ライフサイエンスコーパス: adipose tissue

1 ociated with abdominal adiposity function in adipose tissue.

2 gh the lymph node capsule into the perinodal adipose tissue.

3 negatively with expression of FGF21 in human adipose tissue.

4 Ucp1 promoter in subcutaneous inguinal white adipose tissue.

5 factor in the regulation of angiogenesis in adipose tissue.

6 tates communication between the skeleton and adipose tissue.

7 lves immune cell infiltration into expanding adipose tissue.

8 d obesity, and elicits the browning of white adipose tissue.

9 signalling (IRS2) in subcutaneous abdominal adipose tissue.

10 is the insect analog of vertebrate liver and adipose tissue.

11 ncreasing energy-utilizing thermogenic brown adipose tissue.

12 fibroproliferative cells, blood vessels, and adipose tissue.

13 ession levels and M2 macrophage expansion in adipose tissue.

14 the endogenous stereochemistry of 9-PAHSA in adipose tissue.

15 tromal cells are major producers of IL-33 in adipose tissue.

16 effects of maternal diet-induced obesity in adipose tissue.

17 ession of Tfe3, Tf3b, and Ppargamma in white adipose tissue.

18 to prevent excessive de novo lipogenesis in adipose tissue.

19 stem by dampening sympathetic outflow to the adipose tissue.

20 mRNA is linked to cholesterol metabolism in adipose tissue.

21 obesity and had significantly reduced white adipose tissue.

22 of insulin action and JAK/STAT signaling in adipose tissue.

23 ed obesity-induced inflammatory responses in adipose tissue.

24 to avoid fluorodeoxyglucose uptake in brown adipose tissue.

25 with areas of differential methylation in F4 adipose tissue.

26 kines and trafficking of M1 macrophages into adipose tissue.

27 n-related receptor alpha (ERRalpha) in brown adipose tissue.

28 morphogenesis were caused by the absence of adipose tissue.

29 ospholipids, plasma, cholesterol esters, and adipose tissue.

30 AP) is highly expressed in adrenal gland and adipose tissue.

31 ut microbiota and anti-inflammatory state in adipose tissue.

32 21), and activation of signaling pathways in adipose tissue.

33 sition (freezing/melting) in human abdominal adipose tissue.

34 known about the chemicals' effects on adult adipose tissue.

35 ic actions in the liver, skeletal muscle and adipose tissue.

36 nhanced insulin-stimulated Akt activation in adipose tissue.

37 ige adipocyte development in offspring white adipose tissue.

38 and human cultured adipocytes, as well as in adipose tissue.

39 brown adipose tissue and by 'browning' white adipose tissue.

40 th increased insulin signaling in muscle and adipose tissue.

41 ir production of IL-4 in the white and brown adipose tissues.

42 n and liver of mice but not in the thymus or adipose tissues.

43 , exhibited a striking age-dependent loss of adipose tissue accompanied by evidence of adipocyte deat

44 After infection, white adipose tissue accumulated large numbers of pathogen-spe

45 olume, Sost(-/-) mice exhibit a reduction in adipose tissue accumulation in association with increase

46 ts decreased lipogenic pathway in mesenteric adipose tissue after HFD and/or OVX, independent of prev

47 To what extent adipose tissue also contributes to immune surveillance a

48 ncoupling protein 1 expression in both white adipose tissue and 3T3-L1 differentiated adipocytes; in

49 ed hepatic steatosis and oxidative stress in adipose tissue and brain, and improved cognitive functio

50 beta3-adrenergic receptors to activate brown adipose tissue and by 'browning' white adipose tissue.

51 lular mitochondrial density, activates brown adipose tissue and enhances thermogenesis.

52 rogression is facilitated by IL4 secreted by adipose tissue and estrogen receptor-positive and triple

53 with an increase in sympathetic tone of the adipose tissue and expansion of activated macrophages, b

54 Monitoring phase transition in adipose tissue and formation of lipid crystals is import

55 increased numbers of B2 lymphocytes in obese adipose tissue and have shown that high-fat diet-induced

56 increase brain DHA, but increased the DHA in adipose tissue and heart.

57 to increased thermogenic activation of brown adipose tissue and induction of browning in WAT and coul

58 d extravascular fibrin deposits within white adipose tissue and liver as distinct features of mice fe

59 we show that ILC2 are present in para-aortic adipose tissue and lymph nodes and display an inflammato

60 ockdown in 3T3-L1 adipocytes and DHHC7 KO in adipose tissue and muscle decreased Glut4 palmitoylation

61 PPARdelta stimulates fatty acid oxidation in adipose tissue and skeletal muscle and improves dyslipid

62 tant to insulin-stimulated glucose uptake in adipose tissue and skeletal muscle compared with litterm

63 ed an increased number of mast cells in both adipose tissue and the brain.

64 , semiautomated method to quantify abdominal adipose tissue and thigh muscle volume and hepatic proto

65 bility and accuracy for estimating abdominal adipose tissue and thigh muscle volumes and hepatic PDFF

66 Abdominal adipose tissue and thigh muscle were segmented, and thei

67 energy expenditure, hyperplastic brown/white adipose tissues and larger hyperplastic hearts.

68 t of coronary artery calcium score, visceral adipose tissue, and 10-year global cardiovascular diseas

69 sing glucose uptake in cardiac muscle, white adipose tissue, and brown adipose tissue through a mecha

70 n consumption in white adipose tissue, brown adipose tissue, and hepatocytes.

71 protein 1-positive beige adipocytes in white adipose tissue, and increased thermogenesis in mice, whi

72 reduction in noradrenaline concentration in adipose tissue, and restored lipolysis with increased le

73 ans and tissues, including the hypothalamus, adipose tissue, and skeletal muscle.

74 total replacement of the crystalline lens by adipose tissue; and in a third, an anomalous pocket of a

75 ubertal mice overexpressing adiponectin from adipose tissue (APNtg), adiponectin knockouts (APNko), a

76 ATP2, CD36, and G6PC) in liver and abdominal adipose tissues as well as increased IRS1 phosphorylatio

77 with reduced macrophage counts within white adipose tissue, as well as near-complete protection from

78 nnate and adaptive immune system residing in adipose tissues, as well as in the intestine, participat

79 iated with chronic low-grade inflammation of adipose tissue (AT) and an increase of AT macrophages (A

80 hylene (p,p'-DDE), are bioaccumulated in the adipose tissue (AT) and have been implicated in the obes

81 haracterized by body weight loss, atrophy of adipose tissue (AT) and systemic inflammation.

82 The pericardial adipose tissue (AT) contains a high density of lymphoid

83 ss, improves insulin sensitivity, and alters adipose tissue (AT) gene expression, yet the relation wi

84 Adipose tissue (AT) is no longer regarded as an inert li

85 Adoptive transfer of adipose tissue B2 cells (ATB2) from wild-type HFD donor

86 orchestrates lipoprotein processing in brown adipose tissue (BAT) and hepatic conversion of cholester

87 factor Hlx is selectively expressed in brown adipose tissue (BAT) and iWAT, and is translationally up

88 However, the role of brown adipose tissue (BAT) in regulating gestational metabolis

89 a) neurons influences thermogenesis of brown adipose tissue (BAT) independent of ambient temperature

90 Brown adipose tissue (BAT) is an attractive therapeutic target

91 In contrast to white adipose tissue, brown adipose tissue (BAT) is known to play critical roles for

92 Brown adipose tissue (BAT) is regulated by the sympathetic ner

93 Detection and quantification of brown adipose tissue (BAT) mass remains a major challenge, as

94 Brown adipose tissue (BAT) mitochondria exhibit high oxidative

95 aging is routinely used to investigate brown adipose tissue (BAT) thermogenesis, which requires mitoc

96 Brown adipose tissue (BAT) utilizes glucose and free fatty aci

97 ion impairs retinoic acid signaling in brown adipose tissue (BAT), leading to impaired BAT function a

98 r neurons controlling thermogenesis of brown adipose tissue (BAT).

99 CP1 mRNAs were not induced in liver or brown adipose tissue (BAT).

100 ining the integrity of mitochondria in brown adipose tissue (BAT).

101 a robust and specific PD-L1 signal in brown adipose tissue (BAT).

102 determine the thermogenic capacity of brown adipose tissue before environmental cold are unknown.

103 nd after treatment, the volunteers underwent adipose tissue biopsies to measure the total (CD68(+)),

104 Abdominal subcutaneous adipose tissue biopsy samples were collected for microar

105 n glucose metabolism, subcutaneous abdominal adipose tissue blood flow (ATBF), and lipid metabolism i

106 cated within the perivascular compartment of adipose tissue blood vessels.

107 mong various organs: liver, skeletal muscle, adipose tissue, brain, and the endocrine pancreas.

108 White adipose tissue bridges body organs and plays a fundament

109 In contrast to white adipose tissue, brown adipose tissue (BAT) is known to p

110 ed mitochondrial oxygen consumption in white adipose tissue, brown adipose tissue, and hepatocytes.

111 as a powerful regulator for systematic white adipose tissue browning and offer molecular insights int

112 tumor necrosis factor (TNF), was changed in adipose tissue by overfeeding.

113 Brown and beige adipose tissues can catabolize stored energy to generate

114 ice produces well-organized and vascularized adipose tissue, capable of beta-adrenergic-responsive gl

115 ate-limiting transport of insulin across the adipose tissue capillaries is responsible for the slow s

116 n significantly reduced adipocyte apoptosis, adipose tissue collagen and macrophage accumulation as d

117 g had lower thermogenesis in brown and white adipose tissues compared with CON offspring, which was r

118 nsequence, dysfunction of these processes in adipose tissue compartments is tightly linked to severe

119 e, we have shown that at steady state, white adipose tissue contained abundant memory lymphocyte popu

120 For high (0.16%) compared with low (0.06%) adipose tissue content of EPA, the difference in 5-y wei

121 Adipose tissue content of fatty acids was determined by

122 the associations between dietary intake and adipose tissue content of long-chain n-3 PUFAs and subse

123 glycemic index was found.Dietary intake and adipose tissue content of long-chain n-3 PUFAs were neit

124 Adipose tissue content of n-3 PUFAs was not associated w

125 emic control, with increased browning of the adipose tissue, decreased gluconeogenesis, and less hepa

126 ysis probes were implanted into the inguinal adipose tissue depot of C57BL6 mice.

127 abolic response to cold exposure in multiple adipose tissue depots in mice.

128 The neurogenic potential of adipose tissue - derived human mesenchymal stem cells (h

129 ceived intravitreal injections of autologous adipose tissue-derived "stem cells" at one such clinic i

130 We here show that highly suppressive human adipose tissue-derived MSC (AdMSC) display and induce a

131 METHODS AND Adipose tissue-derived MSCs were isolated from atheroscl

132 Visceral adipose tissue-derived serpin (vaspin), serpin A12 of th

133 nexpectedly that GR is dispensable for brown adipose tissue development in mice.

134 le for adipogenesis in culture and for brown adipose tissue development in mice.

135 ersus adipogenic cell expansion during white adipose tissue development, with PDGFRalpha activity coo

136 licated in the regulation of white and brown adipose tissue differentiation.

137 Aging is accompanied by major changes in adipose tissue distribution and function.

138 LOPs) into blood from their storage in inert adipose tissue during rapid weight loss.

139 is accompanied by attrition of dermal white adipose tissue (dWAT) and reduced levels of circulating

140 The abundance of epicardial adipose tissue (EAT) is associated with atrial fibrillat

141 lase 3 (HDAC3) is required to activate brown adipose tissue enhancers to ensure thermogenic aptitude.

142 We conclude that adipose tissue eosinophils play a key role in the regula

143 nced insulin signaling in liver and visceral adipose tissue (epididymal white adipose tissue [WAT]),

144 is study we aimed to define the relevance of adipose tissue ERalpha during high-fat diet (HFD)-induce

145 In epididymal white adipose tissue (eWAT) of PDE3B KO mice on a SvJ129 backg

146 ncreased NEFA storage capacity per volume of adipose tissue exactly compensated for the decrease in f

147 h targeted deletion of EPO receptor in white adipose tissue exhibited sex-differential phenotype in w

148 These mice, which initially developed adipose tissue, exhibited a striking age-dependent loss

149 Adipose tissue expansion progresses rapidly during postn

150 eta disrupts APC niche contact thus blocking adipose tissue expansion.

151 Memory T cells in white adipose tissue expressed a distinct metabolic profile, a

152 rotein 4 and increased subcutaneous inguinal adipose tissue expression of adiponectin, but did not pr

153 ive and anti-inflammatory effects and induce adipose tissue (fat) to produce the vaso-protective prot

154 from obesity-induced glucose intolerance and adipose tissue fibrosis.

155 l vascular fraction from periprostatic white adipose tissue from obese HiMyc mice at 6 months of age

156 ssed a distinct metabolic profile, and white adipose tissue from previously infected mice was suffici

157 predominant stereoisomer that accumulates in adipose tissues from transgenic mice where FAHFAs were f

158 known that 17-beta estradiol (E2) regulates adipose tissue function and VEGFA expression in other ti

159 adipose tissue neutrophils and M2-polarized adipose tissue gene expression.

160 mal fat accumulation in both white and brown adipose tissues, glucose intolerance and insulin resista

161 d oxidation in mouse brown, beige, and white adipose tissues; however, the cellular basis of this dua

162 d causes and consequences of obesity-related adipose tissue hypertrophy and hyperplasia for health, c

163 As) were increased and transplantation of Tg adipose tissue improved glucose tolerance in recipient m

164 high perinatal n-6/n-3 ratios, subcutaneous adipose tissue in 14-day-old wild-type pups receiving lo

165 ration, except minimal infiltration in white adipose tissue in animals treated with the highest BRD33

166 Maternal FO consumption enriched chick adipose tissue in EPA and DHA and reduced adiposity by p

167 To further investigate phase-transition in adipose tissue in microscopic level, an identical coolin

168 g growth factor beta1 (TGF-beta1) in mammary adipose tissue in obese mice activates SMAD3 signaling,

169 Expansion of adipose tissue in response to a positive energy balance

170 The role of adipose tissue in sensing and responding to emotional st

171 omic analysis of subcutaneous inguinal white adipose tissue in the absence of Egr1 identifies the mol

172 ssue; and in a third, an anomalous pocket of adipose tissue in the central vitreous.

173 nhibition suppressed macrophage migration to adipose tissue in vitro.

174 onhepatic organs, including skin, brain, and adipose tissue, in neonatal rats without and after VA su

175 superficial layer of abdominal subcutaneous adipose tissue, increased visceral adipose tissue, marke

176 onical WNT ligand, has been shown to promote adipose tissue inflammation and insulin resistance in an

177 etabolic/immune regulator linking obesity to adipose tissue inflammation and insulin resistance.

178 fibroblasts expressing cadherin-11 regulate adipose tissue inflammation and thus highlight cadherin-

179 Adipose tissue inflammation is a central pathological el

180 ymph nodes, but the role of stromal cells in adipose tissue inflammation is unknown.

181 significantly improved blood lipid profile, adipose tissue inflammation, and aortic stiffness of LCR

182 ameliorates adiposity, insulin sensitivity, adipose tissue inflammation, and arterial stiffness and

183 Consistent with reduced adipose tissue inflammation, cadherin-11-deficient mice

184 f the Ankrd26 gene, which contributes to the adipose tissue inflammatory secretion profile induced by

185 ated adiponectin, mulitilocular subcutaneous adipose tissue (inguinal WAT) with upregulated oxidative

186 or of adipose tissue lipolysis, and impaired adipose tissue insulin action results in unrestrained li

187 h with prediabetes, the relationship between adipose tissue insulin sensitivity (ATIS) and beta-cell

188 In contrast, adipose tissue insulin sensitivity (suppression of free

189 Therefore, any observations of altered adipose tissue insulin sensitivity with extended morning

190 period, we collected data on peripheral and adipose tissue insulin sensitivity, fecal microbiota com

191 ance to the antilipolytic effect of insulin (adipose tissue IR [Adipo-IR]) in a large group of subjec

192 Human breast adipose tissue is a heterogeneous cell population consis

193 ever, the ability of E2 to regulate VEGFA in adipose tissue is currently unknown.

194 Adipose tissue is distributed in depots throughout the b

195 Therefore, JAK2 in adipose tissue is epistatic to liver with regard to insu

196 Hypertrophic remodeling of white adipose tissues is associated with overexposure of lean

197 t body, a counterpart of mammalian liver and adipose tissues, is the metabolic center, playing a key

198 s genes for fat distribution are enriched in adipose tissue itself.

199 nd UCP1 protein expression in inguinal white adipose tissue (iWAT), a common site for emergent active

200 during increased hepatic lipogenesis only if adipose tissue lipid storage capacity is preserved.

201 eased GLUT4, increased ChREBP and markers of adipose tissue lipogenesis.

202 Insulin is a key regulator of adipose tissue lipolysis, and impaired adipose tissue in

203 in their livers and profound suppression of adipose tissue lipolysis, which decreases delivery of FA

204 Epicardial adipose tissue located close to the atrial wall can chan

205 cal pathways and mechanisms in (involuntary) adipose tissue loss as well as its systemic metabolic co

206 d postnatally in subcutaneous inguinal white adipose tissue lost thermogenic gene expression and mult

207 h demonstrated ER stress-induced rewiring of adipose tissue macrophage polarization by IRE1alpha acti

208 Here we show that adipose tissue macrophages regulate the age-related redu

209 In db/db-PI3Kgamma(-/-) mice, the number of adipose tissue macrophages was similar to control, but d

210 cutaneous adipose tissue, increased visceral adipose tissue, marked IR, dyslipidemia, and fatty liver

211 Posterior left atrial adipose tissue mass is significantly larger in patients

212 dy was to assess whether an increased atrial adipose tissue mass posterior to the left atrium is rela

213 Furthermore, the addition of the adipose tissue mass to the multiple variable analysis si

214 The posterior left atrial adipose tissue mass was quantified on computed tomograph

215 The adipose tissue mass was significantly larger in patients

216 999A mice exhibited low body weight, reduced adipose tissue mass, and increased lifespan, similar to

217 Bone marrow adipose tissue (MAT) is negatively associated with bone

218 ncreasing appreciation for the importance of adipose tissue-mediated signals in HF development and fu

219 Insulin regulates adipose tissue metabolism through direct effects on adip

220 ty and, to a lesser extent, the promotion of adipose tissue neutrophil recruitment and M1 polarizatio

221 as similar to control, but displayed reduced adipose tissue neutrophils and M2-polarized adipose tiss

222 Our data indicate that APCs direct adipose tissue niche expansion via a PPARgamma-initiated

223 TL) analyses by using abdominal subcutaneous adipose tissue of 770 extensively phenotyped participant

224 RK 1/2 signaling in 3T3-L1 adipocytes and in adipose tissue of mice.

225 in HDAC-deficient adipocytes as well as the adipose tissue of obese animals and humans.

226 ssion of a human-specific miRNA in the brown adipose tissue of one mouse in vivo can also regulate it

227 decreased in both subcutaneous and visceral adipose tissue of TRPC1 KO mice fed a HF diet and exerci

228 es involved in metabolic pathways in gonadal adipose tissue of WT and APNko, but this effect of DHT w

229 Circulating MIR122 entered muscle and adipose tissues of mice, reducing mRNA levels of genes i

230 stress in the thyroid, but not in the brown adipose tissue or liver.

231 GFRalpha activation inhibits embryonic white adipose tissue organogenesis in a tissue-autonomous mann

232 However, the function of PDGFRalpha during adipose tissue organogenesis is unknown.

233 BS adipocytes, which are considered of white adipose tissue origin can shift towards a brown/beige ad

234 ectively), as did the EPA and DHA content in adipose tissue (P < 0.0001 and P < 0.0001, respectively)

235 sive metabolic cross-talk between the liver, adipose tissue, pancreas and skeletal muscle.

236 reased the mean degree of DNA methylation in adipose tissue, particularly in promoter regions.

237 In adipose tissue, PER2 mRNA rhythms were delayed by 0.97 +

238 r energy metabolism, but their role in white adipose tissue physiology remains incompletely understoo

239 wild-type female mice, suggesting that white adipose tissue plays an integral role in mediating the m

240 Chronic inflammation in adipose tissue, possibly related to adipose cell hypertr

241 Poor vascularization during adipose tissue proliferation causes fibrosis and local i

242 Adipose tissue RBP4 expression and secretion remained in

243 itivity, and losing superficial subcutaneous adipose tissue remained neutral except for an associatio

244 l role for calpains in mediating HFD-induced adipose tissue remodeling by influencing multiple functi

245 Adipose tissue represents a critical component in health

246 Our results suggest that white adipose tissue represents a memory T cell reservoir that

247 In agreement with these findings, adipose-tissue-resident macrophages did not express TH.

248 In adipose tissue, rs11161721 is significantly associated w

249 Subcutaneous adipose tissue samples were collected and analyzed for g

250 e variable cellular composition of colon and adipose tissue samples, highlighting one use of these ce

251 VAT and subcutaneous adipose tissue (SAT) samples obtained from subjects unde

252 llowed by deep sequencing) analyses in brown adipose tissue showed that EBF2 binds and regulates the

253 Adipose tissue-specific AnkB-KO mice develop obesity and

254 Mice with brown adipose tissue-specific genetic ablation of HDAC3 become

255 We found that adipose tissue-specific overexpression of Id1 causes age

256 Here we show that mice with an adipose-tissue-specific knockout of the microRNA (miRNA)

257 Obesity impairs the relaxant capacity of adipose tissue surrounding the vasculature (PVAT) and ha

258 critical capacity for thermogenesis in brown adipose tissue that can be rapidly engaged upon exposure

259 NKT cells from adipose tissues that do not express PLZF and those from

260 Although it is primarily an adipose tissue, the omentum also contains lymphoid aggre

261 ect of overfeeding on the DNA methylation in adipose tissue.The DNA methylation of 4875 Cytosine-phos

262 Regional sympathectomy compromises adipose tissue thermogenesis, and renders mice susceptib

263 diac muscle, white adipose tissue, and brown adipose tissue through a mechanism that was partially in

264 microRNA 140 (miR-140) expression in mammary adipose tissue through a novel negative-feedback loop.

265 Falpha signaling and lipid metabolism in the adipose tissue through modulation of Lys(63) ubiquitinat

266 the physiology and thermogenic properties of adipose tissue to reduce obesity even when mice are fed

267 Adipose tissue triacylglyceride (TAG) and glucose uptake

268 Dietary protein restriction increases adipose tissue uncoupling protein 1 (UCP1), energy expen

269 d spectroscopy were used to measure visceral adipose tissue (VAT) and liver fat fraction (LFF) (total

270 on on food intake, body weight, and visceral adipose tissue (VAT) mass; plasma, lipids (cholesterol a

271 ion of some adipogenesis markers in visceral adipose tissue (VAT) of HFD-fed M-JAK2(-/-) mice.

272 and the main specific end point was visceral adipose tissue (VAT).

273 calorimetry was performed and visceral white adipose tissues (VWAT) were assessed for inflammatory ce

274 each gram increase of posterior left atrial adipose tissue was associated with 1.32 odds ratio of ha

275 Induction of recall responses within white adipose tissue was associated with the collapse of lipid

276 ll activation in the spleen and perivascular adipose tissue was blunted in Tcrdelta(-/-) mice (P<0.01

277 An increase in adipose tissue was independently associated with AF and

278 sensitivity of the skeletal muscle and white adipose tissue was lower in HFHS than control dams.

279 ng mice from body weight loss and muscle and adipose tissue wasting.

280 the study and biochemical analyses of white adipose tissue (WAT) and liver were performed.

281 2 knockdown also led to loss of dermal white adipose tissue (WAT) and markedly decreased abdominal WA

282 preferential expansion of subcutaneous white adipose tissue (WAT) appears protective.

283 on (SVF) under conditions that promote white adipose tissue (WAT) browning in mice.

284 White adipose tissue (WAT) can undergo a phenotypic switch, kn

285 phosphatase, was induced in epididymal white adipose tissue (WAT) in response to diet-induced obesity

286 otype (CD45-CD34(+)) resident in human white adipose tissue (WAT) is known to promote the progression

287 on of beige adipocytes is increased in white adipose tissue (WAT) reflects a potential strategy in th

288 Glucocorticoids promote lipolysis in white adipose tissue (WAT) to adapt to energy demands under st

289 s a rapid and persistent remodeling of white adipose tissue (WAT), an increase in energy expenditure

290 and programming of beige adipocytes in white adipose tissue (WAT).

291 activator Prdm16 regulates browning of white adipose tissue (WAT).

292 erest in thermogenesis and browning of white adipose tissue (WAT).

293 nd visceral adipose tissue (epididymal white adipose tissue [WAT]), reduced WAT inflammation, elevate

294 In adipose tissues, we observed a significant increase in R

295 stasis, and higher body weight and abdominal adipose tissue weight were observed in male offspring of

296 that expressed KCP in the kidney, liver, and adipose tissues were resistant to developing high-fat di

297 activity was selectively attenuated in JAK2L adipose tissue, whereas hepatic insulin signaling remain

298 nsporting fat, TGs also act as stored fat in adipose tissue, which is utilized during insufficient ca

299 oved lipid profile, losing deep subcutaneous adipose tissue with improved insulin sensitivity, and lo

300 The omentum is a visceral adipose tissue with unique immune functions.