ライフサイエンスコーパス: adenosine triphosphatase

1 t the KIF5-SNPH coupling inhibited the motor adenosine triphosphatase.

2 ccurs on the level of the vacuolar-type H(+)-adenosine triphosphatase.

3 the proton pumping enzyme vacuolar-type H(+)-adenosine triphosphatase.

4 n ATP7B, which encodes a copper-transporting adenosine triphosphatase.

5 pression of cell surface-associated vacuolar adenosine triphosphatases.

6 requires Vps4, a member of the AAA family of adenosine triphosphatases.

7 uses phosphorylation of plasma membrane H(+)-adenosine triphosphatase 2 at Ser(899), mediating the in

8 ession of a critical proteasome subunit, non-adenosine triphosphatase 4 (PSMD4), was reduced in old m

9 ntermediates (AIs) that contain the cellular adenosine triphosphatase ABCE1 (ATP-binding cassette pro

10 ATP-binding cassette (ABC) adenosine triphosphatases actively transport a wide vari

11 Observations on the adenosine triphosphatase activities and protease suscept

12 Motor adenosine triphosphatase activities and their associatio

13 that reduces contractility by decreasing the adenosine triphosphatase activity of the cardiac myosin

14 y interfered with the microtubule-stimulated adenosine triphosphatase activity of the kinesin motor,

15 A lack of VPS4 adenosine triphosphatase activity reduced budding by up

16 dehydrogenase, inhibition of membrane Na /K adenosine triphosphatase activity, inactivation of membr

17 ) generated by microtubule-activated kinesin adenosine triphosphatase activity.

18 ssed for Na/K/2Cl cotransporter and Na+,K(+)-adenosine triphosphatase activity.

19 stimulated its double-stranded DNA-dependent adenosine triphosphatase activity.

20 ori represses expression of the gastric H, K-adenosine triphosphatase alpha-subunit (HKalpha), which

21 on markers including GATA4, GATA6, and H+/K+-adenosine triphosphatase and abnormal expression of memb

22 fect the distribution and abundance of the P-adenosine triphosphatase and Pinformed 1 auxin efflux fa

23 taining of the cells both with anti-H(+)K(+)-adenosine triphosphatase antibodies and with Texas Red-l

24 olated flagella when exogenous ubiquitin and adenosine triphosphatase are added, suggesting that the

25 es are conserved members of the AAA+ family (adenosine triphosphatases associated with diverse cellul

26 uride's cyclohexylurea group, which binds to adenosine triphosphatase (ATP)-sensitive K(+) (K(ATP)) c

27 d H(+) secretion by the nongastric H(+)/K(+) adenosine triphosphatase (ATP12A) acidified airway surfa

28 We found that the vacuolar H(+)-adenosine triphosphatase ATPase (v-ATPase) is necessary

29 and dissociation between the electron donor adenosine triphosphatase (ATPase) (Fe-protein) and its t

30 The vesicular adenosine triphosphatase (ATPase) acidifies intracellula

31 /X comprised of the Mre11 nuclease and Rad50 adenosine triphosphatase (ATPase) active sites dimerizes

32 n acridine orange fluorescence and H(+),K(+)-adenosine triphosphatase (ATPase) activity in isolated t

33 se IV substrate inactivated bile canalicular adenosine triphosphatase (ATPase) activity in normal but

34 Na,K-adenosine triphosphatase (ATPase) activity is elevated i

35 gold electron microscopy and kinetics of the adenosine triphosphatase (ATPase) activity of purified R

36 -expressing cells, stimulates Pgp-associated adenosine triphosphatase (ATPase) activity, and causes c

37 of IFN-gamma on T84 barrier function, Na+,K+-adenosine triphosphatase (ATPase) activity, and certain

38 ion) and displays low microtubule-stimulated adenosine triphosphatase (ATPase) activity.

39 l-permeable, specific inhibitor of myosin II adenosine triphosphatase (ATPase) activity.

40 nique relative orientation of the N-terminal adenosine triphosphatase (ATPase) and C-terminal nucleas

41 es of 60 and 130 nM in microtubule-dependent adenosine triphosphatase (ATPase) and cell-based cytotox

42 sin heavy chain, sarcoplasmic reticulum Ca2+-adenosine triphosphatase (ATPase) and Na+-Ca2+ exchanger

43 ant baculovirus, possesses RNA/DNA helicase, adenosine triphosphatase (ATPase) and single-stranded (s

44 pic properties related to inhibition of Na/K adenosine triphosphatase (ATPase) and stimulation of sar

45 t of SpoIIIE was shown to be a DNA-dependent adenosine triphosphatase (ATPase) capable of tracking al

46 Here we present evidence that the adenosine triphosphatase (ATPase) cycle of the SF1 helic

47 c environment in the catalytic space of gp17-adenosine triphosphatase (ATPase) determines the rate at

48 we functionally characterized the DExD/H box adenosine triphosphatase (ATPase) Dhh1, a critical regul

49 MTBD) is separated from its ring-shaped AAA+ adenosine triphosphatase (ATPase) domain by a 15-nanomet

50 ide exchange factor GrpE in complex with the adenosine triphosphatase (ATPase) domain of Escherichia

51 members of the conserved Microrchidia (MORC) adenosine triphosphatase (ATPase) family, which are pred

52 are covalent inhibitors of the gastric H+,K+-adenosine triphosphatase (ATPase) forming disulfide bond

53 We examined whether ITP can be used by adenosine triphosphatase (ATPase) in human erythrocytes

54 The enzyme F1-adenosine triphosphatase (ATPase) is a molecular motor t

55 ss animal species and cell types, Na(+),K(+)-adenosine triphosphatase (ATPase) is arguably the most p

56 Na,K-adenosine triphosphatase (ATPase) is essential for the r

57 Because Na,K-adenosine triphosphatase (ATPase) is involved in aqueous

58 ntified the chromatin-remodeling nucleosomal adenosine triphosphatase (ATPase) ISWI as a key molecule

59 The SecA adenosine triphosphatase (ATPase) mediates extrusion of

60 hitecture of MetNI reveals two copies of the adenosine triphosphatase (ATPase) MetN in complex with t

61 y component of the system is the proteasomal adenosine triphosphatase (ATPase) Mpa, which captures, u

62 The H,K-adenosine triphosphatase (ATPase) of gastric parietal ce

63 i release (corresponding to the steady-state adenosine triphosphatase (ATPase) rate of actomyosin (AM

64 with acto-myosin function through the myosin adenosine triphosphatase (ATPase) reaction; 1-(5-isoquin

65 Prp5 protein (Prp5p) is an RNA-stimulated adenosine triphosphatase (ATPase) required for presplice

66 Mutations in the AAA adenosine triphosphatase (ATPase) Spastin (SPG4) cause a

67 Katanin, a member of the AAA adenosine triphosphatase (ATPase) superfamily, uses nucl

68 SF (N-ethylmaleimide-sensitive factor) is an adenosine triphosphatase (ATPase) that contributes to a

69 p97 is a hexameric AAA+ adenosine triphosphatase (ATPase) that is an attractive

70 necessary for packaging in such viruses: the adenosine triphosphatase (ATPase) that powers DNA transl

71 ctivity of the hepatocyte basolateral Na+,K+-adenosine triphosphatase (ATPase) was also decreased by

72 1) Hsp90 facilitates both recruitment of the adenosine triphosphatase (ATPase)-activating cochaperone

73 ombin significantly reduces the rate of Na,K-adenosine triphosphatase (ATPase)-mediated ion transport

74 ereas Kar2p reciprocally activated the Lhs1p adenosine triphosphatase (ATPase).

75 rboxy terminal two thirds is comprised of an adenosine triphosphatase (ATPase)/helicase domain.

76 ium iodide symporter, and hydrogen potassium adenosine triphosphatase [ATPase]) showed reduced expres

77 lectron microscopy structure reveals how the adenosine triphosphatases (ATPases) form a closed spiral

78 In eukaryotes, P-type adenosine triphosphatases (ATPases) generate the plasma

79 DNA-dependent adenosine triphosphatases (ATPases) participate in a bro

80 previously unrecognized subfamily of P-type adenosine triphosphatases (ATPases) that may have diverg

81 volved from ring-shaped hexameric AAA-family adenosine triphosphatases (ATPases), dynein's large size

82 member of the microrchidia (MORC) family of adenosine triphosphatases (ATPases), has been shown to b

83 , an inhibitor of endoplasmic reticulum Ca2+-adenosine triphosphatases (ATPases).

84 er complexes belonging to the AAA+ family of adenosine triphosphatases (ATPases).

85 Here we show that rotary adenosine triphosphatases (ATPases)/synthases from Therm

86 d a peptide cleaved from a2 isoform vacuolar adenosine triphosphatase called a2NTD.

87 report that in living cells the cytoplasmic adenosine triphosphatase called ClpV specifically recogn

88 ffinity catalytic site 1 of chloroplast F(1) adenosine triphosphatase (CF(1) ATPase) were characteriz

89 al outer membrane by the p97/Cdc48-Ufd1-Npl4 adenosine triphosphatase complex is essential for mitoch

90 s and the neuron-specific proton pump and V0 adenosine triphosphatase component V100.

91 nteraction of BRG1-also known as SMARCA4, an adenosine triphosphatase-containing chromatin remodeler-

92 RCA (sarcoplasmic-endoplasmic reticulum Ca2+ adenosine triphosphatase) corrected [Ca2+]er and mitocho

93 ports of membrane-bound, copper-transporting adenosine triphosphatases (Cu-ATPases) selective for cop

94 e catalysis on membranes and suppress futile adenosine triphosphatase cycles.

95 an inhibitor of the vacuolar proton-pumping adenosine triphosphatase, cytosolic calcein fluorescence

96 on supported Trio-dependent Rac1 activation, adenosine triphosphatase-deficient Hsc70 (D10N) abrogate

97 imulate, in a Rag-, Ragulator-, and vacuolar adenosine triphosphatase-dependent fashion, the transloc

98 tide that is essential for stimulating HSP70 adenosine triphosphatase diverges in candidate orthologu

99 that the conserved Swc2/YL1 subunit and the adenosine triphosphatase domain of Swr1 are mainly respo

100 ured regions within Hebo: a TUDOR domain, an adenosine triphosphatase domain, and a new domain, HEBO,

101 tone acetyltransferase dTip60 as well as the adenosine triphosphatase Domino/p400 catalyze the exchan

102 examined the expression of canalicular ecto-adenosine triphosphatase (ecto-ATPase) and mdr P-glycopr

103 d cells have implicated the canalicular ecto-adenosine triphosphatase (ecto-ATPase) in adenosine trip

104 x, which appears to load/unload RuvBL AAA(+) adenosine triphosphatase from pre-snoRNPs; and (d) a pot

105 riggers translocation of the proton pump, HK-adenosine triphosphatase, from cytoplasmic tubulovesicle

106 of fusiform cells, and it inhibited H(+)K(+)-adenosine triphosphatase gene expression.

107 al transformation and inhibition of H + K + -adenosine triphosphatase gene expression.

108 th factor for 7-16 hours stimulates H(+)K(+)-adenosine triphosphatase gene expression.

109 caused by mutations in a copper-transporting adenosine triphosphatase gene, ATP7A.

110 le with the coiled coil emerging from Smc3's adenosine triphosphatase head.

111 eport that the Caenorhabditis elegans P-type adenosine triphosphatase homolog, TAT-1, is critical for

112 Studies examining the development of H, K-adenosine triphosphatase in infants and the role of ente

113 riphosphate through the action of Na(+)/K(+) adenosine triphosphatases in an integrated in vitro lipi

114 s in autophagy, we silenced VCP or expressed adenosine triphosphatase-inactive VCP.

115 hibition was combined with the vacuolar H(+)-adenosine triphosphatase inhibitor bafilomycin A1 or wit

116 adenosine 5'-triphosphate and ARL-67156, an adenosine triphosphatase inhibitor, and were attenuated

117 The use of MLC kinase (MLCK) and myosin adenosine triphosphatase inhibitors led us to propose a

118 Proton pump (H(+)/K(+)-adenosine triphosphatase) inhibitors (PPIs) are widely u

119 The a2 isoform vacuolar adenosine triphosphatase is found on the surface on many

120 We have found that Swr1, a Swi2/Snf2-related adenosine triphosphatase, is the catalytic core of a mul

121 Genetic differences in sodium-potassium adenosine triphosphatase (Na(+)-K(+)ATPase) could explai

122 ly believed that sodium, potassium-activated adenosine triphosphatase (Na+, K+-ATPase) is localized o

123 he vacuolar-type (V-type) sodium ion-pumping adenosine triphosphatase (Na+-ATPase) from Enterococcus

124 hyperplasia with increased sodium-potassium-adenosine triphosphatase (Na,K-ATPase) activity, attenua

125 ty of lens epithelium to synthesize new Na,K-adenosine triphosphatase (Na,K-ATPase) catalytic subunit

126 The rate of Na,K-adenosine triphosphatase (Na,K-ATPase) dependent potassi

127 Sodium and potassium-exchanging adenosine triphosphatase (Na,K-ATPase) in the kidney is

128 + channels, the basolateral sodium potassium-adenosine triphosphatase (Na,K-ATPase), and possibly chl

129 Inhibitors of sodium-potassium-activated adenosine triphosphatase (Na-K-ATPase) have been implica

130 reviously reported that the sodium potassium adenosine triphosphatase (Na/K-ATPase) can effect the am

131 that the alpha1 subunit of sodium potassium adenosine triphosphatase (Na/K-ATPase), acts as a recept

132 ular bile acid transporter Ca2+, Mg(2+)-ecto-adenosine triphosphatase, now facilitates such studies.

133 cytochrome c oxidase, inhibition of F(1)F(0) adenosine triphosphatase, or replacement of all mtDNA-en

134 ligase Hrd1, its partner Sel1, the cytosolic adenosine triphosphatase p97, and degradation by the pro

135 Many members of the SWI2/SNF2 family of adenosine triphosphatases participate in the assembly/di

136 eased expression of the plasma membrane Ca2+-adenosine triphosphatase (PMCA) efflux pump.

137 isk-like structures through a unidirectional adenosine triphosphatase polymerization, primed with a s

138 inding portion binds the polypeptide, and an adenosine triphosphatase portion facilitates substrate e

139 H(+)K(+)-adenosine triphosphatase protein and gene expression wer

140 portional disassembly of both ezrin and Na/K adenosine triphosphatase proteins from their cytoskeleta

141 nts are irreversible inhibitors of the H+/K+ adenosine triphosphatase proton pump.

142 ion at the final common pathway of the H+/K+ adenosine triphosphatase proton pump.

143 ulation of the sarcoplasmic reticulum Ca(2+)-adenosine triphosphatase pump and by augmented levels of

144 Curcumin is a nontoxic Ca-adenosine triphosphatase pump inhibitor that can be admi

145 omerization preceding it may be limiting the adenosine triphosphatase rate.

146 ion and canalicular localization of the ecto-adenosine triphosphatase remained unchanged.

147 mic reticulum (SR) and reuptake by the Ca(2+)adenosine triphosphatase SERCA.

148 Abundance of sarcoplasmic reticulum Ca(2+) adenosine triphosphatase (SERCA), Na(+)/Ca(2+) exchanger

149 Sarcoplasmic/endoplasmic reticulum Ca(2+) adenosine triphosphatase (SERCA)2a, a critical regulator

150 bits the cardiac sarcoplasmic reticular Ca2+-adenosine triphosphatase (SERCA2a) pump.

151 sion of sarcoplasmic reticulum Ca(2+) uptake adenosine triphosphatase (SERCA2a), phospholamban (PLB),

152 to be catalyzed by katanin, a heterodimeric adenosine triphosphatase that can remove tubulin subunit

153 Mtr4 is an essential RNA-dependent adenosine triphosphatase that is required for all of the

154 om pH 7 to 3, and the gene encoding a P-type adenosine triphosphatase that may catalyze NH4+/H+ excha

155 d in WD, encodes a multitransmembrane domain adenosine triphosphatase that traffics from the trans-Go

156 ted the effect of directly inhibiting myosin adenosine triphosphatase using 2,3-butanedione monoxime

157 protons, together with reduced vacuolar H(+)-adenosine triphosphatase (V-ATPase) activity, accounts f

158 ysosomes, leading to the removal of vacuolar adenosine triphosphatase (V-ATPase) and the neutralizati

159 et was enriched for subunits of the vacuolar adenosine triphosphatase (V-ATPase) complex, a proton pu

160 tion through inhibition of the vacuolar H(+)-adenosine triphosphatase (V-ATPase) increased the lumina

161 The vesicular adenosine triphosphatase (v-ATPase) is a proton pump tha

162 that the V0 domain of the vacuolar-type H(+)-adenosine triphosphatase (V-ATPase) is directly implicat

163 , lysosomes never recycle vacuolar-type H(+)-adenosine triphosphatase (V-ATPase) or neutralize to for

164 g sites of the vacuolar proton-translocating adenosine triphosphatase (V-ATPase), cysteine scanning m

165 the Ragulator complex, and the vacuolar H(+)-adenosine triphosphatase (v-ATPase).

166 We show that the neuronal vacuolar-type H(+)-adenosine triphosphatase V0 subunit a1 (V100) can regula

167 Binding to the AAA adenosine triphosphatase valosin-containing protein (VCP

168 The AAA-adenosine triphosphatase Vps4 disassembles and recycles

169 utant deficient in a presumptive proteasomal adenosine triphosphatase was attenuated in mice, and exp

170 ndent functions of human copper-transporting adenosine triphosphatases (Wilson's and Menkes disease p