コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
4 2, 3, 12, 17, 25, and 31 in sets of normal, adenomatous, and malignant tissues from 46 patients with
5 with fundic gland polyp (FGP) formation and adenomatous change, which are similar to those observed
6 use colon epithelium is sufficient to induce adenomatous changes and the generation of Paneth-like ce
13 ly history of colorectal cancer, or interval adenomatous findings could explain only a minority of su
14 essed to two types of more advanced lesions, adenomatous gland forming lesion (Type 1) and atypical g
15 thyroid mass, especially co-existing with an adenomatous goiter, should prompt a work-up for thyroid
16 invasive precursor lesions known as atypical adenomatous hyperplasia (AAH) and bronchioloalveolar car
19 ced the development of premalignant atypical adenomatous hyperplasia lesions at 6 wk after urethane i
20 s(G12D) model displayed features of atypical adenomatous hyperplasia, adenocarcinoma in situ, and inv
23 ly after parathyroidectomy, 100 to 300 mg of adenomatous or hyperplastic diseased parathyroid tissue
25 Using flow cytometric analysis of resected adenomatous parathyroid glands, we have isolated and cha
26 DA) model built to discriminate healthy from adenomatous parathyroid tissue was able to correctly cla
28 ate endoscopic prediction of hyperplastic or adenomatous polyp histology could reduce procedural time
31 ssion analysis, age over 60 years old, male, adenomatous polyp, current smoking and heavy alcohol con
32 types in a murine model of colon cancer, the adenomatous polyposis (APC) mutant (Apc (716/+)) model.
33 ications of MMVTx for patients with familial adenomatous polyposis (FAP) and the technical feasibilit
35 amatically altered for the worse in familial adenomatous polyposis (FAP) because these patients harbo
37 identified as a candidate gene for familial adenomatous polyposis (FAP) but further study identified
38 tumors often occur in patients with familial adenomatous polyposis (FAP) coli who have germ line muta
39 rative colitis (UC) in 37 patients, familial adenomatous polyposis (FAP) in 12 patients, and colonic
40 of thyroid cancer in patients with Familial adenomatous polyposis (FAP) in a prospective study of th
44 rliest colonic tissue alteration in familial adenomatous polyposis (FAP) patients, we present the hyp
46 atous polyposis coli (APC) underlie familial adenomatous polyposis (FAP), an inherited cancer syndrom
47 ive colitis, indeterminate colitis, familial adenomatous polyposis (FAP), and a select group of patie
48 atorial chemoprevention efficacy in familial adenomatous polyposis (FAP), signal of benefit from imag
54 to iPSCs derived from patients with familial adenomatous polyposis (FAP-iPSCs) harboring germline mut
55 testinal tissues from patients with familial adenomatous polyposis (n = 18) or sessile serrated adeno
58 in animals and in patients with the familial adenomatous polyposis by downregulating beta-catenin sig
60 e conditionally expressed a mutant allele of adenomatous polyposis coli (APC(cKO)) in murine uterine
61 n on chronic hypoxia-induced PH, we used the adenomatous polyposis coli (Apc(Min/+)) mouse, where red
65 contribution of the Wnt-regulating proteins adenomatous polyposis coli (APC) and APC2 in the pathoge
66 ested that specific GSK3 substrates, such as adenomatous polyposis coli (APC) and collapsin response
67 onal and transcriptional mechanisms and that adenomatous polyposis coli (APC) and GSK3beta, which are
68 expression of the gut tumor suppressor gene adenomatous polyposis coli (Apc) and its role in the oli
70 ther, we find that both the tumor suppressor adenomatous polyposis coli (APC) and the ADP-ribose poly
71 nce microscopy to image the tumor suppressor adenomatous polyposis coli (APC) and the formin mDia1 du
72 firmed an expected loss in the expression of adenomatous polyposis coli (APC) and the transcriptional
73 First, patients with germline mutations in adenomatous polyposis coli (APC) are susceptible to stom
74 polyposis (FAP) but further study identified adenomatous polyposis coli (APC) as responsible for FAP
76 C57BL/6J mice carrying the Min allele of Adenomatous polyposis coli (Apc) develop numerous adenom
79 mice bearing a heterozygote mutation in the adenomatous polyposis coli (APC) gene (Apc(Min/+) mice).
80 ted gene-targeted pigs with mutations in the adenomatous polyposis coli (APC) gene (APC) that are ort
81 of these models involve modification of the adenomatous polyposis coli (Apc) gene and are excellent
82 ased in human cells with deficiencies in the adenomatous polyposis coli (APC) gene and in cells stimu
93 letion in mice with inactivating mutation of adenomatous polyposis coli (APC) gene reduces intestinal
94 ssf1a can cooperate with inactivation of the adenomatous polyposis coli (Apc) gene to accelerate inte
95 n the bladder as conditional deletion of the adenomatous polyposis coli (Apc) gene within the adult b
96 intestinal carcinogenesis is mutation of the adenomatous polyposis coli (APC) gene, which leads to ac
97 requently bear inactivating mutations of the adenomatous polyposis coli (APC) gene, whose product is
118 aling following loss of the tumor suppressor adenomatous polyposis coli (APC) is thought to initiate
121 esis that initiation of colorectal cancer by adenomatous polyposis coli (APC) mutation is mediated by
125 nt/beta-catenin pathway activation caused by adenomatous polyposis coli (APC) mutations occurs in app
127 (CRCs), an initiating mutation occurs in the adenomatous polyposis coli (APC) or beta-catenin gene, a
128 n that have been associated with loss of the adenomatous polyposis coli (APC) or constitutive activat
131 nestin expressing NeuN positive neurons and adenomatous polyposis coli (APC) positive mature oligode
132 ibition of GSK-3beta and accumulation of the adenomatous polyposis coli (APC) protein at the plus end
136 inked to deficiencies in mismatch repair and adenomatous polyposis coli (APC) proteins, diet, inflamm
139 is functionally important for cell migration.Adenomatous polyposis coli (APC) regulates the localizat
140 gene targeting in mice, we demonstrate that adenomatous polyposis coli (APC) serves an essential fun
141 ne such pathway the tumor-suppressor protein adenomatous polyposis coli (APC) targets RNAs to cell pr
142 ting factor-responsive cells were defined by adenomatous polyposis coli (APC) time-of-flight mass cyt
144 In turn, KIF17 participates in localizing adenomatous polyposis coli (APC) to the plus ends of a s
151 ce with IEC-specific allelic deletion of the adenomatous polyposis coli (Apc) tumor suppressor locus,
152 atase 2A (PP2A) and must be protected by the adenomatous polyposis coli (APC) tumor suppressor protei
154 rcinomas contain truncating mutations in the adenomatous polyposis coli (APC) tumor suppressor, a neg
155 in signaling is negatively controlled by the adenomatous polyposis coli (APC) tumor suppressor, which
156 calculating the in vivo mutation rate of the adenomatous polyposis coli (APC) tumor-suppressor gene i
157 ollows a genetic pathway whereby loss of the adenomatous polyposis coli (APC) tumour suppressor and a
158 RNAs in the granules associate with the adenomatous polyposis coli (APC) tumour suppressor and t
161 In this study, the tumor suppressor protein adenomatous polyposis coli (APC) was found to be importa
164 ne the genetic relationship between MPC1 and Adenomatous polyposis coli (APC), a key tumor suppressor
166 approaches suggest that the tumor suppressor adenomatous polyposis coli (APC), a regulator of Wnt sig
172 atenin destruction complex components Axin1, adenomatous polyposis coli (APC), and GSK3beta were also
173 forms a complex with axin (axis inhibitor), adenomatous polyposis coli (APC), casein kinase 1alpha (
174 Together with its direct binding partner adenomatous polyposis coli (APC), EB1 can stabilize micr
177 in acromegaly patients induced colon p53 and adenomatous polyposis coli (APC), reversing progrowth GH
178 e designed against beta-catenin (Ctnnb1) and adenomatous polyposis coli (Apc), two commonly mutated g
179 ical Wnt signaling by targeting the gene for Adenomatous Polyposis Coli (Apc), which controls Wnt sig
180 e, we show that the tumor suppressor protein adenomatous polyposis coli (APC), which is a known MT-as
181 o develop colitis-associated and spontaneous adenomatous polyposis coli (APC)-related tumors of the i
187 ation suppresses beta-catenin activity in an adenomatous polyposis coli (APC)/glycogen synthase kinas
189 Arm is targeted for proteolysis by the Axin/Adenomatous polyposis coli (Apc1 and Apc2)/Zeste-white 3
190 V integration site family (WNT)/beta-catenin/adenomatous polyposis coli (CTNNB1/APC) pathway has been
193 sly in germ cells for proper localization of Adenomatous polyposis coli 2 and E-cadherin at the hub-G
196 G-protein signaling (RGS) domain that binds adenomatous polyposis coli and Galpha subunits, thereby
197 atenin and the destruction complex component adenomatous polyposis coli at a similar SLS motif to the
199 iation domain family 1 gene RASSF1A, and the adenomatous polyposis coli gene APC in tumors and in his
200 Mice carrying and non-sense mutation in Adenomatous polyposis coli gene at site R850, which desi
207 ling induced by loss of the tumor suppressor adenomatous polyposis coli or casein kinase 1alpha uncov
208 Most information about the roles of the adenomatous polyposis coli protein (APC) and its binding
210 Here we report that the tumour suppressor adenomatous polyposis coli protein (APC) directs the loc
212 ows that the actin-nucleating ability of the adenomatous polyposis coli protein is required for disas
213 W480 cells stably transformed with wild-type adenomatous polyposis coli showed decreased beta-catenin
215 he microtubule motor cytoplasmic dynein, the adenomatous polyposis coli tumor suppressor protein (APC
217 member 1, insulin-like growth factor 2, and adenomatous polyposis coli) and other solid tumors (e.g.
219 g in T cell lineages by deletion of the Apc (adenomatous polyposis coli) gene causes spontaneous T ce
220 mutation of the Wnt repressor APC (encoding adenomatous polyposis coli) leads to a state of aberrant
221 that contained mutations in either the APC (adenomatous polyposis coli) locus or in an allele of bet
224 ectly targets the tumor suppressor gene APC (adenomatous polyposis coli), thereby affecting Wnt (Wing
225 aditionally attributed to mutations in Axin, adenomatous polyposis coli, and beta-catenin that lead t
226 I, IGF2; tumor suppressor candidate 33, N33; adenomatous polyposis coli, APC; mut-L homolog 1, MLH1;
227 a-catenin pathway mutations, such as loss of adenomatous polyposis coli, are insensitive to this nove
229 -catenin pathway regulatory genes, including adenomatous polyposis coli, GSK3beta, axin 1, beta-caten
231 e glycogen synthase kinase 3beta (GSK3beta)- adenomatous polyposis coli-axin-mediated degradation pat
232 otubule organization and capture dynamics in adenomatous polyposis coli-deficient radial progenitors.
233 ted signaling and glycogen synthase kinase-3/adenomatous polyposis coli-mediated beta-catenin activat
235 intestinal tumors driven by mutations in the adenomatous polyposis coli/beta-catenin pathway and acti
238 erived WNT2 activated canonical signaling in adenomatous polyposis coli/beta-catenin wild-type colon
242 families showed recessive inheritance of the adenomatous polyposis phenotype and progression to CRC i
244 polyposis coli (APC) gene reduces intestinal adenomatous polyposis via Axin/beta-catenin axis and the
245 es of hereditary syndromes, such as familial adenomatous polyposis, a cancer predisposition syndrome
246 , germline mutation of which causes familial adenomatous polyposis, an autosomal intestinal cancer sy
247 rosis, Duchenne Muscular Dystrophy, Familial Adenomatous Polyposis, Hereditary Non-polyposis Colorect
248 yndromes, including Lynch syndrome, familial adenomatous polyposis, MUTYH-associated polyposis, and c
249 ately 30% of families affected by colorectal adenomatous polyposis, no germline mutations have been i
250 A diagnosis of Lynch syndrome, familial adenomatous polyposis, or another genetic syndrome can i
253 on-negative Lynch syndrome, 16 with familial adenomatous polyposis, two with constitutional mismatch
254 opic expression of APC, but not its familial adenomatous polyposis-related truncation mutant, promine
265 diseases, three females (1.6 %) had familial adenomatous polyposis; three patients (1 male and two fe
269 io [OR] = 3.1; 95% CI, 1.7-5.5) or high-risk adenomatous polyps (OR = 3.0; 95% CI, 1.2-8.0), compared
270 7; 95% confidence interval [CI], 1.64-6.47), adenomatous polyps (RR, 2.18; 95% CI, 1.18-4.61) and hea
271 op consensus guidelines for the detection of adenomatous polyps and CRC in asymptomatic average-risk
272 ion of CBS in human biopsies of precancerous adenomatous polyps and show that forced upregulation of
274 y layers of the intestinal wall, cancer, and adenomatous polyps based on the REIMS fingerprint of eac
275 in 7 (6%), hyperplastic polyps in 4 (3.5%), adenomatous polyps in 2(2%), history of ulcerative colit
276 ction of IL10 and have increased colitis and adenomatous polyps in chemical and genetic models of col
277 n-3 PUFAs are associated with lower risk of adenomatous polyps in women, and the association may be
278 urveillance colonoscopy for individuals with adenomatous polyps is based on adenoma histology, size,
280 classified as having no evidence of disease, adenomatous polyps of less than 10 mm, of 10 mm or more,
282 (95% confidence interval (CI): 1.4, 2.5) for adenomatous polyps only, 5.0 (95% CI: 3.3, 7.3) for hype
283 the hypothesis that colonoscopic removal of adenomatous polyps prevents death from colorectal cancer
285 r high grade dysplasia or size > 1 cm or > 3 adenomatous polyps) were found in 98 cases (7 %), low ri
286 characteristics of these tests for detecting adenomatous polyps, advanced adenomas based on size, or
287 be prevented by the detection and removal of adenomatous polyps, and survival is significantly better
288 s of human colorectal carcinoma (CRC) and in adenomatous polyps, indicating its suppression occurs ea
289 can detect cancer early and also can detect adenomatous polyps, thus providing a greater potential f
290 orectal cancer (CRC) patients with normal or adenomatous polyps, we found that both the adenoma and c
291 ed apoptosis, and suppressed angiogenesis in adenomatous polyps, which reduced both tumor number and
298 and two-fold (P = .02), respectively, during adenomatous transformation in normal rectal epithelium.
299 ACF, once postulated as the ACF destined for adenomatous transformation, have been rarely identified
300 273 colorectal glands (126 hyperplastic, 147 adenomatous) were isolated via laser capture microdissec
WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。