptive thermogenesis.
3 resistance to the LPS-induced suppression of adaptive thermogenesis.
4 ory responses are key regulators to suppress adaptive thermogenesis.
5 iation with increased energy expenditure and adaptive thermogenesis.
6 zed for energy expenditure, a process called adaptive thermogenesis.
7 otecting critical organs from hypothermia by adaptive thermogenesis.
8 diture in small mammals and neonates through adaptive thermogenesis.
9 is dissipated as heat in a process known as adaptive thermogenesis.
10 in WAT that drove uncoupled respiration and adaptive thermogenesis.
11 pecific roles for NT-PGC-1alpha in basal and adaptive thermogenesis.
12 gram of alternative macrophage activation in adaptive thermogenesis.
13 etic input to the transcriptional program of adaptive thermogenesis.
14 and oxidative metabolism, and essential for adaptive thermogenesis.
15 c responsiveness does not limit cold-induced adaptive thermogenesis.
16 cription factors to orchestrate a program of adaptive thermogenesis.
17 that plays a major role in the regulation of adaptive thermogenesis.
18 relation to classical physiological views of adaptive thermogenesis.
19 ays a major role in regulating lipolysis and adaptive thermogenesis.
20 receptors to the transcriptional program of adaptive thermogenesis.
21 kable under basal conditions and show normal adaptive thermogenesis, a process previously suggested t
24 Brown adipose tissue (BAT) is essential for adaptive thermogenesis and dissipation of caloric excess
28 the preponderant determinant of the impaired adaptive thermogenesis and hypothermia in cold-exposed D
30 Brown adipocytes (BAs) are specialized for adaptive thermogenesis and, upon sympathetic stimulation
33 ed hepatic insulin sensitivity and increased adaptive thermogenesis, and Them2-/- mice are also resis
37 variant N terminal (NT)-PGC-1alpha regulate adaptive thermogenesis by transcriptional induction of t
39 Diet-induced weight loss is accompanied by adaptive thermogenesis, ie, a disproportional or greater
42 f browning in inguinal WAT and activation of adaptive thermogenesis in brown adipose tissue (BAT).
43 dered a central transcriptional regulator of adaptive thermogenesis in brown adipose tissue (BAT).
46 metabolism, particularly certain aspects of adaptive thermogenesis in brown fat and skeletal muscle,
50 r mechanisms responsible for the compromised adaptive thermogenesis in obese subjects have not yet be
60 aim of this study was to investigate whether adaptive thermogenesis is sustained during weight mainte
64 s a major site of energy dissipation through adaptive thermogenesis mediated by uncoupling protein 1
66 iple aspects of energy metabolism, including adaptive thermogenesis, mitochondrial biogenesis, and fa
67 leads to a marked reduction of BAT-mediated adaptive thermogenesis, obesity and systemic insulin res
70 via type 2 deiodinase (D2) is necessary for adaptive thermogenesis, such that mice lacking D2 (D2KO)
71 , and there is no indication for a change in adaptive thermogenesis up to 1 y, when weight loss is ma
72 dipose-derived cytokine in the regulation of adaptive thermogenesis via a non-adrenergic pathway.
74 zed determinants of adipocyte plasticity and adaptive thermogenesis, which may have potential therape
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