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1 r osseous replacement occurs more often than adipocytic.
2 of PPARgamma at Ser-112 is required for its adipocytic activity, whereas phosphorylation of RUNX2 at
3 actors, resulting in activation of PPARgamma adipocytic and suppression of RUNX2 osteoblastic activit
7 ie, sharing a similar expression profile to adipocytic cells at a corresponding stage of differentia
9 T4 protein and gives rise to a population of adipocytic cells that take up glucose in direct response
12 hanced contractility despite no reduction in adipocytic commitment or increased expression of TGF-bet
13 ted a redirected lineage choice that favored adipocytic commitment over fibroblast or myofibroblast d
14 scular adipocytes exhibit a reduced state of adipocytic differentiation as compared with adipocytes d
16 rget-gene transcription that normally drives adipocytic differentiation of 3T3-L1 cells, lipid accumu
20 Together, these data indicate that terminal adipocytic differentiation was induced in these malignan
21 hese MSCs showed that they were committed to adipocytic differentiation, but unable to terminally dif
22 phenotypic changes that were consistent with adipocytic differentiation, suggesting that the effects
23 ell as functionally in terms of its specific adipocytic differentiation-based response to ET-743.
31 cells (Q-HSCs), which exhibit epithelial and adipocytic features, into myofibroblastic-HSC (MF-HSCs)
32 4% and approximately 100%, respectively, for adipocytic genes, in accordance with an experimental des
34 ersely, bone-resident cells committed to the adipocytic lineage inhibit hematopoiesis and bone healin
35 the molecular identity of the bone-resident adipocytic lineage, and they establish its involvement i
37 can differentiate along the chondrocytic and adipocytic lineages in vivo, these cells were inoculated
38 erentiating to osteocytic, chondrocytic, and adipocytic lineages when stimulated under appropriate co
39 s and the increase in mRNA expression of the adipocytic markers peroxisome proliferator-activated rec
40 sarcomas are compared to their corresponding adipocytic maturing cells, we identified a group of gene
41 the tyrosine protein kinase activity of rat adipocytic membrane fragments in the absence of insulin;
42 multiple cell lineages, such as melanocytic, adipocytic, osteocytic, and chondrocytic lineages, which
43 n members of the osteogenic, myoblastic, and adipocytic pathways, 176 new genes in addition to 28 ori
44 own under osteogenic conditions developed an adipocytic phenotype after 3 days of complete inhibition
47 r results reveal the developmental origin of adipocytic properties and the pathophysiological contrib
49 ase of the myocardium characterized by fibro-adipocytic replacement of myocytes, predominantly in the
50 ancer at the Klb locus was also bound by the adipocytic transcription factor peroxisome proliferator-
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