ライフサイエンスコーパス: adenomatous polyp

1 ell that expands clonally to give rise to an adenomatous polyp.

2 urveillance to patients with newly diagnosed adenomatous polyps.

3 at screening and more aggressive removal of adenomatous polyps.

4 fter cardiac transplantation on the risk for adenomatous polyps.

5 id not correlate with an increased burden of adenomatous polyps.

6 al cancer relies in part on the detection of adenomatous polyps.

7 ctal cancer in the families of patients with adenomatous polyps.

8 30 primary colorectal adenocarcinomas and 24 adenomatous polyps.

9 olonoscopies that identified hyperplastic or adenomatous polyps.

10 cer was prevented by colonoscopic removal of adenomatous polyps.

11 system for differentiating hyperplastic from adenomatous polyps.

12 cially in the context of hyperplastic and/or adenomatous polyps.

13 APC gene and individual risk of metachronous adenomatous polyps.

14 idence of colorectal cancer in patients with adenomatous polyps.

15 antly more biallelic carriers had coexisting adenomatous polyps.

16 d women aged 50-80 y with recently diagnosed adenomatous polyps.

17 ribute to the development and advancement of adenomatous polyps.

18 scopic findings, and of these, 256 cases had adenomatous polyps.

19 pression of haptoglobin in colon cancers and adenomatous polyps.

20 logically proven synchronous or metachronous adenomatous polyps.

21 tions correlated with the presence of >or=20 adenomatous polyps.

22 the incidence of disease through removal of adenomatous polyps.

23 well established for the precursor lesions, adenomatous polyps.

24 and vegetable consumption and first incident adenomatous polyps.

25 4 serrated adenomas, 3 admixed hyperplastic-adenomatous polyps, 10 tubular adenomas, and 6 carcinoma

26 e endoscopists had equivalent miss rates for adenomatous polyps (20% vs. 21%, respectively; P = 0.91)

27 rrated adenomas, 33% of admixed hyperplastic-adenomatous polyps, 30% of tubular adenomas, and 33% of

28 arcinomas (n=41), hyperplastic polyps (n=8), adenomatous polyps (=35), and adenocarcinomas (n=27), us

29 Of the 69 adenomatous polyps, 46 of the 51 that were 6 mm or more

30 TS polymorphisms in a case control study of adenomatous polyps (510 cases and 604 polyp-free control

31 es of 0.03, 0.03, and 0.04 for patients with adenomatous polyps 6 mm or larger, 6-9 mm, and 10 mm or

32 rgo altered expression in the APC(min) mouse adenomatous polyp, a precursor of cancer, as well as in

33 The primary outcome measure was advanced adenomatous polyps (AAPs).

34 ctal carcinomas are believed to originate as adenomatous polyps (adenomas), and the identification an

35 persons who have first-degree relatives with adenomatous polyps (adenomas).

36 characteristics of these tests for detecting adenomatous polyps, advanced adenomas based on size, or

37 In adenomatous polyps, although DNMT1 expression coincided

38 Age over 60 years old, adenomatous polyp and heavy alcohol consumption may affe

39 Colonic epithelial cells in adenomatous polyps and adenocarcinomas had a decreased i

40 X-2) is aberrantly expressed in premalignant adenomatous polyps and colorectal carcinomas and is asso

41 are aberrantly overexpressed in premalignant adenomatous polyps and colorectal carcinomas of humans.

42 op consensus guidelines for the detection of adenomatous polyps and CRC in asymptomatic average-risk

43 Ashkenazi Jewish patients with a history of adenomatous polyps and in 1 (5%) of 20 Ashkenazi Jewish

44 Sulindac causes regression of precancerous adenomatous polyps and inhibits the growth of cultured c

45 ion of CBS in human biopsies of precancerous adenomatous polyps and show that forced upregulation of

46 primary chemoprevention trial who developed adenomatous polyps and then had eradication of polyps af

47 sa from 13 healthy subjects, 9 patients with adenomatous polyps, and 9 with cancer.

48 present study of intact human colon crypts, adenomatous polyps, and adenocarcinomas focuses on subce

49 o assess the associations between NSAID use, adenomatous polyps, and apoptosis.

50 agreed that most colon cancers develop from adenomatous polyps, and it is this fact on which screeni

51 be prevented by the detection and removal of adenomatous polyps, and survival is significantly better

52 RCs with corresponding normal mucosa, and 52 adenomatous polyps (APs).

53 Siblings and parents of patients with adenomatous polyps are at increased risk for colorectal

54 Adenomatous polyps are by definition dysplastic and path

55 Findings from six studies of MTHFR C677T and adenomatous polyps are inconsistent.

56 Adenomatous polyps are precursors to colorectal cancer (

57 Adenomatous polyps are the most common neoplastic findin

58 ugs, designed to decrease the growth rate of adenomatous polyps, are very efficient at lowering colon

59 nguinity, who developed 10 carcinomas and 35 adenomatous polyps at age 23 and duodenal adenocarcinoma

60 ive value both for colorectal cancer and for adenomatous polyps at least 1 cm in diameter.

61 ) for detecting retrospectively identifiable adenomatous polyps at least 1 cm in size.

62 y layers of the intestinal wall, cancer, and adenomatous polyps based on the REIMS fingerprint of eac

63 ccurred in 30% of adenocarcinomas and 34% of adenomatous polyps but not in hyperplastic polyps or nor

64 s elevated in Ashkenazi Jewish patients with adenomatous polyps, but not hyperplastic polyps.The I130

65 e migration of epithelial cells within human adenomatous polyps by determining the distribution of pr

66 esota Cancer Prevention Research Unit (CPRU) adenomatous polyp case-control study.

67 With a case-control design involving adenomatous polyp cases (n = 564), colonoscopy-negative

68 unoreactive cells was strikingly reversed in adenomatous polyps compared with normal mucosa.

69 ssion analysis, age over 60 years old, male, adenomatous polyp, current smoking and heavy alcohol con

70 Given the high rate of adenomatous polyp detection on colonoscopy, the amount o

71 Participants with large-bowel adenomatous polyps diagnosed in the past 6 mo were rando

72 ial appearances, eight had carcinoma, 24 had adenomatous polyps, five had hyperplasia, 11 had fibroid

73 he aim of this study was to evaluate colonic adenomatous polyps for microsatellite instability to det

74 errated adenomas, and 3 admixed hyperplastic-adenomatous polyps from 13 patients with hyperplastic po

75 s from the training set and 102 ICVs and 138 adenomatous polyps from the test set could be segmented

76 A total of 386 ICVs and 67 adenomatous polyps from the training set and 102 ICVs an

77 Adenomatous polyps had surface "grooves" or "sulci." Sen

78 sor lesions to colorectal cancer, colorectal adenomatous polyps have been studied to enhance knowledg

79 ate endoscopic prediction of hyperplastic or adenomatous polyp histology could reduce procedural time

80 of digital chromoendoscopy for prediction of adenomatous polyp histology was 90.1 %.

81 in 7 (6%), hyperplastic polyps in 4 (3.5%), adenomatous polyps in 2(2%), history of ulcerative colit

82 ction of IL10 and have increased colitis and adenomatous polyps in chemical and genetic models of col

83 , including folate, reduce the recurrence of adenomatous polyps in humans, the effect is small.

84 Yet, adenomatous polyps in noncolitic colons are usually remo

85 ted disease characterized by the presence of adenomatous polyps in the colon and rectum, with inevita

86 f energy from fat) diet on the recurrence of adenomatous polyps in the large bowel.

87 n-3 PUFAs are associated with lower risk of adenomatous polyps in women, and the association may be

88 s of human colorectal carcinoma (CRC) and in adenomatous polyps, indicating its suppression occurs ea

89 stores, and iron intake as risk factors for adenomatous polyps, intermediate markers for colorectal

90 urveillance colonoscopy for individuals with adenomatous polyps is based on adenoma histology, size,

91 er, compared with patients with a history of adenomatous polyps, is unknown.

92 Colorectal neoplasms (adenomatous polyps) missed at OC before VC results were

93 hat whereas Apc(Min)(/+) mice developed many adenomatous polyps, mostly in the small intestine, by 3

94 00), in those APC-negative patients with <20 adenomatous polyps (n = 26), or in those with CRC who we

95 Treg cells that expand in adenomatous polyps no longer produce IL-10 and instead s

96 Six patients (19%) had adenomatous polyps; no carcinomas were detected.

97 with a first-time diagnosis of at least one adenomatous polyp of the distal colon or rectum and 502

98 classified as having no evidence of disease, adenomatous polyps of less than 10 mm, of 10 mm or more,

99 It has been suggested that controls with adenomatous polyps of the colon and rectum should be exc

100 nosuppression does not increase the risk for adenomatous polyps of the colon.

101 racterized by the development of hundreds of adenomatous polyps of the colorectum.

102 mor has only been reported to originate from adenomatous polyps of the gastric mucosa in these clinic

103 In 2003-2005, cases with adenomatous polyps only (n = 639), hyperplastic polyps o

104 (95% confidence interval (CI): 1.4, 2.5) for adenomatous polyps only, 5.0 (95% CI: 3.3, 7.3) for hype

105 The study was based on 236 cases with adenomatous polyps or cancer and 409 controls, all colon

106 No differences in detection of adenomatous polyps or frequency of complications were fo

107 ate with sex, age, size, or dysplasia of the adenomatous polyps or with differentiation and Dukes' st

108 inations, but not for patients with low-risk adenomatous polyps (OR = 1.8; 95% CI, 0.9-3.7).

109 io [OR] = 3.1; 95% CI, 1.7-5.5) or high-risk adenomatous polyps (OR = 3.0; 95% CI, 1.2-8.0), compared

110 use aspirin were also not at reduced risk of adenomatous polyps (OR, 1.59; 95% CI, 0.68-3.73).

111 ) carcinomas (P = 0.0001) and 12 of 24 (50%) adenomatous polyps (P = 0.0001).

112 centrations and the prevalence of colorectal adenomatous polyps (precursors to colorectal cancer) in

113 wice daily and the Prevention of Spontaneous Adenomatous Polyps (PreSAP) trial used 400 mg once daily

114 The Adenomatous Polyp Prevention on Vioxx (APPROVe) study as

115 the hypothesis that colonoscopic removal of adenomatous polyps prevents death from colorectal cancer

116 These results imply that development of adenomatous polyps proceeds through a top-down mechanism

117 ion, proposed mechanisms of development from adenomatous polyps, rationale for screening, and screeni

118 ns were detected by direct sequencing in 738 adenomatous polyps removed at baseline from 639 particip

119 7; 95% confidence interval [CI], 1.64-6.47), adenomatous polyps (RR, 2.18; 95% CI, 1.18-4.61) and hea

120 Eighty percent of adenomatous polyps showed no instability.

121 copy and polypectomy because of a history of adenomatous polyps; standardized incidence ratios were 1

122 ed region in colorectal FAP: allelic loss in adenomatous polyps tended to occur when the germline mut

123 sceral adipose tissue (VAT) in subjects with adenomatous polyps, the precursor lesion of colorectal c

124 dergoing complete colonoscopy and removal of adenomatous polyps; they remained in the study for appro

125 can detect cancer early and also can detect adenomatous polyps, thus providing a greater potential f

126 adenocarcinomas, including cases in which an adenomatous polyp was also present, by immunohistochemis

127 s 7.57 (95% CI 1.84-31.4) and with high-risk adenomatous polyps was 3.11 (1.86-5.18).

128 The sensitivity of optical colonoscopy for adenomatous polyps was 87.5 percent, 91.5 percent, and 9

129 0% (81.8-99.1), and for cancer and high-risk adenomatous polyps was 90.1% (84.4-94.0); specificity wa

130 The sensitivity of virtual colonoscopy for adenomatous polyps was 93.8 percent for polyps at least

131 The specificity of virtual colonoscopy for adenomatous polyps was 96.0 percent for polyps at least

132 orectal cancer (CRC) patients with normal or adenomatous polyps, we found that both the adenoma and c

133 ios for cancer, and for cancer and high-risk adenomatous polyps were also calculated.

134 Similar values were found when only adenomatous polyps were considered.

135 The proportion of examinations in which adenomatous polyps were detected by barium enema colonos

136 mpared with 38 (8%) of 497 P/P participants; adenomatous polyps were found in 28 (16%) N/N individual

137 National Polyp Study who had newly diagnosed adenomatous polyps were interviewed for information on t

138 Adenomatous polyps were present in 111 of 120 gene carri

139 In the group >3 years posttransplant, adenomatous polyps were present in 16%, hyperplastic pol

140 In the group <3 years posttransplant, adenomatous polyps were present in 25%, hyperplastic pol

141 Neoplastic lesions (mostly adenomatous polyps) were detected in 23.5% of screened s

142 r high grade dysplasia or size > 1 cm or > 3 adenomatous polyps) were found in 98 cases (7 %), low ri

143 ed apoptosis, and suppressed angiogenesis in adenomatous polyps, which reduced both tumor number and