ライフサイエンスコーパス: adipogenic

1 and 2 of the 7 predicted negatives were also adipogenic.

2 urface expression of N-cadherin and enhanced adipogenic ability supports this view.

3 Thus, the adipogenic action of exogenous UAG in tibial marrow is d

4 ease TTF-1 expression contributes to its pro-adipogenic action.

5 The adipogenic activity in house dust occurred at concentrat

6 This study sought to evaluate the adipogenic activity of indoor house dust extracts and a

7 vels (<20 mug of dust/well), and significant adipogenic activity was also exhibited by 28 of the SVOC

8 ng lipid and glucose metabolism with reduced adipogenic activity, that may be used as a model for a s

9 oxPi, and asked how well the ToxPi predicted adipogenic activity.

10 zone, suggesting that LT175 may have a lower adipogenic activity.

11 progenitor cell line that lacks osteogenic, adipogenic and angiogenic potential but is capable of di

12 tial for Wnt secretion, alleviates both anti-adipogenic and anti-lipogenic effects of Hh in cell cult

13 study, for the first time, reveals the brown adipogenic and browning effects of apelin and suggests a

14 aired osteogenic differentiation, but normal adipogenic and chondrogenic differentiation.

15 e embryonic stem cells survival, is robustly adipogenic and induces postnatal adipose tissue formatio

16 Expression levels of adipogenic and lipogenic genes in adipose tissues were a

17 ere capable of colony-forming efficiency and adipogenic and osteo/dentinogenic differentiation under

18 nalogous beta-catenin-independent defects in adipogenic and osteogenic differentiation, and knockdown

19 DDT enhanced both adipogenic and osteogenic differentiation, which was con

20 cter of transduced hMSCs was investigated by adipogenic and osteogenic differentiation.

21 re we specify the cellular identities of the adipogenic and osteogenic lineages of the bone.

22 contributing exclusive differentiation along adipogenic and osteogenic lineages, respectively.

23 witch in the commitment decision between the adipogenic and osteogenic lineages.

24 levels, and FM 550 exposure with changes in adipogenic and osteogenic pathways.

25 from human neck fat and characterized their adipogenic and thermogenic differentiation.

26 ation by increasing the expressions of brown adipogenic and thermogenic transcriptional factors via t

27 ls are more osteogenic, softer gels are more adipogenic, and cell spreading areas increase with the s

28 In an adipogenic assay, both enantiomers increased the express

29 including desmoplakin, predominantly in the adipogenic but not fibrogenic subsets.

30 0 knockout ADSCs have dramatically decreased adipogenic capabilities associated with downregulation o

31 Excessive RA-mediated activity impedes the adipogenic capability of ASCs at early but not late stag

32 However, hiPSC-BAPs display a low adipogenic capacity compared to adult-BAPs when maintain

33 hymal stromal stem cells (MSCs) to study the adipogenic capacity of BADGE and BPA and evaluated their

34 Thus, the weight-reducing (DI) increased adipogenic capacity of preadipocytes and shifted their s

35 besity demonstrates long-term effects on the adipogenic capacity of progenitor cells in offspring adi

36 enchymal stem cells (mBMSCs) to evaluate the adipogenic capacity of ToxCast chemicals.

37 , which renders progenitor cells with a high adipogenic capacity.

38 This study compares several adipogenic cell culture systems under a variety of condi

39 t the importance of balancing stromal versus adipogenic cell expansion during white adipose tissue de

40 NAs can redirect mesenchymal stem cells into adipogenic cell fate with concomitant up-regulation of k

41 ate that Wnt/beta-catenin signaling controls adipogenic cell fate within the lower dermis, which pote

42 ssed in adipose tissues and murine and human adipogenic cell lines and is localized in the mitochondr

43 423 in stromal vascular cells and cloned low adipogenic cells dramatically increased their adipogenic

44 of Zfp423 and TGF-beta between low and high adipogenic cells is associated with the DNA methylation

45 Zfp423 knockdown by shRNA in high adipogenic cells largely prevented their adipogenic diff

46 verexpression of Nat8l in immortalized brown adipogenic cells strongly increases glucose incorporatio

47 10.7+/-138.4%, respectively, P<0.05) in high adipogenic cells.

48 mplicate Rb1 as necessary for maintenance of adipogenic characteristics in fully differentiated adipo

49 Of these 9 adipogenic chemicals, 3 activated PPARgamma, and 1 activ

50 tocol significantly impacts the detection of adipogenic chemicals, and therefore, influences reproduc

51 ficant impact on potencies and efficacies of adipogenic chemicals.

52 e potential for tri-lineage differentiation (adipogenic, chondrogenic, and osteogenic).

53 by treating confluent preadipocytes with the adipogenic cocktail, which activates transcription facto

54 adult-BAPs when maintained in a traditional adipogenic cocktail.

55 Thus adipogenic commitment and differentiation is regulated b

56 l feedback regulator of both white and beige adipogenic commitment and differentiation, and resistanc

57 intracellular Wisp2 protein for BMP4-induced adipogenic commitment and PPARgamma activation was verif

58 rotein, the latter regulating precursor cell adipogenic commitment and PPARgamma induction by BMP4.

59 t embryonic day 14.5 (E14.5), when the early adipogenic commitment is initiated.

60 ted state, whereas cytosolic WISP2 regulates adipogenic commitment.

61 ased on lineage tracing that mural cells are adipogenic, contrasting with the conclusions of a recent

62 The standard protocol for full in vitro adipogenic conversion of committed preadipocytes, such a

63 knockdown in 3T3-L1 preadipocytes inhibited adipogenic conversion.

64 ression of PPARgamma2, is able to rescue the adipogenic defect caused by DPP8/9 inhibition in preadip

65 a agonist pioglitazone partially rescued the adipogenic defect in CGL cells.

66 dipogenic genes, our method recapitulates an adipogenic developmental pathway through successive meso

67 latory regions to repress expression of anti-adipogenic developmental regulators.

68 Last, ORO in adipogenic differentiating cells was positively correlat

69 preadipocytes significantly increases their adipogenic differentiation ability.

70 f tribbles homolog 3 (Trib3) that suppressed adipogenic differentiation and inflammatory responses by

71 iR-130 during DIO may contribute to impaired adipogenic differentiation and obesity-related metabolic

72 ocyte progenitors in the fat of offspring to adipogenic differentiation and subsequent depletion, whi

73 Genetic ablation of HDAC9 improves adipogenic differentiation and systemic metabolic state

74 ngs suggest that APCDD1 positively regulates adipogenic differentiation and that its down-regulation

75 e of the PPARgamma2 locus after the onset of adipogenic differentiation and the mechanisms by which i

76 enic occurred in the early stage of terminal adipogenic differentiation and was highly correlated wit

77 l stromal cells indicated that Dab2 promoted adipogenic differentiation by modulation of MAPK (Erk1/2

78 MSCs, with enhanced osteogenic and decreased adipogenic differentiation capacities, as compared with

79 aracter demonstrated by their osteogenic and adipogenic differentiation capacity and their proliferat

80 Induction of either osteogenic or adipogenic differentiation could be validated in bmMSC c

81 EPI also accelerated and amplified adipogenic differentiation detected by increasing the ad

82 Impaired adipogenic differentiation during diet-induced obesity (

83 gesting that miR-130 contributes to impaired adipogenic differentiation during DIO by repressing APCD

84 We hypothesized that MO enhances adipogenic differentiation during fetal development thro

85 n summary, MO enhanced Zfp423 expression and adipogenic differentiation during fetal development, at

86 ery because its DR makes cells more prone to adipogenic differentiation even in the absence of the ad

87 potential of Sca1(+) fibroblasts to undergo adipogenic differentiation ex vivo.

88 regulator of preadipocyte proliferation and adipogenic differentiation in ASAT.

89 analysis showed that Zfp423 regulates early adipogenic differentiation in fetal progenitor cells.

90 liver, obesity, adipositis, and myogenic and adipogenic differentiation in muscle tissue in the HFrD

91 ithin this lineage significantly reduced its adipogenic differentiation in the context of exogenous,

92 ce of Wnt/beta-catenin signaling controlling adipogenic differentiation in the developing reticular d

93 of osteoblast differentiation, T63 inhibited adipogenic differentiation in the pluripotent mesenchyma

94 us and visceral adipose tissues and impaired adipogenic differentiation in vitro Mechanistically, we

95 and (iii) exhibit markedly increased ex vivo adipogenic differentiation into brown adipocytes.

96 HFD-induced impaired adipogenic differentiation is associated with elevated e

97 lture, before or after stiffening, such that adipogenic differentiation is favoured for later stiffen

98 esults in decreased myogenic activity, while adipogenic differentiation is significantly increased.

99 Adipogenic differentiation of 3T3-L1 cells is abolished

100 Conversely, activation of GPR30 enhanced adipogenic differentiation of 3T3-L1 preadipocytes.

101 Deletion of GPR30 reduced adipogenic differentiation of adipose tissue-derived str

102 ntribution of short-chain fatty acids to the adipogenic differentiation of adipose-derived stem cells

103 DEG-BA into mouse ears markedly enhanced the adipogenic differentiation of ADSCs, leading to dermal a

104 Atrial myocardium secretome induces the adipogenic differentiation of adult mesenchymal epicardi

105 es osteoblast differentiation and suppresses adipogenic differentiation of AMSCs.

106 and enzymatic hydrolysates (CPHs) to inhibit adipogenic differentiation of C3H10T1/2 murine mesenchym

107 ubstrate deformations, or the osteogenic and adipogenic differentiation of human adipose-derived stro

108 difications that occur during osteogenic and adipogenic differentiation of mouse bone marrow-derived

109 knockdown of HOTAIR inhibited or stimulated adipogenic differentiation of MSCs, respectively.

110 omoter, which is expected to durably elevate adipogenic differentiation of progenitor cells in adult

111 er-donor variability of their osteogenic and adipogenic differentiation potential, as well as their a

112 gths and osteoblastogenic, chondrogenic, and adipogenic differentiation potentials.

113 encing GREM1 and/or adding BMP4 during white adipogenic differentiation reactivated beige/brown marke

114 transcription factor Prep1 is a repressor of adipogenic differentiation since its down-regulation (DR

115 sate demonstrated a higher reduction in anti-adipogenic differentiation through quantitation by oil-r

116 Here we report a novel role for APCDD1 in adipogenic differentiation via repression of Wnt signali

117 The BA-DEG-BA-induced adipogenic differentiation was mediated via peroxisome p

118 Adipogenic differentiation was not affected by the prese

119 dipogenic cells dramatically increased their adipogenic differentiation, accompanied with the inhibit

120 es from these mice likewise exhibit impaired adipogenic differentiation, and this phenotype persists

121 ted LMNA p.R482W mutation is known to impair adipogenic differentiation, but the mechanisms involved

122 educed cellular cholesterol is important for adipogenic differentiation, evidenced by increased induc

123 e that a chronic high-fat diet (HFD) impairs adipogenic differentiation, leading to accumulation of i

124 enhanced, whereas an miR-130 mimic blunted, adipogenic differentiation, suggesting that miR-130 cont

125 Following adipogenic differentiation, the expression of peroxisome

126 (FTO-4) mice exhibit increased potential for adipogenic differentiation, while MEFs derived from FTO

127 D6, Ppargamma2 and Cebpalpha expression, and adipogenic differentiation, yet had no effect on C/EBPbe

128 tiated adipocytes with blunted expression of adipogenic differentiation-specific genes.

129 nzyme required for PPARgamma2 activation and adipogenic differentiation.

130 (HDAC9), an endogenous negative regulator of adipogenic differentiation.

131 d C3H10T1/2 mesenchymal stem cells, promoted adipogenic differentiation.

132 hibitor of Wnt signaling, a key regulator of adipogenic differentiation.

133 igh adipogenic cells largely prevented their adipogenic differentiation.

134 s-mediated overexpression of APCDD1 enhanced adipogenic differentiation.

135 g transcription factor that is essential for adipogenic differentiation.

136 , Lipe, Plin1, Pparg, and Cebpa genes during adipogenic differentiation.

137 ffspring, which was correlated with enhanced adipogenic differentiation.

138 Wnt proteins, was found to prevent the anti-adipogenic effect of 5-Aza-dC in 3T3-L1 preadipocytes an

139 further demonstrated that the mitogenic and adipogenic effect of ghrelin were mainly dependent on th

140 Finally, the adipogenic effect of ibm-infused UAG was completely abol

141 e transgene showed a blunted response to the adipogenic effects of a high fat diet.

142 In the present study, we reveal the adipogenic effects of Ang(1-7) through activation of Mas

143 We sought to test the adipogenic effects of BADGE in a biologically relevant c

144 Surprisingly, the adipogenic effects of intra-bone marrow (ibm)-infused ac

145 The anti-adipogenic effects of yerba mate, chlorogenic acid, quer

146 ting DNA binding of transcription factors to adipogenic enhancers, in particular C/EBPbeta.

147 family, member 10B (Wnt10b)/beta-catenin and adipogenic ERK/MAPK signaling pathways.

148 ion sites on C/EBPbeta, enhances these early adipogenic events.

149 ptide secreted by atrial myocytes is a major adipogenic factor operating at a low concentration by bi

150 induce the expression of Wnt6, a known anti-adipogenic factor, in fat depots of the mouse.

151 s of Bmp7 expression, a critical early brown adipogenic factor.

152 on profiling identified keratinocyte-derived adipogenic factors that are induced by beta-catenin acti

153 ramme and enter into smooth muscle and brown adipogenic fates.

154 We show a novel pro-adipogenic function of AGEs in replicative senescent pre

155 GE (RAGE) expression is required for the pro-adipogenic function of AGEs in senescent preadipocytes.

156 We report that JMJD6 is required for adipogenic gene expression and differentiation in a mann

157 on of G9a in mouse adipose tissues increases adipogenic gene expression and tissue weight.

158 e lamin A p.R482W hot spot mutation prevents adipogenic gene expression by epigenetically deregulatin

159 pocytes showed not only greater induction of adipogenic gene expression during differentiation but al

160 BADGE exposure elicited an adipogenic gene expression profile, and its ability to i

161 s indicate dual roles for JMJD6 in promoting adipogenic gene expression program by post-transcription

162 osteogenic and decreased PPARgamma-dependent adipogenic gene expression.

163 ancer binding protein-alpha, which drive the adipogenic gene program, was markedly suppressed by CTRP

164 To elucidate further components of the adipogenic gene regulatory network, we performed a large

165 ipocyte differentiation for induction of the adipogenic gene transcription program, including the key

166 proliferation and their expression of a key adipogenic gene, peroxisome proliferator-activated recep

167 positively associated with the expression of adipogenic genes (PPARgamma and IRS1) in both visceral a

168 , C/ebpalpha and Ppargamma, as well as other adipogenic genes at both the mRNA and protein levels.

169 transcriptional programs: the expression of adipogenic genes common to both brown fat (BAT) and whit

170 ired for stimulation of several GR-regulated adipogenic genes in 3T3-L1 preadipocytes by glucocortico

171 on of nucleosomal H4K5 and H3 to re-activate adipogenic genes to reverting adipogenesis.

172 to induce adipogenesis and the expression of adipogenic genes was not blocked by known PPARgamma anta

173 arg) and white (Fabp4, Pnpla2, AdipoQ, Fasn) adipogenic genes, and glucose metabolism genes (Glut4, I

174 earlier and increased expression of specific adipogenic genes, consequent to the increased response o

175 Without overexpression of exogenous adipogenic genes, our method recapitulates an adipogenic

176 activation of C/EBPbeta-primed enhancers of adipogenic genes.

177 hylated arsenic (DMA3+, </= 2 muM) decreased adipogenic hormone-induced adipogenesis in a concentrati

178 trast, more lipid deposition was observed in adipogenic-induced smoker PDLSC.

179 edly bypassed by prolonged treatment with an adipogenic inducer, 3-isobutyl-1-methylxanthine (IBMX).

180 c differentiation even in the absence of the adipogenic inducers.

181 7a and miR-27b were down-regulated following adipogenic induction of human adipose-derived stem cells

182 with overexpression of the MIR355 gene after adipogenic induction.

183 Cs readily differentiated into adipocytes on adipogenic induction.

184 ine 3T3-L1 preadipocytes were used to assess adipogenic induction.

185 ing frizzled-like sequences bound the potent adipogenic inhibitor, Wnt10b, in vitro.

186 Therefore, miR-155 deletion increases adipogenic, insulin sensitivity, and energy uncoupling m

187 These studies indicate an adipogenic lifestyle alone is sufficient for the develop

188 lly expressed Rev-erbalpha responded to both adipogenic ligand and fibrogenic transforming growth fac

189 enitor identity and potency, promoting white adipogenic lineage commitment.

190 -fat diet feeding activates expansion of the adipogenic lineage, an effect that is significantly enha

191 transcription factor committing cells to the adipogenic lineage, with exceptionally dense CpG sites i

192 promoting their differentiation along fibro-adipogenic lineages while inhibiting myogenesis.

193 Additionally, an elevated expression of the adipogenic marker genes PPARgamma and Cebpalpha with a c

194 up-regulate expression of cyclin D3 and two adipogenic markers (CCAAT/enhancer binding protein alpha

195 tissues, leading to diminished expression of adipogenic markers and adipocyte differentiation.

196 red lipid accumulation and expression of key adipogenic markers in differentiating progenitors expose

197 that pairs H3K4me3 with H3K9me3 to maintain adipogenic master regulatory genes (Cebpa and Pparg) exp

198 Our data suggest that miR-27 is an anti-adipogenic microRNA partly by targeting PHB and impairin

199 s the ability of lamin A to repress the anti-adipogenic miR-335, providing a potential molecular mech

200 eregulating long-range enhancers of the anti-adipogenic MIR335 microRNA gene in human adipocyte proge

201 uencing, we revealed that bta-miR-23a was an adipogenic miRNA mediating bovine adipogenesis in skelet

202 c variants of ternatin, a cytotoxic and anti-adipogenic natural product whose molecular mode of actio

203 the mechanisms underlying the regulation of adipogenic or osteoblastogenic development focus on tran

204 w stromal cells) to a metabolically stressed adipogenic pathway that induces synthesis of a hyalurona

205 he commitment of adipocyte progenitors to an adipogenic pathway.

206 induced to differentiate down osteogenic or adipogenic pathways by controlling the content of foulin

207 erved simultaneous expression of osteogenic, adipogenic, pericytic, and hematopoiesis-supporting gene

208 the cooperative DNA binding behavior of the adipogenic peroxisome proliferator-activated receptor ga

209 genesis by decreasing the recruitment of the adipogenic peroxisome proliferator-activated receptor ga

210 ur data show that Nat8l impacts on the brown adipogenic phenotype and suggests the existence of the N

211 rt of genes was selected to characterize the adipogenic phenotype in primary cell cultures from three

212 ransient knockdown of Rb1 led to loss of the adipogenic phenotype.

213 tion, a propensity for cell death, and a pro-adipogenic phenotype.

214 assessment represent crude estimates of the adipogenic potential because of the disruption of the in

215 ased impact on insulin secretion and reduced adipogenic potential but with preservation of anti-infla

216 Thus, altered adipogenic potential caused by Ube2l6 knockdown is likel

217 sults highlight the extensive differences in adipogenic potential in various fat depots.

218 Adipogenic potential of preadipocytes with knockdown or

219 ed a cell-autonomous role in restraining the adipogenic potential of preadipocytes.

220 ound that glycated BSA restores the impaired adipogenic potential of senescent preadipocytes in vitro

221 The impaired adipogenic potential of senescent preadipocytes is a hal

222 ation of the aP2 lineage greatly reduces the adipogenic potential of SVF cells.

223 d hyperplastic adipose tissue, with enhanced adipogenic potential of the stromal vascular fraction an

224 N-cadherin expression or migration or confer adipogenic potential to immortalized RB1(+/+) calvarial

225 pression of Zfp521 in cells greatly inhibits adipogenic potential, whereas RNAi-mediated knock-down o

226 ased phagocytic capacity, and acquisition of adipogenic potential.

227 ca1(+), non-myogenic, and exhibiting a fibro/adipogenic potential.

228 n-6/n-3 ratios on offspring adipogenesis and adipogenic potential.

229 genic support function (PC-M), while lacking adipogenic potential.

230 exhibited reduced myogenic (Myf5 and -6) and adipogenic (Pparg, Cebpa, and Lep) gene expression, wher

231 Brown adipogenic precursor activity in embryos was confined to

232 tein-4 (BMP4) plays a key role in regulating adipogenic precursor cell commitment and differentiation

233 kers, suggesting the presence of multipotent adipogenic precursor cells.

234 in mesenchymal stem cells, fibroblasts, and adipogenic precursor cells.

235 results suggest that the need for MCE in the adipogenic process is independent from the requirement f

236 te that polyamines are required early in the adipogenic process.

237 range of activation of genes involved in the adipogenic process.

238 data suggest a role for Foxc1 in inhibiting adipogenic processes in CAR progenitors.

239 constellation of markers diagnostic of fibro/adipogenic progenitor cells and were often associated wi

240 f the type 1A BMP receptor (Bmpr1a) in brown adipogenic progenitor cells leads to a severe paucity of

241 c progenitor cells, embryonic stem cells and adipogenic progenitor cells.

242 e hair follicles, suggesting a new source of adipogenic progenitor cells.

243 Maternal obesity reduces adipogenic progenitor density in offspring adipose tissu

244 al pathway through successive mesodermal and adipogenic progenitor stages.

245 cytes, we isolated satellite cells and fibro/adipogenic progenitors (FAPs) from muscle; satellite cel

246 Upon injury, muscle-resident fibro/adipogenic progenitors (FAPs) proliferated and gave rise

247 ulates a population of muscle-resident fibro/adipogenic progenitors (FAPs) that play a supportive rol

248 results identify perivascular cells as fibro/adipogenic progenitors in WAT and show that PDGFRalpha t

249 However, whether aP2 is expressed in adipogenic progenitors is controversial.

250 s deletion does not impair the classic brown adipogenic program but rather induces premature activati

251 ates AKT signaling to drive and maintain the adipogenic program in the skin.

252 contributing to the control of the multistep adipogenic program that determines the number of precurs

253 Accordingly, Nck2 deficiency promotes an adipogenic program that not only enhances adipocyte diff

254 Here we use the dynamic adipogenic program that occurs during hair growth to unc

255 vity is required for up-regulating the brown adipogenic program.

256 R1), fundamentals for the PPARgamma-mediated adipogenic program.

257 n association with the implementation of the adipogenic program.

258 The inherent adipogenic properties of human adipose-derived stem cell

259 The adipogenic property of the atrial secretome was enhanced

260 Fatty acid-binding protein 4 (FABP4) is an adipogenic protein and is implicated in atherosclerosis,

261 reased adipose PHD levels and decreased anti-adipogenic protein levels by increasing their ubiquitina

262 inhibitor) prevented the degradation of anti-adipogenic proteins and retarded RIAD.

263 cription factors to induce the expression of adipogenic proteins leading to the accumulation of lipid

264 ociated with increases in the levels of anti-adipogenic proteins such as GATA-3, KLF-2, and transcrip

265 nd that this leads to the commitment of anti-adipogenic proteins to the ubiquitination-proteasomal pa

266 iquitination/proteasomal degradation of anti-adipogenic proteins.

267 cellular matrix turnover and shedding of the adipogenic regulator DLK1, but that in adipose tissue in

268 ed the expression and activity of the master adipogenic regulator peroxisome proliferator-activator r

269 paralleled by upregulated expression of the adipogenic regulator PPARG and its co-activator PPARGC1B

270 ramuscular adipogeneic commitment as an anti-adipogenic regulator which acts by targeting ZNF423.

271 ntrols the expression of most early and late adipogenic regulators, identifying ZEB1 as a central tra

272 iber repair, chronic muscle inflammation and adipogenic replacement of dysferlinopathic muscle.

273 deficient mice reduced muscle inflammation, adipogenic replacement of myofibers, and improved muscle

274 2 does not result in chronic inflammation or adipogenic replacement of the myofibers.

275 rlin exhibit chronic muscle inflammation and adipogenic replacement of the myofibers.

276 TTF-1 interacts with PPFP to inhibit the pro-adipogenic response to pioglitazone, and that the abilit

277 ntiation, coinciding with enhanced export of adipogenic RNAs.

278 mediated via enhanced expression of the pro-adipogenic short isoform of RUNX1T1, which enhanced adip

279 ymal stem cells independently of major human adipogenic signals through C/EBPdelta, C/EBPalpha and pe

280 Here, we show that adipogenic stimuli trigger nuclear translocation of S6K1

281 evelopmental caveats of using antibiotics in adipogenic studies.

282 a at enhancers controlling the expression of adipogenic target genes and continued differentiation.

283 In contrast, the master adipogenic TF peroxisome proliferator-activated receptor

284 We identified 22 novel pro-adipogenic TFs and characterized the top ranking TF, ZEB

285 lt human atrial epicardial cells were highly adipogenic through an epithelial-mesenchymal transition

286 bits PPFP target gene expression and impairs adipogenic trans-differentiation.

287 s chromatin opening and binding of the early adipogenic transcription factor C/EBPbeta to PPARgamma p

288 ng activin A, blunted fat loss, and enhanced adipogenic transcription factor expression within 3 week

289 Gcn5/PCAF inhibits expression of the master adipogenic transcription factor gene PPARgamma, thereby

290 ced lipid accumulation and inhibited the key adipogenic transcription factor peroxisome proliferative

291 ment or by siRNA knockdown of the master pro-adipogenic transcription factor peroxisome proliferator

292 BDE-47 modestly activated the key adipogenic transcription factor peroxisome proliferator-

293 Thy1 decreases the activity of the adipogenic transcription factor PPARgamma by more than 6

294 istically, Fyn regulates the activity of the adipogenic transcription factor signal transducer and ac

295 CAAT/enhancer-binding protein alpha, a major adipogenic transcription factor, and therefore, they wer

296 Here we show that C/EBPbeta, a key pro-adipogenic transcription factor, is PARylated by PARP-1

297 fasting insulin and increased expression of adipogenic transcription factors but lack glucose intole

298 esis by directly promoting the expression of adipogenic transcription factors CCAAT/enhancer-binding

299 F6 acts by exerting translational control of adipogenic transcription factors like C/EBPbeta, C/EBPde

300 PC-A cells were not osteogenic or adipogenic under standard differentiation conditions and