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1 and was augmented after administration of an adenosine antagonist.
2 y provide leads for the development of novel adenosine antagonists.
3 s for the development of novel, selective A3 adenosine antagonists.
4 f the potent and highly selective racemic A1-adenosine antagonist 1,3-dipropyl-8-[2-(5,6-epoxynorborn
5 After reinstatement, application of the A1 adenosine antagonist 1,3-dipropyl-8-cyclopentylxanthine
6 IBMX; phosphodiesterase type 5 inhibitor and adenosine antagonist, 10 micromol/L) and phorbol myrista
7 aken together, our findings suggest that the adenosine antagonist 125l-3-(4-azidophenethyl)-1-propyl-
9 Group 1 received a 10-min infusion of the adenosine antagonist 8-sulfophenyltheophylline (0.9 mg/k
12 as increase in affinity for the nonxanthine adenosine antagonist 9-chloro-2-(furyl)[1,2,4]triazolo[1
13 strains also differed in the ability of the adenosine antagonist caffeine (50 mg/kg, i.p.) to prolon
15 ade of A1 adenosine receptor action with the adenosine antagonist caffeine can prevent hypoxia-induce
16 havioral synergy observed following combined adenosine antagonist-D1 dopamine agonist and combined D1
17 onist ZM 241385 (10 nmol/L) but not the A(1) adenosine antagonist DPCPX (5 micromol/L) abolished the
18 gents, including vasopressin antagonists and adenosine antagonists, hold promise for the future, and
20 ctural features of xanthine and non-xanthine adenosine antagonists to BTH4 (7) suggests a high degree
21 llyl-8-cyclohexylxanthine (DAX) are xanthine adenosine antagonists which activate chloride efflux fro
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