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1 ges are entirely dissociable from changes in addictive behavior.
2 derstanding the neurobiological mechanism of addictive behavior.
3 plays a critical role in the development of addictive behavior.
4 d depression-like behavior and bHRs prone to addictive behavior.
5 Excessive sun tanning can result in addictive behavior.
6 hereby causing development and expression of addictive behavior.
7 mon vertebrate model system for the study of addictive behavior.
8 gmental area (VTA) are pivotally involved in addictive behavior.
9 he formation of drug-associated memories and addictive behavior.
10 has an important role in the development of addictive behavior.
11 ug-induced changes in dendritic spines drive addictive behavior.
12 er than stopping cigarette smoking, a highly addictive behavior.
13 y relevant for the control of stress-related addictive behavior.
14 tress that may help to initiate and maintain addictive behavior.
15 ine-induced aberrant synaptic plasticity and addictive behavior.
16 promotes the development and persistence of addictive behavior.
17 that likely contribute to the maintenance of addictive behavior.
18 understanding of their efficacy in reducing addictive behavior.
19 he FAAH-endocannabinoid system in regulating addictive behavior.
20 elucidating the biological underpinnings of addictive behavior.
21 pendent remodeling that may underlie certain addictive behavior.
22 ion in target regions in an effort to dampen addictive behaviors.
23 entifying new therapeutic targets for opiate addictive behaviors.
24 a role in the association between stress and addictive behaviors.
25 ased stress reactivity and irritability) and addictive behaviors.
26 rapeutics to better understand and influence addictive behaviors.
27 xpression in neural circuits responsible for addictive behaviors.
28 atal dysfunction, is a key characteristic of addictive behaviors.
29 fy specific circuit components that underlie addictive behaviors.
30 (LH), are involved in reward processing and addictive behaviors.
31 onal response that is linked to long-lasting addictive behaviors.
32 in determining who does and does not develop addictive behaviors.
33 Acb) dopamine mediates several motivated and addictive behaviors.
34 A (PKA) pathways are strongly implicated in addictive behaviors.
35 us accumbens modulates both motivational and addictive behaviors.
36 ribute to the acquisition and maintenance of addictive behaviors.
37 could be related to those that contribute to addictive behaviors.
38 t overeating of palatable food is similar to addictive behavior and characterized by dysregulation of
40 -analysis, representing 643 individuals with addictive behaviors and 609 healthy control individuals.
44 c neurotransmission plays a critical role in addictive behaviors, and recent evidence indicates that
46 ce use or gambling; participants at risk for addictive behaviors; and studies using the same patient
48 mesolimbic dopamine system, suggesting that addictive behaviors are encoded by changes in the reward
49 gnificance of learning factors in persistent addictive behavior as well as the neurobiological basis
50 ociated memories is critical for maintaining addictive behaviors, as presentation of drug-associated
52 ber of brain systems have been implicated in addictive behavior, but none have yet been shown to be n
53 (PFC) contribute to several core aspects of addictive behaviors, but the underlying neuronal process
54 re central to pain control, drug reward, and addictive behaviors, but underlying circuit mechanisms h
57 d aggression, impulsivity, and proclivity to addictive behaviors compared with low-novelty reactive r
58 et region amygdala, a key area implicated in addictive behavior, differs depending on the GATA4 genot
59 ased sensitivity to environmental events and addictive behaviors during adolescence, whereas the pauc
61 the synaptic plasticity associated with drug addictive behaviors; however, the causal role of the CRE
62 s depressive-like effects and contributes to addictive behavior in male nonhuman primates and rodents
65 ne and may contribute to an understanding of addictive behavior including disruptions in sleep and ci
66 self-administration drives the appearance of addictive behavior is unclear, but the mechanistic expla
67 anding the neural basis of this control over addictive behavior may aid in the development of treatme
68 ormones in the development and expression of addictive behaviors may provide new insights into treatm
69 opamine D1 receptors has profound effects on addictive behavior, movement control, and working memory
70 defense functions; the partially selfish and addictive behavior of the defense systems; and coupling
71 asticity and its synaptic concomitants drive addictive behaviors or whether they reflect homeostatic
72 Substantial evidence from rodent models of addictive behavior points to the involvement of the vent
74 e synaptic changes associated with facets of addictive behavior, supporting partial coincident neurol
75 ibute to impulsivity, which is a hallmark of addictive behaviors that underlie compulsive drug seekin
76 st the role of NMDAR-dependent plasticity in addictive behavior, we genetically inactivated functiona
77 king behavior, novelty-seeking behavior, and addictive behavior, we hypothesized alterations of the f
78 ompulsivity occurs during the development of addictive behavior, which provides insights into the gen
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