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1 sence or absence of monoclonal antibodies to adenylate cyclase toxin.
2 e, with the exception of the mutants lacking adenylate cyclase toxin.
3 depending on whether the bacteria expressed adenylate cyclase toxin.
4 e a primary in vivo target of the Bordetella adenylate cyclase toxin.
5 rains, but only in the absence of functional adenylate cyclase toxin, a B. pertussis toxin that has b
6 t advances in understanding the functions of adenylate cyclase toxin, a type 1 secretion system (T1SS
8 ious studies indicate that B. bronchiseptica adenylate cyclase toxin (ACT) and the type III secretion
10 criptionally express cyaA, the gene encoding adenylate cyclase toxin (ACT) in other pathogenic Bordet
13 with Bordetella pertussis, and the secreted adenylate cyclase toxin (ACT) is essential for the bacte
17 B. pertussis uses pertussis toxin (PT) and adenylate cyclase toxin (ACT) to kill and modulate host
18 The catalytic domain of Bordetella pertussis adenylate cyclase toxin (ACT) translocates directly acro
19 ertussis and B. bronchiseptica, which encode adenylate cyclase toxin (ACT), are functionally intercha
21 filamentous hemagglutinin (FHA) and for the adenylate cyclase toxin (ACT), which blocks neutrophil f
22 l toxins, including pertussis toxin (PT) and adenylate cyclase toxin (ACT), which have both been show
23 ent of whooping cough, secretes and releases adenylate cyclase toxin (ACT), which is a protein bacter
27 ir function, we tested the effect of FITC on adenylate cyclase toxin activity, an important extracell
30 d Bvg-regulated virulence factors, including adenylate cyclase toxin and filamentous hemagglutinin, d
33 ion of neutralizing monoclonal antibodies to adenylate cyclase toxin converted a serum that previousl
34 ding domain (RD) of the Bordetella pertussis adenylate cyclase toxin CyaA fused to the C terminus of
35 genetically detoxified Bordetella pertussis adenylate cyclase toxin (CyaA) as a delivery system for
36 everal virulence factors, among which is the adenylate cyclase toxin (CyaA) that plays a crucial role
37 ordetella pertussis produces a cell-invasive adenylate cyclase toxin (CyaA) which is related to the R
39 analysis showed that cyaA, the gene encoding adenylate cyclase toxin (CyaA), was the most downregulat
41 r lacking virulence factors-pertussis toxin, adenylate cyclase toxin, dermonecrotic toxin, filamentou
42 This allowed the engineering of recombinant adenylate cyclase toxin from Bordetella pertussis for th
47 strains lacking filamentous hemagglutinin or adenylate cyclase toxin, nor when purified PT was admini
49 ns deficient in filamentous hemagglutinin or adenylate cyclase toxin were incapable of proliferation
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