ライフサイエンスコーパス: adenopathy

1 There was no palpable axillary adenopathy.

2 dard of care for the sampling of mediastinal adenopathy.

3 nciclovir, can control symptoms and decrease adenopathy.

4 tumour in patients who present with axillary adenopathy.

5 rall survival (OS) among patients with bulky adenopathy.

6 inct), calcification, growth, and associated adenopathy.

7 or suspected lung carcinoma and mediastinal adenopathy.

8 ng patients with lung cancer and mediastinal adenopathy.

9 for pretracheal, hilar, and high pretracheal adenopathy.

10 t cancer in patients with malignant axillary adenopathy.

11 examination were known did not have palpable adenopathy.

12 ce of supraclavicular or contralateral hilar adenopathy.

13 pulmonary abnormalities (150 of 1558, 9.6%), adenopathy (103 of 1558, 6.6%), renal lesions (101 of 15

14 61%), and presented with massive mediastinal adenopathy (70%).

15 autoimmune disease but has little effect on adenopathy, administration of anti-CD40L to MRL-lpr mice

16 lso associated with the severity of inguinal adenopathy among men with hydrocele.

17 Additional findings included adenopathy and adrenal and pulmonary metastases.

18 ted in the 1st year of life with symptoms of adenopathy and hepatosplenomegaly.

19 Chest radiograph revealed hilar adenopathy and left pleural effusion; computed tomograph

20 ember 1997, 38 women with malignant axillary adenopathy and negative mammographic and physical examin

21 However, mediastinal adenopathy and pleural effusions were significantly more

22 apoptosis manifests as chronic, nonmalignant adenopathy and splenomegaly; the expansion of an unusual

23 cyte apoptosis permits chronic, nonmalignant adenopathy and splenomegaly; the survival of normally un

24 In the past, hilar adenopathy and/or mediastinal mass have been considered

25 bin <10 g/L, platelets <100 x 10(9)/L, bulky adenopathy and/or organomegaly, symptomatic hyperviscosi

26 s, syndromes 4 ("phobia-apraxia"), 5 ("fever-adenopathy"), and 6 ("weakness-incontinence") involved w

27 invasive breast cancer, no palpable axillary adenopathy, and 1 or 2 sentinel lymph nodes containing m

28 primary breast cancer, no palpable axillary adenopathy, and 1 or 2 sentinel lymph nodes containing m

29 al T1-T2 invasive breast cancer, no palpable adenopathy, and 1 to 2 SLNs containing metastases identi

30 m right upper lobe mass, without mediastinal adenopathy, and a 6-cm cystic mass in the spleen.

31 op severe hypergammaglobulinemia, lymph node adenopathy, and autoimmune disease.

32 s with fludarabine-refractory disease, bulky adenopathy, and del(17p) CLL.

33 pleural effusions, mediastinal and cervical adenopathy, and no gastrointestinal lesion.

34 uated for parenchymal opacities, cavitation, adenopathy, and pleural effusions.

35 infected patients with mediastinal and hilar adenopathy at Bellevue Hospital Center.

36 The adenopathy at US was hyper- and/or isoechoic relative to

37 as a key technique for sampling mediastinal adenopathy but variable yields are reported.

38 led well-resolved spikes in fever, rash, and adenopathy diagnoses, with the maximum incidence occurri

39 C) and 11 (92%) of 12 patients with palpable adenopathy experiencing at least a 50% reduction in the

40 compared to 12% of the wild type, developed adenopathy, hepatosplenomegaly, and circulating lymphobl

41 A pattern of spiculated axillary adenopathy in a patient with breast cancer appears to be

42 effective in the diagnosis of intrathoracic adenopathy in HIV-infected patients, and is particularly

43 On examination, she had adenopathy in the left axillary and supraclavicular regi

44 ted extraintestinal findings (eg, mesenteric adenopathy in two patients, perianal and enterocolic fis

45 tomography of the chest showing mediastinal adenopathy led to a presumptive diagnosis of inhalationa

46 galy, retroperitoneal adenopathy, periportal adenopathy, mesenteric adenopathy, thickening of the ome

47 brospinal fluid (CSF) pleocytosis, and hilar adenopathy more frequently than NMOSD (p < 0.05); CSF hy

48 nonseminomatous germ cell tumor (NSGCT) with adenopathy more than 2 cm, multiple masses, elevated ser

49 with periappendiceal fat stranding (n = 14), adenopathy (n = 6), appendolith(s) (n = 4), or fluid (n

50 ar consolidation (n = 3), and/or mediastinal adenopathy (n = 8).

51 Seventeen patients with metastatic cervical adenopathy of unknown primary origin were referred for F

52 2 WG registries to obtain evidence of hilar adenopathy or mediastinal masses.

53 latelet count less than 100 x 10(9)/L, bulky adenopathy or organomegaly, symptomatic hyperviscosity,

54 All of these patients had bulky adenopathy or organomegaly.

55 Mediastinal adenopathy or pleural effusions were classified as typic

56 tases were often associated with mediastinal adenopathy or suspected extrapulmonary nonnodal metastas

57 and neck and stage IV disease (N2 or N3 neck adenopathy) or recurrent local-regional disease and who

58 ally-defined resection margins and no portal adenopathy; other factors included presence of < or = 4

59 NS) vs MYD88(L265P)CXCR4(WT) presented with adenopathy (P < .01), further delineating differences in

60 of posterior cervical, inguinal or axillary adenopathy, palatine petechiae, splenomegaly, or atypica

61 sis were hepatosplenomegaly, retroperitoneal adenopathy, periportal adenopathy, mesenteric adenopathy

62 fever, aphthous stomatitis, pharyngitis, and adenopathy (PFAPA) syndrome.

63 ondary or other findings (mediastinal edema, adenopathy, pleural effusion, or a sternal or lung abnor

64 diographs demonstrated mediastinal widening, adenopathy, pleural effusions, and air-space disease.

65 3.1 [95% CI, 1.6-5.9]), inguinal or axillary adenopathy (specificity range, 0.82-0.91; positive LR ra

66 gs of lymphoproliferative disorders, such as adenopathy, splenomegaly, thymic enlargement, and hepato

67 ases with the presence of posterior cervical adenopathy (summary specificity, 0.87; positive LR, 3.1

68 atopoiesis with progressive splenomegaly and adenopathy, surviving only slightly longer than TGF-beta

69 denopathy, periportal adenopathy, mesenteric adenopathy, thickening of the omentum and the mesentery,

70 Adenopathy (two patients) and vascular invasion (one pat

71 ancreatic ductal dilatation, local invasion, adenopathy, vascular invasion, vascular encasement, meta

72 g patients with unresectable (N3) NSCLC when adenopathy was not present on CT imaging and appears to

73 gocytosis, histologic profile, age, sex, and adenopathy) were significantly associated with survival.

74 All patients had multifocal adenopathy, which was massive in some patients; 14 of 15

75 omography images showed enlarged mediastinal adenopathy with increased [(18)F]fluorodeoxyglucose upta

76 ion as LABC with ipsilateral supraclavicular adenopathy without evidence of distant disease, received

77 The adenopathy worsened despite withdrawal of immunosuppress