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1 This was due to an increase in the amount of adenine nucleotide translocase.
2 source, ATP depletion, or inhibition of the adenine nucleotide translocase.
3 tage-dependent anion channel (porin) and the adenine nucleotide translocase.
4 of porin, mitochondrial creatine kinase, and adenine nucleotide translocase.
5 hondria is known to be cardioprotective, and adenine nucleotide translocase 1 (ANT1) is a key mediato
7 ondria, to interact with and dephosphorylate adenine nucleotide translocase 1 (ANT1), a central molec
8 nd several mitochondrial proteins, including adenine nucleotide translocase 1 (ANT1), were more oxidi
9 eviously cloned cDNAs derived from the mouse adenine nucleotide translocase-1 and -2 genes (Ant1 and
11 olve saturated fatty acid stimulation of the adenine nucleotide translocase 2 (ANT2), an inner mitoch
12 us-purified voltage-dependent anion channel, adenine nucleotide translocase and the fusion protein we
13 bility transition pore that is formed by the adenine nucleotide translocase and the voltage-dependent
14 increased oxygen consumption independent of adenine nucleotide translocase and uncoupling proteins,
15 ease of the apparent molecular weight of the adenine nucleotide translocase (ANT) by up to 1.2 kDa.
16 een mitochondrial creatine kinase (MtCK) and adenine nucleotide translocase (ANT) can play an importa
20 n maleimide is known to attack Cys159 of the adenine nucleotide translocase (ANT) in a CAT-sensitive
21 nding puzzle is that in permeabilized cells, adenine nucleotide translocase (ANT) is less accessible
24 oside and bongkrekic acid, inhibitors of the adenine nucleotide translocase (ANT) that lock the trans
26 mely voltage-dependent anion channel (VDAC), adenine nucleotide translocase (ANT), and hexokinase II
27 or alpha induced RIP-dependent inhibition of adenine nucleotide translocase (ANT)-conducted transport
28 ibits all ADP-ATP-using reactions except the adenine nucleotide translocase (ANT)-mediated mitochondr
34 ds, these mitochondria also showed a greater adenine nucleotide translocase-catalysed proton conducta
37 d increased proton transport activity of the adenine nucleotide translocase (dependent on fatty acids
38 cle depended on electron transport chain and adenine nucleotide translocase functionality, but it was
39 This study provides evidence for a role of adenine nucleotide translocase in the mechanism underlyi
41 ependent anion channel (outer membrane), the adenine nucleotide translocase (inner membrane) and cycl
42 ge-dependent anion channel (outer membrane), adenine nucleotide translocase (inner membrane) and cycl
43 ation, increased levels of the mitochondrial adenine nucleotide translocase stress-sensitive B (SesB)
44 the voltage-dependent anion channel and the adenine nucleotide translocase were similar in the two m
45 ction of both the F(1)F(O)-ATPase and of the adenine nucleotide translocase, which delivers nucleotid
46 ation/knockdown-induced dysregulation in the adenine nucleotide translocase, which results in a slowe
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