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1 ein 4.1B (DAL-1, differentially expressed in adenocarcinoma of the lung).
2 tment disease-free survival of patients with adenocarcinoma of the lung.
3 8)F-FDG PET could be a prognostic factor for adenocarcinoma of the lung.
4 ated with adult-onset pulmonary fibrosis and adenocarcinoma of the lung.
5 uated in an orthotopic model of bronchogenic adenocarcinoma of the lung.
6 d tumor progression of tobacco smoke-induced adenocarcinoma of the lung.
7 ted with the presence of a k-ras mutation in adenocarcinoma of the lung.
8  lung transplant in a patient with a primary adenocarcinoma of the lung.
9 le for the observed rise in the incidence of adenocarcinoma of the lung.
10 ly and significantly associated with risk of adenocarcinoma of the lung.
11 riably, fall into the spectrum of peripheral adenocarcinomas of the lung.
12  42% of large cell carcinomas, and in 11% of adenocarcinomas of the lung.
13 hat protein 4.1B/differentially expressed in adenocarcinoma of the lung-1 (DAL-1) expression in menin
14 e study population comprised 4 patients with adenocarcinomas of the lung, 3 with squamous cell carcin
15           DAL-1 (differentially expressed in adenocarcinoma of the lung)/4.1B is a tumor suppressor g
16                                              Adenocarcinoma of the lung, a leading cause of cancer de
17           Inhibition of Notch-1 signaling in adenocarcinoma of the lung (ACL) cells causes apoptosis
18 d 27 smoking patients diagnosed with primary adenocarcinoma of the lung and searched for chromosomal
19  identified in a screen as down regulated in adenocarcinoma of the lung and was mapped to chromosome
20            It is preferentially expressed in adenocarcinomas of the lung and has been investigated as
21 enetic correlations for the subcategories of adenocarcinoma of the lung are now a topic of increasing
22 s should be considered for all patients with adenocarcinoma of the lung at diagnosis, regardless of c
23  the gene expression profiles of 154 primary adenocarcinomas of the lung, colon, and ovary.
24 d multistep carcinogenesis model, peripheral adenocarcinoma of the lung develops from noninvasive pre
25 ngthened when the analysis was restricted to adenocarcinoma of the lung (ETS-adjusted odds ratio = 1.
26                                           In adenocarcinomas of the lung, FANCF promoter methylation
27 formed to perform multiplexed assays testing adenocarcinomas of the lung for drivers in 10 genes to e
28 d States, approximately 15% of patients with adenocarcinoma of the lung harbor activating EGFR mutati
29 ic carcinogen vinyl carbamate, which induces adenocarcinoma of the lung in all animals within 16 week
30                                     Mucinous adenocarcinoma of the lung is a subtype of highly invasi
31                           The development of adenocarcinoma of the lung is believed to proceed from i
32                                              Adenocarcinoma of the lung is more strongly associated w
33                                              Adenocarcinoma of the lung is the most common form of lu
34 4.1 gene, DAL-1 (differentially expressed in adenocarcinoma of the lung) located on chromosome 18p11.
35 .47; Ptrend = 0.03) and was not observed for adenocarcinomas of the lung (multivariate RR = 0.91, 95%
36 arcinoma is a distinctive subtype of typical adenocarcinoma of the lung that tends to metastasize wid
37 -causing gene in a large family with IPF and adenocarcinoma of the lung to a 15.7 Mb region on chromo
38 ociation of waist circumference with risk of adenocarcinoma of the lung was found.
39 named DAL-1 (for Differentially expressed in Adenocarcinoma of the Lung), was shown to suppress growt
40  hundred patients with histologically proven adenocarcinoma of the lung were included in this retrosp
41                   Autopsy revealed papillary adenocarcinoma of the lung with numerous metastatic lesi
42 day, the incidence of SQCLCs is surpassed by adenocarcinomas of the lung with a shift towards periphe

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