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1 and compared to control mice carry wildtype Adenomatous polyposis coli gene.
2 t is caused by inactivating mutations in the Adenomatous polyposis coli gene.
3 lon cancer cells that have a mutation in the adenomatous polyposis coli gene.
4 eficient mice carrying the Min allele of the adenomatous polyposis coli gene.
5 eted by human colon cancer cells with mutant adenomatous polyposis coli genes.
6 or the germ-line ApcMin mutation in the Apc (adenomatous polyposis coli) gene.
7 (FAP) and the site of mutations in the APC (adenomatous polyposis coli) gene.
8 sis is caused by a germ-line mutation in the adenomatous polyposis coli gene and is characterized by
9 iation domain family 1 gene RASSF1A, and the adenomatous polyposis coli gene APC in tumors and in his
10 e intestinal neoplasia (Min) mutation of the adenomatous polyposis coli gene (Apc) and homozygous for
11 adic colon adenomas acquire mutations in the adenomatous polyposis coli gene (APC) and show defects i
12 By using mice with germline mutations in the adenomatous polyposis coli gene (Apc) and/or DNA mismatc
14 point mutations in a 121 bp sequence of the adenomatous polyposis coli gene (APC) in human lymphobla
18 t human colorectal cancers, mutations in the adenomatous polyposis coli gene (APC) or CTNNB1 constitu
19 inoma but lacks an inherited mutation of the adenomatous polyposis coli gene (APC) that is associated
20 a segment polarity gene, patched (PTCH), the adenomatous polyposis coli gene (APC), beta-catenin, and
21 ue-1 (hMLH1), and the tumor suppressor genes adenomatous polyposis coli gene (APC), p53, and DPC4.
25 posis that have proximal 5' mutations of the adenomatous polyposis coli gene are more likely to have
27 g in T cell lineages by deletion of the Apc (adenomatous polyposis coli) gene causes spontaneous T ce
28 line mutation in a gene on chromosome 5 (the adenomatous polyposis coli gene) causes familial adenoma
30 olyposis because of germline mutation of the adenomatous polyposis coli gene is characterized by deve
33 ta-catenin degradation complex comprising of adenomatous polyposis coli gene product (APC) and serine
34 its other negative regulators, such as axin, adenomatous polyposis coli gene product (APC), and glyco
35 eta-catenin by inhibiting its binding to the adenomatous polyposis coli gene product and subsequent g
36 in, where carrier proteins like axin and the adenomatous polyposis coli gene product APC interact wit
40 ts from 7 families that had mutations in the adenomatous polyposis coli gene that were located toward
42 comparatively rare missense variants in the adenomatous polyposis coli gene, which is responsible fo
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