ライフサイエンスコーパス: adjacent cell

1 GF signaling in trans from the surface of an adjacent cell.

2 h that each cell interacted with exactly one adjacent cell.

3 g the anticlinal pavement cell walls between adjacent cells.

4 w bacteria to exit from host cells to infect adjacent cells.

5 ane proteins that form gap junctions between adjacent cells.

6 all positively charged ions to cross between adjacent cells.

7 raction between the contractile ring and the adjacent cells.

8 elium disrupts AJs but only when depleted in adjacent cells.

9 f the plasma membrane, which are engulfed by adjacent cells.

10 to the apoplasm, and subsequent import into adjacent cells.

11 ght asymmetries in the Dl or N activities of adjacent cells.

12 passage of ions and small molecules between adjacent cells.

13 enic requirements and negative regulation by adjacent cells.

14 es specifying the probabilities of moving to adjacent cells.

15 align themselves with similar structures in adjacent cells.

16 ch, enabling them to trans-activate Notch on adjacent cells.

17 sion follows reception of the Wnt8 ligand by adjacent cells.

18 l and are aligned with similar structures in adjacent cells.

19 ve lost viability when the fungus moved into adjacent cells.

20 n the extracellular matrix or the surface of adjacent cells.

21 ular channels that connect the cytoplasms of adjacent cells.

22 forms due to a difference in Notch levels in adjacent cells.

23 gnate receptors expressed within the same or adjacent cells.

24 the proximodistal junctional region between adjacent cells.

25 may be involved in the communication between adjacent cells.

26 asm; however, mutant virus did not spread to adjacent cells.

27 cal microtubules and actin filaments between adjacent cells.

28 innervation may influence gene expression in adjacent cells.

29 in from the cytoplasm to the cell surface of adjacent cells.

30 elopment by regulating communication between adjacent cells.

31 iate the gated transfer of molecules between adjacent cells.

32 shape by linking the actin cytoskeletons of adjacent cells.

33 unctional hemichannels, one from each of the adjacent cells.

34 e TMV replication immediately after entry to adjacent cells.

35 ing ions and small molecules to move between adjacent cells.

36 ing appears necessary for synchronization of adjacent cells.

37 syndecan-2-mediated inside-out signaling to adjacent cells.

38 ion of proteins and their ability to move to adjacent cells.

39 ssion of nectin-1 in trans on the surface of adjacent cells.

40 m and propelled by actin-based motility into adjacent cells.

41 one cell may activate signaling processes in adjacent cells.

42 nt cells by interconnecting the cytoplasm of adjacent cells.

43 with its transmembrane ligands expressed on adjacent cells.

44 specific sites of contact (plaques) between adjacent cells.

45 hrinA1, which is anchored to the membrane of adjacent cells.

46 migrates and accumulates at lower levels in adjacent cells.

47 tion and the coalescing of cell cytoplasm of adjacent cells.

48 le tethers that bind to surface receptors on adjacent cells.

49 erentiation and promote the lignification of adjacent cells.

50 f attachment of the myofilament apparatus to adjacent cells.

51 s and their complex interaction with MCTs in adjacent cells.

52 ell adhesion by linking the cytoskeletons of adjacent cells.

53 ons, here termed listeriopods, and spread to adjacent cells.

54 ent cell separation and hydraulic changes in adjacent cells.

55 r protoplast and at the radial walls between adjacent cells.

56 walls and interconnect the cytoplasm between adjacent cells.

57 orm by defective cytokinesis or by fusion of adjacent cells.

58 te electrical and metabolic coupling between adjacent cells.

59 en Vm depolarization and spike discharges in adjacent cells.

60 ol, and oxidation to 11-cis-retinal occur in adjacent cells.

61 evels of WUS protein in the rib meristem and adjacent cells.

62 oplasm and, ultimately, spread directly into adjacent cells.

63 many cell diameters to directly contact non-adjacent cells.

64 ght to be transmembrane ligands expressed on adjacent cells.

65 bacteria through the cell cytoplasm and into adjacent cells.

66 ile ActA is required for bacterial spread to adjacent cells.

67 e receptor activated by ligands expressed on adjacent cells.

68 lasma membrane protrusions that project into adjacent cells.

69 creases were frequently observed in pairs of adjacent cells.

70 ls and/or coordination of this process among adjacent cells.

71 idly diffused from the microinjected cell to adjacent cells.

72 induces ectopic neuronal differentiation in adjacent cells.

73 her Gn1-R acts non-autonomously to influence adjacent cells.

74 llular matrix, or tethered to the surface of adjacent cells.

75 correlation was found between the events in adjacent cells.

76 the rib meristem and lower nuclear levels in adjacent cells.

77 lation was found between the firing sites in adjacent cells.

78 preserving stable adherens junctions between adjacent cells.

79 omeostasis, supporting communication between adjacent cells.

80 the host cytosol and for bacterial spread to adjacent cells.

81 g channels that penetrate the septum between adjacent cells.

82 mediate intercellular communication between adjacent cells.

83 chemical cues in both stimulated tissues and adjacent cells.

84 both within the plasma membrane and between adjacent cells.

85 r by the adhesion protein surface-exposed in adjacent cells.

86 hanically couples the actin cytoskeletons of adjacent cells.

87 itude and diversity between HCV-infected and adjacent cells.

88 by interacting in the intercellular space of adjacent cells.

89 escaped and gained access to the cytosol of adjacent cells.

90 ls controlling behavioral output relative to adjacent cells.

91 e amnioserosa are apparently asynchronous in adjacent cells.

92 to translocate toxic effector proteins into adjacent cells.

93 axons and failed to spread from axon tips to adjacent cells.

94 trongly guided by physical forces imposed by adjacent cells.

95 evolution and mediates communication between adjacent cells.

96 spread from the silencing initiation site to adjacent cells.

97 nautonomously regulated by the properties of adjacent cells.

98 on the surface, and wide connection with the adjacent cells.

99 mpact the expression of these genes in these adjacent cells.

100 herin that forms homodimeric bridges between adjacent cells.

101 sin, and intercellular adhesions interfacing adjacent cells.

102 transfer of ions and small molecules between adjacent cells.

103 with both the extracellular matrix (ECM) and adjacent cells.

104 through linked hemichannels from each of two adjacent cells.

105 e ability of HIV-1 to mediate fusion between adjacent cells, a process referred to as "fusion-from-wi

106 Adjacent cells adopted alteration in their expression pr

107 nt, they also function as extensions between adjacent cells along cell-cell junctions.

108 ons that allow exchange of molecules between adjacent cells also seem to have an important role.

109 of an electrical axo-axonal coupling between adjacent cells and a reciprocal inhibition between the m

110 junction channels span the membranes of two adjacent cells and allow the gated transit of molecules

111 lls direct the spindle orientations of their adjacent cells and are consistent with a mechanism of or

112 nd messengers, and small metabolites between adjacent cells and are formed by two unrelated protein f

113 he mid-pericarp layer (MPL), are larger than adjacent cells and are highly branched.

114 dium and these vesicles can communicate with adjacent cells and even cells of the immune system.

115 cribed in glioma cells can be transferred to adjacent cells and induces targeted inhibition of protei

116 two processes: cell-to-cell movement between adjacent cells and long-distance movement that allows th

117 components, and possibly other proteins, in adjacent cells and may therefore directly mediate these

118 dherins function to promote adhesion between adjacent cells and play critical roles in such cellular

119 lasma membrane protrusions that project into adjacent cells and resolve into vacuoles from which the

120 Cellular response to physical cues from adjacent cells and the extracellular matrix leads to a d

121 interactions with other surface molecules on adjacent cells and through these interactions initiate s

122 sion of sessile epithelial cells attached to adjacent cells and to the basement membrane into dissoci

123 tution by stem cells, paracrine effects from adjacent cells, and circulating anti-aging molecules may

124 y measuring gap sizes, angles formed between adjacent cells, and curvatures of cell surfaces at gaps.

125 ular expansion and coordination of growth of adjacent cells, and defects in karyokinesis and cytokine

126 ion, intracellular multiplication, spread to adjacent cells, and induction of brisk inflammatory resp

127 th the cell surface, can act on unstimulated adjacent cells, and is specifically inhibited by TIMP-3,

128 ell-fate decisions--including cell death--in adjacent cells, and raise the possibility that they do s

129 ic organization, membrane apposition between adjacent cells, and structure and distribution of pili a

130 this localization is not coordinated between adjacent cells, and the crescents subsequently are lost

131 1 in PECs can also affect gene expression in adjacent cells, and this effect is abolished by the T164

132 chsous (Ds), which form heterodimers between adjacent cells; and the Golgi kinase Four-jointed (Fj),

133 move laterally, they are pushed sideways by adjacent cells; and when cells move forward, they are pu

134 hesions of cells to extracellular matrix and adjacent cells are mediated by integrins and VE-cadherin

135 polarized'), or when tight junctions between adjacent cells are opened, heregulin-alpha activates its

136 Remarkably, the sarcomeres of adjacent cells are precisely paired across the junctiona

137 Fate maps show that polarizing region and adjacent cells are specified in parallel through a serie

138 ane channels, which connect the cytoplasm of adjacent cells, are composed of connexins.

139 ular matrix proteins and counterreceptors on adjacent cells, are implicated in synapse maturation and

140 tem, and find that they are not expressed in adjacent cells as would have been expected.

141 m of an infected cell suddenly flows into an adjacent cell, as visualized through wild-type MV-expres

142 Groups averaging three adjacent cells at a later stage can contribute progeny w

143 Shigella protrusions invade adjacent cells at junctions where three cells meet, the

144 rectly through information transmission from adjacent cells at the peripheral innervation area.

145 secreted Nkx3.1 protein can translocate into adjacent cells, bind to the regulatory sequence of Nkx3.

146 n the overexpressing cell and in immediately adjacent cells but not in cells farther away.

147 rmed membrane protrusions that extended into adjacent cells, but the protrusions rarely resolved into

148 olipases C (PLCs) also resulted in spread to adjacent cells, but these triple mutants became trapped

149 influence the effects of mineral coatings on adjacent cells, but to date it has not been practical to

150 g the propagation of second messengers among adjacent cells by connexin43-gap junctions.

151 ving epithelial cell to adapt to the loss of adjacent cells by dedifferentiating and proliferating.

152 litates transport of virus infection between adjacent cells by modifying plasmodesmata.

153 hannels (GJCs) that connect the cytoplasm of adjacent cells by the end-to-end docking of two Cx hemic

154 r matrix (ECM), or cell-surface molecules on adjacent cells can activate signaling pathways that comm

155 region of the tailbud, very small groups of adjacent cells can contribute progeny to the neural tube

156 we discuss recent work that shows that tumor-adjacent cells can respond to genotoxic stress by activa

157 Strikingly, adjacent cells can take up DEK secreted from synovial ma

158 systematic map of the visual field, in which adjacent cell clusters represent adjacent points of visu

159 lls indicated the presence of Cx31.9 between adjacent cells, consistent with its localization to gap

160 res maintenance of adhesive contacts between adjacent cells, coordination of polarized cell protrusio

161 ructs causes induction of ectopic Endo 16 in adjacent cells, demonstrating further that Pmar1 control

162 In this way, cycling cells favor regrowth in adjacent cells deprived of polyamines.

163 The molecular mechanism to understand why adjacent cells develop into bile ducts or grow actively

164 sition zymogen granules, or communicate with adjacent cells, disrupting pancreatic function.

165 Although adjacent cells express both Dl ligand and N receptor, si

166 lt in a checkerboard spatial pattern whereby adjacent cells express opposing levels of Notch and Delt

167 is of cortical peritubular capillaries, with adjacent cells expressing pericyte-like markers; we spec

168 However, bacteria in adjacent cells failed to multiply and were found in doub

169 rm and are required for the specification of adjacent cell fates.

170 that is critical for inducing and patterning adjacent cell fates.

171 e cell lineage as the guard cells or from an adjacent cell file.

172 fter breakdown of primary cell walls between adjacent cell files at the site of detachment.

173 Delimiting LAX3 expression to two adjacent cell files overlying new LRP is crucial to ensu

174 al cells while the outer cell, together with adjacent cells, forms the primary parietal layer.

175 erent ability of adherent cells to move into adjacent cell-free areas, thus providing information on

176 aA expressed by hair cells normally prevents adjacent cells from adopting the same cell fate, and tha

177 els that physically connect the cytoplasm of adjacent cells, gap junctions permit transport of a dive

178 diate fusion between the plasma membranes of adjacent cells, HIV-1 enters through an endocytic pathwa

179 rmation and movement of VRCs was repeated in adjacent cells in 3-4 h vs. 20 h from primary infected c

180 By acting as a dynamic link between adjacent cells in a monolayer, adherens junctions (AJs)

181 vesicular stomatitis virus (VSV) spread, to adjacent cells in a monolayer.

182 OA cartilage reflect the increased number of adjacent cells in clusters that are able to interact dir

183 scopy confirmed disorganized capillaries and adjacent cells in cortical peritubular spaces in mutant

184 ting the transfer of small molecules between adjacent cells in culture, indicating its potential role

185 tion cell death suppress RNA interference in adjacent cells in Drosophila melanogaster.

186 induced a physical separation of the TJs of adjacent cells in immunofluorescence staining of the TJ

187 t neurons, only JHM.SD spread efficiently to adjacent cells in the absence of CEACAM1a.

188 Hh), a protein which induces a thin strip of adjacent cells in the anterior compartment to express th

189 -localize in the same cells but localized in adjacent cells in the colon.

190 Although cytoplasmic bridges between adjacent cells in the filaments of nitrogen-fixing cyano

191 sin-dependent cell contraction or cross-link adjacent cells in the monolayer.

192 g rapid exchange of Tf molecules between two adjacent cells in the monolayer.

193 actions, and (2) interactions are limited to adjacent cells in the mosaic.

194 of cyclic gene expression coordination among adjacent cells in the presomitic mesoderm.

195 ected by three promoters and this results in adjacent cells in the same tissue containing different i

196 lcholine that triggers the depolarization of adjacent cells in the spinal cord.

197 glycolysis, and provides a source of FA for adjacent cells in the vessel wall or tissues.

198 separated by a completed peptidoglycan wall, adjacent cells in these chains were often compartmentali

199 osely with the extracellular matrix and with adjacent cells including pericytes and smooth muscle cel

200 ceptors to ephrinB ligands on the surface of adjacent cells initiates signaling cascades that regulat

201 oordinating growth and communication between adjacent cells is a critical yet poorly understood aspec

202 tems indicate that localised signalling from adjacent cells is a general mechanism for defining the p

203 icles into axons and extracellular spread to adjacent cells is fundamentally important for identifyin

204 Gap junction communication between adjacent cells is known to be vital during embryogenesis

205 t that homophilic interaction of Echinoid on adjacent cells is necessary for its function.

206 The resulting polarization in adjacent cells is opposite to that within the stimulated

207 issemination of virus from infected cells to adjacent cells is promoted by the polymerization of acti

208 ROS production in adjacent cells is triggered by extracellular ROS signals

209 h activity and duct lineage specification in adjacent cells lacking jag1b and jag2b expression.

210 in the stele, the SHR protein moves into an adjacent cell layer, where it specifies endodermal cell

211 The vertebrate liver and heart arise from adjacent cell layers in the anterior lateral (AL) endode

212 h whether GLK proteins are able to influence adjacent cell layers, we used tissue-specific promoters

213 The ensuing engagement of Fas by FasL on adjacent cells leads to apoptosis.

214 Mechanical cross-talk between adjacent cells leads to enhancement of matrix reorganiza

215 ells; and why junctional buttressing between adjacent cells may benefit renal epithelium in maximizin

216 y growing cells, the resistance to stress in adjacent cells mechanically isolates rapidly growing cel

217 the talks revealed how interactions between adjacent cells mediated by Notch regulate development an

218 response to laser pulsing, forming pores in adjacent cell membranes through which cargo is gently dr

219 metrically, instructing unequal responses in adjacent cell membranes to establish molecular asymmetry

220 sical association between Integrin alpha5 on adjacent cell membranes.

221 cell by a mechanism involving the fusion of adjacent cell membranes.

222 hotoreceptors share the same origin of a few adjacent cells near the center of mitotic domain 20, sug

223 echanism by which mechanical stresses signal adjacent cells not only within the epithelium, but other

224 as a diffusible signal for the induction in adjacent cells of genes encoding cellular protectants su

225 s distributes in a concentration gradient to adjacent cells of the anterior compartment.

226 ng a negative regulator, CLAVATA3 (CLV3), in adjacent cells of the central zone (CZ).

227 ted specifically at the interface of the two adjacent cells of the developing sporangium, the mother

228 idney, while the tip cells interact with the adjacent cells of the metanephric mesenchyme, inducing t

229 ust interact with connexin(s) present in the adjacent cells of the nonpigmented epithelium.

230 thod as selected cells could differ from the adjacent cells or could belong to the same cell populati

231 g with assembly into connexons, docking with adjacent cells or gating properties of the gap junction.

232 role in the development and/or regulation of adjacent cells or tissues.

233 ls throughout the infection or can spread to adjacent cells over time.

234 pithelial cells in culture make contact with adjacent cells, PLAC-24 is specifically recruited to the

235 eractions between Eph kinases and ephrins on adjacent cells play a central role in neuronal patternin

236 ell by transfer from the polar septum to the adjacent cell pole where SpoIIE is protected from proteo

237 nd neuronal migration, inhibit the mixing of adjacent cell populations in the developing optic lobe.

238 al signalling restricts the intermingling of adjacent cell populations, whereas unidirectional activa

239 ur that can stabilize a distinct identity of adjacent cell populations.

240 essential for the docking of connexons from adjacent cells, preventing the formation of functional g

241 sing cells to GCV, and its overexpression in adjacent cells protects them from bystander cell death.

242 nal (R3) activates the Notch receptor in the adjacent cell (R4) via the ligand Delta, resulting in R3

243 Prior to activation, Itgalpha5 expressed on adjacent cells reciprocally and non-cell-autonomously in

244 e model based solely on interactions between adjacent cells reproduced 99% of the spatial structure a

245 F S/L gene lost adherence to the stratum and adjacent cells, resulting in an altered plaque morpholog

246 ction foci progressively expanded locally to adjacent cells, resulting in meningitis, choroiditis, en

247 the contractile acto-myosin cytoskeletons of adjacent cells, serving as a tension-transducer.

248 ization at specific sites of contact between adjacent cells showed significant reduction in the high-

249 ade side-to-side contacts, FrzCD clusters in adjacent cells showed transient alignments.

250 the EEC model and interacting with multiple adjacent cells simultaneously.

251 between the hemopexin domain of MT1-MMP and adjacent cell surface molecules is responsible for outsi

252 tivity is linked to Frizzled activity on the adjacent cell surface.

253 membrane-linked ephrin-A ligands residing on adjacent cell surfaces.

254 in paracrine or autocrine interactions with adjacent cell surfaces.

255 ifies differences between Frizzled levels on adjacent cell surfaces.

256 signaling can keep the rapid oscillations in adjacent cells synchronized.

257 osphorylation of EGFR on the cell surface of adjacent cells than equivalent levels of mature TGFalpha

258 s and that HAART prevents spread of virus to adjacent cells, the observation that this combination of

259 idly from one cell and immediately invade an adjacent cell, thereby minimizing exposure to the extrac

260 ddition to serving as a rigid anchor between adjacent cells, this study implicates desmosomal cadheri

261 spreading of prosensory specification to the adjacent cells through an intercellular mechanism.

262 tructure to epithelial tissues by connecting adjacent cells through homophilic E-cadherin interaction

263 lls activate Enhancer of split expression in adjacent cells through Notch signalling.

264 ses move from the initially infected cell to adjacent cells through plasmodesmata (PDs).

265 ne protrusions that resolve into vacuoles in adjacent cells, through a poorly understood mechanism.

266 id residues from the gangliosides present on adjacent cells, thus creating cell to cell interactions.

267 ligands for HVEM when presented in trans by adjacent cells, thus forming a bidirectional signaling p

268 ith localized inhibition of outgrowth of the adjacent cell to form interdigitating lobes and indentat

269 veloped particle could potentially infect an adjacent cell to mediate the cell-to-cell spread that is

270 stem cell-inducing transcription factor into adjacent cells to activate a negative regulator, thereby

271 The ability of previously-adjacent cells to adopt radically different fates could

272 Cell surface connexons dock between adjacent cells to allow for gap junctional intercellular

273 nnels that directly connect the cytoplasm of adjacent cells to allow for the intercellular transfer o

274 but also forces the lateral membrane of the adjacent cells to bend towards the initiating cells, the

275 n, particularly pheomelanin, photosensitizes adjacent cells to caspase-3 independent apoptosis, and t

276 lop into oogonia that enter meiosis, recruit adjacent cells to form follicle-like structures, and lat

277 bility in individual cells or couple between adjacent cells to form gap junctions and thereby provide

278 We propose that the micromeres induce adjacent cells to form SMCs, possibly by presenting a li

279 on molecules that interact with neurexins on adjacent cells to promote glutamatergic and GABAergic sy

280 iggered rapid migration and proliferation of adjacent cells to restore their density.

281 The ability of adjacent cells to stimulate HSV gene expression in neuro

282 binds to transmembrane receptors located on adjacent cells triggering a tyrosine kinase cascade.

283 The floorplate and adjacent cell types are absent in gli1-/-;gli2-/- mice,

284 e gradient into discrete transitions between adjacent cell types is the genetic toggle switch, compos

285 evelopment involves the formation of several adjacent cell types required for normal male fertility.

286 esigned to infer directional signals linking adjacent cell types within a complex tissue.

287 ected cGAS(+)STING(-) cells can migrate into adjacent cells via gap junctions to function in trans in

288 teroid-receptor positive cells but rather in adjacent cells via paracrine signaling involving several

289 scent proteins provides a way to distinguish adjacent cells, visualize cellular interactions and perf

290 lular movement of the VRCs and the spread to adjacent cells was blocked by inhibitors of filamentous

291 by microinjection, and their diffusion into adjacent cells was recorded photographically.

292 as accompanied by movement of the protein to adjacent cells, where it directly activated homeotic tar

293 r, moves from the stele to a single layer of adjacent cells, where it enters the nucleus.

294 tiated by trans binding between cadherins on adjacent cells, which is followed by the clustering of c

295 rs are delivered, but not in nonhaustoriated adjacent cells, which show high PR1::GUS expression leve

296 of the extracellular domains of cadherins on adjacent cells, with the cytoplasmic regions serving to

297 paracrinely to stimulate the mitogenesis of adjacent cells within the nascent DRG.

298 The cords appeared to invade adjacent cells within the plane of the monolayer.

299 ong growth-derived maximal tensile stress in adjacent cells would mechanically isolate rapidly growin

300 e hypothesis that HIV infection of common or adjacent cells would stimulate replication and spread of