戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 and hydrolysis of ATP, cAMP, AMP, and NAD to adenosine.
2  MBF to the same level as a standard dose of adenosine.
3 te than resting indices and does not require adenosine.
4 with the 5' splice site and the branch-point adenosine.
5  on MBF due to hypercapnia was compared with adenosine.
6 nitive performance do not explicitly include adenosine.
7 hancing the production of the ATP derivative adenosine.
8  increase in the generation of extracellular adenosine.
9 A binding tunnel that typically contains the adenosine 2'OH of the RNA-cap moiety.
10 t requires the following: Gs-protein-coupled adenosine 2A receptor activation, new TrkB synthesis, an
11 tection from IRI through bone marrow-derived adenosine 2a receptors.
12 th pharmacological and genetic inhibition of adenosine-2A receptor (A2AR) in culture and Lrp4 mutant
13 endent activating transcription factor (ATF)-adenosine 3',5'-monophosphate (cAMP) response element-bi
14                                   The cyclic adenosine 3',5'-monophosphate receptor protein (CAP), a
15 displayed reduced aggregation in response to adenosine 5'-diphosphate and epinephrine, but variable a
16 numerous agonists (collagen-related peptide, adenosine 5'-diphosphate, A23187, thrombin, or U46619).
17 negative and positive allosteric regulators, adenosine 5'-monophosphate (AMP) and fructose 1,6-bispho
18 tivity was stimulated to a greater extent by adenosine 5'-O-(thiotriphosphate) (ATPgammaS) than with
19 of the sulfate reductive pathway key enzyme, adenosine 5'-phosphosulfate reductase (APR, EC 1.8.99.2)
20                                          The adenosine 6-minute infusion with rubidium-82 injection a
21 ins alter gene expression both by catalyzing adenosine (A) to inosine (I) RNA editing and binding to
22                                              Adenosine, a key extracellular signaling mediator, regul
23 ce of heterosynaptic plasticity; blockade of adenosine A1 receptors abolished it.
24 ncreases the concentrations of adenosine and adenosine A1 receptors in specific regions of the brain.
25 inoid receptors present in the striatum, ie, adenosine A2A receptor (A2AR) and cannabinoid CB1 recept
26 e describe the spontaneous reconstitution of adenosine A2A receptor (A2AR) during the de novo formati
27                                          The adenosine A2A receptor (A2AR) has long been implicated i
28 e reduced oxygen-induced neural apoptosis by adenosine A2A receptor (A2AR)-dependent mechanism, as re
29 sity and morphology, and upregulation of the adenosine A2A receptor (A2AR).
30                       Here, we show that the adenosine A2a receptor (ADORA2A) promotes hypoxia-induci
31 esulted in discovery of potent and selective adenosine A2A receptor antagonists bearing substituted 1
32 s- and Golf-coupled dopamine D1 receptor and adenosine A2A receptor in the brain and other organs, el
33                                 The cerebral adenosine A2A receptor is an attractive therapeutic targ
34 ntially therapeutic dopamine D1 receptor and adenosine A2A receptor ligands with functionally selecti
35 2 (CK2) negatively regulates dopamine D1 and adenosine A2A receptor signaling in the striatum.
36 ha), from dopamine D1 receptor-expressing or adenosine A2a receptor-expressing medium spiny neurons (
37 on of Th1 and Th2 cells in vitro depended on adenosine A2A receptors, which were also required for th
38 [2,1-f]purine-2,4-dione derivatives as human adenosine A3 receptor (hA3R) antagonists to determine th
39 ic state of this RNA shows guanosine G40 and adenosine A32 close to the G53-U54 cleavage site.
40                                              Adenosine/adenosine receptor-mediated signaling has been
41                   In this work, we show that adenosine (ADO), which is a potent endogenous modulator,
42 hod for determining specificity of synthetic adenosine agonist compounds for the A2A and A2B receptor
43 and allowing us to isolate the most specific adenosine agonist compounds.
44 ts, the stable 3-deaza-3-methoxynaphtylethyl-adenosine analog (3d-Napht-A), and smaller 3-deaza-adeno
45 ine analog (3d-Napht-A), and smaller 3-deaza-adenosine analogs, into DNA oligonucleotides to assess R
46  to test the hypothesis that changes in both adenosine and A1 receptor concentrations can capture cha
47 ttern implies that differences in endogenous adenosine and A1AR availability might be causal for indi
48 airments and increases the concentrations of adenosine and adenosine A1 receptors in specific regions
49                       Specifically, the main adenosine and endocannabinoid receptors present in the s
50                                              Adenosine and functional A1 adenosine receptor (A1AR) av
51 that generate similar voltammograms, such as adenosine and histamine.
52  Mean perfusion defect volumes measured with adenosine and hypercapnia were significantly correlated
53 te, adenosine monophosphate) and nucleoside (adenosine and inosine) levels were quantified by high-pe
54 with endogenous activators of PKA, including adenosine and prostaglandin E2, results in a profound de
55 the quantum yield of 8-DEA-tC mispaired with adenosine and, separately, opposite an abasic site show
56 ridine monophosphate, inosine monophosphate, adenosine, and guanosine) and kokumi (gamma-l-glutamyl-l
57 tinal tract and unveil an important role for adenosine as a regulator of host-microbe interactions.
58 sition for nucleophilic attack of the branch adenosine at the 5' splice site.
59 osition that overlaps with the native ligand adenosine, but its methoxyphenyl group extends to an exo
60                    This study tested whether adenosine caused prolonged asystole after transplantatio
61  subject, (3) stress perfusion and CFR after adenosine compared with dipyridamole, (4) heterogeneity
62 nes, and adenosine injection increased urine adenosine concentration and decreased urine pH.
63  Here we show that, apart from NAD+, another adenosine-containing cofactor FAD and highly abundant ur
64                                Deficiency of adenosine deaminase 2 (DADA2) is caused by biallelic del
65 e repair, we exploit the catalytic domain of Adenosine Deaminase Acting on RNA (ADAR2) that deaminate
66 AC)7 with the Zalpha-binding domain of human adenosine deaminase acting on RNA 1 (ADAR1, hZalphaADAR1
67 n directly inhibits editing, we do show that adenosine deaminase acting on RNA 1 and 2 and fibrillari
68 sine-to-inosine deaminase activity by ADAR2 (adenosine deaminase acting on RNA type 2) to transcripts
69                   In this study, we identify adenosine deaminase that acts on RNA 3 (ADAR3) as an imp
70 eic HSCT that has shown clinical benefit for adenosine deaminase-deficient (ADA-deficient) SCID when
71 o-inosine (A-to-I) RNA editing, catalyzed by Adenosine DeAminases acting on double-stranded RNA(dsRNA
72                           ADAR1 isoforms are adenosine deaminases that edit and destabilize double-st
73                                              Adenosine diphosphate (ADP) enhances platelet activation
74 ownregulate the adenosine triphosphate (ATP)/adenosine diphosphate (ADP) metabolite ratio which stron
75 sphate (ATP) and its first five catabolites: adenosine diphosphate (ADP), adenosine monophosphate (AM
76                                      Protein adenosine diphosphate (ADP)-ribosylation is a physiologi
77 e redox potential and adenosine triphosphate/adenosine diphosphate failed to reach a new steady state
78 uent use of radial access and novel platelet adenosine diphosphate P2Y12 receptor inhibitors.
79 2365 patients enrolled in the Treatment With Adenosine Diphosphate Receptor Inhibitors: Longitudinal
80                     It metabolizes NAD(+) to adenosine diphosphate ribose (ADPR) and cyclic ADPR, reg
81                             Nucleotide (ATP, adenosine diphosphate, adenosine monophosphate) and nucl
82           Soluble apyrase reduced renal ATP, adenosine diphosphate, and adenosine monophosphate, but
83            Cangrelor is a potent intravenous adenosine diphosphate-receptor antagonist that in the CH
84 r 1) prevents it from inhibiting PARP1 [poly(adenosine diphosphate-ribose) polymerase], a critical DN
85                  Prevention of generation of adenosine diphosphoribose in ECs significantly reduced C
86                        Overexpression of the adenosine diphosphoribose insensitive TRPM2 mutant chann
87 y in response to intracellular generation of adenosine diphosphoribose-the TRPM2 ligand.
88    The effects of patient characteristics on adenosine dose required to produce atrioventricular bloc
89 t characteristic significantly predicted the adenosine dose to produce atrioventricular block or dura
90 e mRNA level, a disease-causing guanosine to adenosine (G > A) mutation in the mouse MeCP2 DNA bindin
91     These data support the thesis that local adenosine generated by cells at the injury site is criti
92 denosine levels supported a critical role of adenosine generated by proximal tubule CD73 expression i
93                                Extracellular adenosine generated by the ectonucleotidases CD39 and CD
94 etin and its isomer, while its reaction with adenosine generated kinetin riboside, as confirmed by co
95 cleotidases that control ATP degradation and adenosine generation.
96                                 We show that adenosine harbors bacteriostatic activity against Salmon
97                                              Adenosine has been implicated in pathways that generate
98 ew versatile scaffold for the development of adenosine human (h) receptor antagonists.
99 aCO2) can increase MBF to that observed with adenosine in 3 groups of canines: without coronary steno
100 ding, respectively, and it was decoded as an adenosine in both systems.
101  the pro- and anti-viral effects of methyl-6-adenosine in distinct viral life cycles, the role of 2'
102 ression, reducing the level of intracellular adenosine in endothelial cells, and activating the trans
103 nce exists for the role of immunosuppressive adenosine in promoting tumor growth and spread in a numb
104  we define a novel antimicrobial activity of adenosine in the gastrointestinal tract and unveil an im
105       Therefore, we investigated the role of adenosine in the regulation of V-ATPase in ICs.
106                               Editing of one adenosine in the transcript encoding the glutamate recep
107 was a close agreement of maximal saline- and adenosine-induced coronary flow reserve (intraclass corr
108 ography scans using rubidium-82 with various adenosine infusion protocols identified (1) the protocol
109                                 The 6-minute adenosine infusion with rubidium-82 activation at 3 minu
110 significantly 15.7% higher than the 4-minute adenosine infusion with rubidium-82 injection at 2 minut
111 rs are located in A-IC apical membranes, and adenosine injection increased urine adenosine concentrat
112                                              Adenosine is a Food and Drug Administration-approved dru
113                                           As adenosine is known to cause changes in intracellular cal
114 ing the stem leads to destabilization unless adenosine is the opposing basepair of 8-oxoGua; (ii) mod
115 ucleotidase (CD73), an enzyme that generates adenosine, is functionally important in modifying CB sen
116 al CO2 (PaCO2), particularly with respect to adenosine, is not well characterized because of challeng
117  Here we show that concentrations of ATP and adenosine, its metabolite, fall after treatment of mouse
118                             In contrast, the adenosine kinase inhibitor, 5-iodotubericidin, which inh
119                           We found different adenosine kinase-inactivating (ADK-inactivating) alterat
120 We report that in healthy lowlanders, plasma adenosine levels are rapidly induced by initial ascent t
121 ent studies showed no change in total kidney adenosine levels in treated mice compared with vehicle-t
122 enzymatic activity of CD73 and extracellular adenosine levels supported a critical role of adenosine
123                                        Renal adenosine levels were lower before and after ischemia in
124 sphate, and adenosine monophosphate, but not adenosine levels, during ischemia.
125 stream metabolic changes, such as increasing adenosine levels, which might have effects on seizure co
126 report that methylation at the 6 position of adenosine (m(6)A) in RNA is rapidly (within 2 min) and t
127 on of a methyl group to the N(6) position of adenosine (m(6)A), has been shown to affect splicing, tr
128 ified by methylation of the N(6) position of adenosine (m(6)A).
129 '-deadenylation activity, suggesting that 3'-adenosine may occupy the ATP-binding pocket.
130                                           N6-adenosine methylation (m(6) A) of mRNA is an essential p
131                 We demonstrate that the N(6)-adenosine methyltransferase METTL16 regulates expression
132 6.0 Kcal/mole and for all of them the edited adenosines mis-paired with cytosines on the pre-miRNA st
133                            We speculate that adenosine modulation may provide a mechanism for fine-tu
134 otein 1 (PABP1) were covalently linked to an adenosine moiety as in the AdoMet cofactor to generate t
135 e nucleotide signaling molecule 3',5'-cyclic adenosine monophosphate (3',5'-cAMP) plays important phy
136 ve catabolites: adenosine diphosphate (ADP), adenosine monophosphate (AMP), inosine monophosphate (IM
137                              Cyclic di-3',5'-adenosine monophosphate (c-di-AMP) is a broadly conserve
138                                       Cyclic adenosine monophosphate (cAMP) and protein kinase A (PKA
139 udies exploring the importance of the cyclic adenosine monophosphate (cAMP) cascade in major depressi
140 bunit through formation of a PDE-PKAR-cyclic adenosine monophosphate (cAMP) complex (the termination
141 phodiesterase (PDE4) and elevation of cyclic adenosine monophosphate (cAMP) has emerged as a promisin
142                                       Cyclic adenosine monophosphate (cAMP) is an important mediator
143                         Intracellular cyclic adenosine monophosphate (cAMP) levels tune the voltage r
144  were treated with Angiopoietin 1 and cyclic adenosine monophosphate (cAMP) to vary the Pd of the HUV
145 f phenylalanine, acetylhistidine, and cyclic adenosine monophosphate (cAMP) were found in urine sampl
146 rmore, we demonstrated that Tregs use cyclic adenosine monophosphate (cAMP)-dependent protein kinase
147  chemotaxis towards emitted pulses of cyclic adenosine monophosphate (cAMP).
148 lutamatergic, monoaminergic, calcium, cyclic adenosine monophosphate [cAMP], dopamine- and cAMP-regul
149 culture and decidualized with 8-bromo-cyclic adenosine monophosphate and medroxyprogesterone acetate.
150                       We show that cyclic-di-adenosine monophosphate in live Gram-positive bacteria i
151 n of the cardiac stress marker NR4A1; cyclic adenosine monophosphate levels; and cyclic adenosine mon
152 ional activity and phosphorylation of cyclic adenosine monophosphate response element binding protein
153 ated the functional regulation of the cyclic adenosine monophosphate response element binding protein
154  promoter region as well as increased cyclic adenosine monophosphate response element-mediated transc
155      Nucleotide (ATP, adenosine diphosphate, adenosine monophosphate) and nucleoside (adenosine and i
156 educed renal ATP, adenosine diphosphate, and adenosine monophosphate, but not adenosine levels, durin
157                                           5'-Adenosine monophosphate-activated protein kinase (AMPK)
158  (PTEN) induces activation of the phospho-5' adenosine monophosphate-activated protein kinase and pho
159 adou et al. report that the metabolic sensor adenosine monophosphate-activated protein kinase influen
160 rectly targets the 3' untranslated region of adenosine monophosphate-activated protein kinase subunit
161 c adenosine monophosphate levels; and cyclic adenosine monophosphate-dependent protein kinase A-media
162 ivation of neuronal EP2 receptors and cyclic adenosine monophosphate-dependent protein kinase.
163 m of ICAM-4 activation occurs via the cyclic adenosine monophosphate-protein kinase A (cAMP-PKA)-depe
164     GS-5734 is a monophosphate prodrug of an adenosine nucleoside analog that showed therapeutic effi
165 but very little is known about the effect of adenosine on IC function.
166           Intravenous treatment of mice with adenosine or agonists of ADORA2A and ADORA2B purinergic
167 xes containing AK and each of four different adenosine phosphate ligands.
168                                 CD73-derived adenosine plays an anti-inflammatory role in various org
169 sults suggest that controlling access to the adenosine pocket can contribute to substrate specificity
170 iprocal alterations were made to enlarge the adenosine pocket of EhACK and reduce that of MtACK.
171 njection of liposomal suspensions containing adenosine prevents development of OA in rats.
172 investigate the contribution of CD73-derived adenosine produced by T cells to cardiac remodeling afte
173 uced immunosuppressive mechanisms, including adenosine production.
174 with the 3/6-minute protocol, the 2/4-minute adenosine protocol would potentially have changed 332 of
175                  Adenosine and functional A1 adenosine receptor (A1AR) availability are supposed to m
176 of compound 25 as a potent and selective A2A adenosine receptor (A2AAdoR) antagonist with reasonable
177 MR) spectroscopy with the wild-type-like A2A adenosine receptor (A2AAR) in solution provides a compre
178         Here, we show that engagement of A2A adenosine receptor (A2AR) acts as a checkpoint that limi
179   The cAMP-elevating, Gs protein-coupled A2a adenosine receptor (A2aR) is an evolutionarily conserved
180 overexpress mRNA for dopamine receptor 2 and adenosine receptor 2a in the striatum, markers of medium
181                   Here the authors show that adenosine receptor A2A drives pathological angiogenesis
182 ry synapses from basolateral amygdala via A1 adenosine receptor activation and enhanced inhibitory sy
183 wild-type levels, suggesting a dependence on adenosine receptor activity.
184    The effect of inosine was mimicked by the adenosine receptor agonist NECA and the A2B receptor ago
185 le of enantiospecific recognition at the A2B adenosine receptor and opens new possibilities in ligand
186 ith the synthesis of an A2-subtype selective adenosine receptor antagonist in only two steps.
187 Caffeine (5, 10, or 15 mg/kg), a nonspecific adenosine receptor antagonist, dose-dependently and at h
188 n against OIR, effective therapeutic window, adenosine receptor mechanisms, and neuroglial involvemen
189  of BRAF and MEK in combination with the A2A adenosine receptor provided significant protection again
190                           Stimulation of the adenosine receptor subtype A2B increases the gap junctio
191 ge, new cholesterol-binding sites on the A2A adenosine receptor, a G-protein-coupled receptor that is
192 MD/FEP) in fragment optimization for the A2A adenosine receptor, a pharmaceutically relevant G protei
193 at integrates gap junction coupling into the adenosine receptor-dependent signalling of endothelial c
194 ulfur phasing of the human G protein-coupled adenosine receptor.
195 tent binding across therapeutically relevant adenosine receptors (A1, A2a, and A3) as well as a poten
196 to G protein-coupled receptors including the adenosine receptors (AR), which are involved in a pletho
197 -coupled receptors, the A1, A2A, A2B, and A3 adenosine receptors (ARs).
198  characterize the physiological link between adenosine receptors and the gap junction coupling in end
199                        Because each of these adenosine receptors plays a distinct role throughout the
200 These effects were mimicked by antagonism of adenosine receptors with 8-(p-sulfophenyl) theophylline.
201 or), opioid receptors, adrenergic receptors, adenosine receptors, dopamine receptor, and sphingosine
202                            Here we show that adenosine regulates weight dependence of heterosynaptic
203 e, poxviruses remodel host ribosomes so that adenosine repeats erroneously generated by slippage of t
204 As with 5' untranslated regions that contain adenosine repeats, so-called polyA-leaders.
205                                              Adenosine replacement by intra-articular injection of li
206 , modulation of heterosynaptic plasticity by adenosine represents an endogenous mechanism that transl
207                   2'-O-ribose-methylation of adenosine residues, however, has been shown to inhibit a
208 promoting neuromodulators, noradrenaline and adenosine, respectively.
209 y is active in melanoma patients and whether adenosine restricts the efficacy of clinically approved
210 t within the GC facilitates an extracellular adenosine-rich milieu, which serves to limit TFH frequen
211 ng ADORA2B have been implicated in mediating adenosine's effects in IPF.
212 ogether, our results establish A2AR-mediated adenosine signaling as an intrinsic negative regulator o
213              Previous work demonstrated that adenosine signaling in IECs results in a number of tissu
214 ed antitumor immunity.Significance: Ablating adenosine signaling is found to promote natural killer c
215 is promoted by a cross-talk between HA, CD73/adenosine signaling, and other profibrotic mediators.
216 nforcement signals, mediated by dopamine and adenosine signaling.
217                                              Adenosine signalling via erythrocyte ADORA2B induces PKA
218 ticularly hydrogen peroxide (H2O2), impaired adenosine stimulated wound repair.
219 ght dependence of heterosynaptic plasticity: adenosine strengthened weight dependence of heterosynapt
220                                    Different adenosine stress imaging protocols have not been systemi
221  were compared with those obtained during an adenosine stress test (140 mug/kg/min).
222 aluation of stable angina symptoms underwent adenosine-stress dynamic computed tomography myocardial
223 ter in a pocket surrounding the branch point adenosine, suggesting a competitive mode of action.
224 e fold but remarkably, unlike tuberculosinol adenosine synthase and other cis-prenyl transferases (e.
225 veloped a novel receptor-ligand model of the adenosine system to test the hypothesis that changes in
226                                              Adenosine, the standard therapy for treating supraventri
227 hows that medullary A-ICs respond to luminal adenosine through ADORA2A and ADORA2B receptors in a cAM
228 deletion allows rapid accumulation of plasma adenosine to counteract hypoxic tissue damage in mice.
229 re required for paraspeckles, as well as for adenosine to inosine (A to I) RNA editing of Ctn RNA in
230                                              Adenosine to Inosine (A-to-I) RNA editing is a co- or po
231                                Conversion of adenosine to inosine is a frequent type of RNA editing,
232  and innate immune sensing, and functions of adenosine to inosine modifications in retroviral life cy
233                                              Adenosine-to-inosine (A-to-I) editing of dsRNA by ADAR p
234 of The Cancer Genome Atlas and identified 19 adenosine-to-inosine (A-to-I) RNA editing hotspots.
235                                              Adenosine-to-inosine (A-to-I) RNA editing is a conserved
236                                              Adenosine-to-inosine (A-to-I) RNA editing, catalyzed by
237 alytically inactive Cas13 (dCas13) to direct adenosine-to-inosine deaminase activity by ADAR2 (adenos
238 igate whether angiomiR miR487b is subject to adenosine-to-inosine editing or 2'-O-ribose-methylation
239 residues, however, has been shown to inhibit adenosine-to-inosine editing.
240                                        Since adenosine-to-inosine RNA editing has recently emerged as
241                                              Adenosine-to-inosine RNA editing in transcripts encoding
242 d inoFISH to directly visualize and quantify adenosine-to-inosine-edited transcripts in situ.
243 es adenosine tone) and brain state (elevated adenosine tone increases sleep pressure), modulation of
244                                      Because adenosine tone is a natural correlate of activity level
245 relate of activity level (activity increases adenosine tone) and brain state (elevated adenosine tone
246 c environment in the catalytic space of gp17-adenosine triphosphatase (ATPase) determines the rate at
247 y component of the system is the proteasomal adenosine triphosphatase (ATPase) Mpa, which captures, u
248 necessary for packaging in such viruses: the adenosine triphosphatase (ATPase) that powers DNA transl
249    Sarcoplasmic/endoplasmic reticulum Ca(2+) adenosine triphosphatase (SERCA)2a, a critical regulator
250 tion through inhibition of the vacuolar H(+)-adenosine triphosphatase (V-ATPase) increased the lumina
251 lectron microscopy structure reveals how the adenosine triphosphatases (ATPases) form a closed spiral
252 es are conserved members of the AAA+ family (adenosine triphosphatases associated with diverse cellul
253 a key purinergic enzyme in the hydrolysis of adenosine triphosphate (ATP) and increased CD39 enzymati
254 and accurate method for the determination of adenosine triphosphate (ATP) and its first five cataboli
255 sly showed that patients with BD show normal adenosine triphosphate (ATP) and phosphocreatine levels
256 es on host cells for essential nutrients and adenosine triphosphate (ATP) for a productive infection.
257                           Here, we show that adenosine triphosphate (ATP) has properties of a biologi
258     METHOD: Mitochondrial O2 consumption and adenosine triphosphate (ATP) synthesis rates of osteosar
259 es have demonstrated that in the presence of adenosine triphosphate (ATP) the human RAD51 (HsRAD51) r
260 lectrostatic lock to prevent coordination of adenosine triphosphate (ATP) to the catalytic site.
261                           ABCB4 (MDR3) is an adenosine triphosphate (ATP)-binding cassette (ABC) tran
262 re harboring disease-causing variants in the adenosine triphosphate (ATP)-binding cassette subfamily
263 plex loaded onto DNA directly interacts with adenosine triphosphate (ATP)-bound DnaA and stimulates t
264 al part of the chromatin landscape shaped by adenosine triphosphate (ATP)-dependent chromatin remodel
265 nt study, H2O2 dose-dependently impaired the adenosine triphosphate (ATP)-induced Ca(2+) response, wh
266 fically, ketamine tended to downregulate the adenosine triphosphate (ATP)/adenosine diphosphate (ADP)
267 ic machinery for hydrolysis of extracellular adenosine triphosphate and nicotinamide adenine dinucleo
268 xylic acids to aldehydes using the cofactors adenosine triphosphate and nicotinamide adenine dinucleo
269 r neurons-acetylcholine and a combination of adenosine triphosphate and nitric oxide, respectively.
270 l lumen via the sterol-exporting heterodimer adenosine triphosphate binding cassette subfamily G memb
271                 Sperm motility is powered by adenosine triphosphate but the relative importance of la
272 pressed in liver facilitating the release of adenosine triphosphate from hepatocytes.
273 that adopts two distinct folds, and the post-adenosine triphosphate hydrolysis state of KaiC create a
274 ae condensin is a molecular motor capable of adenosine triphosphate hydrolysis-dependent translocatio
275 asmid segregation events by stimulating ParF adenosine triphosphate hydrolysis.
276                Within the liver vasculature, adenosine triphosphate is converted into pyrophosphate,
277 diac energetic status (phosphocreatine/gamma-adenosine triphosphate ratio, 1.3+/-0.1 versus 1.9+/-0.1
278 ly discovered that absence of ABCC6-mediated adenosine triphosphate release from the liver and conseq
279 gnificantly impairs platelet aggregation and adenosine triphosphate secretion induced by numerous ago
280  and IL18; cells were incubated with LPS and adenosine triphosphate to activate the NLRP3 complex.
281 e leaving group of the native substrate with adenosine triphosphate, enabling sensitive detection via
282     By characterization of a Petunia hybrida adenosine triphosphate-binding cassette (ABC) transporte
283 in; KCNN4, the Gardos channel; and ABCB6, an adenosine triphosphate-binding cassette family member, i
284  JNK1 or JNK2 or treatment with JNK-IN-8, an adenosine triphosphate-competitive irreversible pan-JNK
285 d threaded through the helicase domain in an adenosine triphosphate-dependent manner.
286 eltapsi-driven presequence translocation and adenosine triphosphate-driven import motor activity.
287  host crucial metabolic pathways and produce adenosine triphosphate.
288 ies on oxidative phosphorylation to generate adenosine triphosphate.
289 ide adenine dinucleotide redox potential and adenosine triphosphate/adenosine diphosphate failed to r
290 that DDX3X, like Ded1p, utilizes exclusively adenosine triphosphates to unwind helices, oligomerizes
291 pha1-, and alpha2- adrenergic receptors, and adenosine type 1 receptor (A1R), tethered to Galphas-XL,
292  gene HP1522 in strain 26695, encodes a N(6)-adenosine type III DNA methyltransferase.
293  an isothiazolo[4,3-d]pyrimidine analogue of adenosine ((tz) A), was developed.
294 hways converging in the formation of AMP and adenosine via CD73.
295 der hypercapnia was 2.1 +/- 0.9 mL/min/g and adenosine was 2.2 +/- 1.1 mL/min/g; these were significa
296 ified with carboxymethyl-cytosine instead of adenosine was described, but no biological activity of t
297 a transvenous pacing catheter was placed and adenosine was given following a dose-escalation protocol
298              MBF after the administration of adenosine was significantly greater than that with the m
299 ly significant asystole (>/=12 seconds after adenosine) was quantified.
300 munostimulatory ATP versus immunosuppressive adenosine within the tumor microenvironment.

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。
 
Page Top