ライフサイエンスコーパス: adipose

1 keletal muscle (36.4%), liver (16.1%), brown adipose (29.7%), and bone marrow (32.9%)-and increases o

2 A corresponding reduction in adipose adiponectin production was observed in susceptib

3 ATP citrate-lyase levels in tissues such as adipose and liver, but the impact of diet on acetyl-CoA

4 turn regulate lipid homeostasis in fat body/adipose and the intestine.

5 vascular smooth muscle from small resistance adipose arteries of non-diabetic and clinically diagnose

6 er investigation of their potential roles in adipose biology and in regulating cardio-metabolic trait

7 ese data highlight a new role for ER-beta in adipose biology and its potential to be a safer alternat

8 n receptor beta (ER-beta) and its ligands on adipose biology.

9 uring pregnancy and lactation promoted white adipose browning and thermogenesis in offspring at weani

10 ation in adipose tissue, possibly related to adipose cell hypertrophy, hypoxia, and/or intestinal lea

11 n sensitive, had increased glucose uptake by adipose cells and skeletal muscle in vivo and ex vivo, i

12 6 diminishes responsiveness to type I IFN in adipose cells to promote thermogenic and mitochondrial f

13 articles in 3D volumes and lipid droplets in adipose cells.

14 egulation of metabolic circadian rhythms and adipose core clock genes in mice and characterization of

15 Focal adipose deficiency, such as lipoatrophy, lumpectomy or f

16 timeline of global thermogenic metabolism in adipose depots during acute cold exposure.

17 Adipose-derived (AD) mesenchymal stem cells (MSCs) espec

18 its of its recellularization with autologous adipose-derived adult stem cells remains unclear.

19 Autologous adipose-derived adult stem cells were obtained by electi

20 Adipose-derived lactate likely contributes to high endog

21 n equivalent model, Klar et al. incorporated adipose-derived mesenchymal cells into skin substitutes

22 l cells into skin substitutes and found that adipose-derived mesenchymal cells secreted high levels o

23 Here, we have shown that transplantation of adipose-derived stem cells (ASCs) accelerates the proces

24 The osteogenic differentiation of adipose-derived stem cells (ASCs) was significantly enha

25 mulating osteoblast differentiation of human adipose-derived stem cells (hASC), compared to red (660

26 nts were found to be nondurotactic for human adipose-derived stem cells (hASCs), allowing the present

27 stimulation to induce osteogenesis of human adipose-derived stem cells.

28 Herein, we report the application of adipose-derived stem/stromal cells (ASCs) for engineerin

29 ration in the posttranscriptional control of adipose development and function.

30 the primary source of EPA and DHA, affected adipose development in offspring.

31 ldren, suggesting the possibility to program adipose development through dietary fatty acids before b

32 ietary fatty acid profile programs offspring adipose development.

33 The lower ATIS and diminished adipose DI in IGT versus NGT is in line with the comprom

34 Adipose DI was calculated as ATIS: (1/GlyRa x fasting in

35 in murine macrophages, compared it to the LD adipose differentiation-related protein (Adrp)/perilipin

36 IS, and beta-cell function relative to ATIS (adipose disposition index [DI]) in obese youth with impa

37 Taken together, our data demonstrate that adipose Dnmt3a is a novel epigenetic mediator of insulin

38 evidence of being regulatory hotspots using adipose expression.

39 Most importantly, expression of adipose FA binding protein (A-FABP) in macrophages facil

40 des and adipokines, and varied expression of adipose genes associated with altered insulin response/g

41 l hypothesis which proposes that the reduced adipose glucose uptake in obesity is a physiological dow

42 development as an exogenous factor, while in adipose HFD's impact roughly coincides with the endogeno

43 nal activation of insulin receptor (InR) and adipose lipase brummer (bmm).

44 atrial natriuretic peptide, and thus, induce adipose lipolysis, we studied peripheral and systemic me

45 lved in muscle structure and metabolism, and adipose metabolism and adipogenesis.

46 induced vasodilatation in human subcutaneous adipose microvessels.

47 iche component that promotes wound repair in adipose, muscle, and lung tissues.

48 nt profiles in the mixtures derived from the adipose of each species.

49 We describe similar massive adipose overgrowth with suppressed leptin expression in

50 s on glucose regulation, hyperlipidemia, and adipose pathology; but may not be as effective as behavi

51 defined, and the regulation and function of adipose plasticity in development and physiology can be

52 r findings support an important role for the adipose PPARgamma-adiponectin axis in susceptibility to

53 n of either Atxn1 or Ube2e2 in primary mouse adipose progenitor cells impaired adipocyte differentiat

54 Knowledge of adipose regulation in animals that undergo rapid fat dep

55 Tissue explants can be used to investigate adipose regulation in wildlife species with large fat re

56 The adipose remodeling phenotypes are recapitulated by fat-s

57 y an inflammatory-like phenotype atypical of adipose resident ILC2.

58 ented the traction forces generated by human adipose stem cells (hASCs).

59 ubertal mice overexpressing adiponectin from adipose tissue (APNtg), adiponectin knockouts (APNko), a

60 haracterized by body weight loss, atrophy of adipose tissue (AT) and systemic inflammation.

61 The pericardial adipose tissue (AT) contains a high density of lymphoid

62 ss, improves insulin sensitivity, and alters adipose tissue (AT) gene expression, yet the relation wi

63 Adipose tissue (AT) is no longer regarded as an inert li

64 orchestrates lipoprotein processing in brown adipose tissue (BAT) and hepatic conversion of cholester

65 factor Hlx is selectively expressed in brown adipose tissue (BAT) and iWAT, and is translationally up

66 However, the role of brown adipose tissue (BAT) in regulating gestational metabolis

67 a) neurons influences thermogenesis of brown adipose tissue (BAT) independent of ambient temperature

68 In contrast to white adipose tissue, brown adipose tissue (BAT) is known to play critical roles for

69 Brown adipose tissue (BAT) is regulated by the sympathetic ner

70 Detection and quantification of brown adipose tissue (BAT) mass remains a major challenge, as

71 Brown adipose tissue (BAT) mitochondria exhibit high oxidative

72 aging is routinely used to investigate brown adipose tissue (BAT) thermogenesis, which requires mitoc

73 Brown adipose tissue (BAT) utilizes glucose and free fatty aci

74 ion impairs retinoic acid signaling in brown adipose tissue (BAT), leading to impaired BAT function a

75 a robust and specific PD-L1 signal in brown adipose tissue (BAT).

76 r neurons controlling thermogenesis of brown adipose tissue (BAT).

77 CP1 mRNAs were not induced in liver or brown adipose tissue (BAT).

78 is accompanied by attrition of dermal white adipose tissue (dWAT) and reduced levels of circulating

79 The abundance of epicardial adipose tissue (EAT) is associated with atrial fibrillat

80 nced insulin signaling in liver and visceral adipose tissue (epididymal white adipose tissue [WAT]),

81 In epididymal white adipose tissue (eWAT) of PDE3B KO mice on a SvJ129 backg

82 ive and anti-inflammatory effects and induce adipose tissue (fat) to produce the vaso-protective prot

83 ated adiponectin, mulitilocular subcutaneous adipose tissue (inguinal WAT) with upregulated oxidative

84 nd UCP1 protein expression in inguinal white adipose tissue (iWAT), a common site for emergent active

85 Bone marrow adipose tissue (MAT) is negatively associated with bone

86 ectively), as did the EPA and DHA content in adipose tissue (P < 0.0001 and P < 0.0001, respectively)

87 VAT and subcutaneous adipose tissue (SAT) samples obtained from subjects unde

88 d spectroscopy were used to measure visceral adipose tissue (VAT) and liver fat fraction (LFF) (total

89 on on food intake, body weight, and visceral adipose tissue (VAT) mass; plasma, lipids (cholesterol a

90 ion of some adipogenesis markers in visceral adipose tissue (VAT) of HFD-fed M-JAK2(-/-) mice.

91 and the main specific end point was visceral adipose tissue (VAT).

92 the study and biochemical analyses of white adipose tissue (WAT) and liver were performed.

93 2 knockdown also led to loss of dermal white adipose tissue (WAT) and markedly decreased abdominal WA

94 preferential expansion of subcutaneous white adipose tissue (WAT) appears protective.

95 on (SVF) under conditions that promote white adipose tissue (WAT) browning in mice.

96 White adipose tissue (WAT) can undergo a phenotypic switch, kn

97 s a rapid and persistent remodeling of white adipose tissue (WAT), an increase in energy expenditure

98 and programming of beige adipocytes in white adipose tissue (WAT).

99 The neurogenic potential of adipose tissue - derived human mesenchymal stem cells (h

100 nd visceral adipose tissue (epididymal white adipose tissue [WAT]), reduced WAT inflammation, elevate

101 , exhibited a striking age-dependent loss of adipose tissue accompanied by evidence of adipocyte deat

102 After infection, white adipose tissue accumulated large numbers of pathogen-spe

103 olume, Sost(-/-) mice exhibit a reduction in adipose tissue accumulation in association with increase

104 ts decreased lipogenic pathway in mesenteric adipose tissue after HFD and/or OVX, independent of prev

105 To what extent adipose tissue also contributes to immune surveillance a

106 ncoupling protein 1 expression in both white adipose tissue and 3T3-L1 differentiated adipocytes; in

107 beta3-adrenergic receptors to activate brown adipose tissue and by 'browning' white adipose tissue.

108 with an increase in sympathetic tone of the adipose tissue and expansion of activated macrophages, b

109 Monitoring phase transition in adipose tissue and formation of lipid crystals is import

110 increased numbers of B2 lymphocytes in obese adipose tissue and have shown that high-fat diet-induced

111 increase brain DHA, but increased the DHA in adipose tissue and heart.

112 to increased thermogenic activation of brown adipose tissue and induction of browning in WAT and coul

113 we show that ILC2 are present in para-aortic adipose tissue and lymph nodes and display an inflammato

114 ed an increased number of mast cells in both adipose tissue and the brain.

115 Abdominal adipose tissue and thigh muscle were segmented, and thei

116 Adoptive transfer of adipose tissue B2 cells (ATB2) from wild-type HFD donor

117 determine the thermogenic capacity of brown adipose tissue before environmental cold are unknown.

118 nd after treatment, the volunteers underwent adipose tissue biopsies to measure the total (CD68(+)),

119 Abdominal subcutaneous adipose tissue biopsy samples were collected for microar

120 n glucose metabolism, subcutaneous abdominal adipose tissue blood flow (ATBF), and lipid metabolism i

121 White adipose tissue bridges body organs and plays a fundament

122 tumor necrosis factor (TNF), was changed in adipose tissue by overfeeding.

123 ate-limiting transport of insulin across the adipose tissue capillaries is responsible for the slow s

124 n significantly reduced adipocyte apoptosis, adipose tissue collagen and macrophage accumulation as d

125 nsequence, dysfunction of these processes in adipose tissue compartments is tightly linked to severe

126 e, we have shown that at steady state, white adipose tissue contained abundant memory lymphocyte popu

127 For high (0.16%) compared with low (0.06%) adipose tissue content of EPA, the difference in 5-y wei

128 the associations between dietary intake and adipose tissue content of long-chain n-3 PUFAs and subse

129 glycemic index was found.Dietary intake and adipose tissue content of long-chain n-3 PUFAs were neit

130 Adipose tissue content of n-3 PUFAs was not associated w

131 ysis probes were implanted into the inguinal adipose tissue depot of C57BL6 mice.

132 abolic response to cold exposure in multiple adipose tissue depots in mice.

133 nexpectedly that GR is dispensable for brown adipose tissue development in mice.

134 le for adipogenesis in culture and for brown adipose tissue development in mice.

135 ersus adipogenic cell expansion during white adipose tissue development, with PDGFRalpha activity coo

136 licated in the regulation of white and brown adipose tissue differentiation.

137 Aging is accompanied by major changes in adipose tissue distribution and function.

138 LOPs) into blood from their storage in inert adipose tissue during rapid weight loss.

139 lase 3 (HDAC3) is required to activate brown adipose tissue enhancers to ensure thermogenic aptitude.

140 We conclude that adipose tissue eosinophils play a key role in the regula

141 ncreased NEFA storage capacity per volume of adipose tissue exactly compensated for the decrease in f

142 h targeted deletion of EPO receptor in white adipose tissue exhibited sex-differential phenotype in w

143 Adipose tissue expansion progresses rapidly during postn

144 Memory T cells in white adipose tissue expressed a distinct metabolic profile, a

145 rotein 4 and increased subcutaneous inguinal adipose tissue expression of adiponectin, but did not pr

146 from obesity-induced glucose intolerance and adipose tissue fibrosis.

147 l vascular fraction from periprostatic white adipose tissue from obese HiMyc mice at 6 months of age

148 ssed a distinct metabolic profile, and white adipose tissue from previously infected mice was suffici

149 known that 17-beta estradiol (E2) regulates adipose tissue function and VEGFA expression in other ti

150 d causes and consequences of obesity-related adipose tissue hypertrophy and hyperplasia for health, c

151 As) were increased and transplantation of Tg adipose tissue improved glucose tolerance in recipient m

152 high perinatal n-6/n-3 ratios, subcutaneous adipose tissue in 14-day-old wild-type pups receiving lo

153 To further investigate phase-transition in adipose tissue in microscopic level, an identical coolin

154 g growth factor beta1 (TGF-beta1) in mammary adipose tissue in obese mice activates SMAD3 signaling,

155 Expansion of adipose tissue in response to a positive energy balance

156 The role of adipose tissue in sensing and responding to emotional st

157 omic analysis of subcutaneous inguinal white adipose tissue in the absence of Egr1 identifies the mol

158 nhibition suppressed macrophage migration to adipose tissue in vitro.

159 etabolic/immune regulator linking obesity to adipose tissue inflammation and insulin resistance.

160 Adipose tissue inflammation is a central pathological el

161 ymph nodes, but the role of stromal cells in adipose tissue inflammation is unknown.

162 ameliorates adiposity, insulin sensitivity, adipose tissue inflammation, and arterial stiffness and

163 Consistent with reduced adipose tissue inflammation, cadherin-11-deficient mice

164 or of adipose tissue lipolysis, and impaired adipose tissue insulin action results in unrestrained li

165 In contrast, adipose tissue insulin sensitivity (suppression of free

166 Therefore, any observations of altered adipose tissue insulin sensitivity with extended morning

167 ance to the antilipolytic effect of insulin (adipose tissue IR [Adipo-IR]) in a large group of subjec

168 Human breast adipose tissue is a heterogeneous cell population consis

169 ever, the ability of E2 to regulate VEGFA in adipose tissue is currently unknown.

170 Adipose tissue is distributed in depots throughout the b

171 s genes for fat distribution are enriched in adipose tissue itself.

172 during increased hepatic lipogenesis only if adipose tissue lipid storage capacity is preserved.

173 eased GLUT4, increased ChREBP and markers of adipose tissue lipogenesis.

174 Insulin is a key regulator of adipose tissue lipolysis, and impaired adipose tissue in

175 in their livers and profound suppression of adipose tissue lipolysis, which decreases delivery of FA

176 cal pathways and mechanisms in (involuntary) adipose tissue loss as well as its systemic metabolic co

177 d postnatally in subcutaneous inguinal white adipose tissue lost thermogenic gene expression and mult

178 dy was to assess whether an increased atrial adipose tissue mass posterior to the left atrium is rela

179 Furthermore, the addition of the adipose tissue mass to the multiple variable analysis si

180 The adipose tissue mass was significantly larger in patients

181 Insulin regulates adipose tissue metabolism through direct effects on adip

182 ty and, to a lesser extent, the promotion of adipose tissue neutrophil recruitment and M1 polarizatio

183 TL) analyses by using abdominal subcutaneous adipose tissue of 770 extensively phenotyped participant

184 RK 1/2 signaling in 3T3-L1 adipocytes and in adipose tissue of mice.

185 in HDAC-deficient adipocytes as well as the adipose tissue of obese animals and humans.

186 ssion of a human-specific miRNA in the brown adipose tissue of one mouse in vivo can also regulate it

187 decreased in both subcutaneous and visceral adipose tissue of TRPC1 KO mice fed a HF diet and exerci

188 es involved in metabolic pathways in gonadal adipose tissue of WT and APNko, but this effect of DHT w

189 stress in the thyroid, but not in the brown adipose tissue or liver.

190 BS adipocytes, which are considered of white adipose tissue origin can shift towards a brown/beige ad

191 r energy metabolism, but their role in white adipose tissue physiology remains incompletely understoo

192 wild-type female mice, suggesting that white adipose tissue plays an integral role in mediating the m

193 Adipose tissue RBP4 expression and secretion remained in

194 itivity, and losing superficial subcutaneous adipose tissue remained neutral except for an associatio

195 l role for calpains in mediating HFD-induced adipose tissue remodeling by influencing multiple functi

196 Adipose tissue represents a critical component in health

197 Our results suggest that white adipose tissue represents a memory T cell reservoir that

198 Subcutaneous adipose tissue samples were collected and analyzed for g

199 e variable cellular composition of colon and adipose tissue samples, highlighting one use of these ce

200 llowed by deep sequencing) analyses in brown adipose tissue showed that EBF2 binds and regulates the

201 Obesity impairs the relaxant capacity of adipose tissue surrounding the vasculature (PVAT) and ha

202 critical capacity for thermogenesis in brown adipose tissue that can be rapidly engaged upon exposure

203 Regional sympathectomy compromises adipose tissue thermogenesis, and renders mice susceptib

204 diac muscle, white adipose tissue, and brown adipose tissue through a mechanism that was partially in

205 microRNA 140 (miR-140) expression in mammary adipose tissue through a novel negative-feedback loop.

206 Falpha signaling and lipid metabolism in the adipose tissue through modulation of Lys(63) ubiquitinat

207 the physiology and thermogenic properties of adipose tissue to reduce obesity even when mice are fed

208 Adipose tissue triacylglyceride (TAG) and glucose uptake

209 Dietary protein restriction increases adipose tissue uncoupling protein 1 (UCP1), energy expen

210 each gram increase of posterior left atrial adipose tissue was associated with 1.32 odds ratio of ha

211 Induction of recall responses within white adipose tissue was associated with the collapse of lipid

212 An increase in adipose tissue was independently associated with AF and

213 sensitivity of the skeletal muscle and white adipose tissue was lower in HFHS than control dams.

214 ng mice from body weight loss and muscle and adipose tissue wasting.

215 oved lipid profile, losing deep subcutaneous adipose tissue with improved insulin sensitivity, and lo

216 The omentum is a visceral adipose tissue with unique immune functions.

217 t of coronary artery calcium score, visceral adipose tissue, and 10-year global cardiovascular diseas

218 sing glucose uptake in cardiac muscle, white adipose tissue, and brown adipose tissue through a mecha

219 n consumption in white adipose tissue, brown adipose tissue, and hepatocytes.

220 ans and tissues, including the hypothalamus, adipose tissue, and skeletal muscle.

221 mong various organs: liver, skeletal muscle, adipose tissue, brain, and the endocrine pancreas.

222 In contrast to white adipose tissue, brown adipose tissue (BAT) is known to p

223 ed mitochondrial oxygen consumption in white adipose tissue, brown adipose tissue, and hepatocytes.

224 ice produces well-organized and vascularized adipose tissue, capable of beta-adrenergic-responsive gl

225 emic control, with increased browning of the adipose tissue, decreased gluconeogenesis, and less hepa

226 These mice, which initially developed adipose tissue, exhibited a striking age-dependent loss

227 onhepatic organs, including skin, brain, and adipose tissue, in neonatal rats without and after VA su

228 superficial layer of abdominal subcutaneous adipose tissue, increased visceral adipose tissue, marke

229 cutaneous adipose tissue, increased visceral adipose tissue, marked IR, dyslipidemia, and fatty liver

230 sive metabolic cross-talk between the liver, adipose tissue, pancreas and skeletal muscle.

231 reased the mean degree of DNA methylation in adipose tissue, particularly in promoter regions.

232 In adipose tissue, PER2 mRNA rhythms were delayed by 0.97 +

233 Chronic inflammation in adipose tissue, possibly related to adipose cell hypertr

234 In adipose tissue, rs11161721 is significantly associated w

235 Although it is primarily an adipose tissue, the omentum also contains lymphoid aggre

236 activity was selectively attenuated in JAK2L adipose tissue, whereas hepatic insulin signaling remain

237 nsporting fat, TGs also act as stored fat in adipose tissue, which is utilized during insufficient ca

238 ceived intravitreal injections of autologous adipose tissue-derived "stem cells" at one such clinic i

239 We here show that highly suppressive human adipose tissue-derived MSC (AdMSC) display and induce a

240 METHODS AND Adipose tissue-derived MSCs were isolated from atheroscl

241 Visceral adipose tissue-derived serpin (vaspin), serpin A12 of th

242 ncreasing appreciation for the importance of adipose tissue-mediated signals in HF development and fu

243 Adipose tissue-specific AnkB-KO mice develop obesity and

244 Mice with brown adipose tissue-specific genetic ablation of HDAC3 become

245 We found that adipose tissue-specific overexpression of Id1 causes age

246 ociated with abdominal adiposity function in adipose tissue.

247 th increased insulin signaling in muscle and adipose tissue.

248 gh the lymph node capsule into the perinodal adipose tissue.

249 negatively with expression of FGF21 in human adipose tissue.

250 factor in the regulation of angiogenesis in adipose tissue.

251 Ucp1 promoter in subcutaneous inguinal white adipose tissue.

252 tates communication between the skeleton and adipose tissue.

253 d obesity, and elicits the browning of white adipose tissue.

254 lves immune cell infiltration into expanding adipose tissue.

255 is the insect analog of vertebrate liver and adipose tissue.

256 signalling (IRS2) in subcutaneous abdominal adipose tissue.

257 ncreasing energy-utilizing thermogenic brown adipose tissue.

258 fibroproliferative cells, blood vessels, and adipose tissue.

259 ession levels and M2 macrophage expansion in adipose tissue.

260 the endogenous stereochemistry of 9-PAHSA in adipose tissue.

261 tromal cells are major producers of IL-33 in adipose tissue.

262 effects of maternal diet-induced obesity in adipose tissue.

263 ession of Tfe3, Tf3b, and Ppargamma in white adipose tissue.

264 to prevent excessive de novo lipogenesis in adipose tissue.

265 stem by dampening sympathetic outflow to the adipose tissue.

266 mRNA is linked to cholesterol metabolism in adipose tissue.

267 of insulin action and JAK/STAT signaling in adipose tissue.

268 21), and activation of signaling pathways in adipose tissue.

269 obesity and had significantly reduced white adipose tissue.

270 with areas of differential methylation in F4 adipose tissue.

271 sition (freezing/melting) in human abdominal adipose tissue.

272 known about the chemicals' effects on adult adipose tissue.

273 ic actions in the liver, skeletal muscle and adipose tissue.

274 nhanced insulin-stimulated Akt activation in adipose tissue.

275 ige adipocyte development in offspring white adipose tissue.

276 and human cultured adipocytes, as well as in adipose tissue.

277 brown adipose tissue and by 'browning' white adipose tissue.

278 ect of overfeeding on the DNA methylation in adipose tissue.The DNA methylation of 4875 Cytosine-phos

279 In agreement with these findings, adipose-tissue-resident macrophages did not express TH.

280 Here we show that mice with an adipose-tissue-specific knockout of the microRNA (miRNA)

281 calorimetry was performed and visceral white adipose tissues (VWAT) were assessed for inflammatory ce

282 energy expenditure, hyperplastic brown/white adipose tissues and larger hyperplastic hearts.

283 ATP2, CD36, and G6PC) in liver and abdominal adipose tissues as well as increased IRS1 phosphorylatio

284 g had lower thermogenesis in brown and white adipose tissues compared with CON offspring, which was r

285 predominant stereoisomer that accumulates in adipose tissues from transgenic mice where FAHFAs were f

286 Circulating MIR122 entered muscle and adipose tissues of mice, reducing mRNA levels of genes i

287 NKT cells from adipose tissues that do not express PLZF and those from

288 that expressed KCP in the kidney, liver, and adipose tissues were resistant to developing high-fat di

289 nnate and adaptive immune system residing in adipose tissues, as well as in the intestine, participat

290 mal fat accumulation in both white and brown adipose tissues, glucose intolerance and insulin resista

291 t body, a counterpart of mammalian liver and adipose tissues, is the metabolic center, playing a key

292 In adipose tissues, we observed a significant increase in R

293 n and liver of mice but not in the thymus or adipose tissues.

294 ir production of IL-4 in the white and brown adipose tissues.

295 LD loss via activation of cytosolic lipases adipose triglyceride lipase (ATGL) and hormone-sensitive

296 ion down-modulates LD catabolism mediated by adipose triglyceride lipase (ATGL), the key enzyme for i

297 syndrome, is a highly conserved regulator of adipose triglyceride lipase (ATGL)-mediated lipolysis th

298 Adipose type 1 innate lymphoid cells (AT1-ILCs) promote

299 Yet, the signals and mechanisms that govern adipose vascular niche formation and APC niche interacti

300 how that the assembly and maintenance of the adipose vascular niche is controlled by PPARgamma acting