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1 h 20/40 or better (HR, 1.56; 95% CI, 1.06-2.30; P = 0.024), adjusted for age, sex, and statistically significant covariat
2 h HF was 2.16 (95% confidence interval [CI]: 1.39 to 3.35); adjusted for age, sex, and Charlson comorbidity index; HR: 1.
4 anoma (odds ratio 2.90, 95% confidence interval 1.07-7.90), adjusted for age, sex, body mass index, and statin use.
6 rences were assessed with multivariable regression analyses adjusted for age, sex, body mass index, smoking, physical act
9 year cohort (born before 1940 or born in 1940 or later) and adjusted for age, sex, and race, were used to estimate hazard
10 incident parkinsonism per SD decrease in global cognition, adjusted for age, sex, and study subcohort.
11 hisms (SNPs) with natural log-transformed PTH concentration adjusted for age, sex, season, study site, and principal comp
14 the product and marginal effects of the GRS and SSB intake adjusted for age-, sex-, and cohort-specific covariates, with
15 son-years were calculated, and incidence rate ratios (IRRs) adjusted for age, sex, socioeconomic status, smoking, alcohol
17 However, a Cox proportional hazards model, adjusted for age, sex, and genotype for the Duffy antigen/rec
19 on to OH and SBP variability, using a Cox regression model, adjusted for age; sex; smoking status; alcohol intake; SBP; D
20 mortality was assessed with Cox proportional hazards models adjusted for age, sex, AMD severity, VA, history of cataract
22 Calculations were based on proportional hazards models adjusted for age, sex, race, HIV risk group, calendar year, c
25 cation and IOP were assessed using linear regression models adjusted for age, sex, body mass index, ethnicity, and the me
26 a with those testing negative in logistic regression models adjusted for age, sex, hospital, calendar time, time from ill
28 c melanomas were evaluated using logistic regression models adjusted for age, sex, study center, and primary status (sing
29 cardiovascular mortality, and total mortality in the models adjusted for age, sex, and centre (random effect).
30 95, 2.32), and 2.57 (1.66, 4.04) (P < 0.001 for trend) once adjusted for age, sex, smoking, LDL-cholesterol, BMI, waist c
31 000 person-years were calculated, and incidence rate ratios adjusted for age, sex, and socioeconomic status were estimate
32 s of all outcomes, we used multivariable linear regression, adjusted for age, sex, HIV status, and socioeconomic status.
34 idual plaque bacterial counts in unadjusted models or those adjusted for age, sex, and race (P >0.1 for all).
35 ndependently associated with death or lung transplantation, adjusted for age, sex, and type of PAH.
36 k of progression to dementia and the independent variables, adjusted for age, sex, education, comorbidities, and cognitio
41 Models accounted for familial relatedness and were adjusted for age, sex, total arsenic levels, and population s
42 survival (MSS) estimates up to 5 years after diagnosis were adjusted for age, sex, and 8th edition American Joint Committ
47 d with the use of Cox proportional hazards models that were adjusted for age, sex, body mass index, smoking status, educa
50 nal 7 were also significant in Cox regression analyses when adjusted for age, sex, and N-terminal probrain natriuretic pe
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