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1 DLD-1 colon carcinoma cells with the mutated adenomatous polyposis coli protein.
2  GI tumors in Mlh1 mice express little or no adenomatous polyposis coli protein.
3 ith the second PDZ domain of DLG, similar to adenomatous polyposis coli protein.
4      Most information about the roles of the adenomatous polyposis coli protein (APC) and its binding
5 at RNAi-mediated depletion of two kMAPs, the adenomatous polyposis coli protein (APC) and its binding
6 olding protein Axin and the tumor suppressor adenomatous polyposis coli protein (APC) are critical co
7                       We recently identified adenomatous polyposis coli protein (APC) as a key regula
8         The tumor suppressor function of the adenomatous polyposis coli protein (APC) depends, in par
9    Here we report that the tumour suppressor adenomatous polyposis coli protein (APC) directs the loc
10                                 Axin and the adenomatous polyposis coli protein (APC) interact to dow
11                              We propose that adenomatous polyposis coli protein (APC) is a key coordi
12                           Loss of functional adenomatous polyposis coli protein (APC) leads to uncont
13 , which is required for interaction with the adenomatous polyposis coli protein, bind and inhibit GSK
14 d beta-catenin requires interaction with the adenomatous polyposis coli protein but not with TCF for
15                   We had shown that the APC (adenomatous polyposis coli) protein controls localizatio
16 ixed disulfides with both beta4-spectrin and adenomatous polyposis coli protein in the cytosol.
17 otein EB1, which is a binding partner to the adenomatous polyposis coli protein involved in colon can
18 n from Mlp of a region similar to one in the adenomatous polyposis coli protein involved in EB1 bindi
19 ows that the actin-nucleating ability of the adenomatous polyposis coli protein is required for disas
20            Furthermore, in cells lacking the adenomatous polyposis coli protein required for beta-cat

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