ライフサイエンスコーパス: adenovirus infection

1 e in calpain protein was observed after LacZ adenovirus infection.

2 artments with no effect on the efficiency of adenovirus infection.

3 ive treatment, if possible, in patients with adenovirus infection.

4 n on cell surfaces is required for efficient adenovirus infection.

5 be important in cutaneous wound healing and adenovirus infection.

6 in three of four woodchucks at 2 weeks after adenovirus infection.

7 er to cells that are relatively resistant to adenovirus infection.

8 involved in the induction of JNK activity by adenovirus infection.

9 c cells proceed with a lytic or a persistent adenovirus infection.

10 ne expression in cells that are resistant to adenovirus infection.

11 etically normal cells outside the context of adenovirus infection.

12 ned the activation of this pathway following adenovirus infection.

13 hich were null for both alleles of p53, upon adenovirus infection.

14 s, but not IgG antibody molecules, inhibited adenovirus infection.

15 roducing cells in mouse liver within 24 h of adenovirus infection.

16 normally inhibits DNA replication following Adenovirus infection.

17 HeLa cells, which are commonly used to study adenovirus infection.

18 in the host neutralizing immune response to adenovirus infection.

19 e interact specifically during the course of adenovirus infection.

20 of fusing with autophagosomes which enhance adenovirus infection.

21 nity in humanized mice during a hepatotropic adenovirus infection.

22 yncytial virus infection and higher rates of adenovirus infection.

23 h clinical data on treatment and outcomes of adenovirus infection.

24 reduces cell surface CAR(Ex8) abundance and adenovirus infection.

25 hology associated with replication-defective adenovirus infection.

26 nt of rejection did not increase the risk of adenovirus infection.

27 bial peptides are potent inhibitors of human adenovirus infection.

28 show that this prevents ATR signaling during adenovirus infection.

29 23 patients with KD and 7 of 8 patients with adenovirus infection.

30 n nonpermissive 293 cells can be overcome by adenovirus infection.

31 apical surface and was highly susceptible to adenovirus infection.

32 tep of NHEJ, is degraded as a consequence of adenovirus infection.

33 l cellular function necessary for productive adenovirus infection.

34 on hinders RIDalpha activity during an acute adenovirus infection.

35 1 repair complex (MRN) to promote productive adenovirus infection.

36 and induction of the DNA damage response by adenovirus infection.

37 kDa proteins interact with each other during adenovirus infection.

38 piratory syncytial virus, parainfluenza, and adenovirus infection.

39 is accompanied by reduced susceptibility to adenovirus infection.

40 ellular repair complex is inactivated during adenovirus infection.

41 thus enhancing the cell's susceptibility to adenovirus infection.

42 s the GCN5 acetyltransferase during a normal adenovirus infection.

43 were unchanged after tyrphostin treatment or adenovirus infection.

44 an patients can develop severe, often lethal adenovirus infections.

45 onarily conserved target of E4-ORF3 in human adenovirus infections.

46 njugation, which were not found with control adenovirus infections.

47 hile they are potent, they also risk causing adenovirus infections.

48 are believed to prolong acute and persistent adenovirus infections.

49 little is known about the immune response to adenovirus infections.

50 e as a vector for the transmission of ocular adenovirus infections.

51 as antiviral agents for treatment of serious adenovirus infections.

52 prevalence behind RSV infection (15.7%) and adenovirus infection (10.3%).

53 of late adenovirus mRNA in the late phase of adenovirus infection; (4) repression of host mRNA and tr

54 tuzumab in vivo were at the greatest risk of adenovirus infection (45% probability) regardless of don

55 Adenovirus infection activates cellular DNA damage respo

56 Human adenovirus infection activates intracellular signaling,

57 Here, we show that adenovirus infection activates the c-Jun N-terminal kina

58 ice (transthyretin HNF-3beta) or recombinant adenovirus infection (AdHNF3beta), and observed diminish

59 Adenovirus infections affected 24% of recipients and dev

60 We describe the first case of a fatal adenovirus infection after several years of immunosuppre

61 ctively studied the incidence and outcome of adenovirus infections after SCT using preemptive screeni

62 y(A) sites is found during the late stage of adenovirus infection, after viral DNA replication has be

63 Respiratory adenovirus infection among military recruits is a seriou

64 in the initiation of inflammation following adenovirus infection and adenovirus-mediated gene transf

65 Adenovirus infection and E1A expression sensitize cells

66 Adenovirus infection and expression of E1A induces both

67 he intracellular response that are unique to adenovirus infection and how adenoviral proteins produce

68 inant poly(A) site during the early stage of adenovirus infection and in plasmid transfections when m

69 production of chemokines by monocytes after adenovirus infection and increases monocyte migration.

70 K protein) is synthesized abundantly in late adenovirus infection and is required for efficient lysis

71 ding definition of how p53 is inactivated in adenovirus infection and provides key insights that coul

72 negative cells) increased its sensitivity to adenovirus infection and significantly inhibited its in

73 d to inhibit apoptosis induced by E1A during adenovirus infection and transformation.

74 nsitive to cell stresses, namely heat shock, adenovirus infection and treatment with cycloheximide, w

75 Hence, a possible relationship between adenovirus infection and tyrosine phosphorylation of FAK

76 bronchoalveolar lavage fluid after pulmonary adenovirus infection, and all were significantly elevate

77 I3K) and Akt and their downstream targets in adenovirus infection, and here we report the novel findi

78 We have also discovered that adenovirus infections are tightly regulated by endosome

79 be employed to develop novel drugs to treat adenovirus infection as well as be used as tools for gen

80 ctive therapeutic strategies to treat severe adenovirus infections as well as improved adenovirus vec

81 These lymphocytes are susceptible to adenovirus infection, as demonstrated by reporter green

82 We detected unsuspected mucosal adenovirus infection associated with enteritis as well a

83 These results reveal how adenovirus infection attenuates LMP2 expression, thereby

84 es, including lymphocytes, are refractory to adenovirus infection because they lack the Coxsackie/ade

85 grossly rearranged and stabilized following adenovirus infection, but paclitaxel does not increase t

86 antihexon monoclonal antibody 9C12 inhibits adenovirus infection by blocking microtubule-dependent t

87 ct evidence that human alpha-defensins block adenovirus infection by preventing uncoating during cell

88 These data suggest adenovirus infection can activate beta-cell survival and

89 Together, these data argue that adenovirus infection can result in inhibition of RNAi an

90 ranslation of mRNAs during the late phase of adenovirus infection, can also modulate mRNA export from

91 Adenovirus infections cause significant morbidity and mo

92 s the first report describing an outbreak of adenovirus infection causing diarrhea among adult hemato

93 rexpression of CREM-17X in intact islets via adenovirus infection decreased islet insulin mRNA levels

94 ent with previous studies demonstrating that adenovirus infection depends on attachment of a viral fi

95 pithelial cells obtained from null mice show adenovirus infection efficiency equal to that from wild-

96 alysis from a prospective study of high-risk adenovirus infections following hematopoietic progenitor

97 pulations required stimulation subsequent to adenovirus infection for reporter expression.

98 Numerous outbreaks of adenovirus infection from different types of health care

99 e PDZ1 domain is able to rescue CAR(Ex8) and adenovirus infection from MAGI-1-mediated suppression.

100 findings explain the relative resistance to adenovirus infection from the apical surface.

101 In addition to adenovirus infection, group 3 ILCs were also found in mo

102 tein that is produced at high levels in late adenovirus infection (>24 h postinfection).

103 his study was aimed at investigating whether adenovirus infection has any impact on the potential of

104 m and the metabolic fate of glutamine during adenovirus infection have remained elusive.

105 neumoniae, Mycoplasma pneumoniae, and latent adenovirus infections have been correlated with asthma c

106 Unlike human adenovirus infections, however, mouse adenovirus type 1

107 We demonstrate here that during adenovirus infection, immediate early viral proteins fro

108 rotected mice from a subsequent OVA-encoding adenovirus infection in a CD8(+) cell-dependent manner a

109 of pneumonia developed during an outbreak of adenovirus infection in a chronic psychiatric care facil

110 IgA neutralizes adenovirus infection in a TRIM21- and proteasome-depende

111 GFAT1 and GFAT1Alt expressed by recombinant adenovirus infection in COS-7 cells displayed robust enz

112 cious in minimizing the clinical symptoms of adenovirus infection in rabbit eyes.

113 study, we recommend active surveillance for adenovirus infection in T-cell-depleted SCT and withdraw

114 e the incidence and clinical significance of adenovirus infection in this population.

115 l localization, it affects both adhesion and adenovirus infection in unique ways.

116 fatal disseminated disease resembling human adenovirus infections in immunocompromised patients.

117 lovirus (CMV), Epstein-Barr virus (EBV), and adenovirus infections in immunosuppressed patients.

118 lymphocytes are not generally susceptible to adenovirus infection, in part because of the absence of

119 SERCA2 adenovirus infection increased SERCA2 mRNA expression to

120 Adenovirus infection induced cytotoxic T lymphocytes (CT

121 Adenovirus infection induced the rapid activation of Raf

122 Adenovirus infection induces a cellular DNA damage respo

123 These results demonstrate that adenovirus infection induces a significant stabilizing e

124 E1A expression during adenovirus infection induces apoptosis.

125 sion of the E4-6/7 protein in the context of adenovirus infection induces E2F-1 protein accumulation.

126 We show that adenovirus infection induces global changes in SUMO loca

127 ajor part of the mechanism by which systemic adenovirus infection induces pathology, as opposed to th

128 Adenovirus infection inhibits synthesis and processing o

129 Adenovirus infection is a potentially serious complicati

130 Adenovirus infection is an important cause of morbidity

131 Adenovirus infection is becoming increasingly recognized

132 ha)14iNKT cells during replication-defective adenovirus infection is not known and is the main focus

133 In the nontransplant pediatric population, adenovirus infection is the most common cause.

134 es, which are almost completely resistant to adenovirus infection, is sufficient to facilitate the ef

135 Adenovirus infection leads to increased apoptosis by the

136 n a murine model of intravenous hepatotropic adenovirus infection, liver-primed antiviral CD8(+) T ce

137 enovirus-induced myocarditis, and persistent adenovirus infection may contribute to ongoing cardiac d

138 nscriptional activation in the late phase of adenovirus infection, newly synthesized viral early E1A

139 the AAV2 capsid gene promoter (P40), neither adenovirus infection nor the large Rep protein was requi

140 At the late stage after adenovirus infection, numerous autophagic vacuoles accom

141 Adenovirus infections occur in 5% to 21% of patients fol

142 Adenovirus infection occurs in 10% of pediatric orthotop

143 age at transplantation was a risk factor for adenovirus infection (odds ratio=0.81, 95% confidence in

144 Adenovirus infection of hematopoietic cells frequently r

145 rly dynamics in an experimental system using adenovirus infection of human embryonic kidney (293) cel

146 Efficient adenovirus infection of target cells depends upon the pr

147 One patient had primary graft failure due to adenovirus infection of the donor lungs, and required pr

148 y contribute to the inflammatory response to adenovirus infection of the human cornea.

149 We report a case of adenovirus infection of the renal allograft in a combine

150 effect on transfection by either recombinant adenovirus infection or electroporation.

151 ses a ribosome jumping mechanism during late adenovirus infection or heat shock (stress) of mammalian

152 overexpression of the active form of Akt by adenovirus infection or inhibition of the Akt downstream

153 f the mutant p53(175H) allele by recombinant adenovirus infection or stable transfection also stabili

154 when cells are sensitized to TRAIL either by adenovirus infection or treatment with cycloheximide.

155 patients with KD than in patients with acute adenovirus infections or systemic adverse drug reactions

156 orylation of the ssD-BP also correlates with adenovirus infection, or expression of the adenovirus E4

157 00k protein displaces Mnk1 from eIF4G during adenovirus infection, or in transfected cells.

158 nd ARF levels by DNA damage, recombinant ARF adenovirus infection, or inducible MDM2 expression leads

159 ential for normal development, reproduction, adenovirus infection, or the healing of cutaneous wounds

160 Here, we show that the adenovirus infection process has a significant impact on

161 has been analyzed in detail, the dynamics of adenovirus infection remain largely unknown due to techn

162 s dying a nonapoptotic cell death induced by adenovirus infection repressed macrophage proinflammator

163 Adenovirus infection resulted in a transient enhancement

164 s in the presence of doxycycline followed by adenovirus infection resulted in helper and vector gene

165 ession, DNA damage down-regulates MCL-1, and adenovirus infection resulted in the accumulation of pho

166 , Cryptosporidium, rotavirus, norovirus, and adenovirus infections resulting from indirect wastewater

167 evented activation of p40 transcription with adenovirus infection, resulting in a reduced level of ca

168 Adenovirus infection should be considered when evaluatin

169 om this study, the primary control of falcon adenovirus infections should be based on segregation of

170 d more than cccDNA and mRNA levels following adenovirus infection suggests that the former decline ei

171 During the late stages of adenovirus infection, the 100K protein (100K) inhibits t

172 During adenovirus infection, the emphasis of gene expression sw

173 e estrogen exposure, and through recombinant adenovirus infection, the introduction and stable expres

174 During the early phase of adenovirus infection, the promoter-proximal L1 poly(A) s

175 During adenovirus infection, the viral protein E4orf3 associate

176 Late in an adenovirus infection, the viral proteinase (AVP) becomes

177 ndings validate a novel approach to treating adenovirus infections through the modulation of host cel

178 tion by cycloheximide and at a late stage of adenovirus infection, thus accounting for the loss of RN

179 mplished either through cell treatment or by adenovirus infection to express dominant-negative AMPK,

180 udies we used transgenic mice or recombinant adenovirus infection to increase hepatic expression of f

181 a)14iNKT cells were activated in response to adenovirus infection to induce significant levels of hep

182 We also show that adenovirus infection triggers a response mediated by the

183 We explored the possibility that adenovirus infection triggers signal transduction pathwa

184 During adenovirus infection, viral proteins are expressed that

185 The incidence of adenovirus infection was 19.7% (15 of 76), and the virus

186 FR901228 treatment before adenovirus infection was associated with at least a 10-f

187 Adenovirus infection was classified as either disease or

188 Adenovirus infection was diagnosed in 17 (94%) persons w

189 Recovery from adenovirus infection was marked by elevation of sorbitol

190 Adenovirus infection was very efficient, even at very lo

191 ytoplasm during the late phase of subgroup C adenovirus infection, we have examined the metabolism of

192 Two hallmarks of a successful adenovirus infection were abolished by the NO donors: th

193 age at transplantation is a risk factor for adenovirus infection; whereas cytomegalovirus D+/R- sero

194 Adenovirus infection, which is a waterborne viral diseas

195 lu RNAs and that these RNPs accumulate after adenovirus infection, while levels of SRP9, SRP14, SRP54

196 onors were beta-galactosidase-positive after adenovirus infection with a multiplicity of infection of

197 be phosphorylated by ATR during a wild-type adenovirus infection, with some contribution from ATM an