ライフサイエンスコーパス: adolescent-onset

1 ychiatric disorders with a high incidence of adolescent onset.

2 The disorder has an early childhood or adolescent onset.

3 at-onset groups: childhood onset (ages <12), adolescent onset (ages 12-17), early adult onset (ages 1

4 volumes of adolescents and young adults with adolescent-onset alcohol use disorders to those of healt

5 isms underlying schizophrenia's typical post-adolescent onset also remain elusive.

6 highest detection rate was in those with an adolescent onset and a family history (75%).

7 150-1151del) was associated with juvenile or adolescent onset and mental retardation, whereas we show

8 with bipolar disorder, especially those with adolescent onset and the above-noted factors, appear to

9 Adolescent (35-day-old at start; adolescent-onset) and adult (86-day-old at start) male r

10 rmalities may contribute to the emergence of adolescent-onset as well as early-onset conduct disorder

11 ional anisotropy (FA), have been reported in adolescent-onset bipolar disorder and in youth at famili

12 Although adolescent-onset bipolar disorder is associated with sig

13 oes not support a causal association linking adolescent-onset cannabis problems with young adult depr

14 sitive association between, on the one hand, adolescent-onset cannabis use and dependence and, on the

15 Impairment was concentrated among adolescent-onset cannabis users, with more persistent us

16 ermine whether decline is concentrated among adolescent-onset cannabis users.

17 restore neuropsychological functioning among adolescent-onset cannabis users.

18 ogy of early-onset conduct disorder, whereas adolescent-onset conduct disorder arises as a result of

19 ress; thus, arg(-/-) mice present a model of adolescent-onset dendritic simplification.

20 ts, little is known about the association of adolescent-onset depression and development of T2DM in y

21 blunted neural response to rewards precedes adolescent-onset depression and symptom emergence.

22 ophysiology of depression is associated with adolescent-onset depression.

23 Rates of adolescent onset disorder dropped sharply by the late 20

24 more to recurrence than chronicity of child-adolescent onset disorders.

25 omplications and relapses that are common in adolescent-onset drug addiction.

26 Diagnostic stability was high for child- and adolescent-onset DSM-III-R schizophrenia (positive predi

27 rating that adolescents with both early- and adolescent-onset forms of conduct disorder show impaired

28 Adolescent-onset gambling is associated with more severe

29 Similarly, the adolescent-onset group failed to show significant neural

30 ralimbic cortex was greater in rats from the adolescent-onset groups than adults.

31 bid delinquency were identified: stable low, adolescent-onset high to moderate, stable moderate, and

32 We investigated families with adolescent-onset idiopathic generalized epilepsy (IGE),

33 ound evidence for linkage to chromosome 8 in adolescent-onset IGE families in which JME was not prese

34 families ascertained through a proband with adolescent-onset IGE.

35 ssible candidate for these specific forms of adolescent-onset IGE.

36 omosome 18 locus predisposing to a number of adolescent-onset IGEs.

37 Six patients presented as an adult or adolescent-onset limb-girdle muscular dystrophy, one pre

38 A total of 274 participants with adolescent-onset major depressive disorder were assessed

39 opmental pathways that lead to first-episode adolescent-onset MDD (incident cases) in those at high f

40 us testing of different pathways to incident adolescent-onset MDD composed of contributions from fami

41 Clinical outcomes of adolescent-onset MDD into adulthood compared with contro

42 ial mortality from suicide into adulthood in adolescent-onset MDD patients.

43 cant independent clinical antecedents of new adolescent-onset MDD, but disruptive behavior (beta = -0

44 noid system as a notable research target for adolescent-onset mental health disorders.

45 nt research target for the neuropathology of adolescent-onset mental health disorders.

46 iological research progress in childhood-and adolescent-onset multiple sclerosis have been informed b

47 male adolescents with early-onset (N=36) or adolescent-onset (N=27) conduct disorder and in healthy

48 incubation of nicotine craving occurs after adolescent-onset nicotine self-administration and that n

49 n of nicotine craving is also observed after adolescent-onset nicotine self-administration and that n

50 ther incubation of craving also occurs after adolescent-onset nicotine self-administration.

51 ng whether this incubation is observed after adolescent-onset nicotine self-administration.

52 er 4 (19.4%) female-undiagnosed-wheezers had adolescent-onset non-atopic wheeze, low BDR and BHR, imp

53 d with developmental tooth abnormalities and adolescent onset of a broad range of ectodermal defects.

54 ory circuits is thought to contribute to the adolescent onset of cognitive deficits observed in schiz

55 FSIN development may contribute to the post-adolescent onset of schizophrenia endophenotypes.

56 The disease is characterized by adolescent onset of weakness, and atrophy of thenar and

57 rodevelopmental theory to address causes for adolescent onsets of clinical depressive disorders.

58 ealthy subjects with a longitudinal study of adolescent-onset patients (n = 12, representative subset

59 cross-sectional study of brain structure for adolescent-onset patients (n = 25) and adult-onset patie

60 disorder to 40% in those with a childhood or adolescent onset progressive disorder.

61 The phenotype is characterized by adolescent-onset progressive spastic ataxia with frequen

62 ain function, a trajectory likely underlying adolescent-onset psychiatric conditions such as schizoph

63 ciencies in this circuit are associated with adolescent-onset psychiatric disorders in humans.

64 cutive patients with first-episode child- or adolescent-onset psychosis (mean age at onset=14.2 years

65 Structural abnormalities specific to adolescent-onset schizophrenia in the sensori-motor cort

66 ervations suggest that the neuropathology of adolescent-onset schizophrenia involves larger and wides

67 Adolescent-onset schizophrenia provides an exceptional o

68 Compared with other psychoses, child- or adolescent-onset schizophrenia was associated with signi

69 ocial and symptomatic outcomes of child- and adolescent-onset schizophrenia with those of nonschizoph

70 altered white matter maturation specific to adolescent-onset schizophrenia.

71 tural grey and white matter abnormalities in adolescent-onset schizophrenia.

72 ractional Anisotropy) were carried out on 25 adolescent-onset schizophrenic patients and 25 healthy a

73 roups and reduced right insula volume in the adolescent-onset subgroup.