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1 trial cysts, nabothian cysts, leiomyoma, and adenomyosis.
2 ia and hyperkeratosis, uterine inflammation, adenomyosis, and fibrosis, as well as oviductal smooth m
3 emale D2 receptor -/- mice developed uterine adenomyosis in response to prolonged prolactin exposure.
4 intraluminal blood, and two showed signs of adenomyosis, indicating functioning endometrial tissue;
6 t of the myometrium, these data suggest that adenomyosis may be caused primarily by defects in the fo
8 he groups that developed a high incidence of adenomyosis showed histological evidence of disturbed di
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